the energy system creating life and cancer from inanimate … · 2009. 6. 26. · 144 the energy...

AbstractAll living systems (which include can-

cer) are defined by three characteristics:irreversibility; growth by exponential expan-sion; survive mortality by information trans-fer creating animate successors from lifelessprecursors.

Growth always obeys pure exponentialfunctions at its start which slow with age-ing to obey Gompertzian functions. Hencecancer colonies and multicellular life reachgrowth asymptotes. Irreversibility of thegrowth following transfer of information (asin ovum fertilization) is universally under-stood and demonstrated in pregnancy.

Anaerobic glycolysis specifically exhib-its these three functions which power thegrowth of cancer and all unicellular lifeforms. This system (titled ERex denoting anElectrically conductive Reaction ofEXponential growth) also automaticallycreates intelligence. Adult neurons neithercontain ERex (because they never becomecancerous) nor can their axons transmittheir message information electrically (be-cause they fail to respond to Faraday’s lawsof electromagnetic induction). Glial cellswhich contain ERex become cancerous,MUST be the seat of intelligence, a reactionentitled CRex or the Chemical Reaction of theEXponential growth of cystathionine.Control of anaerobic glycolysis by oxygen(Pasteur’s reaction) (a) permits individualcellular mitotic control so that stable multi-cellular life will result: (b) controls overpro-duction of cystathionine by CRex in glial cells.

A glial cell with failure of the Pasteur re-action reverts either to cancer or Alzheimer’sdisease. These conclusions are based upon ap-proximately 30,000 patients reviewed whilst

being treated with ionizing radiation therapy,approximately 9,000 patients treated withsome combination of UHF radiowaves andionizing radiation together with approxi-mately 1,400 patients treated with intravenousglycolytic blocking agents before UHF therapy.

Introduction: Biological Energy, Storageand Synthesis

Adenosine triphosphate (ATP) is anenergy rich molecule. When one phosphate(phosphorous atom) is removed from ATPan energy-neutral adenosine diphosphate(ADP) molecule remains. The energy re-leased as phosphate is the “pocket money”of the cell which it can use for any biologi-cal function.

Synthesis of ATP from ADP is the func-tion of two separate carbohydrate meta-bolic systems, one using oxygen, calledaerobic glycolysis, and the other withoutoxygen, called anaerobic glycolysis. War-burg experimentally confirmed these twodistinct categories of carbohydrate metabo-lism and gave them simple, rational titles.1

1. Warburg’s “respiration” is aerobicglycolysis. This uses oxygen with glucose toproduce energy in the form of ATP. Activelymetabolic cells as in the liver and kidneyobtain 100 times more energy from respi-ration than from fermentation.

2. Warburg’s “fermentation” is anaero-bic glycolysis and every foetal, stem, can-cer, most glial and some other specializedcells use fermentation to synthesize ATP.This is the only source of energy for celldivision or mitosis. ATP is measured quan-titatively by the formation of lactic acid inthe absence of oxygen. The fermentationsystem is present in the extranuclear cellcomponents.

This identical fermentation reactionoccurs in anaerobic conditions outside thebody when the cell involved is a yeast or-

The Energy System Creating Life andCancer from Inanimate Compounds

J.A.G. Holt, M.B., Ch.B., F.R.C.S., F.R.C.R., F.R.A.N.Z.C.R., D.R.C.O.G.1

N.B. This system was revealed during analysis of clinicalresponses to radiowave therapy in the treatment of cancerand viral diseases. No financial or other support was involved.1. Radiowave Therapy Centre, 2nd Floor, 31 Outram StreetWest Perth, Western Australia 6005

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The Energy System Creating Life and Cancer from Inanimate Compounds

ganism and produces ethyl alcohol andcarbon dioxide.

Discussion: Glycolysis AnalyzedTable 1 (below) records the energy as-

sociated with the various steps of fermen-tation.2 The engine “powering” Table 1 isan interconnected cyclical system of glu-tathione. Glutathione is an essential com-ponent of respiration as shown byHopkins.4,5 Needham and Lehmann, (us-ing egg brei) established interconnectedreactions for both forms of glutathione.6

From fertilization to emergence from theegg shell glutathione is an essential com-ponent of anaerobic glycolysis in the em-bryo. As soon as the chicken foetus can ac-cess oxygen aerobic glycolysis commenceswhile anaerobic glycolysis diminishes.

Ashford and Holmes confirmed two gly-colytic pathways, measuring the respiratoryquotients (RQ) of cancer and other cells.7 Allcancer cells have varying RQ levels less thanone; indicating a combination of both sys-tems. Geiger and Needham and Nowinskiconfirmed these findings.8,9 Ashford andHolmes, Baker and Warburg reported that theCentral Nervous System adult neurones ex-clusively metabolize by aerobic glycoly-sis.7,9,10,11 Their RQ is always one.

There are no cancers which arise fromadult neurones (RQ = 1). All brain canceris from a glial cell (RQ = 0.4 to less than1.0, variable). The conclusion must be thatall adult neurones have lost all their primi-tive anaerobic glycolytic systems. The finalmaturation from a neuroblast to an adultneurone must be the expulsion or destruc-

Phosphorous Total EnergyContent Used or Created

(ATP)

Step 1: 1 glucose ––> 2 glycerose Nil -2Step 2: 2 glycerose + 2 phosphoric acid (2HP) ––>

2 glyceraldehyde phosphate (2GP) +2P = +4Step 3: 2GP + 2HP + 2 Reduced Glutathione (2GSH)––>

2GS-diGlycerophosphoric acid (a glutathionecomplex) + 2H

Step 4: This splits into 1 oxidized glutathione (GSSG)+ pyruvic acid + 4H ions + 4P + 4P = + 8

