advanced trauma life support reviewer · · 2012-08-31– tinnitus, amnesia, irritability,...
TRANSCRIPT
Advanced Trauma Life Support Reviewer
Quality Development Department
Trauma
� Leading cause of death ages 1- 44 years
� 60 million injuries a year
� $ 400 Billion annually
� 2.7 million admissions / year
� 100,000 deaths per year
� 50 % die prior to receiving any medical care
ATLS
� ATLS formed in 1978 to standardize trauma care
� American College of Surgeons
� Adopted in over 40 countries
� Goal is to teach a simplified and standardized approach to the care of trauma patients
� “Golden Hour”
Types of Trauma
� Blunt Trauma
� Penetrating Trauma
� Burns / Cold
� Environmental Exposure
� Hemorrhage is the # 2 cause of death in trauma
ATLS
� ABCDE
� A = Airway
� B = Breathing / Bleeding
� C = Circulation
� D = Disability
� E = Exposure / Environment
Vital Signs
� Physiological status
– Clinical Clues
� Body temperature
� Heart rate
� Blood pressure
� Respiratory rate
� O2 saturation
� Pulse Pressure
Pulse Pressure
� Pulse pressure is the difference between systolic and diastolic blood pressure
� The change in blood pressure seen during a contraction of the heart
� Normal Range: 30 - 40 mmHg
� Systolic pressure - Diastolic pressure
Low / Narrow Pulse Pressure < 25 mmHg
� Significant blood loss
� Low stroke volume
� Tachycardia
� Severe Aortic Stenosis
� Constrictive Pericarditis
� Pericardial Effusion
� Ascites
� Congestive Heart Failure
� Shock
High Pulse Pressure > 40 mmHg– Stiffness of the major arteries
– Aortic regurgitation
– AV malformation
– Atherosclerosis
– Thyrotoxicosis
– Fever
– Pregnancy
– Heart block
– Aortic dissection
– Endocarditis
– Raised intracranial pressure
Phases of ATLS
� Triage – Rapid initial assessment
and management of the injured patient
� Prehospital Phase
� Hospital
� Posthospital Phase
Triage
Multiple Casualties
� Severity of injuries does not exceed the ability of the facility
Mass Casualties
� Injuries Exceed the capabilities of the facility and staff
Prehospital Phase
� Airway maintenance
� Control external frank bleeding and shock
� Immobilization
� Minimize scene time
� Trauma alert criteria
� Transport to the nearest APPRORIATE facility
� DO NO HARM !
Primary Survey
� ABCDE
� Life threatening conditions are identified and managed simultaneously
Airway and C-Spine
� Airway patency
– Jaw thrust or chin lift
� Immobilize C- Spine
� Head and neck not be moved
� Assume a cervical spine injury in any multisystem trauma
– Especially in patients with AMS
� GCS < 8 or airway compromise - ET / KT intubate
Breathing
� Ventilation involves function of the lungs, chest wall and diaphragm
� Expose patients chest and look
– Flail chest
– Tracheal deviation
– Puncture sites
� Percussion, Inspection & Palpation
– Pneumothorax
– Hemothorax
Bleeding
� External hemorrhage must be identified and controlled in the primary survey
� Hemorrhage is the predominant cause of preventable post injury deaths
Tourniquets
� Used in traumatic amputation
� Use in PRE shock conditions
� Beneficial if saving Life vs. Limb
Circulation
� Hypotension is considered hypovolemia until proven otherwise
� Signs and effects of Severe Hypovolemia:
– 1. LOC
– 2. Skin color
– 3. Pulse quality
Circulation
� Control bleeding by direct pressure or operative interventions
� Minimum 2 large caliber IV’s
� Warmed LR, 2 – 3 L bolus
� Blood Type Specific or O neg.
