2nd year-coronary artery disease related to dm( ihm- mrs. cordenillo
TRANSCRIPT
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I. Introduction
Diabetes mellitus is a group of metabolic diseases characterized by high
blood sugar (glucose) levels that result from defects in insulin secretion, or
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predisposition. The rapid modernization and industrialization allows the
individual more time for leisure thus lessening physical activity. This leads to
increasedfood consumption leading to weight gain and obesity.
DM is associated with a markedly increased risk of coronary artery
disease. In the Framingham study, the incidence of coronary artery disease
was increased in diabetic subjects. While the most common cardiac
manifestation in diabetic patients is coronary artery disease, DM also
appears to be strongly linked to heart failure (HF).
Approximately 15 to 25% of patients with HF are diabetics and it has been
suggested that DM may play an important role in the pathogenesis,
prognosis, and response to treatment of HF. The increased risk of
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b. Specific
Upon the completion of this case study, the student shall have:
Described and explained Diabetes Mellitus, Coronary Artery Disease and
Congestive Heart Failure.
Reviewed the anatomy and physiology of the organs involved.
Identified the risk factors contributing to the occurrence of the condition.
Expounded on the laboratory and diagnostic procedures done with the
patient, their purposes, and specific nursing responsibilities before, during
and after the procedure.
Enumerated the different medications administered for the condition, their
i di ti d ifi i ibiliti
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Every cell in the human body needs energy in order to function. The
bodys primary energy source is glucose, a simple sugar resulting from thedigestion of foods which contains carbohydrates (sugars and starches).Glucose from the digested food circulates in the blood as a ready energysource for any cells that need it. Insulin is a hormone or chemical producedby cells in the pancreas, an organ located behind the stomach. Insulin bondsto a receptor site on the outside of cell and acts like a key to open a doorwayinto the cell through which glucose can enter. Some of the glucose can be
converted to concentrated energy sources like glycogen or fatty acids andsaved for later use. When there is not enough insulin produced or when thedoorway no longer recognizes the insulin key, glucose stays in the bloodrather entering the cells.
Anatomy of the pancreas:The pancreas is an elongated, tapered organ located across the back
of the abdomen, behind the stomach. The right side of the organ (called thehead) is the widest part of the organ and lies in the curve of the duodenum(the first section of the small intestine). The tapered left side extends slightlyupward (called the body of the pancreas) and ends near the spleen (calledthe tail).
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The Exterior of the HeartThe heart is the muscle in the lower half of the picture. The heart has
four chambers. The right and left atria are shown in purple. The right and leftventricles are shown in red. Some of the main blood vesselsarteries andveinsthat make up the blood circulatory system are directly connected to
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the heart's left ventricle. The coronary arteries are located on the heart'ssurface at the beginning of the aorta. The coronary arteries carry oxygen-rich
blood to all parts of the heart.
The Interior of the Heart
The SeptumThe right and left sides of the heart are divided by an internal wall of
tissue called the septum. The area of the septum that divides the atria (the
two upper chambers of the heart) is called the atrial or interatrial septum. Thearea of the septum that divides the ventricles (the two lower chambers of theheart) is called the ventricular or interventricular septum.
Heart ChambersThe two upper chambers of the heart are called atria. The atria receive
and collect blood. The two lower chambers of the heart are called ventricles.
The ventricles pump blood out of the heart into the circulatory system to otherparts of the body.
Blood FlowThe arrows in the drawing show the direction that blood flows through
the heart.
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Final Diagnosis: Hypertension, Hypertensive, Coronary Artery Disease
CHF class II w/ pericardial and pleural effusion
Attending Physician: Dr. Belasoto
V. Clinical Assessment
a. Nursing History
Pt is a diagnosed DM for >5 years. A month PTA, had met postprandial
vomiting preceded by dizziness (+) on and off grade 1 bipedal nonpitting edema,
(+) blurring of vision.
b. Past Health Problem/ Status
Diabetic >5 yrs
Nonhypertensive
nonasthmatic
VI. BRIEF SOCIAL, CULTURAL AND RELIGIOUS BACKGROUND
Educational Background:
Unrevealed.
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With equal pulses
No edema
No cyanosis
Cephalocaudal Assessment:Vital Signs:
BP- 140/80 mmHg; T- 36.6C; PR- 78 bpm; RR- 19 rpmGeneral Appearance:
Mrs. M.B was seen lying on her bed with an IV fluid of PNLSS 1l x KVOin the left metacarpal veins and regulated at10 cc/hr.
Assessment:Skin
Dry and warm to touch-With fair skin complexion-With pallor
Head With short hair, black and equally distributedEyes
With dark brown iris
With pale palpebral conjunctivaEars
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contraction.
1/14/10 Differential count1/15/10
Glucose 6.16 mmol/L 4.10-5.90 AbnormalIt indicates thatdont have enoughinsulin in yourbody.
cholesterol 6.49 0-5.20LDL 4.80 1.71-4.60 Abnormal
A cholesterol thatcirculates in the
blood. It canslowly build up inthe inner walls of
the arteries thatfeed the heart andbrain. It indicates
form plaque, athick, hard deposit
that can narrow
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malignant tumor.
