carbohydrate – modified diets for malabsorption syndromes
TRANSCRIPT
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NUTRITIONAL CARE FOR
MALABSORPTION SYNDROMES
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NUTRITIONAL CARE FOR MALABSORPTION SYNDROMES
A. LACTOSE-RESTRICTED DIET
B. POSTGASTRECTOMY DIET
C. GLUTEN- FREE DIETS
- Which relieves the uncomfortable symptoms of lactose intolerance.
- Controls malabsorption that may occur following surgery of the stomach
- Reverse malabsorption caused by celiac disease.
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LACTOSE-RESTRICTED DIETS
Are highly individualized diets that most often limit, but do not exclude, milk and milk products.
Treat malabsorption caused by a deficiency of lactase, the enzyme that splits lactose to glucose and galactose.
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MEDICAL NUTRTION THERAPY FORLACTOSE INTOLERANCE
Clients should test their tolerance for lactose by gradually increasing consumption of lactose-containing foods the point that precipitates symptoms of lactose intolerance- bloating, cramps, and diarrhea.
Include 1 or 2 cups of milk per day, provided that the milk is taken with food and in small at a time
Yogurt contains bacteria that produce lactase and thus help digest lactose.
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POSTGASTRECTOMY DIET
- POSTGASTRECTOMY following surgery that removes all (total
gastrectomy) or part (subtotal or partial gastrectomy) of the stomach.
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POSTGASTRECTOMY DIET
This diet aims: to provide the energy and nutrients
necessary to support recovery and minimize complications associated with gastric surgery.
One complication that can arise when the portion of the stomach containing the pyloric sphincter has been removed, bypassed, or disrupted is the DUMPING SYNDROME.
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DUMPING SYNDROME
Rapid emptying of the stomach contents into the small intestine.
The symptoms that result from the rapid entry of undigested food into the jejunum:
3D’s Diaphoresis Drowsiness Diarrhea
Hypoglycemia (late sign)
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DIETARY MANAGEMENT OF DUMPING SYNDROME
Modifications: Rationale:
Chew foods thoroughly and eat slowly.
Avoid concentrated sweets
Because the body digests and absorbs these carbohydrates rapidly and breaks them down into many particles that draw fluids from the body into the intestine.
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DIETARY MANAGEMENT OF DUMPING SYNDROME
Modifications: Rationale:
SFF
Drink fluids in small amounts about 45 mins., Before or after meals.
To fit the reduced storage capacity of the stomach. THUS, IT PREVENTS
DUMPING OF FOOD INTO THE INTESTINE
To prevent overloading the stomach’s reduced storage capacity and slows the transit of food from the stomach to the intestine.
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DIEATRY MANAGEMENT OF DUMPING SYNDROME
Modifications: Rationale: Dry solid diet
Lie down immediately after eating for 30 to 60 mins.
Be aware that lactose intolerance may develop and add to the problem diarrhea and abdominal pain
Better than liquids as they enter the jejunum less rapidly.
To help to slow transit of food to the intestine. Clients who experience reflux, however, should not lie down after eating.
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DIETARY MANAGEMENT OF DUMPING SYNDROME
Modifications: Rationale:
DIET: HIGH FAT
HIGH CHON
LOW CHO
Delays emptying of the stomach
Increase colloidal osmotic pressure and prevent shifting of plasma
No simple sugars
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POP QUIZ
Management of dumping syndrome is best accomplished by planning to maintain the patient on a:
A. Low CHON, High CHO
B. Low residue, bland diet
C. Fluid intake of at least 500 ml with each meal
D. Small frequent feeding schedule
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GLUTEN – FREE DIETS
GLUTEN A protein found in wheat.
Eliminates certain grains that inflame the intestine and lead to malabsorption for people with CELIAC DISEASE.
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CELIAC DISEASE
Also called Celiac sprue/ Gluten-sensitive enteropathy PERMANENT inability to tolerate dietary
gluten in the small intestine.
A sensitivity to a part of the protein gluten that causes flattening of the intestinal villi and malabsorption.
