help! is it aphasia, apraxia, dysarthria or all of the above??!! jamie l. johnson, ma l/ccc-slp...
TRANSCRIPT
HELP! Is it Aphasia, Apraxia, Dysarthria or ALL of
the Above??!!
Jamie L. Johnson, MA L/CCC-SLPUniversity of Kansas HospitalSeptember 24, 2015
During this session, participants will actively participate indiscussing:
Different types of aphasia and dysarthria.
Define dysarthria and apraxia.
Evaluation in the acute and outpatient settings will be identified.
The role of the SLP in education with family/friends for carryover into the home and community will briefly be discussed.
Standardized Evaluations
•Purpose:▫Assess 4 modalities▫Classification▫Prognostic statement▫Treatment
Fluency
•Average of 3 longest phrases in response to▫What happened to you▫Description of picture (cookie theft)▫Response to emotional question-”Tell me
about your family.” “Do you remember what happened on 911?”
Standardized Evaluations• Classification
▫WAB▫BDAE
• Non-classification▫MTDDA▫PICA▫Functional Assessment of Communication
Skills for Adults (ASHA FACS)▫CADL▫Communication Effectiveness Index (CETI)-
caregiver
Type of Aphasia
Fluent or Nonfluent
Conversational Speech
Auditory comprehension
Repetition Naming Lesion Location
Anomic Aphasia
fluent Fluent, normal utterance length and well-formed sentences
Good for everyday conversation, difficulty with complex syntax
preserved Impairment is hallmark
Acute – outside perisylvian zona (angular gyrus or inferior temporal region), chronic- perisylvian area, posterior tempolateral region
Conduction Aphasia
fluent Fluent with normal utterance length but has paraphasias
Good for casual conversation, difficult with complex syntax
Impairment is hallmark, good spontaneous speech, paraphasias during repetition
Always impaired
Posterior perisylvian lesions affecting supramarginal gyrus in parietal lobe and arcuate fasciculus
Transcortical sensory aphasia (TcSA)
fluent Fluent with normal utterance length, but semantic paraphasias, anomia
Significantly impaired
Preserved Severely impaired
Extrasylvian regions involving POT junction region; posterior and deep to Wernicke’s area; sensory info doesn’t reach language areas
Wernicke’s aphasia
Fluent Fluent, easily articulated speech of normal utterance length, semantic and phonemic paraphasias, verbal output excessive and rapid but empty
Severely impaired at single-word level, difficulty with complex syntax and multi-step commands, unaware of inability to produce coherent speech
Significantly defective, cannot even repeat single words
Paraphasic and severe anomia
Large posterior perisylvian lesions encompassing Wernicke’s area and extending superiorly into inferior parietal region
Transcortical motor aphasia (TcMA)
Nonfluent Little attempt to produce spontaneous speech, mute, speech is reduced in length
Good for most conversational interaction, difficulty with complex syntax
Preserved, but absence of spontaneous speech
Relatively preserved
Extrasylvian regions of left frontal lobe; dorsolateral frontal lesions located anterior or superior to Broca’s area, supplementary motor areas, cingulate gyrus
Broca’s aphasia
Nonfluent
Slow, halting speech production, utterances are of reduced length with simple grammar
Good for conversational speech, difficulty with complex syntax
Limited to single words and short phrases
Impaired to some degree, especially for low frequency words
Broca’s area causes transient disruption of speech production and fluency; persistent Broca’s aphasia from larger perisylvian lesions encompassing more of the left frontal lobe
GLOBAL APHASIA• VERBAL EXPRESSION
▫ NON-FLUENT▫ Severely Impaired▫ *Automatic speech may
be preserved• COMPREHENSION
▫ Severely Impaired• REPETITION
▫ Impaired• WRITING
▫ Impaired• READING
COMPREHENSION▫ Impaired
• LARGE LEFT DOMINANT LESION▫ Involving Broca’s and
Wernicke’s areas
BROCA’S APHASIA• VERBAL EXPRESSION:
▫ NON-FLUENT (4words or less)
▫ Slow effortful▫ Perseverations▫ “Telegraphic speech”
• COMPREHENSION:▫ Relatively preserved
• REPETITION▫ Poor
• WRITING ▫ Parallels expression
• READING COMPREHENSION▫ Relatively spared
• ANTERIOR PORTION OF THE LEFT
HEMISPHERE
TRANSCORTICAL MOTOR APHASIA• VERBAL EXPRESSION
▫ NON-FLUENT• AUDITORY
COMPREHENSION:▫ Intact
• REPETITION▫ Intact
• SIMILAR TO BROCA’S WITH ABILITY TO REPEAT
• Anterior and Superior to Broca’s area
• Watershed, borderzones
*MIXED NON-FLUENT• Resembles Broca’s but
auditory comprehension below 50 percentile.