Date 1/20/10
Chest PA
Xray findings:
Follow up study done, as compared with the previous chest x-ray taken
12/17/09 shows haziness in both lung bases obliterating the hemidiaphragm andcostophillic sulci.
There is a regression of haziness in the ( R ) upper lung field. The rest of
the findings are unchanged.
Impression:
Bilateral pleural effusionAtherosclerotic Aorta
Bibasal pneumonia
Cardiomegaly, borderline
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Hyperglycemia
Dyslipidemia
Atherosclerosis
Deposition of Fats in
the Aorta
Narrowing and
Blockage of the Aorta
Chronic High Blood
Glucose
Increase Osmotic
Pressure
Fluid Goes to
vascular space
High Blood Volume
Hypertension
Viscous Blood
Poor Circulation
Low Blood Supply
Nerve Damage
NeuropathyLow Blood to
Coronary Artery
Peripheral Nervous
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Trade
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Names
&
Dosage
Generic
Name
Classification Action Indication Side Effect Contraindication Nursing
Responsibility
140 mg.
ODsimvastatin cholesterol-
lowering drug
antihyperlipidermic
reduces cholesterolby inhibiting anenzyme in the liver(HMG-CoAreductase) that isnecessary for theproduction ofcholesterol.In the blood,statins(HMG-CoAreductase) lowertotal and LDL("bad") cholesterolas wellas triglycerides
In patients withcoronary heartdisease,diabetes,peripheral vesseldisease, or historyofstroke or othercerebrovasculardisease andnonfatalmyocardialinfarction (heartattack)
headache,nausea,vomiting, diarrhea, abdominalpain, muscle pain,and abnormalliver test.Mostserious potentialside effects areliver damage andmuscleinflammation orbreakdown.
Pregnantwomen, fungalbyproducts,lactation
Ask the pt. if theyare allergic to it.Limit alcoholicbeverages of thept.Give in theevening; thehighest rates ofcholesterolsynthesis arebetween midnightand 5 am
10 mg
t
a
b
O
D
Norvasc Antianginal
Antihypertensive
Calcium channel
blocker
Decreases cardiacoxygenconsumption,Increased deliveryof oxygen tocardiac cells, blockthe transport of
calcium into thesmooth musclecells lining thecoronary arteriesand other arteriesof the body.
Chronic stableangina, alone or incombination withother drugs.Essentialhypertensive,alone or in
combination withotherantihypertensives
Allergies toamlodipine, heartblock,lactation,use cautiously withheart failure
headache andedema (swelling) of the lowerextremities,dizziness
Assess skinlesions, edema,baselineECG,ausculation,GI normal output.
Monitor patient
carefully(BP,cardiacrhythm andoutput)
40 mg Clexane Anticoagulants,
Antiplatelets &
-treats blood clots
-treats certain types
renal impairment:
Treatment of
Haemorrhage,
thrombocytopenia
majorblood
Assess for tingling
weakness,
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IX. Nursing Management (NCP)
Problem 1
Assessment Nursing Diagnosis Planning Intervention Evaluation
Objective
= 6.16 mmol/dL
(glucose)
> weakness
Risk for infection r/tglucose level
After 1 hour of nursing
interventions,
the patient will identify
interventions to prevent
risk of infection
Independent
>Stress proper handhygiene by washing handsand using sanitizers.
>Increase intake of Vit C> Observe for signs ofinfection and inflammation.
>Maintain aseptictechnique for IVinsertion procedure,administration ofmedications, andprovidingmaintenance and sitecare. Rotate IV sitesas indicated.
>Provide skincare by keeping the skin
dry,linens dry andwrinkle free.
After 1 hour of
nursing interventions,
the patient have
identified interventions
to prevent risk of infecti
like being aware of eati
foods that will decrease
the risk of infections.
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Problem 2
Assessment Nursing Diagnosis Planning Intervention Evaluation
SubjectiveSadto matambok koya.Galing ky sangnagmaskit ko sangdiabetes nga ni gulpiakon pagniwang, asverbalized
ObjectiveWt. 110 lbs
Non-elastic skin
Imbalanced Nutrition:Less Than Body
requirements related toincreased metabolicneeds caused by the
disease
After the nursingintervention:The patient will be-encourage the patient toeat well balanced diet-maintain blood glucose
level-Achieve and maintainreasonable weight
Independent
>Discouraged beveragesthat are caffeinated orcarbonated> Encouraged low fat dietand high fiber foods
>Achieve and maintainreasonable weight>Practiced goodnutritionalhabits>Use less salt>Eat less sugar>Eat less fat>Eat more complexcarbohydrate
Goal partially met asevidenced by patient arethreatened by the factorsoccur. Avoided drinkingcarbonated drinks. Eatwell.
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Problem 3