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Lecture 2B Fluid & electrolytes (Chapter 7) Integumentary System (chapters 44-45)

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Lecture 2B. Fluid & electrolytes (Chapter 7) Integumentary System (chapters 44-45). Structure & Function of the Integumentary System. 2 regions Epidermis Dermis. Epidermis. Location: Outermost part Melanin Color Protects from UV light Keratin Water repellent. Epidermis. Function - PowerPoint PPT Presentation

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Page 1: Lecture 2B

Lecture 2B

Fluid & electrolytes (Chapter 7) Integumentary System (chapters 44-

45)

Page 2: Lecture 2B

Structure & Function of the Integumentary System

• 2 regions– Epidermis– Dermis

Page 3: Lecture 2B

Epidermis

• Location:– Outermost part

• Melanin– Color– Protects from UV light

• Keratin– Water repellent

Page 4: Lecture 2B

Epidermis

• Function– Protect!

Page 5: Lecture 2B

Dermis

• Location– Deeper layer

• Contains– Blood vessels– Nerve endings– Lymphatic vessels– Hair follicles– Sebaceous glands– Sweat glands

Page 6: Lecture 2B

Skin Assessment

• History– C/O

• Onset• Duration• Characteristics• Relief factors• Exacerbation

– Changes• Skin• Meds

Page 7: Lecture 2B

Skin Assessment

• Assess all skin areas– Redness– Swelling– Lesions– Pain

• Measure lesions

Page 8: Lecture 2B

Common skin lesions

• Macule, patch– Flat, nonpalpable change

in skin color.– Macule < 1 cm– Patch > 1 cm – i.e. freckles, Mongolian

spots

Page 9: Lecture 2B

Common skin lesions

• Papule, plaque– Elevated, solid, palpable

mass with circumscribed border.

– Papule < 0.5 cm– Plaque > 0.5 cm– i.e. moles, warts,

psoriasis

Page 10: Lecture 2B

Common skin lesions

• Nodule, tumor– Elevated, solid palpable

mass extending deeper into the dermis than a papule

– Nodule• 0.5 – 2cm

– Tumor• > 2cm

Page 11: Lecture 2B

Common skin lesions

• Vesicle, bulla– Elevated, fluid filled,

round/oval shaped, palpable mass with thin translucent walls

– Vesicle• < 0.5 cm

– Bulla• >0.5 cm

– i.e. herpes simplex, chicken poxs, burns

Page 12: Lecture 2B

Common skin lesions

• Wheal– Elevated, often reddish,

irregular borders, caused by diffuse fluid in the tissue rather than free fluid in a cavity

– i.e. • Insect bites, hives

Page 13: Lecture 2B

Common skin lesions

• Pustule– Elevated pus-filled

vesicle or bulla with circumscribed border.

– i.e. acne, impetigo, carbuncles

Page 14: Lecture 2B

Older skin

• Normal changes– i Subcutaneous tissue– Dermal thinning– i Elasticity– i Turgor– i Hair and nail growth

Page 15: Lecture 2B

Common diagnostic test for integumentary disorders

• Biopsy– Skin sample – To rule out malignancy

• Nrs. Responsibilityconsent form signedSuppliesApply dressingSend specimen to the

lab

Page 16: Lecture 2B

Pressure ulcers

• AKA– Decubitus ulcers

• Ischemic lesions• Caused by

– External pressure– Friction– Shear

Page 17: Lecture 2B

Pressure ulcer development

Pressure

i blood flow

i oxygen

ischemia

necrosis

ulceration

Page 18: Lecture 2B

High Risk Areas for Pressure ulcers

• Bony prominence– Heels– Greater trochanter– Sacrum– Ischia– Shoulder

Page 19: Lecture 2B

Usual pressure ulcer locations• Over Bony Prominences

1. Occiput 2. Ears 3. Scapula 4. Spinous Processes 5. Shoulder 6. Elbow 7. Iliac Crest 8. Sacrum/Coccyx 9. Ischial Tuberosity 10. Trochanter 11. Knee 12. Malleolus 13. Heel 14. Toes

Page 20: Lecture 2B

Other locations…• Any skin surface subject

to excess pressure• Examples include skin

surfaces under: – Oxygen tubing – Urinary catheter drainage

tubing – Casts – Cervical collars

Page 21: Lecture 2B

Pressure Ulcers from other sources of pressure

• Boots/boot straps• Heel protectors/protector straps• Oxygen tubing• Stockings• Any device that can lead to pressure induced

ischemia on the skin

Page 22: Lecture 2B

High risk clients: pressure ulcers

• Immobile• Elderly• Incontinence• Nutritional deficit• Smoking

Page 23: Lecture 2B

Complications

• Pain

Page 24: Lecture 2B

Pain with Pressure Ulcers

• 59% report some degree of pain• Only 2% receive pain medication

within 4 hours of dressing change• 45% report pain as distressing or

horrible

Page 25: Lecture 2B

Complications

• Pain• Infection

Page 26: Lecture 2B

Infection COMPLICATIONS

• Sepsis

• Localized infection

• Cellulitis

• Osteomyelitis

Page 27: Lecture 2B

Complications

• Pain• Infection• Quality of life• Cost• Death

Page 28: Lecture 2B

Mortality• 40% die per year• 60% die within 1 year

after hospital discharge

Page 29: Lecture 2B

Prevention!!!General Skin Care

• Assess• Clean & Dry• Avoid massage• i Pressure• Well balanced nutrition

Page 30: Lecture 2B

Protect skin from Moisture

• Clean• Moisturize• Barriers• Bowel & Bladder

program

Page 31: Lecture 2B

Pressure Reduction

• Rehabilitation h mobility• Repositioning • Pressure reduction devices• Float Heels• No sliding

