ADRENAL GLAND

&THYMUS GLAND

AMIT KUMAR SHUKLAM.A. SEM-1ST

D.A.V. PG COLLEGE VARANASI

ADRENAL GLAND

ADRENAL GLAND

ADRENAL MEDULLAADRENAL CORTEX

GLUCOCORTICOIDS

MINERALOCORTICOIDS

SEX HORMONES

EPINEPHRINE

NOR-EPINEPHRINE

FUNCTION OF HORMONES GLUCOCORTICOIDS

MONITOR & REGULATE CARBOHYDRATES, PROTEINS & FAT METABOLISM RAISE BLOOD GLUCOSE LEVEL

REDUCE THE EFFECT OF INFLAMMATION OR SWELLING(CORTISOL) STIMULATE THE PRODUCTION OF GLUCOSE FROM FATS & PROTEINS-

GLUCONEOGENESIS(CORTISOL) MINERALOCORTICOIDS

REGULATE SALT & WATER BALANCE, LEADING TO INCREASE OF BLOOD VOLUME & PRESSURE REABSORB SODIUM & EXCRETE POTASSIUM(ALDOSTERONE)

SEX HORMONES STIMULATE REPRODUCTIVE ORGANS & BRING ABOUT SEX CHARACTERISTICS

EPINEPHRINE & NOR-EPINEPHRINE PRODUCE ‘FIGHT’ OR ‘FLIGHT’ RESPONSE

INCREASE HEART RATE BLOOD FLOW TO MOST SKELETAL MUSCLES

INCREASE CONCENTRATION OF GLUCOSE LEVEL IN THE BLOOD DECREASE FLOW TO DIGESTIVE ORGAN

ROLE IN STRESS RESPONSE ADRENAL CORTEX PROVIDE A SLOWER, BUT LONG-TERM STRESS RESPONSE

ADRENAL MEDULLA PROVIDE A RAPID, BUT SHORT-TERM SRESS RESPONSE

MALFUNCTION OF ADRENAL GLAND ADRENAL CORTEX

HYPER-SECRETION OF GLUCOCORTICOIDS CAUSE “CUSHING’S SYNDROME” HYPO-SECRETION OF GLUCOCORTICOIDS CAUSE “ADDISON’S DISEASE”

ADRENAL MEDULLA HYPER – SECRETION OF EPINEPHRINE & NOR- EPINEPHRINE CAUSE:

HYPERTENSION HYPERGLYCEMIA NERVOUSNESS BLOOD CLOTTING SWEATING

SPEECH EFFECTED

THYMUS GLAND

RELEASING HORMONE- THYMOSINS

THYMOSINS STIMULATE PRODUCTION AND MATURATION OF “T LYMPHOCYTES” THYMUS GLAND IS MOST ACTIVE DURING CHILDHOOD

T LYMPHOCYTES RESPONSIBLE FOR SPECIFIC DEFENSES AGAINST PARTICULAR INVADER

THANK YOU