technical aspects of chemical warfare defense lesson topic 4.1
TRANSCRIPT
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Technical aspects of chemical warfare defense
Lesson topic 4.1
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Enabling Objectives
Perform buddy aid and self aid procedures Select the treatments of chemical warfare
agents Classify chemical agents by physical state,
physiological action or tactical use Select the physiological symptoms of
chemical warfare agents
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Chemical Defense Terms, Classifications and definitions
Hydrolysis Decomposition in water Formation of a new substance Changes/dilutes
Detoxification Rate at which the body will counteract
effects of a poisonous substance Exposure time Dose & concentration
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Chemical Defense Terms, Classifications and definitions
Persistence How long the agent is effective after
used Physical properties Volatility, gas, solid or liquid
Weather conditions Wind speed, heat, rain, etc.
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Chemical Defense Terms, Classifications and definitions
dissemination Explosive/nonexplosive munitions
Percutaneous enter through skin
Non-percutaneous respiratory tract, ingestion, injure the
eyes but not the skin
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Chemical Defense Terms, Classifications and definitions
Tactical use How agents are used against personnel
Physical State stored or disseminated
Physiological action physical effect
Cumulative Repeated exposures (additive effect)
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Chemical Defense Terms, Classifications and definitions
Casualty agents agents cause death or severe injury
Incapacitating agents agents Produce temporary physiological
and/or psychological effects Physical state
Solid Liquid Gas or vapor
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Chemical Agents
Nerve Tactical use
Casualty agent
Deadliest
GA, GB, and GD:
Agents for quick casualties
VX: Agents for delayed casualties
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Nerve
Dispersed
Artillery, mortar, rocket
Aircraft spray
Bombs/bomblets & missiles
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GA Colorless to light brown liquids
GB Colorless liquids, no odor in pure state
GD Colorless liquids, Fruity odor of camphor
VX: Amber colored oil
Physical state
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Disseminated in persistent & non-persistent form
G Agents, Liquid form 1 - 2 daysV Agents, 1 - 2 weeks, persistent
Persistency
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General Physiological action Absorbed through any body surface Impacts entire nervous system Inhibits or blocks the action of
cholinesterase Results in a build up of acetylcholine Must have an even balance of these two
enzymes for the muscle system to function
Death due to respiratory failure
Physiological actions
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Mild symptoms pinpointed pupils/blurred vision Runny nose Salivation/drooling Difficulty in breathing Excessive sweating
Physiological actions
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Physiological Actions
Severe symptoms Nausea & Vomiting Cramps Involuntary defecation & urination Twitching & jerking of muscles Headaches Strange/confused behavior Violent convulsions Cessation of breathing, death
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Rate of action Rapid, death may occur within 15 minutes
Rate of detoxification Slight, body can detoxify slowly Cumulative
Nerve agent
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Nerve Agent Pyridostigmine bromide Pretreatment Tablet (NAPP) 21 tablets 7 day supply 1 tablet every 8 hours
Protects enzyme in the body from nerve agents
Antidote enhancer
Nerve agent Pretreatment
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If you miss a dose, it will not be made up Do not take 2 tablets at once
Start over, 1 tablet every 8 hoursTaking more than 1 tablet at a time
does not provide additional protectionIt may be more hazardous if there is
exposure to a nerve agent
NAPP
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NAPP OVERDOSE SYMPTOMS
Abdominal crampsNauseaDiarrheaOccasional skin rashWeaknessDimmed or blurred vision due to
pinpointed pupilsIf any of the signs occur; consult
medical personnel
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Atropine auto injector Stored in cool, dry spaces
Carried in mask carrier when issued
Issued 3 auto injectors
Nerve agent antidotes
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Antropine Injector
Characteristics Yellow end is safety cap
Green body with yellow stripes
Green end is injector end
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Antropine Injector
Administration Remove the yellow safety cap & press the
green injector end into the outer thigh
Hold for 10 seconds Place used Auto injector in front smock
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Administration
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Nerve agent antidotes
2-Pam Chloride, (Enzyme reactivation) Gray ridged safety cap
Black end is injector end
Stored with atropine 3 Auto injectors will be issued
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Remove gray ridged safety cap
Press black injector end to the outer thigh
Same area as the atropine injection
Hold for 10 seconds Place used Auto injector in front smock
2PAM Chloride
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CANA
CANA (Convulsion Antidote for Nerve Agents) 10mg Diazepam Black end is injector end Gray safety cap Ridges of the CANA injector distinguish
it from other injectors 1 injector issued Buddy aid only
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2 PAM Chloride & Atropine auto injectors
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Chemical Agents
Blister agents Tactical use: casualty agent
Causes incapacitation
Death by infection
Could supplement nerve agents
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Main groups Mustards (H) Lewiste (L) Phosgene oxime (CX)
Dispersed by Artillery, mortar, rockets Aircraft spray Bombs/bomblets
Blister
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Most found as liquids
Colorless to dark-brown
Oily droplets
Extremely persistent
Physical state
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Effect the eyes, the lungs & form blisters on the skin
Eyes are more vulnerable than the skin
Breathing passages very vulnerable
Initially irritates cells
Produces cell poisoning due to infections
Symptoms vary depending upon agent
Physiological actions
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Mustards: H (Levinstein) HD (Distilled), HN (Nitrogen)
No immediate painRedness of skin will appear at the
exposed areas onlyOccurs 4 - 6 hours after contactThin skinned & warm moist areas are
very susceptibleConcentration of agent & exposed
are important factors
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Eye exposure causes severe inflammation or damage temporary or permanent
Water filled blisters similar to burn blisters will appear within 1 - 2 days
Healing time ranges from 3 - 8 weeks
HD burns will heal more slowly
Mustards: H (Levinstein) HD (Distilled), HN (Nitrogen)
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Inhalation of mustard agents will damage the respiratory track which will then produce symptoms similar to those of choking agents (Discussed later).
