congestive heart failure sagar naik
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CONGESTIVE HEART FAILURE Sagar Naik, PT
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CONGESTIVE HEART FAILURE
Sagar Naik, PT
Heart failure is a pathophysiologic state in which an abnormality of
cardiac function is responsible for the failure of the heart to pump blood
at rate commensurate with the requirements of the metabolizing tissues
or can do so only from an abnormally elevated filling pressure.
It can be thought of as a clinical syndrome comprising a constellation of
symptoms and signs attributable to cardiac dysfunction.
Etiology:
Heart failure is frequently, but not always, caused by a defect in myocardial
contraction.
It may result from
• Primary abnormality in the heart muscle
Cardiomyopathies
• Extramyocardial abnormalities
Coronary atherosclerosis
• Abnormalities of the heart valves
Mitral stenosis
A similar clinical syndrome may be present without any detectable abnormality of
myocardial function.
• Acute hypertensive crisis
• Rupture of aortic valve cusp
• Massive pulmonary embolism
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In addition, conditions associated with impairment of filling of the ventricles
like tricuspid or mitral stenosis, constrictive pericarditis and endocardial
fibrosis can lead to heart failure in presence of normal myocardial function.
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CONGESTIVE HEART FAILURE Sagar Naik, PT
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Precipitating Causes:
• Infection
• Anemia
• Thyrotoxicosis, Pregnancy & Obesity
• Arrhythmias
• Rheumatic, viral, & other forms of myocarditis
• Infective endocarditis
• Physical, dietary, fluid, environmental, & emotional excesses
• Systemic hypertension
• Myocardial infarction
• Pulmonary embolism
• Drugs induced
Classification of Heart Failure:
Heart failure can be classified or described in several ways, which are as follows:
High Output VS Low Output Heart Failure:
The low output heart failure i.e., heart failure with low cardiac output is
seen in patients with heart failure secondary to
• Ischaemic heart disease
• Dilated Cardiomyopathy
• Valvular & Pericardial diseases
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• Hypertension
The high output heart failure i.e., heart failure with high cardiac output is
seen in patients with
• Hyperthyroidism
•Anemia
• Pregnancy
• Arteriovenous fistulas
• Beriberi physio4all...
CONGESTIVE HEART FAILURE Sagar Naik, PT
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• Paget’s disease
In clinical practice, however, low-output and high-output HF cannot always be
readily distinguished.
The normal range of cardiac output is wide [2.2 to 3.5 (L/min)/m2]; in manypatients with so-called low-output heart failure, the cardiac output may actually be
just within the normal range at rest (although lower than it had been previously),
but it fails to rise normally during exertion.
On the other hand, in patients with so-called high-output heart failure, the output
may not exceed the upper limits of normal (although it would have been elevated
had it been measured before heart failure supervened); rather, it may have fallen to
within normal limits.
Acute VS Chronic Heart Failure:
The acute heart failure is the sudden development of a large myocardial
infarction or rupture of a cardiac valve in a patient who previously was
entirely well.
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Chronic heart failure is typically observed in patients with dilated
cardiomyopathy or multivalvular heart disease that develops or
progresses slowly.
Acute heart failure is usually predominantly systolic, and the sudden reduction
in cardiac output often results in systemic hypotension without peripheral
oedema.
In contrast, in chronic heart failure, arterial pressure is ordinarily well
maintained until very late in the course, but there is often accumulation of
oedema.
Right-Sided VS Left-Sided Heart Failure:
Many of the clinical manifestations of heart failure result from the accumulation of
excess fluid behind either one or both ventricles.
This fluid usually localizes upstream to (behind) the ventricle that is initially
affected.
Patients in whom the left ventricle is hemodynamically overloaded (e.g., aortic
stenosis) or weakened (e.g., postmyocardial infarction) develop dyspnoea and
orthopnoea as a result of pulmonary congestion, a condition referred to as
left-sided heart failure.
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In contrast, when the underlying abnormality affects the right ventricle primarily
(e.g., congenital valvular pulmonic stenosis or pulmonary hypertension secondary
to pulmonary thromboembolism), symptoms resulting from pulmonary
congestion are uncommon, and edema, congestive hepatomegaly, and
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systemic venous distention, i.e., clinical manifestations of right-sided heart
failure, are more prominent.
When heart failure has existed for months or years, such localization of excess fluid
behind the failing ventricle may no longer exist.
