the cardiovascular system
DESCRIPTION
The Cardiovascular System. Chapters 15-18. The Heart. Location: Thoracic cavity Behind sternum Between the lungs. The Heart:. Protective sac PERICARDIUM Layers of heart tissue Epicardium Myocardium Endocardium. The Heart. Function Double pump Right - PowerPoint PPT PresentationTRANSCRIPT
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The Cardiovascular System
Chapters 15-18
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The Heart
• Location:– Thoracic cavity– Behind sternum– Between the lungs
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The Heart:
• Protective sac– PERICARDIUM
• Layers of heart tissue– Epicardium– Myocardium– Endocardium
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The Heart
• Function– Double pump– Right
• Receives blood from the body (pumps blood to)
• Lungs– Left
• Receives blood from the lungs
• body
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The Heart: Blood flow
• Inferior & Superior Vena cava
• Right Atrium – Tricuspid valve
• Right Ventricle – Pulmonary valve
• Pulmonary Arteries – Pulmonary arterioles – Pulmonary capillaries – Pulmonary venules
• Pulmonary veins
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The Heart: Blood flow
• Pulmonary veins • Left Atrium
– Bicuspid (mitral) valve • Left ventricle • Aorta
– Aortic Valve• (body)• Arteries• Capillaries • Vein
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The Heart: Blood flow
• Veins • Inferior & Superior Vena cava
• Right Atrium – Tricuspid valve
• Right Ventricle • Pulmonary Arteries
– Pulmonary arterioles – Pulmonary capillaries – Pulmonary venules
• Pulmonary veins
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Heart Beat
• Lub-dub; lub-dub• First sound– S1 (lub)– “AV valves” close– (valves between the
atriums & ventricles)– Tricuspid– Bicuspid
• Second sound– S2 (dub)– “semilunar valves” close– Aortic valve– Pulmonary valve
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Conduction System
• Cardiac muscle does not need the nervous system to generate an electrical impulse
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Conduction System
• Heart beat is controlled by special cells in the myocardium –“Conduction system”
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Conduction Systemhttp://www.youtube.com/watch?v=te_SY3MeWys
• Sinoatrial (SA) node – AKA: Pacemaker
• Internodal pathways • Atrioventricular (AV) node • Bundle of His • Purkinje fibers
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Conduction SystemElectorcardiogram (EKG)
• http://www.pennmedicine.org/encyclopedia/em_DisplayAnimation.aspx?gcid=000001&ptid=57
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Cardiac Cycle
• Contraction & relaxation of the heart =– 1 heart beat
• Diastole– Ventricles relax
• Systole– Ventricles contact
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Normal Heart Rate
• 70-90 / minute• > – tachycardia
• < – bradycardia
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Cardiac output
• Stroke Volume (SV)–Amount of blood
pushed from the heart with each heart beat –@ 70 mL
• Cardiac Output (CO)–Amount of blood
pumped in 1 min.• CO = HR (pulse) x SV–@4-8 L/min
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Peripheral Vascular System• Network of blood vessels
that carry blood to peripheral tissues and then return it to the heart
• Arteries – carry blood away from the
heart• Capillaries• Veins
– carry blood towards the heart
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Capillaries
• Where Oxygen & nutrients are exchanges
• Very permeable
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Arteries & Veins
• Aorta – Arteries
• Arterioles– Capillaries
• Venules– Veins
• Superior & Inferior Vena Cava
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Blood vessel structure
• Inner layer – Slick surface
• Middle layer– Smooth muscle
• Out layer – Protection
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Blood Vessel Structure
• Smooth muscle function– Constriction
• Narrowing– Dilation
• Widening
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Blood Vessel Structure
• Veins have something Arteries don’t have!– Valves
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Blood Pressure (BP)
• Force exerted by blood against the walls of the arteries
• SYSTOLIC– Pressure exerted when the
heart contracts• DIASTOLIC– Pressure when the heart is
filling
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Blood Pressure (BP)
• Optimal Blood Pressure–<120 / 80
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Blood
• Oxygenated–Blood that is carrying
oxygen O2• Deoxygenated–Blood that is not
carrying oxygen–Carrying CO2
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Oxygenated & Deoxygenated
• Inferior & Superior Vena cava
