infant bowel obstruction 2005

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    Infant Bowel Obstruction

    Robert W. Letton, Jr., MD

    Associate Professor of Surgery

    Pediatric SurgeryOklahoma University Health Sciences Center

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    Question 1?

    Why do Pediatric Surgeons

    always make such a big dealout of a little yellow orgreen emesis?

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    Answer

    Because unlike when Stansees Wendy in Southpark,

    it usually meansbowel obstruction or

    necrosis in ourpatients!

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    Goals

    Discuss the work-up and management of

    the child with potential bowel obstruction

    Recognize the common causes of bowelobstruction in children

    Discuss surgical management of common

    causes of bowel obstruction

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    History

    Birth History

    Feeding History

    Formula intolerance

    Emesis

    Bilious vs non-bilious

    Bowel Habits

    passage of meconium

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    History

    Antecedent episodes

    Irritable, lethargic

    History of inguinal hernia

    Family history

    Hirschsprungs

    Recent immunization or URI

    Intussusception

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    Physical Exam

    General state of hydration

    Obvious source of sepsis

    meningitis, strep throat, otitis, pneumonia, UTI

    Inspect abdomen

    scaphoid or distended, discolored

    Auscultate Palpate

    masses, tenderness, peritonitis

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    Physical Exam

    Must remove diaper

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    Physical Exam

    Must perform rectal exam, not just look!

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    Ancillary Studies

    CBC, Lytes, UA, +/- Blood Cx, +/- ABG

    Acute abdominal series

    left lateral decub, KUB, CXR

    Contrast Study

    From above or below??

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    Initial Management

    NG or OG to low wall suction (NPO!!)

    Hydrate and replace losses

    10 cc/kg of crystalloidIS NOT AN

    ADEQUATE BOLUS!!

    Antibiotics if suspect perforation or necrosis

    Consult surgeon and/or transfer to

    appropriate facility

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    Bowel Obstruction

    Diagnosis often age specific

    Bilious vomiting in the infant and child is a

    surgical emergency until proven otherwise

    Difficult to tell when volvulus is present

    Child may look surprisingly good until its

    too late

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    Etiology of Bowel Obstruction

    Atresias

    Hirschsprungs

    Malrotation Volvulus

    Intussusception

    Incarcerated Hernia Perforated appendix

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    Atresia

    Usually presents the first few days of life

    Child may feed well for a day or two with

    distal atresia

    Duodenal atresia often diagnosed on

    antenatal U/S

    Atresias can occur anywhere in GI tract

    from pharynx to anus

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    Atresias

    Esophageal: aspirate feeds immediately, OG

    tube wont pass (non-bilious, but still bad)

    Duodenal: bilious vomiting immediately,double bubble on KUB with absence of

    distal gas, Downs Syndrome

    Jejunal: usually present 1st 24 hours, largedilated proximal loop or loops

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    Atresias

    Ileal: may take 24-48 hours before bilious

    emesis

    Colonic: rare, may present with biliousemesis after 2-3 days

    Anal: should be diagnosed at birth, often a

    perineal fistula is labeled normal

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    Obvious Obstruction

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    Atresias may be multiple

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    Jejunal Atresia

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    Apple Peel Deformity (IIIb)

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    Imperforate Anus: Anal atresia

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    Hirschsprungs Disease

    Congenital colonic aganglionosis

    Physiologic obstruction

    May present first few days to weeks of life

    Short segment disease often tolerated for

    months

    Starts at anus and extends proximally a

    variable distance

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    Hirschsprungs

    Delayed passage of meconium at birth

    Meconium plug syndrome, small left colon

    syndrome, Downs syndrome Often present with distension and diarrhea at 2-4

    weeks of life

    May or may not have emesis

    Profoundly distended abdomen with dilated bowel

    Fever and WBCs with colitis

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    Hirschsprungs

    Rectal exam may seem normal until

    withdraw finger

    Explosive release of liquid stool almostdiagnostic

    Barium enema while dilated

    Irrigate and dilate until decompressed

    Suction rectal biopsy

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    Hirschsprungs Disease

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    Barium Enema

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    Treatment

    NO WAY!

