infant bowel obstruction 2005
TRANSCRIPT
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Infant Bowel Obstruction
Robert W. Letton, Jr., MD
Associate Professor of Surgery
Pediatric SurgeryOklahoma University Health Sciences Center
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Question 1?
Why do Pediatric Surgeons
always make such a big dealout of a little yellow orgreen emesis?
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Answer
Because unlike when Stansees Wendy in Southpark,
it usually meansbowel obstruction or
necrosis in ourpatients!
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Goals
Discuss the work-up and management of
the child with potential bowel obstruction
Recognize the common causes of bowelobstruction in children
Discuss surgical management of common
causes of bowel obstruction
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History
Birth History
Feeding History
Formula intolerance
Emesis
Bilious vs non-bilious
Bowel Habits
passage of meconium
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History
Antecedent episodes
Irritable, lethargic
History of inguinal hernia
Family history
Hirschsprungs
Recent immunization or URI
Intussusception
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Physical Exam
General state of hydration
Obvious source of sepsis
meningitis, strep throat, otitis, pneumonia, UTI
Inspect abdomen
scaphoid or distended, discolored
Auscultate Palpate
masses, tenderness, peritonitis
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Physical Exam
Must remove diaper
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Physical Exam
Must perform rectal exam, not just look!
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Ancillary Studies
CBC, Lytes, UA, +/- Blood Cx, +/- ABG
Acute abdominal series
left lateral decub, KUB, CXR
Contrast Study
From above or below??
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Initial Management
NG or OG to low wall suction (NPO!!)
Hydrate and replace losses
10 cc/kg of crystalloidIS NOT AN
ADEQUATE BOLUS!!
Antibiotics if suspect perforation or necrosis
Consult surgeon and/or transfer to
appropriate facility
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Bowel Obstruction
Diagnosis often age specific
Bilious vomiting in the infant and child is a
surgical emergency until proven otherwise
Difficult to tell when volvulus is present
Child may look surprisingly good until its
too late
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Etiology of Bowel Obstruction
Atresias
Hirschsprungs
Malrotation Volvulus
Intussusception
Incarcerated Hernia Perforated appendix
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Atresia
Usually presents the first few days of life
Child may feed well for a day or two with
distal atresia
Duodenal atresia often diagnosed on
antenatal U/S
Atresias can occur anywhere in GI tract
from pharynx to anus
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Atresias
Esophageal: aspirate feeds immediately, OG
tube wont pass (non-bilious, but still bad)
Duodenal: bilious vomiting immediately,double bubble on KUB with absence of
distal gas, Downs Syndrome
Jejunal: usually present 1st 24 hours, largedilated proximal loop or loops
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Atresias
Ileal: may take 24-48 hours before bilious
emesis
Colonic: rare, may present with biliousemesis after 2-3 days
Anal: should be diagnosed at birth, often a
perineal fistula is labeled normal
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Obvious Obstruction
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Atresias may be multiple
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Jejunal Atresia
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Apple Peel Deformity (IIIb)
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Imperforate Anus: Anal atresia
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Hirschsprungs Disease
Congenital colonic aganglionosis
Physiologic obstruction
May present first few days to weeks of life
Short segment disease often tolerated for
months
Starts at anus and extends proximally a
variable distance
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Hirschsprungs
Delayed passage of meconium at birth
Meconium plug syndrome, small left colon
syndrome, Downs syndrome Often present with distension and diarrhea at 2-4
weeks of life
May or may not have emesis
Profoundly distended abdomen with dilated bowel
Fever and WBCs with colitis
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Hirschsprungs
Rectal exam may seem normal until
withdraw finger
Explosive release of liquid stool almostdiagnostic
Barium enema while dilated
Irrigate and dilate until decompressed
Suction rectal biopsy
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Hirschsprungs Disease
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Barium Enema
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Treatment
NO WAY!