• Right Atrium – Tricuspid valve
• Right Ventricle – Pulmonary valve
• Pulmonary Arteries – Pulmonary arterioles – Pulmonary capillaries – Pulmonary venules
• Pulmonary veins
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Oxygenated & Deoxygenated
• Pulmonary veins • Left Atrium
– Bicuspid (mitral) valve • Left ventricle • Aorta
– Aortic Valve• (body)• Arteries• Capillaries • Vein
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The Heart: Blood flow
• Veins • Inferior & Superior Vena cava
• Right Atrium – Tricuspid valve
• Right Ventricle • Pulmonary Arteries
– Pulmonary arterioles – Pulmonary capillaries – Pulmonary venules
• Pulmonary veins
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Cardiac Assessment
• Health History– Chest pain– Breathing problems
• Short of breath– Changes in energy levels– Medication– Life style
• Alcohol intake• Exercise• Smoking• Illicit drugs
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Cardiac Assessment
• Skin Color– Pallor
• Pale– Cyanosis
• Blue
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Cardiac Assessment
• Vital Signs• Peripheral pulses• Capillary refill• Edema?• Auscultate the heart
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Diagnostic Tests
• Lipid profile– Cholesterol– Triglycerides– High-density lipoproteins
(HDL’s)– Low-density lipoproteins
(LDL’s)
• Assess risk for atherosclerosis & coronary heart disease
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Diagnostic Tests
• Serum Cardiac Markers(Cardiac enzymes)– Creatine phosphokinase– CK-MB– cTnT
– cTn1
• Heart muscle cells that are dead or damaged release these proteins.
• Increased levels = heart damage
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Diagnostic Test
• Electrocardiogram (ECG)– Record of the electricity
of the heart
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ECG
• P-wave–Arial
depolarization
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ECG
• QRS complex• Ventricular
depolarization
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ECG
• T-wave• Ventricular
repolization
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ECG
• P-R interval
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ECG
• P-R interval• Period of time
from –SA node to–AV node &
Bundle of His
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Imaging Techniques
• CT scan– 3-D X-ray machine
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Imaging Techniques
• MRI scan– Magnetic resonance
imaging
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MRI: Rules
• No METAL in the room with the machine
• Assess for –Metal implants–Claustrophobia
• http://www.youtube.com/watch?v=7g5UVrOt2CI
• http://www.youtube.com/watch?v=6BBx8BwLhqg&NR=1
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Imaging Techniques
• Angiography–INVASIVE• Insertion into an
artery–X-rays +
fluoroscopy
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Imaging Techniques
• Angiography– INVASIVE– Risks – • Bleeding• Clot
– Assess:• Insertion site• Pedal pulses
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WARNING: Angiography
–Closely monitory the client, the insertion site, the extremity after the procedure. Immediately report evidence of bleeding, pain or a pale pulseless extremity to the charge nurse & physician
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Coronary Heart Disease
• AKA– Coronary Artery Disease
• Definition– Narrowing of the
arteries that supply blood to the heart muscles
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Arteriosclerosis & Atherosclerosis
Arteriosclerosis• Arteries that are
– Thick– Non-elactic
Atherosclerosis• Plaque buildup in the
arteries• #1 cause of CHD
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CHD: Risk Factors
Changeable• Smoking• Obesity• Physical inactivity• High fat diet• High blood pressure
– Hypertension / HTN• High blood lipids
– Hyperlipidemia• Diabetes Mellitus
Non-changeable• Age• Gender• Race• Heredity
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Atherosclerosis: Pathophysiology
• Narrow arteries • i blood flow • ISCHEMIA– Not enough blood or
oxygen for their metabolic needs
• Infarction– Tissue death
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S&S of atherosclerosis
• Due to – ISCHEMIA
• Angina• Myocardial Infarction
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Atherosclerosis: IDT Interventions
• Quit smoking• Diet– Low fat
• Exercise• Control BP• Control DM
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Atherosclerosis: IDT Interventions
• Medications– Cholesterol-Lowering
Drugs• Statins
• Nursing Implications– Monitor • Serum lipid levels• Liver
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Angina Pectoris
• “Chest pain when there is a temporary imbalance between myocardial blood supply and demand”.
