radiological signs in chest medicine part 1

Radiological Signs in Chest Medicine

Gamal Rabie Agmy MD FCCP Professor of chest Diseases Assiut university

S Curve of

Golden

When there is a mass

adjacent to a fissure the

fissure takes the shape

of an S The proximal

convexity is due to a

mass and the distal

concavity is due to

atelectasis Note the

shape of the transverse

fissure

This example represents

a RUL mass with

atelectasis

Pulmonary Artery

Overlay Sign

This is the same concept

as a silhouette sign If

you can recognize the

interlobar pulmonary

artery it means that the

mass seen is either in

front of or behind it

This is an example of a

dissecting aneurysm

Achalasia of esophagus

Inhomogeneous cardiac density

Right half more dense than left

Density crossing midline (right black

arrow)

Right sided inlet to outlet shadow

Right para spinal line (left black

arrow)

Barium swallow below Dilated

esophagus

Feeding vessel sign

Radiographic Signs of Pneumomediastinum

Subcutaneous emphysema

Thymic sail sign

Pneumoprecardium

Ring around the artery sign

Tubular artery sign

Double bronchial wall sign

Continuous diaphragm sign

Extrapleural sign

Air in the pulmonary ligament

Incomplete Border Sign

(Pregnant Lady Sign) The incomplete border sign is

useful to depict an

extrapulmonary mass on

chest radiograph

An extrapulmonary mass will

often have a inner well defined

border and an ill-defined outer

margin This can be attributed

to the inner margin being

tangential to the x-ray beam

and has good inherent

contrast with the

adjacent lung On the other

hand the outer margin is

enface or partially enface with

the x-ray beam and merges

with the pleural or chest wall

thus the border is obscured

Ginkgo leaf sign bull The ginkgo leaf sign is a chest plain radiography

appearance which is seen at extensive subcutaneous

emphysema of the chest wall Air outlines the fibers of

the pectoralis major muscle and creates a branching

pattern that resembles the branching pattern in the

veins of a ginkgo leaf

Juxtaphrenic peak sign

The juxtaphrenic peak sign refers to the peaked or

tented appearance of a hemidiaphragm which can

occur in the setting of lobar collapse It is caused by

retraction of the lower end of diaphragm at an inferior

accessory fissure (most common) major fissure

or inferior pulmonary ligament It is commonly seen

in upper lobe collapse but may also be seen in middle

lobe collapse

CT angiogram Sign

Identification of vessels within an

airless portion of lung on contrast-

enhanced CT

The vessels are prominently seen

against a background of low-

attenuation material

Associated with

bronchoalveolar cell carcinoma

lymphoma

infectious pneumonias

Fallen Lung Sign

This sign refers to the appearance

of the collapsed lung occurring

with a fractured bronchus

The bronchial fracture results in

the lung to fall away from the

hilum either inferiorly and laterally

in an upright patient or posteriorly

as seen on CT in a supine patient

DD

Pneumothorax causes a lung to

collapse inward toward the hilum

Luftsichel Sign

bullGerman for sickle of air (luft air sichel

crescent)

bullParamediastinal lucency due to

interposition of lower lobe apex between

mediastinum and shrunken upper lobe

bullOccurs more commonly on the left than in

the right

Halo Sign

CT shows nodular consolidation associated with a halo of ground-glass

opacity (GGO) in both apices resulting from invasive pulmonary

aspergillosis

This halo represents hemorrhage

When seen in leukemic patients is highly suggestive of the diagnosis of

invasive pulmonary aspergillosis

CT Halo Sign

Reverse Halo Sign

bullCentral ground-glass opacity surrounded

by denser consolidation of crescentic or

ring shape at least 2 mm thick

bullFirst described by Voloudaki in 1996

bullKim in 2003 used the term reverse halo

bullFound to be relatively specific for crypto-

genic organizing pneumonia (COP)