� Hypovolemic Shock should NOT be treated with pressors, steroids or HCO3
Disability
� Rapid neurologic evaluation is done at the end of the primary survey
� LOC = AVPU
� A = ALERT
� V = responds to VOCAL stimuli
� P = responds to ONLY PIANFUL stimuli
� U = UNRESPONSIVE to ALL stimuli
Exposure / Environmental Control
� The patient should be completely undressed
� Cover the patient with warm blankets to prevent hypothermia
� Intravenous fluids should be warmed and a warm
environment maintained
Adjuncts to Primary Survey and Resuscitation
� ECG
� NG / OG
� Foley is Contraindicated if– Blood at the meatus
– Perineal ecchymosis
– Blood in the scrotum
– High riding or nonpalpable prostate
– Pelvic fracture
� Monitor
� Labs, UA, X-rays, CT, MRI
Secondary Survey
Secondary Survey
� Head to toe evaluation
� After primary survey is completed
� Resuscitate efforts are established
� Patient has normalization of vital signs
� GCS score and complete neuro exam
� X-rays and special procedures
� “Tubes in every orifice”
Secondary Survey History
� A = Allergies
� M= Medications
� P= Past Illness / Pregnancy
� L = Last meal
� E = Events / Environment leading to injury
Interventional Therapeutics
� IVF– NS / LR / Hespan /
Blood Products
� Needle decompression
� Cricothyroidectomy
� Chest Tubes
� Preicardiocentesis
� Permissive Hypotension
� DPL
� Intubation
IV Fluids
IVF Resuscitation
� Fastest way to deliver, and distribute fluids
Side Effects
� Lowers Hemoglobin
� Deactivates Clotting factors
� Decreases Coagulopathy
� Decreased clotting factors
� Impaired clotting factor function
� Increased bleeding time
Colloids vs. Crystalloids
� Colloids contain larger insoluble molecules – blood
� The most common crystalloid fluid is NS– Sodium chloride at 0.9% concentration – Close to the concentration in the blood – isotonic
� Ringer's lactate is a isotonic solution – For large volume fluid replacement
� A solution of 5% dextrose in water, D5W, is used if there is at risk of low blood sugar or high sodium– D5W is especially used in mixing and infusing medications
due to its hypotonic properties
Intravenous therapy
NS
� Na = 154 and Cl = 154
LR
� Na = 130
� Cl = 109
� Lactate = 28 mmol / L
� K+ = 4 mmol / L
� Ca2+ = 1.5 mmol / L
D5W
� Glucose = 278 mmol / L
Permissive Hypotension
� Hypotension protective
� BP decrease allows clots to stabilize
� Hypotension “good” in other conditions
– Aortic dissection
– Uncontrolled GI bleed
– Extremity bleeding
Permissive Hypotension
� Minimal IVF before hemorrhage control
– Don’t pop the clot!
� Hemodilution by IVF
– Decreases oxygen carrying capacity
– Decreases clotting factor function
� IV fluids activate inflammatory mediators
� Secondary organ damage
Hemorrhagic Shock
� Sudden and rapid loss of significant amounts of blood
� Hypovolemic shock resulting from acute hemorrhage – Hypotension
– Tachycardia
– Oliguria
– Pale, cold, and clammy skin
Battlefield Medicine -Tourniquets
� Improves Long term Survival
� Apply pre shock
� 90 % mortality if placed after shock symptoms
� 10 % mortality pre shock
� Improvised tourniquets should not be used
Hemorrhagic Shock Classifications
Pre Hospital Recommendations
� No fluids if normotensive
� If hypotensive, controlled IVF until goal:
– Radial pulse SBP = 80
– Mentation (non-head injured patient)
– MAP 40–60 mmHg
� Controlled fluid administration
� Small boluses 25 – 500 ml
� Minimum MAP > 60mmHg to maintain CPP
General Trauma Overview
Head Trauma
Physical Examination Head Injury
� # 1 cause of traumatic death
� Visual acuity
� Ear Trauma
– Halo test
� Raccoons Eyes
� Pupillary size
� Ocular damage
– Remove contact lenses
– Lens dislocation
� Penetrating Injury
Head Exam Coup Contra Coup Brain Injury
� A specific area of brain injury located directly opposite to the site of impact to the head that results from linear violent collisions of the brain with the skull.
