deconditioning syndrome.ppt
TRANSCRIPT
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DECONDITIONING SYNDROME BY BED REST
NURYANI SIDARTA
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AIM
• TO KNOW AND UNDERSTAND THE COMPLICATION OF BED REST
• TO PREVENT THE COMPLICATION OF BED REST
• TO KNOW THE PATHOLOGICAL CONDITION THAT MAY INDUCE DECONDITIONING SYNDROME
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INTRODUCTION
• BED REST HAS BEEN ROUTINELY AND OFTEN CASUALLY PRESCRIBED, DURING A PERIOD OF INJURY AND RECOVERY.
• CAUSES A GENERALIZED DECONDITIONING OF THE HEALTY SUBJECT INAL, INVOLVING MOST OF THE PHYSIOLOGICAL SYSTEMS OF THE BODY, INCLUDING THE CARDIOVASCULAR, PULMONARY, GASTROINTESTINAL, HORMONAL, AND SKELETAL SYSTEMS.
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DECONDITIONING
AS REDUCED CAPACITY OF THE MUSCULOSKELETAL AND OTHER BODY
SYSTEMS (DeLisa)
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DECONDITIONING
THE MULTIPLE CHANGES IN ORGAN SYSTEM PHYSIOLOGY THAT ARE INDUCED BY
INACTIVITY AND REVERSED BY ACTIVITY
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IMMOBILITY
MUSCULOSKELETALMUSCULOSKELETAL
CARDIOVASCULAR
CARDIOVASCULAR
SKINSKIN
NEUROLOGICALEMOTIONAL
NEUROLOGICALEMOTIONAL
GASTOINTESTINAL
GASTOINTESTINAL
GENITOURINARYGENITO
URINARY
RESPIRATORYRESPIRATORY
ENDOCRINEENDOCRINEBODY COMPOSITION
BODY COMPOSITION
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CARDIOVASCULAR SYSTEM
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CARDIAC :↓ STROKE VOLUME
↑ HEART RATE↓ CARDIAC OUTPUT
↓ VO2 MAX
CARDIAC :↓ STROKE VOLUME
↑ HEART RATE↓ CARDIAC OUTPUT
↓ VO2 MAX
CARDIOVASCULAR
CARDIOVASCULAR
NEUROVASCULAR:•ORTHOSTATIC INTOLERANCE
NEUROVASCULAR:•ORTHOSTATIC INTOLERANCE
FLUID BALANCE:• ↓ PLASMA VOLUME
•↓ TOTAL BODY VOLUME•↓ RED BLOOD CELL MASS
FLUID BALANCE:• ↓ PLASMA VOLUME
•↓ TOTAL BODY VOLUME•↓ RED BLOOD CELL MASS
BLOOD COAGULATION:• ↓ CALF BLOOD FLOW•↑ VENOUS THROMBOSIS•↑ BLOOD FIBRINOGEN
BLOOD COAGULATION:• ↓ CALF BLOOD FLOW•↑ VENOUS THROMBOSIS•↑ BLOOD FIBRINOGEN
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CARDIO SYSTEMCARDIO SYSTEM
STROKE VOLUME ↓ 30%STROKE VOLUME ↓ 30%
HR ↑ ½ BEAT/MIN/DAY FRO 3-4 WKHR ↑ ½ BEAT/MIN/DAY FRO 3-4 WK
CARDIAC OUTPUT UNCHANGED OR SLIGHTLY DECREASED
CARDIAC OUTPUT UNCHANGED OR SLIGHTLY DECREASED
MAXIMUM OXYGEN CONSUMPTIONDECREASED UP TO 28%
MAXIMUM OXYGEN CONSUMPTIONDECREASED UP TO 28%
ORTHOSTATIC HYPOTENSION
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VAS CULAR
SYSTE M
VAS CULAR
SYSTE M
↓ BLOOD FLOW IN MUSCLE DUE TO ↓MUSCULAR PUMPING
↓ BLOOD FLOW IN MUSCLE DUE TO ↓MUSCULAR PUMPING
↑ BLOOD VISCOSITY RESULTED FROM INCREASED HT AND BLOOD VISCOSITY
INCREASED PLATELET ADHESIVENESSINCREASED PLATELET ADHESIVENESS
INCREASED FIBRINOGEN LEVEL
INCREASED RISK FOR DVT AND PULMONARY EMBOLISMINCREASED RISK FOR DVT AND PULMONARY EMBOLISM
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TILTING TABLE
