egfr mutation-induced alternative splicing of max contributes to growth of glycolytic tumors in...
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Figure 2 Cell Metabolism , DOI: ( /j.cmet ) Copyright © 2013 Elsevier Inc. Terms and Conditions Terms and ConditionsTRANSCRIPT
EGFR Mutation-Induced Alternative Splicing of Max Contributes to Growth of Glycolytic Tumors in Brain Cancer
Ivan Babic, Erik S. Anderson, Kazuhiro Tanaka, Deliang Guo, Kenta Masui, Bing Li, Shaojun Zhu, Yuchao Gu, Genaro R. Villa, David Akhavan, David Nathanson, Beatrice Gini, Sergey Mareninov, Rui Li, Carolina Espindola Camacho, Siavash K. Kurdistani, Ascia Eskin,
Stanley F. Nelson, William H. Yong, Webster K. Cavenee, Timothy F. Cloughesy, Heather R. Christofk, Douglas L. Black, Paul S. Mischel
Cell Metabolism Volume 17, Issue 6, Pages 1000-1008 (June 2013)
DOI: 10.1016/j.cmet.2013.04.013
Copyright © 2013 Elsevier Inc. Terms and Conditions
Figure 1
Cell Metabolism 2013 17, 1000-1008DOI: (10.1016/j.cmet.2013.04.013) Copyright © 2013 Elsevier Inc. Terms and Conditions
Figure 2
Cell Metabolism 2013 17, 1000-1008DOI: (10.1016/j.cmet.2013.04.013) Copyright © 2013 Elsevier Inc. Terms and Conditions
Figure 3
Cell Metabolism 2013 17, 1000-1008DOI: (10.1016/j.cmet.2013.04.013) Copyright © 2013 Elsevier Inc. Terms and Conditions
Figure 4
Cell Metabolism 2013 17, 1000-1008DOI: (10.1016/j.cmet.2013.04.013) Copyright © 2013 Elsevier Inc. Terms and Conditions
Cell Metabolism 2013 17, 1000-1008DOI: (10.1016/j.cmet.2013.04.013) Copyright © 2013 Elsevier Inc. Terms and Conditions