Step 5: GSSG + pyruvic acid + 4H ions ––> 2GSH + NilLactic acid

Notea) Overall energy produced is ............................................................................................................ +6b) 1 doubling to 2 can be binary OR exponential (Naperian) mathematics,but 4 doubling to 8 can only be exponential mathematics. Since life exclusively grows by an exponentialmathematical function, Steps 2, 3 and 4 must be the creation of life.c) Because Step 1 requires energy the exponential increase of ATP from Steps 2 to 4 is “hidden” and thereforeignored by conventional biochemists.2

d) In Step 4 GSSG is a single molecule (which may split to 2GSH), therefore all other substrates must be in aminimum of 2 molecules each.e) In a closed container (brewing alcohol) then Step 5 becomes:GSSG + pyruvic acid + 4H ions ––> 2GSH + ethyl alcohol + carbon dioxide.f ) Glucose is not a substrate for fermentation, hence its conversion to glycerose and/or mannononose in Step 1.3

Table 1: Reactions Involving Glycerose or Mannononose

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tion of their anaerobic glycolytic systems.This explains the phenomenon of spon-

taneous cure of malignant neuroblastomatain children. Some sufferers, between six and10 years old are cured by necrosis, calcifi-cation or reabsorption of proven malignantcells. At one stage, addition of vitamin B12

may increase (? initiate) this process. Per-sonal experiences of clinical observation ofa self-curing childhood neuroblastomaconfirm the 1 in 100,000 rarity of suchcure.12

Table 2 sets out the mechanismwhereby glutathione, cycling between itstwo forms, generates hydrogen ions. SeeTable 2, below; Illustration 1 (p. 144)

ERex Characteristics and the Pasteur ReactionHydrogen ions are the mechanism of

transfer between generations. In cancer,lactic acid accumulates in increasing quan-tities proportional to the extent of the dis-ease. In a closed bottle the yeast cell actsas cancer and with each generation of yeastcells there is regular production of glutath-ione. The reaction will only proceed within aclosed container and not even yeast, whichis a low form of unicellular life, can have a

perfect structure without its aerobic reac-tions. Yeast cells degenerate bizarrely inbrewing and they may die for lack offermentable sugar. They are amazingly regen-erated in the presence of oxygen.1

The fermentation of a three carbonsugar to alcohol is identical to fermenta-tion in a cancer cell. The cancer and theyeast cells use fermentation energy to di-vide exponentially. Cancer kills its victimby an explosion of exponential growth ofuseless cells from a single cell with a dam-aged, inoperative, Pasteur reaction. Fer-mentation will always burst its containerif sufficient sugar is added before the bot-tle is sealed. Cells have their anaerobic gly-colytic systems controlled by aerobic gly-colytic energy and cancer is caused whenthe control is broken. Yeast cells have bothsystems and each is autonomous. Thereforeyeast cells can be “regenerated” (ie they arenot true cancer cells) by oxygen although acancer cell is irreparable its other aerobicsystems continue to function. The synchro-nous perfection between aerobic energy tointerpret the genetic information whichwould ensure perfect daughter cells is de-stroyed by mitosis at such a forced accel-

The functions of Glutathione energize the “motor” that doubles 4 ATP to 8 ATP.

Two different reactions each with 100% completion create the cycle of2 GSH ––> GSSG ––> 2 GSH. The two reactions are:

2 GSH ––> GSSG + 2 hydrogen IonsGSSG + 2 hydrogen Ions ––> 2 GSH.

This cycle creates irreversibility because cycling either “forward” or “backward” produces anidentical result.13

This system is irreversible and always 100% complete per cycle. Its product is exponentialquantities of ATP (energy) with time when combined with Table 1 reactions. Hydrogen iontransfer procreates these basic life reactions between generations.Tables 1 and 2 can be integrated into a complex power generator of ATP (Illustration 1). Thissystem demonstrates the essential place for the hydrogen ions from phosphoric acid.14

Table 2: The Glutathione Cycle.

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Illustration 1. ERex or the electrical reaction creating exponential growth.

The glutathione cycle and the phosphorous based energy creating cycle is best simplifiedas two meshing gear wheels. On the right is the power source of the electrically conductiveglutathione cycle, 2GSH ––> sulfydryl complex ––> GSSG and 2 H ions ––> 2GSH. On theleft the “gearing” which squashes glyceraldehyde phosphate with extra phosphorus. Thisproduces an intermediate sulfhydryl complex with diphosphoglyceric acid. This breaksdown into pyruvic acid, hydrogen ions and double the quantity of phosphorus previouslycontained with the glycerose molecule. The disulphide, GSSG is reduced to GSH and thecycle is continuous. Each cycle doubles the energy source, phosphorus, stored as ATP orAdenosine Tri-Phosphate. 434 MHz UHF targets 2GSH-GSSG system of the right hand gearwheel causing it to revolve faster when it resonates and/or fluoresces as the “E” part ofER

ex.15,16 The direct target of ionising (x-rays etc) radiations must be the Sulfhydryl group

because “Rex

” represents exponential growth and x-rays “kill” by a negative exponentialdestruction. Therefore x-rays “hit” only this sulfhydryl group. In cancer the pyruvic acid isconverted to lactic acid by the spare hydrogen ions whilst in fermentation these ionsconvert it to ethyl alcohol and carbon dioxide. Pasteur was correct in his firm opinion thatbrewing was a “vital Process.” Both systems only operate in total anoxia.16

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eration that “cancer” creates its own geneticchanges. Cancer diagnosis possibly may befrom abnormal genetic material but pre-vention and therapy of cancer by geneticmanipulation is impossible.17

Illustration 2 (above) represents a dif-ferent way of presenting Illustration 1 tomake it easier to understand how the Pas-teur reaction alters the biochemical proc-

esses which create life.This is the explanation of the origin of

the increasing content of pyruvic acid inthe cancer patient with disease progression.Pyruvic acid is not metabolized for energyand this unused product appears to be re-sponsible for wasting of cancer patients.Gold has shown that it is possible to useagents which convert the pyruvic acid back

Illustration 2. Pasteur’s “Vital” Start of Life.

