1008-1600 stiles-msk diagnoses not to be...
TRANSCRIPT
Musculoskeletal Diagnoses
Not To Be Missed
Bradford H. Stiles, MD, FAAFP
Disclosures
• I have a horrible deep sea fishing addiction
• My golf handicap is my swing
• No financial disclosures
What not to miss
• Scaphoid fracture
• Scapholunate dissociation
• Skier’s thumb
• Jersey finger
• Central slip rupture
• Little Leaguer’s elbow
• Distal biceps rupture
• Pectoralis major tear
• Complete rotator cuff tear
• SCFE
• Legg-Calve-Perthes
• Patella/Quad tendon tear
• Osteochondritis dessicans
• Achilles tendon rupture
• Lisfranc injury
• Jones fracture
• Cauda equina syndrome
Scaphoid fractures
� Most commonly fractured carpal
bone
� MOI: FOOSH injury (also assoc
w/distal radius, lunate and radial
head fxs)
� 3 zones: distal pole, waist,
proximal pole
� Waist: 80%, proximal pole: 15%,
distal pole: 5%
� Tenuous blood supply from distal
end only
� High incidence of non-union &
AVN in more proximal fractures
Presentation/exam
• Classic history
• May not have any swelling
• Tenderness at anatomic
snuffbox
• Positive axial load test of
1st MC
• Pain with
flexion/extension
Normal radiographs
Radiographs
• PA, lateral, oblique
(standard) and scaphoid
views
• Negative initial x-ray
doesn’t rule out a fx
• Rule of thumb: if positive
hx/MOI and snuffbox
TTP, treat as fracture, re-
image in 7-10 days
Treatment• Nondisplaced middle/proximal third fxs immobilized in long arm
thumb spica cast; distal third ND fxs may be immobilized in short arm
thumb spica cast
– Distal 1/3 fxs � 4-6 weeks
– Middle 1/3 and waist fxs � 10-12 weeks (may change to short arm thumb
spica cast after 6 weeks)
– Proximal 1/3 fxs � 12 -20 weeks
• May consider ORIF for proximal 1/3 fractures
• All displaced fractures need Ortho referral
• Risk of proximal AVN & severe radiocarpal OA if not treated properly
Scapholunate dissociation
• Most common ligament injury of the wrist• Commonly missed; long term complication is severe:
radiocarpal OA• MOI = FOOSH• Exam: tender over S-L area; positive scaphoid shift (Watson’s)
test (palpable clunk during palpation of scaphoid tubercle while patient goes from flexion/ulnar deviation to extension/radial deviation)
• Radiographs: standard AP may be normal; need “clenched fist”view; > 3mm S-L gap is positive (“Terry Thomas, Leon Spinks, David Letterman” sign); may have “ring sign”
• Refer to Ortho Hand surgeon; generally require reconstruction
Ring sign and scapholunate angle
Skier’s Thumb
• Sprain/tear of the UCL of the first
MCP joint
• AKA Gamekeeper’s thumb
• May have bony avulsion
• Get x-rays if possible before
testing
• Pain and swelling at site of UCL
• Stress testing of joint in 30° of
flexion
• May need MRI to look for Stener
lesion
Stener lesion
• Proximal end of the UCL
becomes trapped
superficial to the adductor
pollicis aponeurosis
• Requires surgical repair
Treatment
• If Stener lesion not present,
thumb spica splint for 4-6 weeks
• If Stener lesion is present, refer
to Hand Surgery
• Long term complication from
improper treatment is
osteoarthritis
Jersey finger
• Flexor digitorum
profundus tendon
rupture/avulsion
• FDP flexes the DIP joint
• MOI: forced extension
with active DIP flexion
(e.g., grabbing a jersey)
• Ring finger most
common
• Inability to flex DIP joint
• Tendon may retract into
palm
• All require urgent
surgical management
Central slip avulsion of the
extensor tendon� MOI: forced flexion of extended
PIP joint
� Swelling of PIP joint, tender on
dorsal side
� Unable to extend PIP joint
against resistance
� X-ray to assess for large avulsion
� Splint PIP joint in full extension
(leave DIP/MCP joints free) for 6-
8 weeks
Boutonniere deformity
• Late complication of central slip
injury
• Lateral bands drift volar to
rotation axis becoming PIP
flexors with DIP hyperextension
(proximal phalanx “buttonholes”
dorsally
• Deformity presents after several
weeks
• Usually requires surgery
Biceps Tendon Rupture• 90+% are proximal
• Distal ruptures require early diagnosis
• Majority require surgical repair due to significant loss of
supination strength
• Usually acute injury
• DDx: anterior capsule strain, coronoid process fracture,
lateral antebrachial cutaneous n. entrapment
• PE: biceps tendon not palpable in antecubital fossa
Distal Biceps Tendon Rupture
• X-rays to r/o avulsion fractures
• MRI to confirm complete tear
• Early surgical repair due to retraction/scarring
Pectoralis Major Injury
• Internal rotator and flexor of
humerus
• Two heads: sternal and clavicular
• Tear can occur at bony insertion
medially, M-T junction laterally
or in muscle belly (rare)
• MOI: usually acute pull/snap from
heavy lifting
• Rupture of inferior sternal head
easy to miss
• May see defect in “power prayer”
position
• MRI useful if diagnosis is unclear
• Surgical repair is dependent on
strength requirements for
sport/occupation
• Surgical outcomes better with
earlier repair, but late
reconstruction is possible
Complete Rotator Cuff Tear• Complete tears will often retract and scar down, making surgical
repair difficult if delayed
• More common in adults > 40 years old
• Often diagnosed as “tendonitis”
• Obvious weakness with RC testing (empty-can, drop-arm or lift-off
tests)
• Diagnostic injection can help to differentiate tear from
tendonitis/bursitis
• Untreated complete tears increase risk of GH osteoarthritis
Slipped capital femoral epiphysis (SCFE)
• Femoral head slips inferior and posterior to femoral neck