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DIETARY MANAGEMENT OF CELIAC DISEASE
Modifications: Rationale:
HIGH Calories HIGH CHON
Gluten free diet
Low Fat
To compensate for weight loss
Gluten is the toxic factor
Impaired fat absorption
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TREATMENT FOR CELIAC DISEASE
Lifelong adherence to a gluten free diet.
Serves as the primary treatment for celiac disease.
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ELIMINATE B-R-O-W
B BARLEY
R RYE
O OAT
W WHEAT
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POP QUIZ
Which of the statement needs further teaching, if made by a parent of a two year old child recently diagnosed with celiac disease?
A. “I don’t have to make any major dietary changes until my child reaches puberty.”
B. “I have to keep my child on a gluten-free diet to prevent episodes of diarrhea.”
C. “I should read the labels on any processed foods I offer to my child.”
D. “I will need to notify my child’s school about the special diet.”
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DIETARY MANAGEMENT OF DISEASES OF THE:
GALLBLADDER
PANCREAS
LIVER
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GALLBLADDER
LIVER
PRODUCES BILE (CHOLESTEROL, BILE SALTS, BILIRUBIN &
H2O)
BILE DUCT GALLBLADDER
REMAINS UNTIL NEEDED FOR FAT DIGESTION
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GALLBLADDER
CHOLELITHIASIS The formation or presence of stones in
the gallbladder or CBD.
CHOLECYSTITIS Is the inflammation of the gallbladder.
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PREDISPOSING FACTORS: 5 F’S
FEMALE
FAT (OBESE)
FAIR (CAUCASIAN)
FORTY
FERTILE (MULTIGRAVIDA)
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CHOLELITHIASIS WITH CHOLECYSTITIS
DIETARY MANAGEMENT: RATIONALE:
IV fluids and electrolytes – Clear liquid
Low calorie
To rest inflamed gallbladder
To prevent and correct dehydration and electrolyte abnormalities
For obese patients
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CHOLELITHIASIS WITH CHOLECYSTITIS
Dietary management: Rationale:
LOW FAT, BLAND To prevent stimulation and contraction of the gallbladder which causes pain.
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High fat diet enters into the duodenum
Stimulates the pancreas
To release CHOLECYSTOKININ(a hormone is carried by way of the bloodstream to the
GALLBLADDER)
Forces to contract to release BILE
Needed for the EMULSIFICATION OF FATS
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POP QUIZ
Which of the following factors, if noted in patient’s history, would indicate a predisposition for developing cholecystitis?
A. Hypertension
B. Depression
C. Obesity
D. Childlessness
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PANCREAS
FUNCTIONS Secretes the enzymes necessary for the
digestion of CHO, CHON and FAT , together with bicarbonate-rich juices that provide the optimal pH necessary to activate these enzymes.
Stores digestive enzymes in an inactive form to protect itself from digestion.
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PANCREATITIS
The pancreas becomes inflamed, digestive enzymes are activated within the pancreas, and the enzymes damage the pancreas itself.
The blood picks up some of these enzymes; thus serum amylase and lipase rise and serve as indicators of pancreatitis
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CAUSES:
Alchohol abuse Drugs (antihypertensives, diuretics, oral
contraceptives) Biliary Obstruction Autoimmune Intestinal Diseases Unknown
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ACUTE PANCREATITIS
Most often develops as a consequence of gallstones or alcoholism
Sudden severe abdominal pain, n/v and diarrhea often accompany acute pancreatitis
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CHRONIC PANCREATITIS
When severe pancreatitis or repeated episodes of pancreatitis permanently damage the pancreas, absorption, especially of fat, becomes permanently damage.
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DIETARY MANAGEMENT OF PANCREATITIS
Modifications: Rationale:
NPO during acute phase
IVF Therapy
NGT
To prevent stimulation pancreatic stimulations.
To manage fluid deficit
To remove gastrin from the stomach and secretin from the duodenum and help relieve pain and digestion.
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DIETARY MANAGEMENT OF PANCREATITIS
Modifications: Rationale:
Alcohol is restricted
Low Fat, bland
SFF
Enzyme replacements
To prevent further damage to the pancreas.
To control steatorrhea and prevent stimulation for bile production.
Avoid distention.