The Electrical Reaction of Exponentiality: ERex

Which Creates Life and Cancer. A rearrange-ment of Illustration 1. Pasteur’s reaction is defined as the control of anaerobic glycolysis byaerobic glycolysis. In simple terms the energy from 3-carbon sugars either glycerose ormannononose obtained when “burnt” in an atmosphere without oxygen (before the worldhad developed oxygen in the atmosphere) is autonomous and titled ER

ex. As soon as oxygen

developed in the world’s atmosphere control of this reaction is simple. The reaction whichis denoted ER

ex creates exponential growth of energy, is irreversible and therefore is superior

to all other chemical reactions in the universe. It therefore also has the characteristics ofintelligence and would force evolution to create developmental changes to adapt to thechanging environment. Such changes were impossible until oxygen appeared in the world’satmosphere and ER

ex could be controlled into inactivity yet relaxed when needed. The

atmosphere changes on earth would indicate the geological era of the first multicellular life.

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into a usable form. The wasting of the pa-tient can thus be partly or completely con-trolled. Clinically, it appears that this sys-tem controls the cancer. Such is not thecase because the cancer continues toprogress exponentially, although for a timethe wasting disappears.18

Illustration 3 (above) when viewed inconjunction with Illustration 2 shows ex-actly how the aerobic or oxygen-burningglucose-consuming cycles act to controlERex. ERex units must be in very close asso-ciation with specific mitochondria in theextra nuclear cell constituents of the cyto-

plasm because when the system is in per-fect order the pyruvic acid is irreversiblyabsorbed into that mitochondria and usedas a nutrient for the aerobic citric acid cy-cle followed by the aerobic phospho-gluconate cycles.2

The cause of cancer must lie in themitochondria which subserve aerobic glyco-lysis.1 To preserve ERex during active mito-sis when the nuclear membrane is brokenthe surrounding extra nuclear cytoplasm ismade temporarily anoxic, as shown byStern.19 The nucleus is always anoxic.20

The proof of the location of ERex in

Illustration 3. To show how the autonomous circular progression of ERex

is stopped whenoxidised glutathione (GSSG) can no longer be reduced to glutathione (2GSH). The dam-age creating cancer must occur within the starkly outlined representation of the mito-chondrion attached to an ERex unit. When ERex is active to power cell mitosis the wholecell becomes anoxic. Between mitoses only the nucleus is anoxic as demonstrated byStern.19,20 This system is resistant to x-rays and normal cell repair is grossly enhancedbecause repair is stimulated. This reaction controls repair until perfection is restoredwhen it ceases: overall control of perfection to original genetic “blue-print” is the re-sponsibility of the phosphogluconate and citric acid aerobic glycolytic systems in therelevant mitochondria.

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Illustration 4. Deposition of electromagnetic energy in cancer cells reveals the extra-nuclear site of the electrically conductive ERex. Normal cells with inactive ERex are un-altered. Secondary cancer (breast primary) in the upper end of a femur. This lady re-quested a trial of UHF 433-434 MHz in isolation to relieve the pain in her bones. Treatedwith a moving field method exposing her skin to approximately 40 milliwatts per squarecentimetre for 90 minutes. Four days later this specimen was obtained.

NotesA - (Arrows) Normal inflammatory cells.B - (Squares) The normal, undamaged bone cellsC - The dead cancer cells have a featureless grey colour.D - (Circles) Coagulation of the rims of many cancer cells are visible but the nucleus inthe centre will not stain. This is the poached egg appearance of a patient treated withelectrical hyperthermic method in which adequate electromagnetic radiation energy isdelivered to heat the cell above 42°C. The periphery of the cell (cytoplasm) contains theelectrically conductive part of the cell (ERex) which heats selectively and coagulates. Thisperipheral coagulation prevents dyes penetrating to the nucleus. In this secondary can-cer every cancer cell was killed and is direct proof of the cancericidal effect of 434 MHzradiation. No similar proof exists for any cytotoxic chemical yet discovered!

the cytoplasm or extra nuclear cell con-stituent is shown in Illustration 4,(above with its explanatory caption).A “closed” bone (ie a secondary in rigidbone) is the only site to demonstrate thiseffect. Soft tissue cancer has such vascu-lar cooling that electrical heating cannotcoagulate it.

In an earlier publication a patientwith a fungating cancer of the left breast/

axilla was shown before and after x-raytherapy.21 Illustration 5A/5B (p.148)shows the clinical features and a biopsybefore treatment. There are 2% to 5% ofthe cancer cells in mitosis. After the biopsyin Illustration 5 this patient received 434MHz UHF as part of a course of combinedtherapy and Illustration 6A (p.149) is of asecond biopsy one hour after cessation ofthe first day’s UHF exposure. 100% of the

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Illustration 5A shows a biopsy taken from an ulcerative breast cancer as in 5B. Thereare approximately 400 billion cancer cells and only 2% to 5% of these cells are in mitosis.Treated with x-rays in air (normal body temperature) 30 doses of 200 rads per day wouldkill approximately 3.33% (only one in 30) when each cell has 10 ERex targets per cellwhich must all be killed for that cell to die. The target number per cell for any cancercan be calculated from responses to x-ray therapy with and without UHF exposure.

Illustration 5A Illustration 5B

First Biopsy:Mitotic rate - 2-5% and therefore growth obeys N

T/No =

eA/α(1-e-αΤ).X-ray response to 6,000 rads over 30 days is: N

R/No = (1- (1-e-200/165)10)30 after 30 doses of

200 rads. The sensitivity of the one or two active ERex units is 165 rads and all inactiveERex units exceeds 2,000 rads (their Do values).So NR/No = 0.9666% approximately and only 3.33% are killed.

cancer cells are in mitosis. The clinical cureseveral years later after combined therapyis demonstrated in Illustration 6B. (p.149)

ERex

: Stem Cells, Brain Cells and IntelligenceThe electrical characteristics of cancer

have been measured in humans and the re-sultant chart is presented as Appendix 1 (p.157). Table 3 (p. 150) indicates the prob-able sites of ERex in a multicellular organ-

ism based upon the electrical character-istics. The human testis has similar elec-trical characteristics to cancer. Electro-magnetic radiation (both ionizing andnon-ionizing) will cause sterility. The ovumis considerably more resistant to these typesof radiation and in place of it containing anyERex systems it contains the 2,000 or morenucleoli, none of which are transmitted viathe sperm.22-24 The foetal cells which respire

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Illustration 6A After 434 MHz radiation on the cancer of Illustration 5 the mitotic rateis 100%. All the cancer cells are dividing and the problem of radioresistance to x-raytherapy disappears. Using 434 MHz UHF before 160 rads x-rays for 23 days in air,normothermically, potentially can kill far in excess of 400,000 billion cancer cells.A gross overkill without damage to the normal is curative. The healing stimuli of UHF onthe normal Pasteur’s reactions of the local and distant stem cells heal the ulcer as inIllustration 6B (see Appendix 3 for full details).