Reverse Halo Sign

Seen in other conditions

bullWegenerrsquos granulomatosis

bulllymphomatoid granulomatosis

bullparacoccidiodomycosis

bullneoplastic (metastasis)

bullinvasive aspergillosis

bulllipoid pneumonia

Tree-in-Bud Sign

bull

Pearl ring sign

Steeple sign

The steeple sign (also called wine bottle

sign) refers to tapering of the

upper trachea on a frontal chest

radiograph reminiscent of a church

steeple The appearance is suggestive

of croup which should be obvious

clinically A corresponding lateral x-ray

would show narrowing of the subglottic

trachea and ballooning of the

hypopharyn

Cancer Breast

Larger right breast Inverted nipple

Radiation Fibrosis of

Lung

Right lung smaller

Right hemithorax smaller

Paramediastinal fibrosis

Primary lobule

A primary pulmonary lobule is defined

as the lung unit distal to the respiratory

bronchioles It is significantly smaller

than an acinus and is composed of

alveolar ducts alveolar sacs and

alveoli

It has been estimated that

each secondary pulmonary lobule is

composed of 30-50 primary lobules

Pulmonary Acinus

Pulmonary acinus is commonly defined

as the portion of lung distal to

a terminal bronchiole and supplied by a

first-order respiratory bronchiole or

bronchioles

Each secondary pulmonary

lobule contains 3-12 acini and adjacent

acini are separated by

incomplete intralobular septae

Secondary lobule

Basic anatomic unit of pulmonary

structure and function

1-2 cm and is made up of 5-15

pulmonary acini

Supplied by a small bronchiole

(terminal bronchiole) in the

center that is parallelled by the

centrilobular artery

Pulmonary veins and lymphatics

run in the periphery

Two lymphatic systems

central network

peripheral network

The terminal bronchiole in the center divides into respiratory bronchiolies with acini that contain alveoli Lymphatics and veins run within the interlobular septa

