chapter 14
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Chapter 14. Training for Sport. Chapter 14 Overview. Optimizing training: a model Overreaching Excessive training Overtraining Tapering for peak performance Detraining. Training for Sport: Introduction. Positive stress: training that causes improvements in exercise performance - PowerPoint PPT PresentationTRANSCRIPT
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Training for Sport
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CHAPTER 14 CHAPTER 14 OverviewOverview
• Optimizing training: a model
• Overreaching
• Excessive training
• Overtraining
• Tapering for peak performance
• Detraining
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Training for Sport: IntroductionTraining for Sport: Introduction
• Positive stress: training that causes improvements in exercise performance– Major training adaptations in 6 to 10 weeks– Depends on volume and intensity of training– Quantity training versus quality training
• Rate of adaptation genetically limited– Too much versus just right varies– Too much training performance and injury
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Training for Sport: IntroductionTraining for Sport: Introduction
• Must balance volume and intensity– Must include rest– Correct balance enhances performance
• Overtraining performance decrements– Chronic fatigue, illness– Overuse injury, overtraining syndrome
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Optimizing Training: A ModelOptimizing Training: A Model
• Must include progressive overload– Progressively stimulus as body continually adapts– Stimulates continuous improvements
• Undertraining: insufficient stimulus– Adaptations not fully realized– Optimal performance not achieved
• Overtraining: loss of benefits– No additional improvements– Performance decrements, injury
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Optimizing Training: A ModelOptimizing Training: A Model
• Undertraining: off-season
• Acute overload: average training load
• Overreaching: decrement, then benefit
• Overtraining: maladaptations– Performance decrements– Overtraining syndrome, excessive training
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Figure 14.1Figure 14.1
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OverreachingOverreaching
• Systematic attempt in overstressing body for short period of training– Allows body to adapt to stronger stimulus– Not same as excessive training– Caution: easy to cross into overtraining
• Short performance decrement followed by improved performance and function
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Excessive TrainingExcessive Training
• Volume and/or intensity to an extreme – For years, many athletes undertrained– As intensity/volume , so did performance– But more is better is not true after a point
• Example: swim training 3 to 4 h/day no better than 1 to 1.5 h/day
• Can lead to strength, sprint performance
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Excessive TrainingExcessive Training
• Another swim study: single versus multiple daily training sessions
• No evidence that more is better– Similar heart rate and blood lactate improvements– No additional improvements from 2 times/day
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Figure 14.3Figure 14.3
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Excessive TrainingExcessive Training
• Training volume should be sport specific
• Value of high-volume training questionable– In some sports, half the volume may maintain
benefits and risk– Low intensity, high volume inappropriate for sprint-
type performance
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Excessive TrainingExcessive Training
• Intensity and volume inversely related– If volume , intensity should – If intensity , volume should – Different emphasis different fitness results– Applies to resistance, anaerobic, and aerobic
training
• Intensity + volume negative effects
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OvertrainingOvertraining
• Unexplained in performance, function for weeks, months, or years
– Cannot be remedied by short-term training, rest– Putative psychological and physiological causes– Can occur with all forms of training: resistance,
anaerobic, aerobic
• Not all fatigue product of overtraining
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Overtraining SyndromeOvertraining Syndrome
• Highly individualized, subjective
• Symptoms– Strength, coordination, capacity– Fatigue– Change in appetite, weight loss– Sleep and mood disturbances– Lack of motivation, vigor, and/or concentration– Depression
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Overtraining SyndromeOvertraining Syndrome
• Can be intensity or volume related
• Psychological factors– Emotional pressure of competition stress– Parallels with clinical depression
• Physiological factors– Autonomic, endocrine, and immune factors– Not a clear cause-and-effect relationship but
significant parallels
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Figure 14.4Figure 14.4
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Overtraining Syndrome: Sympathetic Overtraining Syndrome: Sympathetic Nervous System ResponsesNervous System Responses
• Increased BP
• Loss of appetite
• Weight loss
• Sleep and emotional disturbances
• Increased basal metabolic rate
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Overtraining Syndrome: PNS Overtraining Syndrome: PNS ResponsesResponses
• More common with endurance athletes
• Early fatigue
• Decreased resting HR
• Decreased resting BP
• Rapid heart rate recovery
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Overtraining Syndrome:Overtraining Syndrome:Endocrine ResponsesEndocrine Responses
• Resting thyroxine, testosterone
• Resting cortisol
• Testosterone:cortisol ratio– Indicator of anabolic recovery processes– Altered ratio may indicate protein catabolism– Possible cause of overtraining syndrome
• Volume-related overtraining appears more likely to affect hormones
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Figure 14.5Figure 14.5
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Overtraining Syndrome:Overtraining Syndrome:Endocrine ResponsesEndocrine Responses
• Blood urea concentration
• Resting catecholamines
• Outside factors may influence values– Overreaching may produce same trends– Time between last training bout and resting blood
sample critical– Blood markers helpful but not definitive diagnostic
tools
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Overtraining Syndrome:Overtraining Syndrome:Neural and Endocrine FactorsNeural and Endocrine Factors
• Overtraining stressors may act primarily through hypothalamic signals– Can lead to sympathetic neural activation– Can lead to pituitary endocrine cascade
• Hormonal axes involved– Sympathetic-adrenal medullary (SAM) axis– Hypothalamic-pituitary-adrenocortical (HPA) axis
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Overtraining Syndrome:Overtraining Syndrome:Immune ResponsesImmune Responses