Illustration 6A Illustration 6B

Second Biopsy:Mitotic rate is 100% and therefore growth obeys N

T/N

o = eAT.

X-ray response to 3,680 rads over 23 days is:Residual/original size or N

R/N

o =

e-160.23/100 = e-37 after 23 doses of 160 rads when UHF has

sensitised all ERex units to 100 rads or less for their Do value.

When NR/No = e-37 the theoretical cancer cell kill is 10,000 times greater than 400 billioncancer cells without injury to normal tissues. UHF also has a simultaneous healing stimu-lation - hence the near perfect result.

anoxically all contain ERex. As they ma-ture the stem cells will have controlledERex systems thereby rendering them-selves available for repair facilities. Oncea cell is fully adult, as in the specific caseof mature neurons, the ERex systems dis-appear and cancer cannot arise fromthem. Active stem cells must contain con-trollable ERex systems. Adult cancer onlyarises from stem cells whose ERex is dam-

aged. Because the respiratory quotient ofbrain glial cells can be below one andmany must be continuously active theyare the cells most likely to be affected(? damaged) by stray electromagnetic ra-diation. Table 3 (p.150)

IntelligenceThe ability to adapt to environment is

the definition of intelligence. Adaptation

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is associated with survival. Compared withevery chemical reaction in the universe onlylife is able to adapt by evolution. The physi-cal characteristics of an exponential, irre-versible reaction create intelligence. Evolu-tion is “pushed” by ERex and must be ofLamarckian form, rather than according toDarwin’s chance theory. Evolution is an au-tomatic method for the selection of condi-tions which will allow the exponentiality toexert maximum adaptation to environment.

Any system which creates productsin rapidly increasing quantities is supe-rior to simple chemical reactions suchas sodium plus chlorine forming com-mon salt. All such simple chemical re-actions are reversible. For example in-vest money in a very generous bank pro-viding 100% compounded interest. Themost intelligent choice is to have the

interest paid exponentially so that inone year $100 becomes $271.83. Theleast intelligent choice is to opt for sim-plicity of annual interest so that in oneyear $100 becomes $200.

The Site of Intelligence in the BrainAdult neurons cannot contain ERex be-

cause they only respire using aerobic glyco-lysis. Glial cells vary in their respiratory quo-tient from 1.0 to lower values, eg 0.4, whichindicates that they are using ERex for fermen-tation as well as respiring with aerobic gly-colysis.7,25,26

Glial cell ERex alone creates the brain’sintelligence functions, but glial cell mitoseswould require an expanding skull vault. Bio-chemical evidence from invertebrates to thehighest sophistication of mammals shows acorresponding increase in the proportion of

Table 3. Possible cellular sites of autonomous glycolysis (cancer).

Site ERex Genes Nucleoli Mitochondrial PossibleActivity Cancer

Precursors

Testis, Sperm Precursors Yes 1/2 set none Intermittent Yes & spermcount effects

Sperm Yes 1/2 set none Intermittent PossibleOvum No 1/2 set 2,000 + Intermittent NoFoetal Cells (Anoxic) Yes Full Yes Continuous YesFoetal Cells (Stem) Yes Full Yes Continuous YesFoetal Cells (adult type) No Full Variable numbers None NoAdult (Stem) Yes Full Yes Intermittent YesAdult (Non Stem- No Full Variable numbers None Noeg neurons)Any age, Stem and Non- Yes Full Yes Continuous Yes & intelligenceStem, brain glial cells defects and

Alzheimer’s “cancer”

All mitochondrial bodies use electron transport systems and are therefore specific recipients ofEMF energy. The only vital system which controls cancer is the aerobic conversion of reduced tooxidised glutathione and the destruction of pyruvic acid in the citric acid and phosphogluconatecycles.

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Table 4. The sensitivity of ERex to radiation under various clinical applications

X-ray sensitization of ERex units occurs at 434 MHz because they fluoresce and resonate at thisfrequency. Each cancer cell always has one or two active and up to 30 or more inactive in itsextra-nuclear component.

The Do (Dzero) value is the cancer’s sensitivity to x-rays. It is the dose to kill 1/e of a colony (35%).

Low dose = very sensitive.High dose = very resistant.

1. Do values: (approximate for each active ERex unit) 160 rads - normothermic.

2. 140 to 155 rads - Whole body wax bath heating to 41.8°C.3. 90-100 rads minimum after 434 MHz UHF stimulation. Effect lasts 20/30 minutes.4. UHF increases x-ray response from NR/No = (1-(1-e-D/Do)x)y toNR/No = e-D.y/Do. ie 1,000 to 10,000 or more cell kill.5. Tourniquet anoxia for limb cancers. Inactive D

o value = 10,000 rads total or 6 x 1650 or 3 x

2500 rads daily in vivo are tolerated when anoxic.