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• ↑FORCED VITAL CAPACITY•↑ TOTAL LUNG CAPACITY
(SLIGHT)•RESIDUAL VOLUME
UNCHANGED•↑ RESPIRATORY RATE•VITAL CAPACITY
UNCHANGED•FUNCTIONAL RESIDUAL CAPACITY UNCHANGED•PULMONARY EMBOLISM
(POSSIBLE)
• ↑FORCED VITAL CAPACITY•↑ TOTAL LUNG CAPACITY
(SLIGHT)•RESIDUAL VOLUME
UNCHANGED•↑ RESPIRATORY RATE•VITAL CAPACITY
UNCHANGED•FUNCTIONAL RESIDUAL CAPACITY UNCHANGED•PULMONARY EMBOLISM
(POSSIBLE)
RESPIRATORYRESPIRATORY
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TREATMENTTREATMENT
• EARLY MOBILIZATION• FREQUENT POSITION CHANGES• INCENTIVE SPIROMETRY• DEEP BREATHING• ADEQUATE HYDRATION• GOOD DENTAL CARE• CHEST PERCUSSION WITH POSTURAL DRAINAGE• ANTIOBIOTIC
• EARLY MOBILIZATION• FREQUENT POSITION CHANGES• INCENTIVE SPIROMETRY• DEEP BREATHING• ADEQUATE HYDRATION• GOOD DENTAL CARE• CHEST PERCUSSION WITH POSTURAL DRAINAGE• ANTIOBIOTIC
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MUSCLES :•ATROPHY
DECR STRENGTHDECR ENDURANCE•CONTRACTURE
•DECREASED STRENGTHOF TENDON & LIG
MUSCLES :•ATROPHY
DECR STRENGTHDECR ENDURANCE•CONTRACTURE
•DECREASED STRENGTHOF TENDON & LIG
BONE :OSTEOPOROSIS
BONE :OSTEOPOROSIS JOINTS:
•CARTILAGE- DEGENERATION
•FIBROFATTY TISSUE INFILTRATION
•SYNOVIAL ATROPHYANKYLOSIS
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CONTRACTURE SEQUELAE
HIP JOINT FLEXION COMPENSATORY LORDOSIS, KNEE FLEXION, SHORT STEPS
HIP JOINT FLEXION COMPENSATORY LORDOSIS, KNEE FLEXION, SHORT STEPS
HIP JOINT EXT ROTATION EXCESSIVE STRESS ON MEDIAL KNEE LIG.HIP JOINT EXT ROTATION EXCESSIVE STRESS ON MEDIAL KNEE LIG.
ANKLE PLANTAR FLEXION GENU RECURVATUM ABSENCE OF HEEL STRIKEANKLE PLANTAR FLEXION GENU RECURVATUM ABSENCE OF HEEL STRIKE
SHOULDER FLEXION, ADDUCTION CAN NOT REACH BACK POCKET, COMB HAIRAND INTERNAL ROTATION OR REACH ABOVE SHOULDER LEVEL
SHOULDER FLEXION, ADDUCTION CAN NOT REACH BACK POCKET, COMB HAIRAND INTERNAL ROTATION OR REACH ABOVE SHOULDER LEVEL
WRIST & FINGER FLEXION WEAKEND GRIP CANNOT OPEN HAND TO GRASPWRIST & FINGER FLEXION WEAKEND GRIP CANNOT OPEN HAND TO GRASP
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DECREAESED MUSCLE STRENGTH
• STRENGTH DECREASES BY 0.5-1.7%/DAY• MUELLER : 1.0-1.5% OF ISOMETRIC STRENGTH
PER DAY OF BED REST OVER 2 WEEKS• STRENGTH LOSS GENERALLY GREATED IN LEGS
THAN ARMS• ANTIGRAVITY MUSCLES LOSS MORE
STRENGTH
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TENDON AND LIGAMENTSTENDON AND LIGAMENTS
• DECREASED TENSILE STRENGTH• INCREASED COLLAGEN TURN OVER• DECREASED COLLAGE MASS• HISTOLOGIC CHANGES :
1. LONGITUDINAL STRESS TO CONNECTIVE TISSUES FOSTERS PARALLEL LIGAMENT OF FIBERS
2. WITH BED REST, NEWLY FORMED COLLAGEN IS LAID DOWN IN HAPHAZARD ARRANGEMENT
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CALCIUM METABOLISM
• MAINTENANCE OF A SKELETON CAPABLE OF RESISTING THE MECHANICAL FORCES APPLIED DURING ACTIVITY IS DEPENDENT ON THE INTTERMITTEN APPLICATION OF THESE SOME FORCES.