• Incidence 2/100,000 children; may be bilateral (20-40%)
• More common in boys (mean age 13 years) than females (mean age 11 years)
• Associated with period of rapid growth, obesity
• Highest risk group is African-American boys
• Present with painful limp; pain usually in groin but may be in anterior thigh or knee
(referred pain)
• Radiographic diagnosis
– Klein’s line, “ice cream off the cone”
• Surgery required
Legg-Calve-Perthes
• Avascular necrosis of femoral head leading to collapse and flattening of femoral head
• Etiology unknown
• Males >> females
• Most common in boys 4 – 10 years old
• Often painless, but will develop limp; easily diagnosed on x-ray
• Goal of treatment is containment of femoral head in acetabulum
– Bracing
– Physical therapy
• Blood supply generally returns over several months, leading to new bone growth
• In children under age 6 years old with appropriate treatment, greater chance of ending up with
normal hip joint
Femoral neck stress fracture• Often misdiagnosed or missed
• Extreme risk of displacement
• Result of overuse/repetitive stress
• Common in athletes, military recruits
• History of recent increased activity (frequency or intensity)
• Tension vs. Compression side
• Must get x-rays if suspicious; may take 2-4 weeks for x-rays to be positive
• Usually present with groin pain or anterior thigh pain with any weight-bearing
activity
• Further work-up required
if x-rays negative but
suspicious history
• Bone scan can be positive
within 24 hours of injury
• MRI extremely sensitive
• Treatment is dependent on
location, compression vs. tension
side
– Nondisplaced compression side
stress fractures treated
conservatively with NWB until
fracture is healed (6-8 weeks);
serial radiographs essential to
monitor for any worsening
– All tension side stress fractures are
treated surgically
Patellar/Quad Tendon Rupture
• Most common in 3rd & 4th decades of life
• Increased risk after ACL reconstruction using patellar tendon graft
• Risk factors: trauma, steroid use, quinolone use, DM, RA/SLE, chronic
tendonitis
• Inability to fully extend leg; focal deformity often present
• X-rays may show avulsion off patella or patellar migration
(inferior/superior); consider contralateral films for comparison
• Knee immobilizer (straight leg) and urgent Ortho referral
• Need to be repaired in first few weeks
Osteochondritis dissecans (OCD)
• Most common in the knee, but can also be seen in ankle,
elbow or any large joint
• Repetitive microtrauma/overuse leads to subchondral bone
death and subsequent articular cartilage fragmentation
• 20%-30% have bilateral involvement (knees)
• Radiographic diagnosis; MRI is the gold standard and used
for classification
OCD MRI Classification
• 4 stages based on MRI
• Stage I: low signal intensity
• Stage II: hypointense rim (no separation of lesion)
• Stage III: high signal intensity with underlying cystic
changes (instability)
• Stage IV: dislocation of fragment into the joint space
OCD Treatment
• Stage I: non-operatively with restricted weight-
bearing x 6-8 weeks
• Stage II: Non-operative if growth plates still open; in
adults it depends on the size of the lesion
• Stage III-IV: surgery
• Missed diagnosis can lead to permanent damage and
early degenerative joint disease
ACHILLES TENDON RUPTURE
• Classic patient: 30-40 y/o
male playing basketball (or
tennis)
• Classic history: “pushing off,
felt like I was shot in the back
of my lower leg”; loud snap
often heard across the court
• Rupture occurs in “watershed”
area 2-6 cm above insertion
Diagnosis
• Palpable defect
• Swelling/ecchymosis
possible
• Positive Thompson’s
test
• Imaging studies
obtained to r/o
avulsion fracture
Thompson Test
Treatment
• Casting vs. surgery
• Can treat non-operatively with serial casting
starting in equinus; long-leg cast required for the
first month
• Surgical repair allows for quicker weightbearing
• 6 months recovery time
LISFRANC INJURY
• Classic forced
plantar flexion injury
(trip down stairs)
• Midfoot injury
• Easily missed
• Beware of the
“burrito foot”
• WB films required
• Radiographic findings
can be very subtle
• Low threshold for MRI if
exam suspicious
• Refer to Ortho
foot/ankle specialist
• Midfoot DJD and chronic
foot pain if missed
JONES FRACTURE
• Proximal 5th MT diaphyseal fracture
• 0.5cm – 1.5cm from the proximal tip of the
5th MT
• Distinguish between tuberosity fractures
and Jones fracture
• “Jonesland” is area of very poor vascular
supply; high rate of non-union
Treatment
• Tuberosity avulsion fractures/stress fractures
treated in walking boot or hard-sole shoe
• True Jones fracture requires either prolonged
NWB cast immobilization (8-12 weeks) or
surgical ORIF
• Missed diagnosis leads to nonunion and chronic
foot pain
• Terminal end of the spinal cord is at L1-2
• Below this spinal canal is filled with the L2-S4 nerve roots, or cauda
equina (“horse tail”)
• Syndrome results from sudden reduction in volume of lumbar
spinal canal
– Central disc herniation
– Epidural abscess or hematoma
– Trauma/fracture with retropulsion
• Onset may be sudden or over hours
Cauda Equina Syndrome
• Symptoms
– Pain out of proportion; radicular pain/numbness bilaterally but worse on one
side
– Saddle anesthesia
– Lower extremity weakness/paralysis
– Urinary or bowel incontinence or urinary retention
• Surgical emergency
– STAT MRI
– STAT spine surgery evaluation
– Missed diagnosis can lead to permanent nerve damage and paralysis
Questions?