All ERex are active: Since the Pasteur reaction is rendered anoxic it is inactive and NOT damagedby these doses. Any frequency will maintain ERex activity and with these doses increase in x-raysensitivity is unnecessary.

cystathionine in the brain. Cystathionine istherefore expressed in the brains of animal lifein exponential proportion to its evolutionarystatus on the tree of life.27

The probability is that ERex uses expo-nential energy production to convert me-thionine into cystathionine, which is itsproven route of synthesis.28

Out of Body ExperiencesAccidental poisoning of humans with

DL-methionine DL-sulfoximine producesout of body experiences and, in animals,feral inane behaviour. The sufferers haveno recollection of real events but all willrecall having died, gone to heaven andreturned to earth. During the attack alltheir senses remain normal and test nor-mal physiologically but the intellectualmind is completely shut off in its fantasyworld. In animals the symptoms are simi-lar; the animal can run, walk and bark but

appears completely insane.29

This syndrome occurs in humans.Women are susceptible to experience “outof life experiences” with doses of methio-nine sulfoximine in excess of 0.25 mg/kgday and men in excess of 0.6 mg/kg day.All symptoms of higher intellectual dis-ruption can be relatively quickly correctedby intravenous injections of methionine.The original report was occasioned by theaccidental poisoning of Indians with a giftof spoiled grain donated by the British gov-ernment to them to ease the starvationafter World War II. The grain was spoilt bynitrogen trichloride as a fumicide. The ac-tive ingredient causing the symptoms wasmethionine sulfoximine.30

The complete lack of any physical abnor-mality as a result of methionine sulfoximineingestion indicates that the neurones areunaffected. The glial cells which become can-cerous must be the site of the misfunction

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and of higher intellectual functions.Glial cells become cancerous. They

therefore contain the intelligent ERex

which is normally controlled by the Pas-teur reaction. When this link is brokensimple glial cancer arises. The intelligencemust also be mediated by ERex but it doesnot cause cancer. We therefore do notknow the controlling factors which pre-vent the development of intelligence andhave to assume that one’s intelligencecould increase exponentially with time forall our life.

Thoughts from our sensory systems arepresented to all areas of our brains. The in-formation processed exponentially by theappropriate sensory areas could increase theircystathionine content. The intelligence inte-grated from this conversion creates “knowl-edge” from mere “information.” Without aninput circuit which processes informationexponentially computer intelligence is impos-sible using binary calculations.

Alzheimer’s disease then assumes thesame basis as cancer. It is an “exponen-tial” creation arising from the glial cells.It is the faulty overproduction of amyloidmaterial in place of the biochemical pre-cursors of cystathionine. Amyloid dis-ease grows exponentially and causesdeath in an identical manner to Alzhe-imer’s disease. The label of CRex stand-ing for the chemical reaction ofexponentiality has been applied to thismechanism which creates intelligence.Uncontrolled activity of ERex causes can-cer. Uncontrolled activity of CRex overproduces abnormal amino acid and pro-tein complexes and thus destroys normalintelligence functions of the central nerv-ous system. Such irregular abnormal syn-theses create tangles of proteins andamino acids which are called amyloidbeta fragments. It is a chemically created“cancer” of the brain.31

Amyloid tangles can occur in otherparts of the body, particularly the liver,the kidneys as a result of chronic sepsis

as was common in the days of tuber-culotic abscesses. This extra-central nervoussystem origin also progressed exponentiallyto death if untreated surgically.

The treatment of the disease usingidentical methods (but including methio-nine sulfoximine) to that applied for can-cer coupled with 434 MHz UHF has beensuccessful in improving a few patientswith Alzheimer’s disease and the methoddeserves great effort to enlarge and im-prove it.31

The Electrical Characteristics of the Brainand Nerves

If you are blindfolded and someonewaves a strong magnet around your head youcannot feel or notice any sensation. This issufficient to prove that the neurones and theiraxons do not conduct information by elec-tric currents. Sensations cannot be inducedwhich prove Faraday’s laws of electromag-netic induction apply to nerve axons.32 Theelectric currents that can be measured run-ning along the neuronal axons are far tooslow to permit our precise rapid coordinatedmuscular movements. If Galvani’s currentstransmitted information along nerve axonsthen hand/foot coordination would differ by1/30th to 1/10th of a second34

If the brain and its peripheral nerves areas electrically conductive as a lightning con-ductor then a lightning strike would auto-matically burn out the entire brain and nerv-ous network. Localized paralysis rarely oc-curs.33 Temporary paralysis in lightning strikeinjures only the glial cells and if they fail torecover then death ensues. In cases of deathby electrocution the electrical activity of thepacemaker and bundle of His in the heartwhich utilized true electrical conduction isdamaged (burnt out) and destroyed anddeath occurs from heart failure. Hence deathafter judicial electrocution is only declaredwhen the heartbeat has ceased. The electro-cardiogram during life displays the electri-cal impulses from the pacemaker along thebundles of His to the heart muscle.

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The electroencephalogram and otherelectrical measurements of brain activity areonly recording our glial cells activity, plus thecurrents of Galvani from the very slow so-called “nerve” currents.34 The first stages ofconduction in the optic nerve (a retinalimage transformed into a signal) occurs infemtoseconds and has been measured us-ing ultra fast lasers.35 Galvani’s currents mustbe due to recovery associated with activityand unrelated to true nerve function trans-mission which is an entirely “chemical” non-ionized system of information transfer. Afemtosecond is 10-15 seconds, one millionbillionths of a second.

Life in an Extremely Radioactive WorldERex creates life: it must tolerate large

ionizing radiation dosages in the anoxic in-tensively radioactive environment of primi-tive earth. The tolerance of glutathione wasdemonstrated by Woodward.36 Proof of suchtolerance in humans is well documented.The tourniquet method of treating limbcancers developed by van den Brenk andpractised by the author will prevent approxi-mately 95% of amputations in young pa-tients because it cures their limb cancers.The ingenuity of the system is that providedthere has been no spread above the tourni-quet all the cancer can be isolated and ren-dered anoxic. The pressures required are ap-proximately 450 mm Hg for a leg tumourand 350 mm Hg for an arm tumour.37

Having placed the tourniquet in positionand inflated it the arterial blood is preventedfrom entering the limb. The limb must thenbe kept warm because it will cool rapidly andwithout continuous warmth the oxygen willnot be burnt in the limb. Some form of elec-trical heating must be used but any frequencywill suffice and a 50 or 60 hertz electric blan-ket is adequate. Approximately 30 to 40 min-utes later the blanket is removed and the limbis viewed with a telethermographic device tosee if any hot spots in the course of the majorarteries can be seen. If not then the patientis treated as soon as practicable. Any slight

leak from under the tourniquet can be disas-trous and require amputation in months oryears to come.

