ecg notes
DESCRIPTION
ECG NotesTRANSCRIPT
By ChugaHaeYo 6767
atrial_flutter multiple p wave -
-regular rhythm at rate 2:1 , 3:1 , 4:1 of atrial rate
-SAWTOOTH appearance
causes
Embolism -
MS & MR -
TS & TR -
Heart failure -
IHD -
Pericarditis -
After cardiac surgery -
Thyrotoxicosis-
COPD-
Alcohol -
Digoxin toxicity-
Idiopathic -
clinical picture
Palpitation -
( Fatigue & headache & dizziness & syncope ( due to ⬇CO -
Investigations
-ECG
RFT -
Serum electrolytes-
Blood glucose -
TFT-
( drug level ( if in digoxin-
(Plasma troponin ( if suspected MI-
CXR-
( ECO ( to assist function of lt & Rt ventricles-
MX
If Acute flutter*
( DC cardioversion (20-100j(
If pt stable*
procainamide-
BETA Blocker or Ca channel blocker ( to control Rate ( -
( Amiodarone ( chemical cardioversion-
Radio frequency catheter ablation to prevent recurrent*
atrial fibrillationabsence Pwave-
Irregular rhythm-
causes ( same atrial flutter (
Pulmonary embolism -
MS & MR-
-TS & TR
Heart failure-
IHD -
Pericarditis -
After cardiac surgery -
-Thyrotoxicosis
COPD -
Alcohol-
Digoxin toxicity-
Idiopathic-
clinical picture (same atrial flutter (
. Palpitation -
(Fatigue & headache & dizziness & syncope ( due to ⬇CO-
C/p of underlying cause-
Investigations
-ECG-
RFT-
Serum electrolytes-
Blood glucose-
TFT-
( drug level ( if in digoxin-
( Plasma troponin ( if suspected MI-
CXR -
( ECO ( to assist function of lt & Rt ventricles-
MX
* mx underlying cause
If Acute flutter*
( DC cardioversion (200j -
If pt stable*
( BETA Blocker or verapamil ( to control Rate-
Amiodarone ( chemical cardioversion (-
anticoagulant or antiplatelet *
( warfarin (INR 2-3-
For pt with " Risk Factors "
( VHD . HTN . IHD . CCF . age>75(
- aspirin
For pt without "Risk Factors" and pt C/I for warfarin
ventricular tachycardia
-No wave
-QRS wide >3mm
rapid regular rhythm -
No Twave-
causes
.MI-
. Myocarditis -
Dilated Cardiomyopathy-
. Hypertrophic Cardiomyopathy -
. Chronic IHD-
. -Mitral valve prolapse
. Hypokalemia-
. Digitalis toxicity-
. -CCF
investigations
.ECG-
( . CXR (Cardiomegaly-
. RFT-
. Cardiac Enzymes ( MI (-
. Serum electrolytes-
(. ABG ( Hypoxia + Acidosis-
( . ECO (assist function of Lt & Rt ventricles-
Mx
Acute hemodynamic unstable*
DC cardioversion -
Acute hemodynamic stable*
1 Sotalol or Amiodarone or Procainamid
BETA blocker or Ligoncocain 2
IV mg. Sulfate (for all pt especially if have risk of hypomagnesaemia( 3
treat underlying cause*
(for recurrent ( Radio frequency ablation and or ICD implantation*
Ventricular fibrillation
-No Pwave
NO Twave-
QRS wide >3mm-
Rapid Irregular Rhymes -
causes
MI-
Hypokalemia-
electric Shock-
clinical picture
loss of consciousness-
pulse absent-
respiratory ceases-
Mx
(Electrical defibrillation (if not available >> CPR-
- if survivor of VF & cause isn't reversible >> implantable CVD =Cardio Vascular Defibrillator
Rt_Axis_Deviation
+ >110
Causes
☆ Children
☆ Tall & Then adult
☆ PE
☆ ASD
☆ VSD
☆ ant. Lat. MI
☆ chronic lung disease
lt_Axis_Deviation->30
Causes
☆ LBBB
☆ obese
☆ hyperkalemia
☆ Emphysema
abnormal_Twave
Tall Twave
DDx >>>
▪ Acute STEMI
Hyperkalemia ▪
inverted or low flat Twave
DDx>>
IHD ▪
Myocarditis▪
Digitalis▪
Hypocalcaemia▪
Hyperkalemia ▪
Hypomagnesaemia▪
UwaveDDx>>>
▪ Hypokalemia
Drugs ( Amiodarone , Quinindine , Sotalol ...etc(▪
ECG_changes_in_HYPERKALEMIAflat Pwave - wide QRS–
- peaked Twave short QT interval-
DDx_of_STsegment_elevetion -Acute STEMI
- Prinzmetal's angina
- pericarditis
- LV aneurysm
- LBBB
- cocaine abuse
#NB
.Pericarditis diffuse in all leads
. MI and angina in some leads
طريق عن بينهم انفرق
-cardiac enzymes
في عالية تكون ح MIالي
- timing
عكس 20فقط anginaال تختفي و ساعة MIدقيقة نص حتى يستمر
imp_localization_of_MI☆ ant. MI >> V1 - V4
☆ lat. MI >> I - aVL - V5 -V6
☆ ant. Lat. MI >> I - aVL - V4 - V6
☆ inf. MI >> II - III - aVF
☆ ant. Septal >> V1- V4
☆ septal MI >> V1-V2