What is a TBI?
� Sudden damage to the brain by an external force
2 Types� Closed Head Injury
– Head forcefully collides with another object
� No skull injury
� Open Head Injury– Object fractures the skull and
debris enters the brain � Brain tissue damage
TBI Severity Levels
� Mild
– Change in the mental status at the time of the injury
– Concussion.
� Moderate
– Loss of consciousness last for minutes to hours
– confused for days or weeks
� Impairments can be temporary or permanent.
� Severe
– Unconscious state for days, weeks, or months
� Impairments are permanent.
Concussion Grades
� Grade 0 results when the head is struck or moved rapidly– Post injury headache and difficulty with concentration
� Grade 1 concussions the patient may appear stunned or dazed– No LOC, and sensory difficulties clear < 1 minute
� “I got my bell rung” description from the athlete
� Grade 2 Headache, cloudy senses > 1 minute, and no LOC – Tinnitus, amnesia, irritability, confusion, or dizziness
� Grade 3 concussions experience a LOC < 1 minute– Not comatose, with the same symptoms as a grade 2 concussion
� Grade 4 concussions experience a LOC > 1 minute– Exhibits the symptoms of the grade 2 and 3 concussions
Post-Concussion Syndrome
� Decreased Processing Speed
� Short Term Memory Impairment
� Concentration/Attention Deficit
� Irritability/Anxiety/Depression
� Fatigue/Sleep Disturbance
� General Feeling of “Fogginess”
� Academic Difficulties
� Persistent headache
� Intolerance of Lights and Noise
Concussion Treatment
� Transport ALL concussions
� Head CT Scan required
� Grades 2 – 4
– Most serious
� Needs neurologic specialists
Differential diagnosis
� Subdural hematoma
� Epidural hematoma
Subdural and Epidural Hematomas
Subdural Hematomas
� Due to the shearing forces of acceleration
� The blood vessels on the surface of the brain are torn and begin to bleed
� No LOC
� Slow developing injury
� Treatment < 4 hours lowers mortality
Epidural hematomas
� Faster progressing injury– LOC
– Then a lucid moment
– Rapid decline in mental function
� Most are associated with a skull fracture
� Surgical intervention is a must to prevent death
Signs of Increased ICP
� Visual difficulties
� Vomiting
� Dyspnea
� Decreased pulse
CPP = MAP - ICP
CPP: Cerebral Perfusion PressureMAP: Mean Arterial PressureICP: Intracranial Pressure
Maxillofacial
� Cribriform fractures
� OG Tube ONLY
� Le Fort fracture– 3 types
� Bilateral horizontal fracture of the maxilla
Maxillofacial Le Fort fractureLe Fort I fracture� Horizontal segmented fracture of the alveolar process of the
maxilla, the teeth are contained in the detached portion
Le Fort II fracture� Fracture of the maxilla, in which the body of the maxilla is
separated from the facial skeleton and the separated portion is pyramidal in shape; the fracture may extend through the body of the maxilla down the midline of the hard palate, through the floor of the orbit, and into the nasal cavity
Le Fort III fracture� Entire maxilla and one or more facial bones are completely
separated from the craniofacial skeleton; such fractures are almost always accompanied by multiple fractures of the facial bones
Cervical Injuries
Cervical Spine and Neck
� Head and spinal cord injury # 1 cause of traumatic death
� Head and Maxillofacial trauma are presumed to have an unstable C-spine
� C-spine immobilization
� Absence of neurologic deficit does not exclude injury
� DO NOT EXPLORE Zone 1 Penetrating injuries– The Platysma
Spinal Cord Injury Definitions
Spinal Shock
� Flaccidity and loss of reflexes after spinal cord injury, variable duration