Providing no oxygen is present in thelimbs they will tolerate 10,000 rads in fourdaily treatments of 2,500 rads each or sixdaily treatments of 1,700 rads. The maxi-mum single dose given and tolerated by alower limb of a patient of van den Brenk was6,000 rads in one application. Very few signsof these dosages are evident when the can-cer has been cleared five, 10 or 20 years later.Some skin fibrosis and loss of hair is usuallythe only sign of treatment.37,38,39 The Pasteurreaction is the sensitive part because it maybe damaged in its mitochondria. Howeverif it is completely inactivated by the anoxiacreated from the tourniquet then it is com-pletely protected.

The autonomous mitosis of ERex is al-ready completely anaerobic and does notrequire oxygen. In these limb cancers mostare sarcomata, or cancers of soft tissues, andit can be calculated that the x number (thenumber of ERex units per cell) may be as highas 30 or more in each cell. Only one or twoof these will be active at any one time buteven with the effectiveness of treatmentusing these extremely large doses all shouldbe simultaneously active.

Anoxic Therapy versus UHF TherapyWithout the EMR of any frequency

(electric blanket) from 50 Hz to at least 915MHz the massive doses of x-rays given willbe no more effective than without the tour-niquet. Using the mathematical equationsin Appendix 3, (p.158) three single doses of2,500 rads applied to a 15 cm diameter can-cer has a theoretical cell kill many billiontimes greater than without the electric blan-ket.37,40 This cell kill could be achieved usingUHF before x-ray therapy but the tourniquetpreserves the limb normality via the Pasteurreaction’s repair function.

After setting up the patient for radio-therapy irradiation the blanket should be re-placed between the x-ray generator and the

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cancer. The necessity of an “electric” blanketstimulus from any of these frequencies indi-cates the danger of cancer activation in everypatient exposed to any frequency stray elec-tromagnetic radiation.

Pasteur’s Reaction - The Sites whereRadiation may be Damaging

By contrast 434 MHz UHF before con-ventional x-ray therapy has no effect upon thePasteur reaction in the normal cells and pro-vided the x-ray dose does not exceed 200 radson each day of application the Pasteur’s reac-tion will only be damaged in a very small per-centage of cells. This damage is directly re-sponsible for the long term sequelae of x-raytherapy - low grade fibrosis, skin changes incolour, hairlessness and defective sebum se-cretion, muscular fibrosis, long term failureof some part of the kidney, liver and brain.These sequelae can only arise from damageto the citric and/or phosphogluconate aero-bic glycolysis.

The expressed paranoia of the dangersof ionizing radiation mostly occurs fromdamage directly to Pasteur’s reaction control-ling ERex. High doses (full courses of radio-therapy for cancer) very, very rarely cause can-cer. In the radiotherapy records between 1935to 1985 two patients (from in excess of 80,000records scrutinized) probably suffered fromx-ray induced cancers in the previouslytreated areas.41 Single doses less than 600 rads(eg 150 to 200 rads for keloids and other non-malignant diseases, such as ankylosingspondylitis) very rarely create cancer andtherefore only occasionally destroy the Pas-teur system.

From 1,470 patients with non-malig-nant diseases, 249 patients were irradiatedfor ankylosing spondylitis, only one devel-oped a proven lymphatic leukaemia andthree suffered chronic hematological non-malignant changes.41 This often curativetreatment for such spondylitis ceased in theearly 1980s as a result of ill advised activi-ties of the “Green” movement.

The Pasteur system is radio-resistant

compared with active ERex systems which

have a Do value (sensitivity value in radsto reduce ERex

numbers to 37% of the preirradiated value) of a minimum of 95 + 5rads when under the influence of 434MHz; 145-155 rads when non electricallyheated to 41.8°C; 160 + 5 rads at 37°C (un-changed by any hypo or hyperbaric oxy-gen pressure up to four atmospheres ab-solute).40 (Table 4, p.152) Any mitochon-drial activity involving ion transfer pathways(eg other mitochondria) may be of similarradiosensitivity. Suitable systems such asvan den Brenk’s need to be identified toprove this statement.

434 + 2 MHz specifically causes cancerto fluoresce and resonate. It is the ERex unitswhich exhibit this interaction and are spe-cifically stimulated for approximately 30 to60 minutes after exposure to UHF. Havingstimulated the ERex units to activity there isa further peak between 22 and 26 hours latersuch that 24 hours after exposure to UHFan almost identical increase in radiosensi-tivity occurs. In the United States where 434MHz is illegal under the FDA rules (915 MHzis their legal frequency which has no sucheffects) the 24 hour effect allows legal expo-sure from amateur 434 MHz generatorswhich are not under a doctor’s control. 24hours later the radiotherapist can treat thepatient and obtain the results desired.42,43,44

This provides proof of the non-hermal ef-fect of 434 + 2 MHz at 37°C.

Appendix 2 (p.158) details the resultsof the author’s trial treating malignantnon Hodgkin’s lymphomata solely withUHF and x-ray therapy compared with atrial demanded by the oncologists of UHFplus or minus x-ray therapy when the pa-tients were synchronously given cytotoxicchemotherapy during the UHF exposure.The results speak for themselves. A con-trolled double blind trial was consideredunethical by the Cancer Council of West-ern Australia on 29 August 1974 yet re-fused such a trial comparing UHF beforex-rays and x-rays in isolation.

155


Illustration 7. The spectrum reflected from a large mass of secondary colon cancer (x-ray A) is typical of all large deposits of living human cancer. This appearance disappearswithin minutes if the patient dies. X-ray B shows reduction of the cancer load after anintravenous injection of L-Glyceraldehyde and UHF applied immediately for 30 minutesto the trunk. Almost immediately after the injection the spectrum changes to a “nor-mal” pattern. X-ray C, three weeks later the cancer has rapidly regrown, the fluid in thechest has increased and death occurred four days later. The spectrum at the time of thex-ray was very similar to the upper tracings.