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Hirschsprungs Disease
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Toxic Megacolon
Severe enterocolitis
Very rare to get with idiopathic constipation
Usually only seen with Hirschsprungs
Disease or Ulcerative Colitis
NG decompression, IV fluids, IV antibiotics
Mortality 20-30% in some studies
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Toxic Megacolon
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Hirschsprungs in an 8 year old
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Malrotation
Normal
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Malrotation
Most often presents during the first fewmonths of life
Infant with acute onset of bilious emesis May be diagnosed on UGI for other reasons
Malrotation is a surgical urgency due to the
possibility of volvulus VOLVULUS IS A SURGICAL
EMERGENCY
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Malrotation
Abdomen usually NOT distended
AAS usually normal
May show bowel obstruction, double-
bubble, or gasless
UGI is definitive diagnostic study
Infant in extremis
resuscitate and operate
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Malrotation
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Malrotation
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Volvulus
Malrotation most common condition
resulting in midgut volvulus
Can have volvulus with normal rotation
omphalomesenteric remnant
internal hernia
Duplication
Adhesive small bowel obstruction
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Midgut Volvulus
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Small Bowel Obstruction
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Meckels
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Duplication/Volvulus
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Duplication
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Intussusception
Inversion of the bowel upon itself
secondary to a lead point
Juvenile intussusception most oftenidiopathic
Also secondary to Meckels
Presents 6 months to 2 years of age
As early as 1 month
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Intussusception
Acute painful episodes followed by periods
of lethargy
When incarcerated progress to continuouslethargy
May or may not have currant-jelly stool
But often stool is heme positive
Rule out with a left lateral decubitus film
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Left-lateral Decubitus Film
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Intussusception
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Intussusception
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Intussusception
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Intussusception
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Bad Intussusception
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Intussusception
7% chance of recurrence after ACEreduction
Usually recur in 48 hours Operative exploration warranted on second
recurrence to R/O pathologic lead point
Recurrence after surgery rare but possible
Post-op intussusception can occur after anysurgery
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Incarcerated Hernia
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Inguinal/Scrotal Anatomy
From Surgery of Infants and Children, Oldham, et. al., 1997
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Inguinal Hernia
From Atlas of Pediatric Surgery, Ashcraft, 1994
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Incarcerated Inguinal Hernia
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Hernia Reduction
From Surgery of Infants and Children, Oldham, et. al., 1997
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Incarcerated Hernia
Most can be reduced in clinic or ED
Bowel usually OK if able to reduce
Surgical consultation if reduction difficult
Repair with 1-2 days of incarceration
Beware the inguinal node in females
incarcerated ovary
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Incarcerated Hernia
If unable to reduce: urgent operative
exploration (NPO)
If able to reduce without sedation: urgentsurgical referral with repair soon
If extremely difficult (sedation, surgical
referral): repair next day Watch child for obstructive symptoms
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Perforated Appendix
Children still die from complications ofperforated appendicitis
Resuscitation is critical Response to surgery variable
Often require multiple procedures,hyperalimentation, prolonged antibiotictherapy
Diagnosis difficult
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AAP Guidelines for Pediatric
Surgical Referral Patients 5 years or younger who may need surgical
care
Infants and children with perforated appendicitis
Seriously injured infants and children
Infants, children, and adolescents with solidmalignancies
Minimally invasive procedures Infants and children with medical conditions that
increase operative risk
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Morbidity
Incidence of Perforation< 1 year old 90-100%
1-2 years old 70-80%
2-5 years old 50%
> 65 years old 50%
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Perforated Appendix
Suspect in children 3-5 years old withhistory suggestive of appendicitis
Bowel obstruction in a 3-5 year oldwithout obvious etiology is perforatedappendix until proven otherwise
Fever > 101.5, WBC > 20 with bands,
diffuse abdominal pain, guarding, SBO onAAS
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Perforated Appendix
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Perforated Appendix
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Resuscitation
NG tube, NPO
20 cc/kg boluses until UOP > 1 cc/kg/hr and
VS stable 1.5-2 times maintenance fluids
Broad Spectrum Antibiotics
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Perforated Appendix
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Summary
Atresias
Hirschsprungs
Malrotation Volvulus
Intussusception
Incarcerated Hernia Perforated Appendix
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Question 2?
Why are Pediatric Surgeons sointerested in flatus?
Contrary to popularbelief, kids with
obstruction can still
have bowelmovements, but theywont pass gas!