Neurogenic Shock
� Impairment of the descending sympathetic pathways in the spinal cord
� Loss of sympathetic innervation to the heart and vascular system - vasomotor tone
– Hypotension and Bradycardia
� Treatment - Vasopressors and Atropine
Spinal Cord Injuries
Brown SéquardSyndrome
� Penetrating trauma
– GSW
� Loss of function on affected side
� Loss of pain and temperature sensation on opposite side
Anterior Cord Syndrome
� Bony fragments or pressure on spinal arteries
� Loss of motor function
� Loss of pain sensation
� Loss of light touch sensation
– Some sparing of light touch, vibration, and proprioreception
Spinal Cord Injuries
Central Cord Syndrome
� Hyperextension of c - spine
� Weakness / parasthesia
– UE > LE
� Preserved function of lower extremities
� Bladder dysfunction
Posterior Cord Syndrome
� Rare
� Hyperextension injury
� Partial loss of proprioreception
� Loss of sensation
– Deep touch
– Vibratory
Chest Trauma
Chest Trauma
� Pain, Dyspnea, or Hypoxia
� 25% of all trauma deaths
� 2/3 of deaths occur after reaching hospital
� 2nd leading cause of death from physical trauma after head and spinal cord injury
Chest Trauma Differential Diagnosis
� Thracheobroncial Injury
� Pneumothorax
� Hemothorax
� Rib / Sternal fractures
� Diaphragm Rupture
� Flail Chest
� Open Chest Wound
� Penetrating Injury
� Pulmonary contusion
Rib Fracture Differential Diagnosis
� 1-3 ribs & Scapula
– High mortality
– Thoracic Injury
� 4-9 ribs
– Intrathoracic injury
� 10 -12 ribs
– Left lower six ribs 20% spleen
– Right lower six ribs 10% liver
– Abdominal Organ Injury
Pneumothorax
� Air in the pleural cavity
� Any injury that disrupts the parietal or visceral pleura
� Unilateral signs:
– ↓movement and breath sounds
– Resonant to percussion
� Confirmed by CXR
Treatment
NRB if < 20 % of chest cavity
Chest Tube if > 20 %
Tension Pneumothorax
� “1 way valve" air leaks into the lung– Air enters is unable to escape the pleural space
� Clinical Diagnosis – Not radiologic– Respiratory distress
– Tachycardia
– Hypotension
– Tracheal deviation to opposite side
– Shift of mediastinum to opposite side
– Decreased LOC
– Neck vein distension - JVD
� Unilateral absence of breath sounds
� Immediate Needle decompression in 2nd space followed by chest tube
Chest Tube Indications
� Pneumothorax– Air in the pleural space
� Pleural effusion– Fluid in the pleural space
� Chylothorax– Lymphatic fluid in the pleural space
� Empyema– Infection of the pleural space
� Hemothorax– Blood in the pleural space
� Hydrothorax– Serous fluid in the pleural space
Massive Hemothorax
� Rapid accumulation of ≥1500 ml blood results in hypoxia
� Shock + absent breath sounds
� Flat or distended neck
Treatment
� Chest decompression # 38 CT
� If 1500 ml evacuated by CT, or > 200 ml/hour continuous loss
� Thoracotomy surgery
Flail Chest
� A segment of chest wall looses continuity with rest of the chest
� Hypoxia from lung injury
Diagnosis
� Asymmetric chest wall movement
� Palpation of crepitus
� Chest X ray
Treatment
� Intubation as indicated
� Analgesia
Cardiac Tamponade
� Pericardium a fixed fibrous structure
– Small amounts of blood can restrict ALL cardiac activity
� Distant heart sounds
� Narrow pulse pressure
Diagnosis
� Pulse Paradoxus
� Beck's Triad
– JVD, Muffled Heart tones, Low BP
� Treatment - Pericardiocentesis
– Requires open pericardotomy
Aortic Rupture
� Blunt trauma involving deceleration forces / injury
� ~90% die within minutes
� Most common site is near the ligamentum arteriosum
Diagnosis:
– Clinical suspicion
– CXR
– Aortography
– Contrast CT or TEE