• WHEN STRESS IS APPLIED TO A BONE, AS IN NORMAL ACTIVITY OR EXERCISE, THE STRAIN IS SENSED, WITH SUBSEQUENT CHANGES IN OSTEOCLAST AND OSTEOBLAST CELLULAR ACTIVITY.
• MAINTENANCE OF A SKELETON CAPABLE OF RESISTING THE MECHANICAL FORCES APPLIED DURING ACTIVITY IS DEPENDENT ON THE INTTERMITTEN APPLICATION OF THESE SOME FORCES.
• WHEN STRESS IS APPLIED TO A BONE, AS IN NORMAL ACTIVITY OR EXERCISE, THE STRAIN IS SENSED, WITH SUBSEQUENT CHANGES IN OSTEOCLAST AND OSTEOBLAST CELLULAR ACTIVITY.
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• IF NO FORCE IS APPLIED TO THE SKELETON, EITHER BECAUSE OF PLASTER IMMOBILIZATION, STRICT BED REST, PARALYSIS, OR WEIGHTLESSNESS OF SPACE FLIGHT, BONE MINERAL IS LOSS BECAUSE THE RATE OF BONE FORMATION FALLS BELOW THE RATE OF BONE MINERAL ABSORPTION.
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STUDIES*
• URINARY CALCIUM INCREASED AT A RATE OF 12% PER WEEK
• LOSSES OF TOTAL BODY CALCIUM IN URINE, SWEAT, AND FECES AVERAGED 0.5% PER MONTHS
• PARALLEL LOSSES IN PHOSPHORUS AND HYDROYPROLINE.
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PREVENTION & TREATMENT
• RELATIVELY INTENSE EXERCISE (ISOTONIC & ISOKINETIC) IN BED HELP MAINTAIN MUSCLE STRENGTH
• DAILY ROM EXERCISE OF MUSCLES DELAYS ATROPHY
• EARLY MOBILIZATION AND WEIGHT BEARING ACTIVITY
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BODY COMPOSITION, METABOLISM, NUTRITION
•↓ LEAN BODY MASS•↑ BODY FAT•NITROGEN LOSS• CALCIUM LOSS•PHOSPHORUS LOSS•SULFUR LOSS•POTASSIUM LOSS
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ALTERED CIRCADIAN RHYTYMALTERED CIRCADIAN RHYTYM
ALTERED TEMPERATURE AND SWEATING RESPONSE
ALTERED TEMPERATURE AND SWEATING RESPONSE
ALTERED REGULATION OF HORMON:PARATHYROID, ADRENAL, ANDROGEN,
THYROID
ALTERED REGULATION OF HORMON:PARATHYROID, ADRENAL, ANDROGEN,
THYROID
IMPAIRED GLUCOSE TOLERANCEIMPAIRED GLUCOSE TOLERANCE
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IMPAIRED GLUCOSE TOLERANCE
• BECAUSE OF INACTIVITY, MUSCLE DEVELOPS INSULIN RESISTANCE THAT MAKE REDUCTION IN PERIPHERAL GLUCOSE UPTAKE WHICH CAUSED HYPERGLYCEMIA FOLLOWED BY HYPERINSULINEMIA
• OCCURS AS SOON AS 3 DAYS AFTER BEDREST• DECREASE 20-50% BY 14 DAYS
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GENITOURINARY
↑ MINERAL EXCRETION↑ MINERAL EXCRETION
↑ POSTVOIDRESIDUALVOLUME
↑ POSTVOIDRESIDUALVOLUME
↑ URINARY TRACT
INFECTION
↑ URINARY TRACT
INFECTION↑ OVERFLOW
INCONTINENCE↑ OVERFLOW
INCONTINENCE
↓ ABILITY TOCONCENTRATE
URINE
↓ ABILITY TOCONCENTRATE
URINE
↓ GFR↓ GFR
↑ CALCULUSFORMATION↑ CALCULUSFORMATION
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GASTROINTESTINAL
↓ FLUID INTAKE↓ FLUID INTAKE
↓ APPETITE↓ APPETITE