E.g. – Patients with long-standing aortic valve disease or systemic hypertension may
develop ankle edema, congestive hepatomegaly, and systemic venous distention
late in the course of their disease, even though the abnormal hemodynamic burden
initially was placed on the left ventricle.
Backward VS Forward Heart Failure:
The concept of backward heart failure contends that in heart failure, one or the other ventricle fails to discharge its contents or fails to fill normally .
As a consequence, the pressures in the atrium and venous system behind the failing
ventricle rise, and retention of sodium and water occur as a consequence of the
elevation of systemic venous and capillary pressures and the resultant transudation
of fluid into the interstitial space.
In contrast, the proponents of the forward heart failure hypothesis maintainthat the clinical manifestations of heart failure result directly from an
inadequate discharge of blood into the arterial system.
According to this concept, salt and water retention is a consequence of diminished
renal perfusion and excessive proximal tubular sodium reabsorption and of
excessive distal tubular reabsorption through activation of the renin-angiotensin-
aldosterone (RAA) system.
Systolic VS Diastolic Heart Failure:
The distinction between these two forms of heart failure, relates to whether the
principal abnormality is the inability of the ventricle to contract normally
and expel sufficient blood (systolic failure) or to relax and/or fill normally
(diastolic failure).
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CONGESTIVE HEART FAILURE Sagar Naik, PT
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The major clinical manifestations of systolic failure relate to an inadequate
cardiac output with weakness, fatigue, reduced exercise tolerance, and
other symptoms of hypoperfusion, while in diastolic failure the manifestations
relate principally to the elevation of filling pressures.
Clinical Features:
Symptoms:
• Dyspnoea on exertion
• Orthopnoea
• Paroxysmal nocturnal dyspnoea
• Cheyne-Stokes respiration (Periodic or cyclic respiration)
• Fatigue & weakness
• Anorexia & Nausea associated with abdominal pain
• Weight loss
• Cerebral Symptoms
Confusion
Difficulty in concentration
Impairment of memory
Headache
Insomnia
Anxiety
Nocturia
Physical Signs:
• Pulse
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Pulse pressure may be diminished (Severe)
Sinus tachycardia
Pulsus alternans
• Raised Jugular Venous Pressure
• 3rd & 4th heart sounds are often audible but are not specific for heart
failure
• Pulmonary Rales physio4all...
CONGESTIVE HEART FAILURE Sagar Naik, PT
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• Cardiac oedema
• Pleural effusion & ascites
• Congestive hepatomegaly & enlargement of spleen may occur
• Cardiac Cachexia
• Laterally displaced apical impulse
Investigations:
• Chest radiograph
Pulmonary venous hypertension (left ventricular failure) seen as dilatation and
engorgement the upper lobe pulmonary veins
Pleural effusion & interlobal thickening
Cardiomegaly
• ECG
• Echocardiogram
• Cardiac catheterization
• Exercise testing with respiratory gas analysis
Differential Diagnosis:
• Non-cardiogenic pulmonary oedema
• Renal insufficiency with fluid overload
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• Hepatic insufficiency
• Anemia
• Thyrotoxicosis
Treatment:
Medical Management:
The treatment of heart failure may be divided into four components:
• Removal of the precipitating cause
• Correction of the underlying cause
• Prevention of deterioration of cardiac function
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• Control of the congestive HF state
General therapeutic measures to be taken by the patient of heart failure are as
follows:
• Restrict salt intake
• Recommend regular, moderate exercise
• Avoid antiarrhythmic agents for asymptomatic arrhythmias
• Avoid non-steroidal anti-inflammatory agents (NSAIDs)
• Provide influenzal and pneumococcal immunization
Diuretics are generally prescribed for all patients with symptoms of heart failure
who have fluid retention.
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All patients with heart failure due to left ventricular systolic dysfunction should
receive an ACE inhibitor , unless they have been shown to be unable to tolerate
these drugs.
All patients with heart failure due to left ventricular systolic dysfunction should
receive β-adrenergic receptor blockers, unless they are unable to tolerate
treatment with these drugs or have a contraindication to their use.
Digoxin is recommended to improve the clinical status of patients with heart failure
and should be used in conjunction with diuretics, ACE inhibitors or β blockers.
Other drugs, which can be used, are as follows:
• Hydralazine & Isosorbide Dinitrate
• Angiotensin Receptor Blockers
• Aldosterone Antagonists
• Calcium Antagonists
• Antiarrhythmic & Device Therapy
• Anticoagulant Therapy
• Positive Inotropic Therapy
Surgical Management:
• Pacemaker
• Heart transplant physio4all...