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    Hirschsprungs Disease

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    Toxic Megacolon

    Severe enterocolitis

    Very rare to get with idiopathic constipation

    Usually only seen with Hirschsprungs

    Disease or Ulcerative Colitis

    NG decompression, IV fluids, IV antibiotics

    Mortality 20-30% in some studies

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    Toxic Megacolon

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    Hirschsprungs in an 8 year old

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    Malrotation

    Normal

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    Malrotation

    Most often presents during the first fewmonths of life

    Infant with acute onset of bilious emesis May be diagnosed on UGI for other reasons

    Malrotation is a surgical urgency due to the

    possibility of volvulus VOLVULUS IS A SURGICAL

    EMERGENCY

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    Malrotation

    Abdomen usually NOT distended

    AAS usually normal

    May show bowel obstruction, double-

    bubble, or gasless

    UGI is definitive diagnostic study

    Infant in extremis

    resuscitate and operate

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    Malrotation

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    Malrotation

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    Volvulus

    Malrotation most common condition

    resulting in midgut volvulus

    Can have volvulus with normal rotation

    omphalomesenteric remnant

    internal hernia

    Duplication

    Adhesive small bowel obstruction

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    Midgut Volvulus

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    Small Bowel Obstruction

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    Meckels

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    Duplication/Volvulus

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    Duplication

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    Intussusception

    Inversion of the bowel upon itself

    secondary to a lead point

    Juvenile intussusception most oftenidiopathic

    Also secondary to Meckels

    Presents 6 months to 2 years of age

    As early as 1 month

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    Intussusception

    Acute painful episodes followed by periods

    of lethargy

    When incarcerated progress to continuouslethargy

    May or may not have currant-jelly stool

    But often stool is heme positive

    Rule out with a left lateral decubitus film

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    Left-lateral Decubitus Film

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    Intussusception

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    Intussusception

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    Intussusception

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    Intussusception

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    Bad Intussusception

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    Intussusception

    7% chance of recurrence after ACEreduction

    Usually recur in 48 hours Operative exploration warranted on second

    recurrence to R/O pathologic lead point

    Recurrence after surgery rare but possible

    Post-op intussusception can occur after anysurgery

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    Incarcerated Hernia

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    Inguinal/Scrotal Anatomy

    From Surgery of Infants and Children, Oldham, et. al., 1997

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    Inguinal Hernia

    From Atlas of Pediatric Surgery, Ashcraft, 1994

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    Incarcerated Inguinal Hernia

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    Hernia Reduction

    From Surgery of Infants and Children, Oldham, et. al., 1997

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    Incarcerated Hernia

    Most can be reduced in clinic or ED

    Bowel usually OK if able to reduce

    Surgical consultation if reduction difficult

    Repair with 1-2 days of incarceration

    Beware the inguinal node in females

    incarcerated ovary

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    Incarcerated Hernia

    If unable to reduce: urgent operative

    exploration (NPO)

    If able to reduce without sedation: urgentsurgical referral with repair soon

    If extremely difficult (sedation, surgical

    referral): repair next day Watch child for obstructive symptoms

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    Perforated Appendix

    Children still die from complications ofperforated appendicitis

    Resuscitation is critical Response to surgery variable

    Often require multiple procedures,hyperalimentation, prolonged antibiotictherapy

    Diagnosis difficult

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    AAP Guidelines for Pediatric

    Surgical Referral Patients 5 years or younger who may need surgical

    care

    Infants and children with perforated appendicitis

    Seriously injured infants and children

    Infants, children, and adolescents with solidmalignancies

    Minimally invasive procedures Infants and children with medical conditions that

    increase operative risk

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    Morbidity

    Incidence of Perforation< 1 year old 90-100%

    1-2 years old 70-80%

    2-5 years old 50%

    > 65 years old 50%

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    Perforated Appendix

    Suspect in children 3-5 years old withhistory suggestive of appendicitis

    Bowel obstruction in a 3-5 year oldwithout obvious etiology is perforatedappendix until proven otherwise

    Fever > 101.5, WBC > 20 with bands,

    diffuse abdominal pain, guarding, SBO onAAS

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    Perforated Appendix

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    Perforated Appendix

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    Resuscitation

    NG tube, NPO

    20 cc/kg boluses until UOP > 1 cc/kg/hr and

    VS stable 1.5-2 times maintenance fluids

    Broad Spectrum Antibiotics

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    Perforated Appendix

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    Summary

    Atresias

    Hirschsprungs

    Malrotation Volvulus

    Intussusception

    Incarcerated Hernia Perforated Appendix

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    Question 2?

    Why are Pediatric Surgeons sointerested in flatus?

    Contrary to popularbelief, kids with

    obstruction can still

    have bowelmovements, but theywont pass gas!