↓ BOWELMOBILITY↓ BOWELMOBILITY
↓ GASTRIC SECRETION
↓ GASTRIC SECRETION
CONSTIPATIONCONSTIPATION
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GI ATLERATIONS• DECREASED TRANSIT TIME• SLOWED BY UP TO 66% IN LYING POSITION
COMPARED WITH STANDING• INCREASED GASTRIC ACIDITY BECAUSE OF
ACTIVATION OF THE SYMPATHETIC SYSTEM INHIBITION OF GASTRIC BICARBONATE SECRETION.
• STOOL CALCIUM LEVEL INCREASED FROM A MEAN OF 797-911 MG PER DAY WITH BED REST
• INTESTINAL ABSORPTION DECREASED FROM 31% WITH BED REST
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PREVENTION & TREATMENT
• USE TOILET OR BEDSIDE COMMODE WHEN POSSIBLE INSTEAD OF BED PAN
• ENCOURAGE EARLY MOBILIZATION• USE HIGH FIBER DIET OR FIBER SUPPLEMENTATION• ENSURE ADEQUATE FLUID INTAKE• DECREASE CONSTIPATING MEDICATIONS SUCH AS
NARCOTICS• CONSIDER STOOL SOFTENERS• GLYCERIN ENEMA DAILY AS NEEDED• LAXATIVES (IF NEEDED)
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SENSORY DEPRIVATION (ATTENTION SPAN, TIME AWARENESS, HAND TO EYE COORDINATION, DEPRESSION,
ANXIETY)↓ BALANCE
↓ COORDINATION↓ VISUAL ACUITY
↑ AUDITORY THRESHOLD
NEUROLOGICAL, EMOTIONAL
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TREATMENT
• STOP MEDICATIONS THAT MAY CAUSE CHANGES IN MENTAL STATUS
• FREQUENTLY ORIENT PATIENT TO TIME AND PLACE
• ENCOURAGE VISITS FROM FAMILY AND FRIENDS
• ENCOURAGE GROUP ACTIVITIES• KEEP PATIENT HEARING AIDS AND GLASSES
EASILY AVAILABLE
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PRESSURE ULCER
PRESSURE ULCER
SKIN
SKIN
EDEMAEDEMA
SUBCUTANEOUS
BURSITIS
SUBCUTANEOUS
BURSITIS
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INTRINSIC:• ABN SKIN SENSATION•ABN MENTAL STATUS •ALTERED CONCIOUSNESS
•ADVANCED AGE•↑LOCAL TISSUE METABOLIC RATE
•PREVIOUS PRESSURE SORE•MUSCLE AND SKIN ATROPHY
•SCARS•EDEMA
•MALNUTRISI & ANEMIA•OBESITY•INFECTION
INTRINSIC:• ABN SKIN SENSATION•ABN MENTAL STATUS •ALTERED CONCIOUSNESS
•ADVANCED AGE•↑LOCAL TISSUE METABOLIC RATE
•PREVIOUS PRESSURE SORE•MUSCLE AND SKIN ATROPHY
•SCARS•EDEMA
•MALNUTRISI & ANEMIA•OBESITY•INFECTION
EXTRINSIC :• DURATION OF PRESSURE•SKIN MACERATION
EXTRINSIC :• DURATION OF PRESSURE•SKIN MACERATION
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PREVENTION PRESSURE ULCERPREVENTION PRESSURE ULCER
• PROPER TURNING• PRESSURE RELIEVING BED• PROPER SKIN CARE & TOILETING• PROPER SEATING• PROPER NUTRITION• SKIN CARE PROCEDURE
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PRESSURE ULCER• ULCUS DECUBITUS• TERM “DECUBITUS” IS DERIVED FROM LATIN
MEANING “LYING DOWN”• CAN RESULT FROM ANY PROLONGED,
UNCHANGED POSITION• ARE CLINICAL MANIFESTATION OF LOCAL TISSUE
DEATH AND CATABOLISM• FOUND MOST FREQUENTLY OVER BONY
PROMINENCE EXPOSED TO COMPRESSING SURFACES