Centrilobular area in blue perilymphatic area in yellow

Raoof S CHEST 2006 129805

Perilymphatic distribution

Centrilobular distribution

Random distribution

Random distribution distribution pattern of

pulmonary nodules where

there pattern of involvement

with respect to the lung lacks

a an architectural

prdominance These nodules

affect the fissures

peribronchovascular

structures and the center of

the secondary pulmonary

nodules They can range from

a number of pathologies

They can have variable

density and may or may not

be uniformly distributed

Some may have

haematogenous origin

Centrilobular distribution Centrilobular lung

nodules refer to an HRCT

chest imaging descriptor for

lung nodules which are

anatomically located centrally

within secondary pulmonary

lobules The term is applied on

the basis of position of the

nodule and not its morphology

Centrilobular nodules can

therefore well defined or poorly

defined


infection with endobronchial

spread

airway spread

of tuberculosis

airway spread of non

tuberculous mycobacterial

infection

airway invasive

aspergillosis

bronchoalveolar

carcinoma with airway spread

subacute hypersensitivity

pneumonitis

bronchiolitides

obliterative bronchiolitis

respiratory bronchiolitis

interstitial lung disease

pulmonary vasculitides

Churg Strauss syndrome

Perilymphatic distribution Perilymphatic lung

nodules refer to type of lung

nodules dependent on their

distribution in relation to

anatomical structures of the

lung These nodules follow

perilymphatic channels and

on imaging are typically

subpleural occur along

fissures interlobular septae

and adjacent to the

bronchovascular bundles

Perilymphatic distribution Nodules in a perilymphathic

distrubution are classically found

with pulmonary sarcoidosis1 but

can also rarely occur with other

conditions such as

silicosis

coal workers pneumoconiosis 3 -

usually simple coal workers

pneumoconiosis

lymphangitis arcinomatosis tends

to be interspersed with

interlobular septal thickening

nodular pulmonary myloidosis

lymphocytic interstitial

pneumonia

ARE NODULES IN CONTACT WITH PLEURA

NO

CENTRILOBULAR

YES

PERILYMPHATIC RANDOM

Hypersensitivity

pneumonitis

HRCT at the level of the

upper lobes reveals an ldquoill-

defined centrilobular nodular

patternrdquo characterised by

micronodules of ground-

glass opacity that are

diffusely distributed

characteristically in the

centre of the pulmonary

lobules

RBILD

HRCT at the level of the upper

lobes exhibits an ldquoill-defined

centrilobular nodular patternrdquo

characterised by micronodules of

ground-glass opacity that are


characteristically in the centre of

the pulmonary lobules In this case

the history of smoking favours the

diagnosis of respiratory

bronchiolitis interstitial lung disease

Miliary TB HRCT at the level of the upper

lobes exhibits a ldquomiliary nodular

patternrdquo characterised by random

micronodules diffusely and

symmetrically distributed within the

lungs having approximately the

same size

Miliary metastatic disease

HRCT at the level of the upper lobes

shows a ldquomilary nodular patternrdquo

characterised by random and

perilymphatic micronodules diffusely

distributed throughout the lungs that

have a more variable size compared

Differential diagnosis of a nodular

pattern of interstitial lung disease

SHRIMP Sarcoidosis

Histiocytosis (Langerhan cell

histiocytosis)

Hypersensitivity pneumonitis

Rheumatoid nodules

Infection (mycobacterial fungal viral)

Metastases Miliary TB

Microlithiasis alveolar

Pneumoconioses (silicosis coal

workers berylliosis)

Ground Glass Nodule

Ground Glass Nodule A ground glass density nodule is a circumscribed area of

increased pulmonary attenuation with preservation of the

bronchial and vascular margins A GGN can be

partly solid (part of the ground-glass opacity completely obscures

the parenchyma)

nonsolid (no completely obscured areas) - pure ground glass

nodules

Although encountered regularly the incidence of cancer in these

nodules has been reported as high as 63 (for partly solid

nodules in one study) Histologically these may represent

cancer - (eg bronchoalveolar carcinoma (BAC) adenocarcinoma

in situminimally invasive adenocarcinoma and invasive

adenocarcinoma)

atypical adenomatous hyperplasia

focal interstitial fibrosis

aspergillosis

focal pulmonary haemorrhages

A 27-year-old man is referred to you for evaluation of an abnormal chest radiograph About 5 months ago he consulted

a doctor because of excessive thirst Evaluation resulted in the diagnosis of diabetes insipidus which responded favorably to desmopressin administered nasally Recently he started to notice shortness of breath when climbing stairs and a chest radiograph was obtained

bullPatient history reveals significant tobacco smoking up to two packs daily for at least 14 years The patient noticed the shortness of breath for at least 2 years and recently he noted a point of tenderness over the chest wall lateral to the

posterior axillary line on the left bullOxygen saturation is 94 while breathing room air and the rest of his vital signs were normal Auscultation reveals only rare crackles without prolongation of the expiratory phase

There is a point of tenderness over the left sixth and seventh ribs in the posterior axillary line and a chest CT scan is obtained

The most likely diagnosis is A Metastatic tumor of unknown primary site B Sarcoidosis C Langerhans cell histiocytosis D Idiopathic pulmonary fibrosis


DECREASED LUNG

ATTENUATION

Lung Cysts

Pulmonary fibrosis (Honeycombing)

Lymphangiomyomatosis

Langerhanscell histiocytosis

Lymphocytic Interstitial Pneumonia (LIP)

Differential Diagnosis

Rough Reticular Fine Reticular

Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP

Usual Interstitial Pneumonia UIP

HRCT Findings

Reticular opacities thickened intra- and

interlobular septa

Irregular interfaces

Honey combing and parenchymal distorsion

Ground glass opacities (never prominent)

Basal and subpleural predominance

Basal and subpleural distribution

UIP

Lymphangioleiomyomatosis

(LAM)

HRCT Morphology

Thin-walled cysts (2mm - 5cm)

Uniform in size rarely confluent

Homogeneous distribution

Chylous pleural effusion

Lymphadenopathy

in young women

Lymphangioleiomyomatosis (LAM)

Tuberous Sclerosis (young man)

Langerhans Cell Histiocytosis

HRCT Findings

Small peribronchiolar nodules (1-5mm)

Thin-walled cysts (lt 1cm)