Mustards: H (Levinstein) HD (Distilled), HN (Nitrogen)
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Lewisite (L)
Immediate pain upon contactEye exposure will produce searing
sensationEYES MUST BE
DECONTAMINATED IMMEDIATELY (WITHIN 1 MIN), OR PERMANENT DAMAGE MAY OCCUR
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Reddening of exposed skin areas will occur within 30 min
Blisters will form in approximately 12 hours with skin burns much deeper
If inhaled, injuries & symptoms similar to choking agents will occur
Death may result within minutes
Lewisite (L)
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Immediate pain on contact (bee sting)Irritation to eyes & nasal membranesExposed skin bleaches in 30 seconds &
will later be surrounded by a red ring/welt
Bleached areas darken in 24 hoursScab formation occurs after 1 weekHealing time, 2 months
Phosgene Oxime (CX)
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Little or no pain at the time of exposure with most blister agents
Lewisite (L) & phosgene oxime (CX) cause immediate pain on contact
All blister agents react almost immediately
Rate of action
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MUSTARD AGENT
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Extremely slow, Effects are cumulative
Self aid/Buddy aid Don mask & clothing Liquid contamination must be washed
from the eye immediately After 2 minutes, treatment is of little use
Rate of detoxification
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Phosgene (CG) & Diphosgene (DP) Tactical use: Casualty agentUsed extensively during WW-I accounted for
80% of all fatal chemical agent casualties Much less effective than nerve agents, but
may be used for quick incapacitating effects
Choking Agents
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Most dangerous member of the group Physical state
Normally found as a colorless gas Odor, new mown hay, grass, or green corn
Phosgene (CG)
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Penetrates lungs causing them to fill with fluid
Permanent lung damage
Effects are confined to the lungs
Physiological action
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Uneasiness & fearSerious attacks of coughing
producing large quantities of white/yellow or bloody frothy fluid
Nausea, vomiting & gastric painBreathing is quick, shallow & painful
Symptoms
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The pulse is fast and faint
Shock followed by death through cardiac arrest or asphyxia
"Dry-land Drowning"
Symptoms
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Choking Agents
Rate of action Immediate or delayed
Rate of detoxification Not detoxified; cumulative
Self aid/Buddy aid Don protective mask
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Tactical use: casualty agent High concentration to cause death Quick casualty effects Typical blood agents
Hydrogen Cyanide (AC)Cyanogen Chloride (CK)
Blood Agents
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Colorless liquids
Odor of bitter almonds
May irritate the eyes & nose
Extremely non-persistent
Physical state
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Absorbed by the mucous membranes & skin
Penetrates lungs without causing injuryFollows the same route as the
oxygenated blood until it reaches the cell tissues
Inhibits the ability of the cell walls to retain oxygen
Death due to respiratory failure
Physiological action
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Increased breathing rate Headaches, dizziness & giddiness Faster pulse rate & pounding of the
heartFlushed skin & lips
Symptoms
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ConvulsionsComaDeathRate of action
Immediate with death occurring from seconds to within fifteen minutes
Advanced symptoms
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Blood Agents
Rate of detoxification Rapid, effects are non-cumulative
Self aid/Buddy aid Don protective mask
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Riot Control/Training Agents
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Riot Control/Training Agents
Temporarily irritating effects
Two categories Vomiting Tearing
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(Adamsite (DM), DA, and DC)
Violent uncontrollable sneezing, cough, nausea, vomiting, and a general feeling of body discomfort
Vomiting compounds
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Large flow of tears & intense eye painTraining & in riot control, although CS
may also be used in combatWarm, moist skin, especially face,
neck, ears & body folds are susceptible to irritation
Tear producing compounds (CS, CN)
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Terms & classificationsNerveblisterBloodChokingRiot Control, Training agents
Summary and Review
CBR