• Chest pain due to i blood/ oxygen to the heart
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Angina Pectoris: S&S
• Pain– Chest– Radiating to
• Neck• Shoulder• Arm • Jaw
– Tight, squeezing, heavy• Shortness of Breath– Dyspnea
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IDT: Angina Pectoris
• WARNING!!!!
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IDT: Angina Pectoris
• Medications– Nitrates– Beta Blockers– Calcium Channel
blockers
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Nitrates
• Action:– Dilate blood vessels – h blood flow to the
heart• E.G.– Nitroglycerin
• Route– Sub-lingual– Patches– Ointment
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Beta-Blockers
• Decrease workload of the heart– i pulse– i BP
• Action– Block adrenalin– Block renin-angiotensin system
• Nrs Implications– Take BP & pulse before– Hold if <50/min
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Calcium Channel Blockers
• Purpose– Treat angina and HTN
• Action– Relax smooth muscle – Vasodilation
• Nrs. Implications– BP & pulse before– Hold if < 50/min
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Myocardial Infarction (MI)
• AKA: Heart attack• i blood / oxygen to the
heart muscle • Ischemia • Infarction / necrosis • i cardiac output• Death
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Myocardial infarction: S&S
• Chest pain• Tachycardia• Short of breath– Dyspnea
• Skin: cool, clammy• Diaphoresis• Anxiety• N&V
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Angina VS. Myocardial InfarctionAnginaProceeded by: stress or exertionRelieved by: rest & nitroglycerineDuration: < 15 minNOT assoc w/: - N/V- Diaphoresis
Myocardial InfarctionProceeded by: anything (anytime)Relieved by: opioids
Duration: > 30 minAssoc w/- N/V- Diaphoresis- dyspnea
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IDT: Myocardial Infarction
• IV (for meds)• Oxygen (via nasal
cannula)• Bed Rest
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Meds: Myocardial Infarction
• Aspirin– Thins the blood–Anticoagulant
• S/E–Bleeding–GI upset
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Meds: Myocardial Infarction
• Pain reliever–Nitroglycerin–Morphine sulfate–Relax blood vessels –Vasodilation –h blood flow – i pain
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Meds: Myocardial Infarction
• Fibrinolytic agents–Drugs that
dissolve clots– S/E• Bleeding
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Heart Failure
• “Inability of the heart to function as a pump to meet the needs of the body”
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Heart Failure
• What affect will heat failure have on Cardiac Output?–decrease
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Heart Failure – compensatory mechanisms
• Cardiac output i• Sympathetic nervous
system –Adrenalin • heart rate• Smooth muscle
contraction
• Cardiac output i• Renin – angiotensin-
aldosterone system– vasoconstriction–Na+ & H2O
retention
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Left side heart
• Left heart pumps blood – BODY!