Bizarre and confluent

Ground glass opacities

Late signs irreversible parenchymal fibrosis Honey comb lung septal thickening

bronchiectasis

1 year later

Peribronchiolar Nodules Cavitating nodules and cysts



Langerhans Cell Histiozytosis

Key Features

Upper lobe predominance

Combination of cysts and noduli

Characteristic stages

Increased Lung volume

Sparing of costophrenic angle

S

M

O

K

I

N

G

after cessation of smoking

Benign lymphoproliferative

disorder Diffuse interstitial infiltration of

mononuclear cells

Not limited to the air ways as

in follicular Bronchiolitis

LIP

= Lymphocytic Interstitial Pneumonia

Sjoumlgren LIP

LIP


Rarely idiopathic

In association with Sjoumlgren‟s syndrome

Immune deficiency syndromes AIDS

Primary biliary cirrhosis

Multicentric Castlemean‟s disease

Sjoegren disease

Dry eye and dry mouth

Fibrosis bronchitis and bronchiolitis

LIP

Overlap

Sarcoid DMPM MXCT

SLE RA (pleural effusion)

Up to 40 x increased risk for lymphoma (mediastinal

adenopathy) and

2 x times increased risk for neoplasma

Young woman Dry mouth Smoker

LAM LIP Histiocytosis

Wegenerbdquos disease

Rheumatoid Arthritis

Emphysema

histopathological definition

hellippermanent abnormal enlargement of

airspaces distal to the bronchioles terminales

and

hellipdestruction of the walls of the involved

airspaces

Centrilobular Emphysema

Panlobular Emphysema

CLE and PLE in one Patient

Fibrosis and Emphysema

LAM Emphysema Fibrosis

LCH Emphysema

Fibrosis Emphysema

Emphysema

Emphysema typically presents as

areas of low attenuation without

visible walls as a result of

parenchymal destruction

EMPHYSEMA

Permanent abnormal enlargement of air

spaces distal to the terminal bronchiole

and accompanied by the destruction of

the walls of the involved air spaces

110

Centrilobular emphysema

Most common type

Irreversible destruction of alveolar

walls in the centrilobular portion of the

lobule

Upper lobe predominance and

uneven distribution

Strongly associated with smoking

Centrilobular (proximal or

centriacinar) emphysema

Found most commonly in the upper lobes

Manifests as multiple small areas of low attenuation without a

perceptible wall producing a punched-out appearance

Often the centrilobular artery is visible within the

centre of these lucencies

112

Centrilobular emphysema due to smoking The periphery of

the lung is spared (blue arrows) Centrilobular artery (yellow

arrows) is seen in the center of the hypodense area

Panlobular emphysema

Affects the whole secondary lobule

Lower lobe predominance

In alpha-1-antitrypsin deficiency but

also seen in smokers with advanced

emphysema

PANLOBULAR EMPHYSEMA

Affects the entire secondary pulmonary

lobule and is more pronounced in the lower

zones

Complete destruction of the entire pulmonary

lobule

Results in an overall decrease in lung

attenuation and a reduction in size of

pulmonary vessels

115


116


Paraseptal (distal acinar)

emphysema

Affects the peripheral parts of

the secondary pulmonary lobule

Produces subpleural lucencies

118

Paraseptal emphysema

Cystic lung disease

Lung cysts are defined as radiolucent areas with a

wall thickness of less than 4mm

Langerhans cell histiocytosis

Lymphangiomyomatosis complicated by pneumothorax

Bronchiectasis

Bronchiectasis is defined as localized bronchial

dilatation (signet-ring sign)

bronchial wall thickening

lack of normal tapering with visibility of airways

in the peripheral lung

mucus retention in the broncial lumen

associated atelectasis and sometimes air

trapping

ABPA glove-finger shadow due to mucoid impaction in central

bronchiectasis in a patient with asthma

Signet-Ring Sign

A signet-ring sign represents an axial cut of a dilated bronchus

(ring) with its accompanying small artery (signet)

Tram Tracks

Bronchial dilation with lack of tapering

HONEYCOMBING

Defined as - small cystic spaces with

irregularly thickened walls composed of

fibrous tissue

Predominate in the peripheral and subpleural

lung regions

Subpleural honeycomb cysts typically occur

in several contiguous layers DD- paraseptal

emphysema in which subpleural cysts usually

occur in a single layer

Indicates the presence of ldquoEND stagerdquo

disease regardless of the cause

129

Honeycombing

Honeycombing is defined by the presence of small cystic

spaces with irregularly thickened walls composed of

fibrous tissue

Causes


1 UIP or interstitial fibrosis

2 Connective tissue disorders

3 Asbestosis

4 Drug induced

5 NSIP (rare)


1 End stage sarcodosis

2 Radiation

3 Hypersensitivity Pneumonitis

4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis

Honeycombing and traction bronchiectasis in UIP

Typical UIP with honeycombing and traction

bronchiectasis in a patient with idiopathic

pulmonary fibrosis (IPF)