investigations_to_confirm_MICardiac Enzymes ( Troponin T& I ( - ⬆⬆⬆⬆ من البداية في حتى 4-2يزدادو ارتفاعهم يستمر و تقريبا 1ساعات سبوعين
Creatin kinase (CK-MB , CK-MM , CK-BB( ،،،،، CK-MB more specific-
في ⬆⬆⬆ يصل 8الى 4يزداد و بعد 24في peakساعات للطبيعي يرجع و 3الى 2ساعةايام
serum myoglobin-
AST ⬆⬆⬆-
لل خالل 48_24في peakيصل للطبيعي يرجع بعدها ساعة72ساعة
LDH ⬆⬆⬆-
لل حتى 4_3في peakيصل ويستمر يوم 14الى 10ايام
(CBC ( leukocytosis-
ECR ⬆-
-CXR
-eco
Mx_of_acute_STEMI
-call for help
- ABC
- O2 for hypoxia
- CBC
- pain relief
* IV morphine
* Nitroglycerin
* B.Blocker
- Antithrombotic
* aspirin
- reperfusion
* PCI ( percutaneous coronary Intervention ( for Stent
* Thrombolytic
* C/I of THROMBOLYTIC
☆ absolute C/I
▪active internal bleeding
▪aortic dissection suspected
▪preior ischemic stroke in last 3months
▪preior hemorrhagic stroke
▪known Intracranial Neoplasm
▪acute active pericarditis
▪pregnancy
▪sever HTN
▪recent surgery for head
DDx_of_ST_Deprssion ▪NSTEMI
▪stable & unstable angina
▪Hypokalemia
▪Digitalis
▪cardiac hypertension
Mx_of_NSTEMI -admission
- call for help
- ABC
- high O2 by mask
- Morphine IV
- Metoclopramide
- sublingual Nitrate
- aspirin chewed
- B.blocker
- Heparin
#before discharge
*Do stress ECG
* Coronary angiography
Rt_ventricular_hypertrophy☆ Rwave in V1 & V2 >5mm
DDX
-PHTN
- pulmonary stenosis
- Hypertrophic cardiomyopathy
lt_ventricular_hypertrophy☆ Rwave in V5 & V6 > 5large square
Ddx
- systemic HTN
- Aortic stenosis
- Hypertrophic cardiomyopathy
DDx_wide_QRS -RBBB
- LBBB
- ventricular extra systole
- ventricular tachycardia
- ventricular fibrillation
#RBBB☆ wide QRS complex
☆ broad notched R wave in V1& V2
( M shaped(
☆ wide and deep Swave in V5 & V6
causes
-IHD
- congenital heart disease (ASD(
- PHTN
- myocarditis
- degenerative conduction system disease
#LBBB
☆ wide QRS complex
☆ broad notched R wave in V5 & V6
( M shaped(
☆ small or absent R wave followed by deep S waves in V1 & V2
causes
It's rare and most commonly seen in IHD
Ventricular_Extrasystole = premature ventricular contraction = PVC
☆ No Pwave
☆ wide QRS (bizzare in shape(
☆ Twave inversely proportion to QRS
☆ prolonged pause after QRS
Heart_block
1st degree HB
☆ PR interval fixed prolongation
☆ No drop beat
☆ QRS normal or narrow
2nd degree HB (MOBITZ I)
☆ PR interval progressive prolongation
☆ Dropped beat (p without QRS(
☆QRS normal or narrow
2nd degree HB (MOBITZ II)= wenckebach phenomenoa
☆ Dropped beat
☆ PR interval normal before dropped beat
☆ Pwave with sinus rhythm
☆ QRS wide
rd HB (complete HB)3
☆ Pwave normal
☆ QRS wide , regular & Slow in rate
☆ PR interval variable ( AV dissociation(
causes
*Acute
- MI
- myocarditis
- Infections
- hyperkalemia
- DRUGS ( Digitalis , BETA blocker , Ca channel blocker & antiarrhythmic drugs(
*chronic
- Idiopathic conduction degeneration
- congenital complete AV block
- Cardiomyopathy
Clinical_pictures
-Asymptomatic
- symptomatic (hemodynamic and unstable(
~ chest pain
~ bradycardia
~ systolic pressure >90mmHg
~ pulmonary Edema
~ dizziness & syncope
investigations
-ECG
- RFT
- serum electrolytes
- TFT
- CXR
( - plasma troponin (MI
- drug level
- ECO
Mx☆ 1st degree HB
>> No need treatment
☆ (2nd degree ( MOBITZ I
if asymptomatic
>> No Need Treatment
if symptomatic
>> Atropine
Or
>> temporary Pacemaker
☆ complete HB
>> permanent Pacemaker
( Except when ass` with transient Inf. MI. Or Asymptomatic congenital
HB(
Supraventreicular_tachycardia
☆ No P wave
☆ QRS narrow >3mm
☆ regular rhythm
#causes
-Anixety
- ⬆⬆ caffee & tobacoo
- Hyperthyroidism
- Alcohol
#Clinical_Pictures
-palpitation
- Breathlessness
- polyuria (sometimes(
#Mx*VAGOTONIC MANEUVERS
. Carotid sinus massage
. Pressure on eyes
. Valsalva maneuver
. Self induced vomiting
. Lowering head btween knee
* DRUGs
. Adenocor
. Verapmil
. BETA blocker
. Digoxin
* DC cardioversion (in emergency
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