early and late signs of increased intracranial pressure
DESCRIPTION
A Presentation of the Early and Late Signs of Increased Intracranial PressureTRANSCRIPT
Early and Late
SUBMITTED BY:Rachel M. Raynog
BSNIII-A8
signs of Increased
Intracranial Pressure
EARLY• Deterioration in the level of
consciousness– Early signs are confusion, restlessness,
lethargy, and disorientation first to time, then to place, and then to person
• Pupillary dysfunction relative to size, shape, and reaction to light:– Early signs are a gradual dilation, a slightly
ovoid shape, and a sluggish response to light ipsilateral to the lesion
• Motor weakness and sensory deficits– Early signs are monoparesis, hemiparesis,
and decreased visual acuity, such as blurred vision, diplopia, and field cuts
EARLY• Headache
-Early signs are slight or vague headache which is the worst upon arising in the morning
• Blood pressure
-Relatively stable in early stages• Pulse
-Relatively stable in early stages• Temperature
- Within normal ranges during compensatory stage
LATE• Deterioration in the level of
consciousness– Later signs are stupor and coma
• Pupillary dysfunction relative to size, shape, and reaction to light:– Later signs are dilation of the ipsilateral
pupil and a non-reactivity to light– Final stage signs are bilateral dilation and
fixation• Motor weakness and sensory deficits– Later signs are hemiplegia, decortication, or
decerebration (either unilateral or bilateral) and triple flexion away from the source of stimuli
LATE• Cranial nerve paralysis• Possible seizures, possible
vomiting (including projective vomiting), and possible papilledema
• Respirations
-acute neurogenic pulmonary edema, adult respiratory distress syndrome, and disseminated intravascular coagulopathy can result from an acute increase in ICP.
LATE• Blood pressure
-Increased blood pressure and a widening pulse in later stages which signal cerebral dysfunction that may be irreversible
oCushing's response (a compensatory response that attempts to provide adequate CPP in the presence of rising ICP) presents as a rising systolic pressure, a widening pulse pressure, and bradycardia and is a late presentation of brain stem dysfunction
oCushing's triad (a very late presentation of brain stem dysfunction) presents as hypertension, usually with a widening pulse pressure, bradycardia, and abnormal respiratory patterns
LATE• Pulse
oIrregular, rapid, and thready in the decompensatory stage
• TemperatureoHigh temperatures can occur during decompensatory stage
• Impaired brain stem reflexes, such as corneal and gag reflexes
END