• Auditory comprehension too good to be Global
WERNICKE’S APHASIA
• VERBAL EXPRESSION▫ FLUENT ▫ Jargon, non-sensical
words, rapid rate▫ Retain sentences but lack
meaning▫ May speak with no
insight into errors• COMPREHENSION
▫ poor• REPETITION
▫ poor
• Temporo-parietal involving Wernicke’s area and adjacent white matter
CONDUCTION APHASIA• VERBAL EXPRESSION
▫ FLUENT ▫ Word finding▫ Paraphasic errors
• COMPREHENISON ▫ Relatively intact
• REPETITION▫ Poor
• RARE
• ARCUATE FASCICULUS AND LEFT PARIETAL
TRANSCORTICAL SENSORY APHASIA• VERBAL EXPRESSION
▫ FLUENT▫ Echolalia
• COMPREHENSION▫ SIMILAR TO
WERNICKE’S-EXCEPT STRONG ABILITY TO REPEAT
REPETITION:▫ Intact
• Watershed PCA/MCA territories, borderzones
• Spares Wernicke’s area
*MIXED TRANSCORTICAL APHASIA
• COMBINATION OF THE TWO TRANSCORTICAL APHASIAS
• AUDITORY COMPREHENSION AND EXPRESSION SEVERELY IMPAIRED
• REPETITION INTACT
ANOMIC APHASIA• VERBAL EXPRESSION
▫ FLUENT▫ Word Finding problems▫ Circumlocutes
• COMPREHENSION ▫ Intact▫ USE OF NON-SPECIFIC
WORDS SUCH AS “THING”
• REPETITION ▫ Intact
• MILD FORM• +Awareness
CROSSED APHASIA• LANGUAGE CENTER NO
IN EXPECTED HEMISPHERE
• • EX. RIGHT-HANDED
PERSON WITH R CVA WITH APHASIA
SUBCORTICAL APHASIA• THALAMUS• INTERNAL CAPSULE• BASAL GANGLIA
• MIRROR CORTICAL LESION APHASIAS
• CAN CO-OCCUR WITH CORTICAL APHASIAS
PRIMARY PROGRESSIVE APHASIA• GRADUAL LOSS OF LANGUAGE-
PRESERVED MEMORY, VISUAL PROCESSING AND PERSONALITY-UNTIL THE END
• BEGINS WITH WORD FINDING PROGRESSES TO IMPAIRED GRAMMAR AND COMPREHENSION
• DYSARTHRIA AND APRAXIA MAY ACCOMPANY
• *STRUCTURAL & PHYSIOLOGICAL ABNORMALITIES IN LEFT HEMISPHERE (FRONTAL, PARIETAL AND TEMPORAL.
• NOT DUE TO NEOPLASTIC, VASCULAR OR METABOLIC ETIOLOGIES NOR INFECTION
• Semantic variant
• Logopenic variant
• Non-fluent Agrammatic variant
INSULA“The Role of the insula in Speech and Language Production”
Oh, A. et al.
Brain and Language 135 (2014) 96-103.
“Prime real estate”
• Responsible for articulatory control
• Direct connections to Broca’s area
• Higher order cognitive aspects of speech-language
• fMRI-exp/rec and production/ perception=Bilateral ant insula
• Speech perception Left dorsal mid-insula
• Expressive language tasks activated left ventral mid=insula
• Mid Insula plays different roles in S/L processing
Differential Diagnosis
•Motor Speech Disorders:•Dysarthria
▫Weakness/paralysis, incoordination, rigidity, involuntary movement
•Apraxia▫Motor planning problem▫Absence of weakness▫Initiation, groping, revisions, inconsistent
Apraxia of Speech
• Messages from the brain to the mouth are disrupted, and the person cannot move his or her lips or tongue to the right place to say sounds correctly, even though the muscles are not weak.
• Apraxia can occur in conjunction with dysarthria or aphasia
• Caused by damage to the parts of the brain that control coordinated muscle movement
Signs or Symptoms of Apraxia of Speech
Know what words they want to say, but their brains have difficulty coordinating the muscle movements necessary to say all the sounds in the words.
Individuals with apraxia may demonstrate: • Difficulty imitating and producing speech sounds• Sound distortions, substitutions, and/or omissions• Inconsistent speech errors• Groping of the tongue and lips to make specific sounds and
words• Slow speech rate• Impaired rhythm and prosody (intonation) of speech• Better automatic speech than purposeful speech• Inability to produce any sound at all in severe cases. • Frustrating
G CODESModifier Impairment NOMS
•0% WNL 7•1-20% MIN 6•20-40 MILD 5•40-60% MILD-MOD 4•60-80% MODERATE 3•80-99% MOD-SEVERE 2•100% SEVERE 1
NOMS MOTOR SPEECH• LEVEL 1: The individual attempts to speak, but speech cannot be understood by
• familiar or unfamiliar listeners at any time.
• LEVEL 2: The individual attempts to speak. The communication partner must assume
• responsibility for interpreting the message, and with consistent and maximal cues,
• the patient can produce short consonant-vowel combinations or automatic words
• that are rarely intelligible in context.
• LEVEL 3: The communication partner must assume primary responsibility for interpreting
• the communication exchange, however, the individual is able to produce short
• consonant-vowel combinations or automatic words intelligibly. With consistent and
• moderate cueing, the individual can produce simple words and phrases intelligibly,
• although accuracy may vary.