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EPIDEMIOLOGY
• 7.7% OF HOSPITALIZED PATIENTS DEVELOP PRESSURE ULCER WITHIN 21 DAYS OF ADMISSION
• ORTHOPEDIC & GERIATRIC : 24%• SCI : 24-59%
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ETIOLOGIC FACTORSETIOLOGIC FACTORS
PRESSURE:•DURATION•INTENSITY
PRESSURE:•DURATION•INTENSITY
SHEAR:•POOR SITTING
POSITION•POOR BED POS•SPASTICITY•SLIDING
INSTEAD OF LIFTING PS
WHEN TRANSFER
SHEAR:•POOR SITTING
POSITION•POOR BED POS•SPASTICITY•SLIDING
INSTEAD OF LIFTING PS
WHEN TRANSFER
FRICTION :• SKIN TEAR•SKIN
ABRASION
FRICTION :• SKIN TEAR•SKIN
ABRASION
SECONDARY FACTORS :• MOBILITY•NUTRION•AGE
•MOISTURE/INCONTINENCE• DIABETES•SMOKING•FEBRIS•IMPAIRED
MENTAL STATUS
SECONDARY FACTORS :• MOBILITY•NUTRION•AGE
•MOISTURE/INCONTINENCE• DIABETES•SMOKING•FEBRIS•IMPAIRED
MENTAL STATUS
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SIX BASIC CONDITION
• SEDENTARY LIFE STYLE• REST BY MEDICAL/SURGICAL ILLNESS• MEDICAL/CAREGIVER NEGLECT WITH PATIENT
RESTRICTED FROM MOBILITY• IMMOBILISATION BODY/PART OF BODY BY
CAST/BRACE (AFTER TRAUMA)• DISUSE BY NEUROMUSCULAR DISORDER
(PARALYSIS)• DISUSE BY WEIGHT
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ULCUS GRADE 1
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ULCUS GRADE 2
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ULCUS GRADE 3
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ULCUS GRADE 4
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PRIMARY PREVENTION
• INDICATES AREA OF HIGH RISK IN SITTING, SUPINE, AND SIDE-LYING POSITIONS
• AVOID SLIDING PATIENT’S BODY ACROSS SURFACES
• PATIENT SHOULD BE REPOSITIONED EVERY 2 HOURS
• INSPECT AND ASSESS SKIN WITH EACH TURNING
• SUPPORT SURFACES
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WOUND MANAGEMENT PRIORITIES
• REDUCE OR ELIMINATE CAUSATIVE FACTORS• PROVIDE SYSTEMIC SUPPORT FOR WOUND
HEALING• INITIATE APPROPRIATE THERAPY• EDUCATION
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SYSTEMIC SUPPORT
• TISSUE PERFUSION AND OXYGENATION• NUTRITIONAL AND FLUID SUPPORT:
1. ALBUMIN2. ZINC : PROTEIN SYNTESIS & REPAIR
3. VIT C : COLLAGEN SYNTHESIS• CONTROL OF SYSTEMIC CONDITIONS AFFECTING
WOUND HEALING
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SYSTEMIC CONDITIONS
• DIABETES MELLITUS• HEMATOPOIETIC ABNORMALITIES
• IMMUNOSUPRESSION• RENAL FAILURE
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INITIATE APPOPRIATE THERAPY
• DEBRIDEMENT• IDENTIFY AND ELIMINATE INFECTION• MAINTAIN MOIST WOUND SURFACE• PROVIDE THERMAL INSULATION• PROTECT HEALING WOUND
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THANK YOU
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