Reflected Spectrum Analysis Lung secondary from Bowel Cancer

A. X-ray of large secondary bowel cancer in lung

B. X-ray one week after L-Glyceraldehyde injection

C. Rapid expansion of cancer three weeks after the temporary improvement

Before Treatment: A Typical pattern

1 Minute after IV Injection of10 GM L-Glyceraldehyde

X-ray Three weeks later: sudden rapiddeterioration: original spectrum again

Typical reflected cancer spectrum

Arrow 1 = ? Fluorescence 434.6/435.2Arrow 2 = ? Fluorescence 436.4/436.6Arrow 3-4 = ? Resonance

Receiving Antenna

Spectrum analyzer

434.584 MHz Output

Meter

Antenna 1

Moving Couch

Patient

Meter

Antenna 2

433.312 MHz Output

Spectrum analyzer- Type 545/IL20 Tektronix.1 MHz/cm 50 traces/sec, 1/8 sec exposure.

Immediate change spectrum afterthe injection

X-Ray one week later

156


The patients treated by the combinedregime of UHF plus low dose x-ray therapyrarely have either immediate reactions orcomplications of treatment, no sequelaeor long term changes in the normal tis-sues due to radiation despite a dramati-cally improved rate of recovery and repairof such complications as metastatic can-cerous fractures.45-50

Inhibition of ERex with L-Glyceralde-hyde

Several patients with widespread can-cer were observed during UHF therapywhen having combined treatment. Spec-trum analysis confirmed the presence ofresonance and fluorescence. Illustration7 (p.155) shows how an intravenous in-jection of L-Glyceraldehyde will almostinstantaneously change the ref lectedspectrum back to normal. Within a shorttime it slowly reverts back to the abnor-mal spectrum. If an adequate dose of L-Glyceraldehyde is given intravenouslythen a very temporary improvement inthe size and symptomatology of the can-cer occurs. This improvement lasts usu-ally two to three days. Since it has beenestablished that L-Glyceraldehyde willspecifically inhibit anaerobic glycolysisthen these findings strongly indicate thatL-Glyceraldehyde completely inhibitsanaerobic glycolysis but does not kill thesystem.51,52 Cancer can only replicate us-ing D-Glyceraldehyde.

The one disease which can be guar-anteed not to respond to such technol-ogy is Legionnaire’s disease. This does notspread exponentially, one patient cannotinfect another and either death or recov-ery ensues. The epidemiology of Legion-naire’s disease is that a group of peopleappear to have been poisoned similarlyand simultaneously in association withwater cooled refrigeration devices. Thesigns and symptoms of Legionnaire’s dis-ease are identical to those of the soldiersthat suffered from phosgene poisoning.

Phosgene is readily produced when elec-trical sparking (eg from a refrigerator mo-tor) occurs in the presence of refrigerantgases such as di-chloro-di-fluoro-meth-ane. A slight leak from the refrigerant cir-cuit into the water bath is the most prob-able cause of Legionnaire’s disease. Wa-ter cannot dissolve phosgene but localizesit around the water bath. This explainswhy Legionnaire’s sufferers never have airpassage symptoms, only sudden edema ofthe alveoli when the gas decays into hy-drochloric acid within them.53,54

The Future for UHF TherapyA non-toxic non-ionizing radiation

method for the control of cancer appearswithin sight. Such methods would de-stroy the electrical reaction of expo-nentiality, ERex.

Every human disease whichprogresses exponentially to death is prob-ably suitable for this method, when fullydeveloped for all such diseases. Cancerand viral infections are obvious. The socalled auto immune diseases appear toprogress exponentially. If it can be shown,as in HIV, that the body cannot mount atrue immune reaction to such diseases asankylosing spondylitis, disseminated scle-rosis, scleroderma and lupus, then themethod might well succeed with them. Apatient who remains free of HIV in 200310 years after treatment was reported afew years ago.21

ConclusionIn summary the complete failure of

radiotherapy to develop from simple uni-form day to day irradiation into a specifictool that will only effect the cancer cell isentirely due to the insistence of physicists,radiotherapists and radiobiologists thatthe interaction of x-rays is a non specificprocess causing uniform ionizationthroughout the area irradiated.55

Ionizing radiation has a specific tar-get which is “half ” of the ER

ex reaction.

157


Appendix 1Chart of the absolute fractional power absorption of human breast cancer compared tothe absorption in the subjects’ opposite normal breast measured at the mirror imagesite. The ratio peaks at 180 MHz and is 5.16. Resonance occurs at 434 + approximately10 to 15 MHz. 27 MHz is UK’s standard medical usage. 915 MHz is used in USA. Theratio of 1.22 is lowest at 1500 MHz + approximately 50 MHz. The error in each curve isthe same at all frequencies at + 0.1 of the ratio figure.This is the average result from five patients who had unilateral breast cancer and whoagreed to this study immediately preceding definitive treatment using radiowaves and x-ray therapy.

0 400 800 1200 1600 2000

Ratio

MegaHz

6/1

5/1

4/1

3/1

2/1

1/1

180

915

1500

43427

UHF also has a specific target which isthe other “half ” of ERex. When UHF isgiven to activate the glutathione cycle andirradiation is given afterwards to kill theactivated sulfydryl complex reaction thetwo specific damaging agents of a cancercell have become identified. The dosagesrequired to eliminate most cancers arewithout morbidity, immediate side effectsor long term sequelae.45-50 Permanent de-

struction of ERex is easy with x-ray therapy

once it is sensitized by UHF. The alter-native to the x-ray is to use intravenousinjections of oxidized glutathione andother similar compounds followed byUHF therapy. Results of such methodshave appeared in other publications.21,56

Experimental confirmation that car-cinogenesis primarily involves glutath-ione is given by Fiala et al.57

158


Appendix 3Growth and X-ray Response Of HumanCancers1. The growth of human malignant cancersis described by Laird as a Gompertz equa-tion of the form.58

NT = NoeA/α (1-e−αT ) Equation E1 orNT/No = eA/α (1-e−αT)

Where No = number of cells at time oNT = number of cells as time T. A and α areconstants for the specific cancer.

