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© 2017 Virginia Mason Medical Center
Soft Tissue Emergencies
C. Craig Blackmore, MD, MPH, FASERVirginia Mason Medical Center
Seattle, WA
© 2012 Virginia Mason Medical Center© 2012 Virginia Mason Medical Center
Soft Tissue Emergencies
T2 Fat Sat
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Soft Tissue Emergencies
• Infections§ Superficial vs deep§ Specific infections
• Hemorrhage• Burns and frostbite• Inflammatory conditions• Miscellaneous
© 2012 Virginia Mason Medical Center© 2012 Virginia Mason Medical Center
Superficial Infections
• Skin injury with extension• Predisposing factors§ Diabetes, immunocompromised§ Circulation (arterial or venous), trauma
• Imaging§ Abscess, extent of infection§ Involvement of bone, joint§ Foreign body
• Ultrasound, radiography, CT, MRI
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Immunosuppressed Abscess
STIR T1 Gad
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Intravenous Drug Use
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Foreign Body
Splinter in thumb
Courtesy of B. Hashimoto, MD
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Deep Infections
• Involvement of muscle, deep fascialcompartments
• Osteomyelitis, septic arthritis• Venous structures (deep)• Intrathoracic/intraabdominal structures
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Muscle AbscessSTIR
STIR
T1 Gad
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Types of Infections
• Animal bites• Marine infections• Pyomyositis• Necrotizing fasciitis (gas/wet gangrene)§ Fournier’s gangrene§ Diabetic foot
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Animal Bites
• 2-5 million per year in USA• Mixed oral flora• Dog (80%) extensive soft tissue damage• Cat (15%) deep puncture• Rodent (3%)• Human (2%) underestimate initial damage
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Animal Bites
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Dog Bite
• Soft tissueswelling only
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Dog Bite
3 weeks
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Dog Bite
2 weeks
STIR
T1 Gad
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Human Bite
8 days later
STIR T1 Gad
T1 Gad
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Rattlesnake Bite
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Marine Infections
• Sea urchins, corals, fish, stingrays• High risk of infection§ Vibrio sp§ Deep, necrotic infections rare
• Local treatment• Foreign body
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Sea Urchin
79 spines removed
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Sea Urchin
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Pyomyositis
• Predisposing factors§ Immunocompromise§ Intravenous drug use§ Trauma
• Staph aureus/MRSA, strep, E. coli• Hematogenous• DDX: muscle injury, hematoma, muscle infarct,
septic arthritis• Myositis progresses to abscess
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Muscle AbscessT1 Gad
STIR
STIR
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Muscle Abscess
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Intravenous Drug Use
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Arm Abscess
STIR T1 Gad
T1 Gad
T1 Gad
T1
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Atypical Infections: MAI
STIR
T1 Gad
T1 GadT1 Gad
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Necrotizing Fasciitis
• Rapidly progressive soft tissue infection• Type 1: Polymicrobial, clostridium§ Diabetes, vascular disease,
immunocompromised§ Fournier’s Gangrene
• Type 2: Group A Strep, Staph Aureus§ Immunocompetent§ “Flesh eating bacteria”
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Necrotizing Fasciitis
• Rapidly Progressive• Spreads along fascial planes§ Less muscle involvement
• Microvascular occlusion, necrosis• Endotoxin release• Mortality of 70-80%
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Necrotizing Fasciitis
• 63 yo obese diabetic female
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Necrotizing Fasciitis
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Necrotizing Fasciitis
• 65 yr old diabetic with ulcer on foot
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Necrotizing Fasciitis4 days later
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Necrotizing Fasciitis
• 66 yr old male with “blood blister” on great toe thatruptured
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Necrotizing Fasciitis
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Hematoma
• May be indistinguishable from infection• Peripheral enhancement• May require aspiration for diagnosis• Anti-coagulants, bleeding dyscrasia• Compartment syndrome
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HematomaSTIRT1 Gad
T1
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Hematoma
STIR
T1
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Hemophilia
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Hemorrhagic Bursitis
PD Fat Sat
PD Fat Sat
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Morel-Levalee
PD Fat Sat PD Fat Sat
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Thermal Injury
• Clinical§ Size, depth§ Age, risk factors
• Imaging§ Integrity of underlying structures§ Vascular supply§ Complications
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Burns
Courtesy of K. Linnau, MD
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Burns
17 year old, 2 months after 3rd degree burn bilateral lower extremity
Courtesy of K. Linnau, MD
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Electrical Burns• Spare skin-higher resistance• Deeper structures at risk
Courtesy of K. Linnau, MD
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Frostbite
• Toes, fingers§ Spare thumb
• Depth of injury• Viability of tissue• Late§ Tissue loss§ Secondary osteomyelitis
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Frostbite5 months after frostbite
8 months 21 months
Courtesy of K. Linnau, MD
16 months
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“Dry” Gangrene
48 year old woman Lupus, Raynaud’s
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Inflammatory Conditions
• Muscle inflammation• Edema, cellular infiltration, cell death, fatty
replacement• Multiple etiologies:§ Inflammatory myositis§ Dermatomyositis§ Rhabdomyolysis§ Overuse
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Inflammatory MyositisSTIR
T2 Fat Sat
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Inflammatory MyositisSTIR
STIR
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Radiation MyositisT1 Gad T1 Gad
T2 Fat Sat
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Myositis
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DermatomyositisSTIR
T1 Fat Sat
T1 Gad
T1 Gad
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Rhabdomyolysis
STIR
T1 Gad
T1 Pre
• Myonecrosis syndrome• Trauma, muscle compression• Overuse• Drugs, toxins, electrolyte
disturbance
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Delayed Onset MuscleSoreness (DOMS)
STIR
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Compartment Syndrome• Increased pressure in anatomically
confined space• Decrease blood flow• Necrosis
• Calf (36%), forearm (10%)
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Compartment Syndrome
Courtesy of K. Linnau, MD
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Leukemia
T1 Gad
T1 Gad
T1 Gad
T1
T2
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Barotrauma
• Navy diver at 50 feet tank failure
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Soft Tissue Emergencies
• Infections§ Superficial vs deep§ Specific infections
• Hemorrhage• Burns and frostbite• Inflammatory conditions• Miscellaneous
© 2012 Virginia Mason Medical Center© 2012 Virginia Mason Medical Center
Thank You!