� Treatment
– Surgical…poor prognosis
Abdomen Trauma
Abdominal Injuries 2 types
Closed
� Bleeding of organs
� Seatbelts can cause internal damage
� Abdomen may become distended w / blood
Open
� Abdominal cavity has been opened
� Very important to watch for infection
� Abdominal organs may protrude out of body
Frequency of Organ Injury
1. Spleen 46%
2. Liver 33%
3. Mesentery 10%
4. Urological 9%
5. Pancreas 9%
6. Small bowel 8%
7. Colon 7%
8. Duodenum 5%
Eviscerated Bowel Treatment
� Position patient on their back – Knees flexed & head & shoulders slightly raised to take the
pressure off the abdomen
� Expose the wound
� DO NOT try to reduce the abdominal organs in the cavity
� Cover the exposed organs with sterile moist gauze
� Cover the gauze with plastic wrap to keep in moisture
� Cover the wrap with abdominal pads to maintain warmth
� Place bulky dressings around protruding organs
� Tape dressings in place
� DO NOT put any pressure on exposed wounds
Diffuse Abdominal Pain Differential Diagnosis
� AAA� Renal artery � Vein thrombosis� Acute appendicitis� Bowel obstruction� Retroperitoneal Bleed� Mesenteric Ischemia � GI tract perforation� Colitis
– Chrons Disease – Ulcerative Colitis
Abdomen Trauma
� A Normal initial exam means nothing
� Unexplained hypotension
– Viscous Injury
– Intra abdominal bleed
� Retroperitoneal Area Injury
– Splenic Bleed
– Renal Laceration
Cullen's Sign
� Superficial edema and bruising in the subcutaneous fatty tissue around the umbilicus
� Ecchymoses in the periumbilical region
� 24 - 48 hours to appear
Abdominal Trauma Signs
Kehr's Sign
� Acute pain in the tip of the shoulder
� Presence of blood or irritants in the peritoneal cavity when a person is lying down and the legs are elevated
� Classical referred pain of a ruptured spleen
Abdominal Trauma Signs
Grey Turner's Sign
– Bruising of the flank, may be indicative of Pancreatic necrosis with retroperitoneal or intra-abdominal bleeding
Cullen's or Turner's sign
– Occurs in approximately 3 % of patients
– Mortality of 37 %
– Retroperitoneal Bleeding
Trauma in Pregnancy
Trauma in Pregnancy
� 34% of traumatic death cases due to MVC
� Amount of blood loss to have clinical signs is greater than nonpregnant
� Pelvic Fracture are highly concern because
� Risk of massive hemorrhage
� Nausea and vomitting
� Supine hypotensive syndrome
� Greater risk of pulmonary edema
� Kleihauer-Betke test
Placental Abruption
� Most likely cause of preterm labor in trauma patients
� Findings
– Contraction
– Fetal tachycardia
– Late deceleration
– Fetal death
Management in Pregnancy� Tachycardia & hypotension are not accurate indicators of shock
� 30% or 2 L blood volume loss - Unstable
� Vasoconstrictive agents
� Use ephedrine & phenylephrine
� Epinephrine & norepinephrine cause
� Uteroplacental vasoconstriction
� Reposition uterus after 20 weeks
Management of Pregnancy
� Non Viable Fetus < 23 wks or < 500 g
� Supportive care
� Close observation
Fetus viable & mother unstable
� Exploratory laparoscopy
� Attempt delivery within 6 minutes after maternal cardiac arrest via C-Section
� CPR continued during and after delivery
Perineum / Rectum
� Examine for Contusions, hematomas, lacerations and urethral bleeding
� High riding prostate
� Blood in bowel lumen
� Vaginal lacerations
Extremity Trauma
Extremity
� Check for Pulses
� Save severed limb and place in plastic bag and place ice in the bag surrounding the limb
� Check capillary refill
� Splint wounds
� Pain Management
Musculoskeletal / Extremity� Bones should be palpated for any deformity
� Examine the patients back !