Inconsistent with UIP

pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)

bullUpper or mid lung

predominance

bullperibronchovascular

predominance

bullextensive ground glass abnormality (extent gt

reticular abnormality)

bullprofuse micronodules

(bilateral predominantly

upper lobes) bulldiscrete cysts (multiple

bilateral away from areas of

honeycombing)

bulldiffuse mosaic

attenuationair trapping (bilateral in three or more

lobes)

bullsubpleural basal

predominance

bullreticular abnormality

bullAbsence of features listed as inconsistent

with UIP pattern


predominance


bullhoneycombing with

or without traction

bronchiectasis

bullAbsence of features

listed as inconsistent

with UIP pattern

Mosiac pattern

Where is the pathology

in the areas with increased density meaning there is ground glass

in the areas with decreased density meaning there is air trapping

Pathology in black areas

Airtrapping Airway

Disease

Bronchiolitis obliterans (constrictive bronchiolitis) idiopathic connective tissue diseases drug reaction

after transplantation after infection

Hypersensitivity pneumonitis granulomatous inflammation of bronchiolar wall

Sarcoidosis granulomatous inflammation of bronchiolar wall

Asthma Bronchiectasis Airway diseases

Airway Disease

what you seehelliphellip

In inspiration sharply demarcated areas of seemingly increased

density (normal) and decreased density

demarcation by interlobular septa

In expiration bdquoblack‟ areas remain in volume and density

bdquowhite‟ areas decrease in volume and increase in

density

INCREASE IN CONTRAST

DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA


Extr Allerg Alveolitis (EAA) HRCT

Morphology

chronic fibrosis

Intra- interlobular septal thickening


Traction bronchiectasis

acute - subacute

acinar (centrilobular) unsharp densities

ground glass (patchy - diffuse)

Pathology in white Areas

Alveolitis Pneumonitis

Ground glass desquamative intertitial pneumoinia (DIP)

nonspecific interstitial pneumonia (NSIP)

organizing pneumonia

In expiration both areas (white and black) decrease in

volume and increase in density

DECREASE IN CONTRAST

DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion

Chronic pulmonary embolism

LOOK FOR

Pulmonary hypertension

idiopathic cardiac disease pulmonary

disease

CTEPH =

Chronic thrombembolic

pulmonary hypertension

Hydrostatic pulmonary

oedema

ldquoseptal patternrdquo characterised by

thickened smoothly interlobular

septae in the right parahilar area

Right pleural effusion is also seen

Lymphangitic carcinomatosis

HRCT of the right lung shows a


diffuse nodular thickening of the

interlobular septae and the right

major fissure

S Curve of

Golden

When there is a mass

adjacent to a fissure the

fissure takes the shape

of an S The proximal

convexity is due to a

mass and the distal

concavity is due to

atelectasis Note the

shape of the transverse

fissure

This example represents

a RUL mass with

atelectasis

Pulmonary Artery

Overlay Sign









dissecting aneurysm





arrow)



arrow)


esophagus

Feeding vessel sign



Thymic sail sign

Pneumoprecardium


Tubular artery sign



Extrapleural sign




useful to depict an


chest radiograph








contrast with the





















lobe collapse

CT angiogram Sign



enhanced CT




Associated with


lymphoma


Fallen Lung Sign









DD



Luftsichel Sign


crescent)





the right

Halo Sign



aspergillosis




CT Halo Sign

Reverse Halo Sign








Reverse Halo Sign








Tree-in-Bud Sign

bull

Pearl ring sign

Steeple sign










hypopharyn

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Pulmonary Artery

Overlay Sign









dissecting aneurysm





arrow)



arrow)