• Circulating cytokines– Mediate inflammatory response to infection and
injury– In response to muscle, bone, joint trauma– Physical stress + rest systemic inflammation
• Inflammation cytokines via monocytes
• May act on brain and body functions, contribute to overtraining symptoms
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Figure 14.6Figure 14.6
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Overtraining Syndrome:Overtraining Syndrome:Immune ResponsesImmune Responses
• Compromised immune function factor in onset of overtraining syndrome
• Overtraining suppresses immune function– Abnormally lymphocytes, antibodies– Incidence of illness after exhaustive exercise– Exercise during illness immune complications
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Figure 14.7Figure 14.7
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Overtraining Syndrome, Fibromyalgia, Overtraining Syndrome, Fibromyalgia, and Chronic Fatigue Syndromeand Chronic Fatigue Syndrome
• Three similar, overlapping syndromes– Notoriously difficult to diagnose– Causes remain unknown
• Similar symptoms– Fatigue– Psychological distress– Endocrine/HPA, neural, and immune dysfunction
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Predicting Overtraining SyndromePredicting Overtraining Syndrome
• Causes unknown, diagnostics difficult• Threshold different for each athlete• Most coaches and trainers use (unreliable)
intuition• No preliminary warning symptoms
– Coaches do not realize until too late– Recovery takes days/weeks/months of rest
• Biological markers have limited effectiveness
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Table 14.1Table 14.1
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Table 14.1 Table 14.1 (continued)(continued)
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Figure 14.8Figure 14.8
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Overtraining SyndromeOvertraining Syndrome
• Treatment– Reduced intensity or rest (weeks, months)– Counseling to deal with stress
• Prevention– Periodization training– Adequate caloric (especially carbohydrate) intake
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Overtraining:Overtraining:Exertional RhabdomyolysisExertional Rhabdomyolysis
• Acute (potentially lethal) condition
• Breakdown of skeletal muscle fibers– In response to unusually strenuous exercise– Often similar to DOMS– Severe cases cause renal failure (protein leakage)– Exacerbated by statin drugs, alcohol, dehydration
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Overtraining:Overtraining:Exertional RhabdomyolysisExertional Rhabdomyolysis
• Signs and symptoms– Severe muscle aches (entire body)– Muscle weakness– Dark or cola-colored urine
• Can reach clinical relevancy– Rare, usually reported in case studies– Requires hospitalization– Precipitated by excessive eccentric exercise
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Tapering for Peak PerformanceTapering for Peak Performance
• Tapering = reduction in training volume/intensity– Prior to major competition (recovery, healing)– 4 to 28 days (or longer)– Most appropriate for infrequent competition
• Results in increased muscular strength– May be associated with contractile mechanisms– Muscles repair, glycogen reserves replenished
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Tapering for Peak PerformanceTapering for Peak Performance
• Does not result in deconditioning– Considerable training to reach VO2max
– Can reduce training by 60% and maintain VO2max
• Leads to improved performance– 3% improved race time – 18 to 25% improved arm strength, power– Effects unknown on team sports, marathons
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DetrainingDetraining
• Loss of training-induced adaptations – Can be partial or complete– Due to training reduction or cessation– Much more substantial change than tapering
• Brief period = tapering
• Longer period = detraining
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DetrainingDetraining
• Immobilization– Immediate loss of muscle mass, strength, power
• Training cessation– Rate of strength and power loss varies
• Causes– Atrophy (immobilization)– Reduced ability to recruit muscle fibers– Altered rates of protein synthesis versus degradation
• Low-level exercise mitigates loss
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DetrainingDetraining
• Muscle endurance quickly– Change seen after 2 weeks of inactivity– Not clear whether the result of muscle or
cardiovascular changes
• Oxidative enzyme activity by 40 to 60%
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Figure 14.9Figure 14.9
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DetrainingDetraining
• Muscle glycogen stores by 40%
• Significant acid-balance imbalance. Exercise test once weekly during detraining showed– Blood lactate accumulation – Bicarbonate – pH
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Figure 14.10Figure 14.10
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Table 14.2Table 14.2
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DetrainingDetraining
• Training only moderate speed, agility
• Detraining only moderate speed, agility– Form, skill, flexibility also lost– Sprint performance still suffers
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DetrainingDetraining
• Significant cardiorespiratory losses
• Based on bed rest studies– Significant submaximal HR– 25% submaximal stroke volume (due to plasma
volume)– 25% maximal cardiac output– 27% VO2max
• Trained athletes lose VO2max faster with detraining, regain it slower
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Figure 14.11Figure 14.11
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DetrainingDetraining
• How much activity is needed to prevent losses in physical conditioning?
• Losses occur when frequency and duration decrease by 2/3 of regular training load
• 70% VO2max training sufficient to maintain maximal aerobic capacity
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Detraining in SpaceDetraining in Space
• Microgravity exposure = detraining– Normal gravity challenges heart and muscles– Detraining may be beneficial in space
• Muscle mass and strength – Particularly postural muscles– Type I, II fiber cross-sectional area – Without muscle stress, bone loss ~4%
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Detraining in SpaceDetraining in Space
• Stroke volume – Less hydrostatic pressure, blood does not pool in
lower extremities– More venous return
• Total blood volume – Plasma volume due to fluid intake, capillary
filtration
– Red blood cell mass – In space beneficial adaptation– On earth orthostatic hypotension
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Detraining in SpaceDetraining in Space
• VO2max immediately postflight– Due to plasma volume and leg strength– Preflight, in-flight VO2max data unknown
• With bed rest, VO2max due to– Total blood volume– Plasma volume and maximal stroke volume
• In-flight exercise essential to preserve astronauts’ long-term health