� Ligament disruption will cause joint instability
� Pelvic fractures can have ecchymosis over the iliac wings, pubis, labia, or scrotum– Pain to palpation to the pubic ring
– Mobility of the pelvis to gentle anterior to posterior pressure suggests pelvic ring Fracture
– If Unstable consider pelvic binder
Musculoskeletal Compartment Syndrome
� Compression of nerves, vessels and muscle inside a closed space - compartment
� This leads to tissue death from the lack of oxygenation as the blood vessels are compressed by the raised pressure in the compartment – Most commonly in the forearm and lower leg
� Striker with pressures > 30 mmHg
� <30 mmHg difference between intracompartmental pressure and DBP is an indication for fasciotomy
Compartment Syndrome
� There are classically 6 "Ps“ :
– Pain out of proportion
– Pallor
– Paralysis
– Pulselessness
– Poikilothermia - failure to thermoregulate
– Paresthesia
� Paresthesia, is a late symptom
� Only the first two are reliable in the diagnosis
Thermal Injuries
Thermal
� Rule of Nines
– Parkland Formula
– Pain management
� Fluid resuscitation is critical < 24 hours
� Airway protection
� Look for singed nasal or facial hairs
� If Uncertain Intubate
Parkland Formula for Treating Burn Victims
� Fluid resuscitation is determined from the percentage BSA involved
� "Rule of 9's" estimates the % BSA
Fluid Requirements for first 24 hours
� TBSA burned(%) x Wt (kg) x 4mL
� Give 1/2 of total requirements in 1st 8 hours
� Give 2nd half over next 16 hours
Neurologic Trauma
Neurologic
� Motor and Sensory Exam
� Revaluation of LOC / GCS
� Tracheal Intubation increases ICP
� Pupillary size
� Paralysis
� Immobilization of entire patient
� Rigid longboard, semi rigid c-collar
Glasgow Coma Scale
Eye opening (E)� – Spontaneous 4� – To speech 3� – To pain 2� – None 1
Verbal response (V)� – Oriented 5� – Confused conversation 4� – Inappropriate words 3� – Incomprehensible sounds 2� – None 1
Best motor response (M)� – Obeys commands 6� – Localizes pain 5� – Normal flexion (withdrawal) 4� – Abnormal flexion (decorticate) 3� – Extension 2� – None (flaccid) 1
Spinal Cord Injuries
Complete injury
� NO motor or sensory impulses pass below lesion
� 50% of injuries
Incomplete injury
� Some signals pass– May have some motor and sensation below level
� Injury > T1 = Quadriplegia
� Injury < T1 = Paraplegia
Cauda Equina Syndrome
� Cauda equina or "horse-tail“ is the mass of nerves after the conus
� Acute loss of function of the lumbar plexus below the termination of the conus of the spinal cord
� Saddle anesthesia
� Lower extremity parasthesia
� Sciatica
� Regeneration possible
� Treatment is surgical decompression
Secondary Survey Adjuncts
� Constant Revaluation
� CPP > 60 mm / Hg
� Adult UO = 0.5 mL / Kg / hour
� Pediatric UO = 1 mL / Kg / hour
� Transfer to a higher level facility if needed
ATLS Summary
� Bleeding control
� C- Spine control
� Splint
� IVF – NS / LR
� Hespan
� Permissive Hypotension
� Blood products
� Pressers if needed
ATLS Summary
� Standard for trauma care for hospitals and advanced paramedical services
� Treat the greatest threat to life first
� Lack of a definitive diagnosis and a detailed history should not slow the application of indicated treatment for life-threatening injury
� Most time-critical interventions performed early
� ATLS improves patient outcomes
Thanks
Please complete the post test and return to administration via interoffice mail or fax to: 352-735-4475 attention Scott Temple
This program is worth 2 CEUs in the Trauma or Electives categories
Please specify on your post test where you would like the CEUs to be inserted.