• LEVEL 4: In simple structured conversation with familiar communication partners,
• the individual can produce simple words and phrases intelligibly. The individual
• usually requires moderate cueing in order to produce simple sentences intelligibly,
• although accuracy may vary.
• LEVEL 5: The individual is able to speak intelligibly using simple sentences in daily routine
• activities with both familiar and unfamiliar communication partners. The individual
• occasionally requires minimal cueing to produce more complex
• sentences/messages in routine activities, although accuracy may vary and the
• individual may occasionally use compensatory strategies.
• LEVEL 6: The individual is successfully able to communicate intelligibly in most activities,
• but some limitations in intelligibility are still apparent in vocational, avocational,
• and social activities. The individual rarely requires minimal cueing to produce
• complex sentences/messages intelligibly. The individual usually uses
• compensatory strategies when encountering difficulty.
• LEVEL 7: The individual’s ability to successfully and independently participate in
• vocational, avocational, or social activities is not limited by speech production.
• Independent functioning
NOMS SPOKEN LANGUAGE COMPREHENSION • LEVEL 1: The individual is alert, but unable to follow simple directions or respond to yes/no
• questions, even with cues.
• LEVEL 2: With consistent, maximal cues, the individual is able to follow simple
• directions, respond to simple yes/no questions in context, and respond to simple
• words or phrases related to personal needs.
• LEVEL 3: The individual usually responds accurately to simple yes/no questions. The
• individual is able to follow simple directions out of context, although moderate
• cueing is consistently needed. Accurate comprehension of more complex
• directions/messages is infrequent.
• LEVEL 4: The individual consistently responds accurately to simple yes/no questions and
• occasionally follows simple directions without cues. Moderate contextual support is
• usually needed to understand complex sentences/messages. The individual is able to
• understand limited conversations about routine daily activities with familiar
• communication partners.
• LEVEL 5: The individual is able to understand communication in structured conversations
• with both familiar and unfamiliar communication partners. The individual
• occasionally requires minimal cueing to understand more complex
• sentences/messages. The individual occasionally initiates the use of
• compensatory strategies when encountering difficulty.
• LEVEL 6: The individual is able to understand communication in most activities, but some
• limitations in comprehension are still apparent in vocational, avocational, and
• social activities. The individual rarely requires minimal cueing to understand complex
• sentences. The individual usually uses compensatory strategies when encountering
• difficulty.
• LEVEL 7: The individual’s ability to independently participate in vocational, avocational,
• and social activities are not limited by spoken language comprehension. When
difficulty with comprehension
NOMS SPOKEN LANGUAGE EXPRESSION • LEVEL 1: The individual attempts to speak, but verbalizations are not meaningful to familiar or• unfamiliar communication partners at any time.
• LEVEL 2: The individual attempts to speak, although few attempts are accurate or appropriate.• The communication partner must assume responsibility for structuring the• communication exchange, and with consistent and maximal cueing, the individual can• only occasionally produce automatic and/or imitative words and phrases that are rarely• meaningful in context.
• LEVEL 3 The communication partner must assume responsibility for structuring the• communication exchange, and with consistent and moderate cueing, the individual can• produce words and phrases that are appropriate and meaningful in context.
• LEVEL 4: The individual is successfully able to initiate communication using spoken language• in simple, structured conversations in routine daily activities with familiar• communication partners. The individual usually requires moderate cueing, but is able to• demonstrate use of simple sentences (i.e., semantics, syntax, and morphology) and• rarely uses complex sentences/messages.
• LEVEL 5: The individual is successfully able to initiate communication using spoken language• in structured conversations with both familiar and unfamiliar communication partners.• The individual occasionally requires minimal cueing to frame more complex sentences• in messages. The individual occasionally self-cues when encountering difficulty.
• LEVEL 6: The individual is successfully able to communicate in most activities, but some• limitations in spoken language are still apparent in vocational, avocational, and social• activities. The individual rarely requires minimal cueing to frame complex sentences.• The individual usually self-cues when encountering difficulty.
• LEVEL 7: The individual’s ability to successfully and independently participate in vocational,• avocational, and social activities is not limited by spoken language skills. Independent• functioning may occasionally include use of self-cueing.
Education
•Family Training•Staff Training•Education materials•Websites•Apps•Home Programs
•Family/Caregiver’s role in Therapy in any setting
REFERENCES• Davis, G. (2007) Aphasiology: Disorders and Clinical Practice.
pages 33-39.• Helm-Estabrooks, N. Albert, M.L., 1991, 2004. Manual of
Aphasia and Aphasia Therapy. 2nd edition, Pro-Ed, Austin Texas.• Johnson, A., Jacobson, B,(2006) Medical Speech Pathology: A
Practitioner's Guide. Thieme Medical Publishers, New York, NY• LaPointe, L. (2001 ) Aphasia and related Neurogenic Language
Disorders. Thieme Medical Publishers, New York, NY• Oh, A. et al. (2014) The Role of the Insula in Speech and
Language Production Brain and Language. 135, 96-103.• www.ASHA.org