Taken with meals Helps digest and absorb
CHON and fat while minimizing steatorrhea.
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POP QUIZ
THE DIET OF THE CLIENT WITH PANCREATITIS SHOULD BE:
A. HIGH FIBER, LOW FAT DIET
B. HIGH CALORIE, HIGH FAT DIET
C. LOW FAT, BLAND DIET
D. HIGH PROTEIN, HIGH FAT DIET
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CYSTIC FIBROSIS
A hereditary disorder characterized by the production of thick mucus(exocrine glands) that affects many organs including the pancreas, lungs, liver, heart, gallbladder, and small intestine.
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CONSEQUENCES OF CYSTIC FIBROSIS Produce secretions of thick, sticky
mucus Impairs the function of lungs and
pancreas
Nutrition implication: Chronic lung disease Malabsorption Loss of electrolytes in sweat
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NUTRITION THERAPY FOR CYSTIC FIBROSIS
Dietary management: Rationale:
High Calories
High CHON
Low Fat
Provide for weight gain.
To compensate for the loss.
Lack of enzyme to digest.
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NUTRITION THERAPY FOR CYSTIC FIBROSIS
Dietary management: Rationale
Enzyme Replacements
Liberal use of salt
Increase fluid intake
To provide deficient pancreatic enzymes
Help control steatorrhea
To replace the losses of electrolytes in the sweat
To liquefy thick secretions
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LIVER
FUNCTIONS:
Metabolizes carbohydrate, protein, and fat so that energy is available to body cells.
Stores glycogen, most vitamins, and many minerals.
Manufactures cholesterol, which serves as a precursor for steroid hormones.
Packages lipids in lipoproteins for transport throughout the body.
Manufactures bile to aid fat digestion.
Makes nonessential amino acid and keeps amino acid composition in the balance for energy use.
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LIVER
Converts amino acid to glucose for energy use.
Converts ammonia from the blood to urea so that it can excreted by the kidneys.
Makes plasma proteins including lipoproteins, clotting and immune system factors, and protein that carry nutrients throughout the body.
Activates many nutrients.
Detoxifies drugs (including alcohol) and other substances that are harmful to the body.
Dismantles worn-out red blood cells and recycles the iron they contain.
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HEPATITIS
Any acute inflammation disease of the liver.
TYPES: ACUTE- viral, drug-induced, toxic CHRONIC- active or persistent
It can be caused by viruses, bacteria or toxic injury to the liver.
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HEPATITIS
2.) TOXIC HEPATITIS Can be caused by drugs, alcohol,
industrial toxins and plant poisons.
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B. CHRONIC
1.) ACTIVE A d/o characterized by continuing hepatic
necrosis, active inflammation and fibrosis which may lead to cirrhosis.
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2.) PERSISTENT
Rare extra hepatic involvement, may be secondary to viral hepatitis.
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MEDICAL NUTTRITION THERAPY
The person with hepatitis:
Good nutrition status
Malnourished
Anorexia
vomiting
Regular, well-balanced diet
High calorie, high protein
SFF
IV fluids replacement
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LIVER CIRRHOSIS
Slow destruction of liver cells and proliferation of fibrous tissues eventually leading to scarring of the liver tissues.
Characterized by:jaundice ascitesCNS dysfunctioncachexiaportal HPN esophageal
varicessplenomegaly
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DIETARY MANAGEMENT OF LIVER CIRRHOSIS
Modifications: Rationale:
High CHON
High CHO Low Fat
Low Na Fluid restriction
To regenerate hepatic cells
To prevent hypoproteinemia
To spare CHON To prevent fatty liver
To prevent ascites and edema
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DIETARY MANAGEMENT OF LIVER CIRRHOSIS
Modifications: Rationale:
SFF
Full liquid diet
Thiamin supplements
Better tolerance
To prevent irritation of esophageal varices.
To prevent complications: WERNICKE-KORSAKOFF
SYNDROME
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POP QUIZ
On admission, a patient is having a symptoms of pancreatitis. Which of the following would have most likely contribute to this diagnosis
A. Increased CHON intake B. Smoking C. Excessive alcohol ingestion D. High cholesterol level
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