esophagus

Feeding vessel sign



Thymic sail sign

Pneumoprecardium


Tubular artery sign



Extrapleural sign




useful to depict an


chest radiograph








contrast with the





















lobe collapse

CT angiogram Sign



enhanced CT




Associated with


lymphoma


Fallen Lung Sign









DD



Luftsichel Sign


crescent)





the right

Halo Sign



aspergillosis




CT Halo Sign

Reverse Halo Sign








Reverse Halo Sign








Tree-in-Bud Sign

bull

Pearl ring sign

Steeple sign










hypopharyn

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure





arrow)



arrow)


esophagus

Feeding vessel sign



Thymic sail sign

Pneumoprecardium


Tubular artery sign



Extrapleural sign




useful to depict an


chest radiograph








contrast with the





















lobe collapse

CT angiogram Sign



enhanced CT




Associated with


lymphoma


Fallen Lung Sign









DD



Luftsichel Sign


crescent)





the right

Halo Sign



aspergillosis




CT Halo Sign

Reverse Halo Sign








Reverse Halo Sign








Tree-in-Bud Sign

bull

Pearl ring sign

Steeple sign










hypopharyn

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Feeding vessel sign



Thymic sail sign

Pneumoprecardium


Tubular artery sign



Extrapleural sign




useful to depict an


chest radiograph








contrast with the





















lobe collapse

CT angiogram Sign



enhanced CT




Associated with


lymphoma


Fallen Lung Sign









DD



Luftsichel Sign


crescent)





the right

Halo Sign



aspergillosis




CT Halo Sign

Reverse Halo Sign








Reverse Halo Sign








Tree-in-Bud Sign

bull

Pearl ring sign

Steeple sign










hypopharyn

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure



Thymic sail sign

Pneumoprecardium


Tubular artery sign



Extrapleural sign




useful to depict an


chest radiograph








contrast with the





















lobe collapse

CT angiogram Sign



enhanced CT




Associated with


lymphoma


Fallen Lung Sign









DD



Luftsichel Sign


crescent)





the right

Halo Sign



aspergillosis




CT Halo Sign

Reverse Halo Sign








Reverse Halo Sign








Tree-in-Bud Sign

bull

Pearl ring sign

Steeple sign










hypopharyn

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure



useful to depict an


chest radiograph








contrast with the





















lobe collapse

CT angiogram Sign



enhanced CT




Associated with


lymphoma


Fallen Lung Sign









DD



Luftsichel Sign


crescent)





the right

Halo Sign



aspergillosis




CT Halo Sign

Reverse Halo Sign








Reverse Halo Sign








Tree-in-Bud Sign

bull

Pearl ring sign

Steeple sign










hypopharyn

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure















lobe collapse

CT angiogram Sign



enhanced CT




Associated with


lymphoma


Fallen Lung Sign









DD



Luftsichel Sign


crescent)





the right

Halo Sign



aspergillosis




CT Halo Sign

Reverse Halo Sign








Reverse Halo Sign








Tree-in-Bud Sign

bull

Pearl ring sign

Steeple sign










hypopharyn

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

CT angiogram Sign



enhanced CT




Associated with


lymphoma


Fallen Lung Sign









DD



Luftsichel Sign


crescent)





the right

Halo Sign



aspergillosis




CT Halo Sign

Reverse Halo Sign








Reverse Halo Sign








Tree-in-Bud Sign

bull

Pearl ring sign

Steeple sign










hypopharyn

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Fallen Lung Sign









DD



Luftsichel Sign


crescent)





the right

Halo Sign



aspergillosis




CT Halo Sign

Reverse Halo Sign








Reverse Halo Sign








Tree-in-Bud Sign

bull

Pearl ring sign

Steeple sign










hypopharyn

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Luftsichel Sign


crescent)