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Left-side heart failure
• i cardiac output• h pulmonary
pressure (backs up)
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S&S: Left Sided Heart Failure
• Activity intolerance• Dizziness/ syncope• ---------• Short of breath• Orthopnea• Cough• Crackles• Tachycardia
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Right side heart
• Right heart pumps blood – Lungs
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Right-side heart failure
• h venous pressure • Edema
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S&S: Right-Side Heart Failure
• Activity intolerance• Edema• Jugular vein distention
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Medications: Heart Failure
• Diuretics– Furosemide / Lasix– Action• h urine output
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Medications: Heart Failure
• Positive Inotropic Agents– Digoxin / Lanoxin
• h contractility (heart contraction strength)
• i pulse– Nursing
• If pulse < 60 hold med
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Hypertension
• AKA: – high blood pressure
• BP – > 140 systolic– > 90 diastolic
• Pg 430 - 438
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Hypertension: Risk Factors
Changeable• Diet
– High Na+– Low K+
• Obesity• Smoking• Alcohol (excess)• Stress• Diabetes
Non-changeable• Family history• Age• Race
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Hypertension: S&S
• “The Silent Killer”– VAGUE!!– H/A– Blurred vision
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IDT Tx: Hypertension
• Monitor BP • No Caffeine• No smoking• Lifestyle changes– Diet– Alcohol / Smoking– Physical Activity– Stress reduction
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Hypertension: Rx
• Broad classification– Anti-hypertensives
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Hypertension: Rx
• Beta-blockers– i the heart rate– i contractility –
• i cardiac output
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Hypertension: Rx
• Calcium Channel blockers– Block Ca+ channels in
arterial smooth muscle – Vasodilation
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Hypertension: Rx
• Diuretics– h urine output – i fluid volume
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Venous Thrombosis
• Thrombo = clot• -osis = abnormal condition• Blood clot (forms on the
wall) of the vein – blockage of blood flow
back to the heart
• Pg 448 - 454
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Thrombophlebitis
• Thrombo = – Clot
• Phlebo – Vein
• -itis– Inflammation
• Inflammation caused by clot in the vein
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Venous thrombosis
• 3 Factors– Venous stasis– h blood coagulation– Vessel wall injury
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Venous thrombosis
• Blood returns to the heart through collateral vessels
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Venous thrombosis
• Thrombus (thrombi) may break loose – Embolus (emboli)
• Thrombi =– Stationary
• Emboli– Mobile
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Deep Vein Thrombi: S&S
• Most common place– Calf
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Deep Vein Thrombi: S&S
• Calf pain• Muscle tenderness• Enlarged calf• h temperature
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Deep Vein Thrombi: Complications
• Pulmonary embolism– Clot that travel to the
lung – Blocks the pulmonary
artery– http://
www.youtube.com/watch?v=I0yJTkW9y9s
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Deep Vein Thrombi: Dx
• Doppler ultrasonography– Used to visualize the
vein and blood flow
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Deep Vein Thrombi: Rx
• NSAID’s• Anticoagulants• Fibroinolytic drugs
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Deep Vein Thrombi: Rx
• NSAID’s– i inflammation
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Deep Vein Thrombi: Rx
• Anticoagulants– Prevent blood clotting– E.G.
• Heparin• Warfarin (Coumadin)• Aspirin
– S/E• Bleeding
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Heparin
• Action–Prevent clot
formation–Prevent clot
growth
• Nrs implications– IV or Sub-cutaneous– Double P dose w/ nrs– Rotate sites – Don’t massage– Report S&S of
bleeding
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Heparin
• Antidote–Protamine Sulfate
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Coumadin
• AKA:–Warfarin
• Action–Prevent clot
formation–*Requires 3-5 days
to reach effective levels
• Nrs implications– Same time each
day– S&S of bleeding
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Coumadin
• Antidote–Vitamin K
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Dx Tests
• PT / PTT– Prothrombin time– Partial prothrombin time• Increased =
– Risk for blood clotting• Decreased
– bleeding
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Deep Vein Thrombi: Rx
• Fibrinolytic drugs– Breaks up clots– S/E
• Bleeding / hemorrhage
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Pulmonary EmboliPrevention of DVT• Leg exercises• X sitting cross legged • Drink fluids• Anticoagulant therapy
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Preventing Complications of Anticoagulant therapy
Hemorrhaging• Wear shoes• Use an electric razor• Use a soft toothbrush• ID band
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S&S of anti-coagulant toxicity
• Blood in urine• Blood in sputum– black stools
• Changes in menstrual flow• Easy bruising• Nose bleeds• Bleeding that does not stop
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Anticoagulant warning (teaching)• Avoid aspirin• Lab work on time• If lactating an infant – P baby for S&S too
• Elderly – Close monitoring