Page 32: Lecture 2B

nutrition and fluid Support

• Dietician• Preferences• Provide assistance & time• Snacks and fluids• Supplements• Assess lab values

Page 33: Lecture 2B

Pressure Ulcer Monitoring and Treatment

Page 34: Lecture 2B

Description of Ulcers

• Stage Ulcer• Location• Size• Wound bed • Granulation tissue

• Necrotic tissue• Wound edges• Drainage• Infection• Pain

Page 35: Lecture 2B

STAGING OF PRESSURE ULCERS

Stage I: Persistent nonblanchable erythema of intact skin.

Page 36: Lecture 2B

STAGING OF PRESSURE ULCERS

• Stage II: Partial-thickness skin loss involving epidermis, dermis, or both. Ulcer is superficial and presents as an abrasion, blister, or shallow crater.

Page 37: Lecture 2B

STAGING OF PRESSURE ULCERS

Stage III: Full-thickness skin loss involving damage or necrosis of subcutaneous tissue that may extend down to, but not through, underlying fascia.

Page 38: Lecture 2B

STAGING OF PRESSURE ULCERS

• Stage IV: Full-thickness skin loss with extensive destruction, tissue necrosis, or damage to muscle, bone, or supporting structures (e.g. tendon, joint capsule). Undermining and sinus tracts may also be present.

Used with permission LWW

Page 39: Lecture 2B

STAGING OF PRESSURE ULCERS

• Unstageable: Full thickness tissue loss in which slough (yellow, tan, gray, green or brown), eschar (tan, brown or black), or both in the wound bed cover the base of the ulcer.

Page 40: Lecture 2B

Granulation tissue

• Intermediate step in healing

• Very fragile • Appearance: Shiny

red & grainy • When inadequate blood

flow exists, granulation tissue may pale in color.

Page 41: Lecture 2B

Slough

• non-viable tissue and requires debridement

• Appearance – stringy mass

• Color– white, yellow/tan, brown

• Becomes thicker and harder to remove

• Easily confused with normal tissues (tendons)

Page 42: Lecture 2B

Eschar• Dead tissue, • Color:

– Tan, brown, black • Leathery, dry hard• Soft, with purulent

discharge– Slimy.

Page 43: Lecture 2B

Prevention

• Reposition – at least every 2 hours (may use pillows, foam wedges)

• Keep head of bed at lowest elevation possible

• Use lifting devices to decrease friction and shear

• Remind patients in chairs to shift weight every 15 min

“Doughnut” seat cushions are contraindicated,may cause pressure ulcers

• Pay special attention to heels (heel ulcers account for 20% of all pressure ulcers)

Page 44: Lecture 2B

PREVENTING HEEL ULCERS

• Assess heels of high-risk patients every day

• Use moisturizer on heels (no massage) twice a day

• Apply dressings to heels:

Page 45: Lecture 2B

PREVENTING HEEL ULCERS

• Have patients wear: Socks to prevent friction (remove at bedtime) Properly fitting sneakers or shoes when in wheelchair

• Place pillow under legs to support heels off bed

• Place heel cushions to prevent pressure

• Turn patients every 2 hours, repositioning heels

Page 46: Lecture 2B

PRESSURE-REDUCINGSUPPORT SURFACES

**Use for all older persons at risk for ulcers**

Page 47: Lecture 2B

Nrs. Dx: Impaired tissue integrity

• Document• Track progress• Do not “reverse stage”

Ulcers do not replace lost muscle, subcutaneous fat, or dermis before re-epithelializing

E.g. Stage IV cannot become stage III

Page 48: Lecture 2B

Dressing

• Keep wound bed moist • Keep surrounding tissue clean & dry• Do not use antiseptic agents

Page 49: Lecture 2B

Types of Dressings

• Gauze• Transparent films• Hydrocolloid• Hydrogel

• Alginates• Foam• Composite

Page 50: Lecture 2B

Nrs. Dx: risk for infection

• Wound cleansing and dressing– frequency when purulent or foul-smelling drainage is first

observed– Avoid topical antiseptics because of their tissue toxicity

• topical antibiotics • Cultures

Page 51: Lecture 2B

Bacterial Infection

• Clinically Infected– redness– purulent drainage– foul odor– edema

Page 52: Lecture 2B

Nrs. Dx: Alt. nutrition, less than body requirements

• nutritional assessment• q day wts• h Protein• Lab

– Albumin

Page 53: Lecture 2B

MANAGEMENT:SURGICAL REPAIR

• used for stage III and IV• Risks to benefits • All wounds with necrotic tissue should be

debrided

Page 54: Lecture 2B

SUMMARY

• Older adults are at high risk for development of pressure ulcers

• Pressure ulcers may result in serious complications

• Techniques that reduce pressure, moisture, friction, and shear can prevent pressure ulcers

• Pressure ulcers should be treated with proper cleansing, dressings, debridement, or surgery as indicated