the right

Halo Sign



aspergillosis




CT Halo Sign

Reverse Halo Sign








Reverse Halo Sign








Tree-in-Bud Sign

bull

Pearl ring sign

Steeple sign










hypopharyn

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Halo Sign



aspergillosis




CT Halo Sign

Reverse Halo Sign








Reverse Halo Sign








Tree-in-Bud Sign

bull

Pearl ring sign

Steeple sign










hypopharyn

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

CT Halo Sign

Reverse Halo Sign








Reverse Halo Sign








Tree-in-Bud Sign

bull

Pearl ring sign

Steeple sign










hypopharyn

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Reverse Halo Sign








Reverse Halo Sign








Tree-in-Bud Sign

bull

Pearl ring sign

Steeple sign










hypopharyn

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Reverse Halo Sign








Tree-in-Bud Sign

bull

Pearl ring sign

Steeple sign










hypopharyn

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Tree-in-Bud Sign

bull

Pearl ring sign

Steeple sign










hypopharyn

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Pearl ring sign

Steeple sign










hypopharyn

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Steeple sign










hypopharyn

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Cancer Breast



Lung

Right lung smaller



Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Primary lobule






alveoli




Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Pulmonary Acinus





bronchioles





Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Secondary lobule




pulmonary acini








central network

peripheral network






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure






Random distribution





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure





a an architectural


affect the fissures

peribronchovascular








Some may have













defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure












defined



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure



spread

airway spread

of tuberculosis



infection

airway invasive

aspergillosis

bronchoalveolar



pneumonitis

bronchiolitides
















and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure











and adjacent to the






conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure





conditions such as

silicosis



pneumoconiosis






pneumonia


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure


NO

CENTRILOBULAR

YES


Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Hypersensitivity

pneumonitis










lobules

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

RBILD


















same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure







same size










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure










SHRIMP Sarcoidosis


histiocytosis)


Rheumatoid nodules






Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Ground Glass Nodule





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure





the parenchyma)


nodules






adenocarcinoma)



aspergillosis









DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure








DECREASED LUNG

ATTENUATION

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Lung Cysts







Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure


Traction

Bronchiectasis

and

Interface

sign

Honey

combing

UIP UIP or NSIP


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure


HRCT Findings


interlobular septa






UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure


UIP


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure


(LAM)

HRCT Morphology





Lymphadenopathy

in young women




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure




HRCT Findings






bronchiectasis

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

1 year later





Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure



Key Features






S

M

O

K

I

N

G




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure




mononuclear cells



LIP


Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Sjoumlgren LIP

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

LIP


Rarely idiopathic





Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Sjoegren disease



LIP

Overlap

Sarcoid DMPM MXCT



adenopathy) and






Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure





Emphysema




and


airspaces






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure






LCH Emphysema

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Fibrosis Emphysema

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Emphysema





EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

EMPHYSEMA





110


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure


Most common type



lobule


uneven distribution









112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure








112









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure









emphysema




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure




zones


lobule



pulmonary vessels

115


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure


116



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure



emphysema




118


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure


Cystic lung disease





Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure



Bronchiectasis








trapping



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure



Signet-Ring Sign



Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Tram Tracks


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure


HONEYCOMBING



fibrous tissue


lung regions







129

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Honeycombing



fibrous tissue

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Causes




3 Asbestosis

4 Drug induced

5 NSIP (rare)



2 Radiation


4Silicosis

4 End stage ARDS

131

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Honeycombing

HRCT showing

subpleural

broncheolectasis






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure






pattern (any one of

seven features)

Possible UIP

pattern (all

three features)

UIP pattern (all

four features)


predominance


predominance







honeycombing)

bulldiffuse mosaic


lobes)


predominance



with UIP pattern


predominance



or without traction

bronchiectasis



with UIP pattern

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Mosiac pattern





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure





Airtrapping Airway

Disease






Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Airway Disease







density


DIFFERENCES

AIRTRAPPING

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Bronchiolitis

obliterans

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Early Sarcoidosis

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Chronic EAA



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure



Morphology

chronic fibrosis




acute - subacute











DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure









DIFFERENCES

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

DI

P

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Cellular

NSIP

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

Mosaic Perfusion


LOOK FOR



disease

CTEPH =




oedema










major fissure

CTEPH =




oedema










major fissure


oedema










major fissure






major fissure

radiological signs in chest medicine part 1

Health & Medicine

silhouette sign

ginkgo leaf sign

juxtaphrenic peak sign

luftsichel sign german

pulmonary artery overlay

chest wall

adjacent lung

mass adjacent