Expressing e- aT as a power series it can beseen that if T is small then

NT = NoeAT.....Equation E2 or NT/No = eAT

As Illustration 5 is converted by UHFto Illustration 6 (less than 5% mitoses to100% mitoses) then 434 MHz UHF changesequation 1 to equation 2 :-

NT/No = eA/α (1-e−αΤ) ––> NT/No = eAT

Appendix 2The Phase One Clinical Trial was con-

ducted at 13 & 21 McCourt Street, WestLeederville, Western Australia and receivedits first patient in February 1974. The pa-tients were divided into three groups:

Group 1 - 18 patients with generalisednon Hodgkin’s lymphoma (some previouslytreated with cytotoxic chemotherapy) andretreated similarly adding UHF therapy.Average age - 54 years. All 18 patients weredead by June 1988 from non Hodgkin’s lym-phoma. Average survival 2.8 years after UHF.12 patients needed one or more transfusions.Sequelae of marrow depression in most. Fivepatients developed central nervous systemdeposits of NHL and required CNS radiation.This was applied prophylactically in a fur-ther three patients. Abandoned after 1984because of the 100% death rate.

Group 2 -12 patients with generalisednon Hodgkin’s lymphoma (some previouslytreated with x–ray therapy). Average age - 49years. Treatment of 434 MHz UHF followedby low dose x-ray therapy. In December 1987six years after patient number 12 was treated- eight were alive, four at 13, 1 at 12, 2 at 11and 1 at 6 years post treatment. One patientlost sight of at 10 years, one dead at 13 yearsin a tractor accident on his farm. Only twopatients died from non Hodgkin’s lymphomaat 11 and 12 years and both declinedretreatment. Haematology of all survivorsnormal. Average total dosages of x-ray therapywas 2750 rads to each area treated. One pa-tient required transfusion, no sequelae wereseen and no patient developed central nerv-ous system deposits.

Group 3-7 patients with retroperito-neal non Hodgkin’s lymphoma. Average age- 47 years. All treated with UHF before x–ray therapy. In December 1987 all 7 pa-tients survived - 3 at 13, 2 at 12, 1 at 11 and1 at 8 years post treatment with normalhaematology in all survivors. Two patientsdeveloped renal impairment, one developeda malignant melanoma of the leg which wassimilarly treated in 1983, one developed

squamous skin cancer and ulcerative coli-tis. Two patients developed secondaries inthe neck and were treated as the abdomenwas at the appropriate times. Five of thesepatients remain alive and well when con-tacted in January 2002, having survived nonHodgkin’s lymphoma free, 1 at 27, 3 at 25and 1 at 22 years post treatment.

Final Conclusions from this Study1. UHF and XRT is a potentially curativemethod (Groups 2 and 3).2. Cytotoxics and UHF are only palliative(Group 1).3. UHF potentiates cytotoxicity (Group 1).4. Non Hodgkin’s lymphoma in CNS is aniatrogenic effect of cytotoxics (Group 1).5. UHF and XRT is the treatment of choicefor all stages, sites and types of non Hodg-kin’s lymphoma. No sequelae occurred inany patient so treated. Most can be un-equivocally cured.45,48,49

159


2. The response of human malignantcancers to ionising (x-rays etc) radiation isdescribed by Johns as similar, diametricallyopposed equations. They have negativeexponential functions.59 Assuming that ina biological system containing N entitiesand a dose dD destroys dN entities then

dN = 1/Do NdD where Do is a constant

Integrating, thus equation becomesNT = Noe-D/Do

Putting D = Do we have N = Noe-1

and thus Do is the dose required toreduce the population to 1/e (or 37%) ofthe original value. This dose Do is the ra-diosensitivity value of the cancer. If it is as-sumed that a multiple hit is required, that isx targets must be hit to destroy a cancer cell,then the survival curve takes the form, NR

being the residual after some No are killedNR/No = (1-(1-e-D/Do)x)y........Equation E3

Where NR is the residual from No after

receiving y doses of radiation of D rads x-rays per day and x is the number of targetsin each cell which must be killed for thecell death. No for in air normothermic x-ray therapy is approximately 160 rads.Simple heat to 41.8°C will reduce No slightlyto 145/150 rads but x is unaltered. (x isthe ERex system, varies from 3 to at least 40or more per cell. All must be killed to killthe cell, only 1 or 2 are active at any onetime in spontaneous cancers).

When inactive ERex has a Do value of at

least 10,000 rads (totally resistant) whenactive the Do value of ERex (x units) becomesa minimum of 95 rads.31 Some or all can betemporarily activated by 434 MHz UHF.Since all are active after adequate 434 MHzUHF, x numbers of 3 to 40 or more becomeunity because x=1. X-ray therapy MUST BEGIVEN either 20 to 30 minutes or 24 hourslater for the optimum maximum response.

The response after UHF changes Equa-tion 3 from

References1. Warburg O et al: On the origin of cancer cells.

Science 123, 1956; 3191: 309-314.2. White A, Handler P, Smith EL: Principles of Bio-

chemistry. 3rd Edition. New York. McGrawHill. 1964; 367-392.

3. Cohen JB: Organic Chemistry for Advanced Stu-dents. London. Edward Arnold. 1907; 327.

4. Hopkins FG: On the auto oxidisable constitu-ent of the cell. Biochem J, 1921; 15: 286-305.

5. Hopkins FG, Elliott KAC: Relation of glutath-ione to cell respiration with special referenceto hepatic tissue. Proc R Soc London, 1931; 109:58-88.

6. Needham J, Lehmann H: Intermediary carbo-hydrate metabolism in embryonic life. v. thephosphorylating cycles. vi. glycolysis withoutphosphorylation. vii experiments on the na-ture of non-phosphorylating glycolysis.Biochem J, 1937; 31: 1210-1254.

7. Ashford CA, Holmes EG: Contributions to thestudy of brain metabolism v role of phosphatesin lactic acid production. Biochem J , 1929; 23:

NR/No = (1-(1-e-D/Do)x)y

to NR/No = e-D.y/Do.................Equation 4

Therefore UHF changes Equation 1 to 2;

NT/No =

eA/α(1-e−αΤ) –––> NT/No

= eAT and

and 3 to 4:

NR/No = (1-(1-e-D/Do)x)y –––> NR/No= e-D.y/Do

And x must represent the ERex systemsin each cancer cell: ERex is the UHF plusthe x-ray target because the growth of thesulfhydryl complex of Illustration 1 growsexponentially and is killed exponentially byUHF and x-rays. As myxomatosis in rab-bits is an exponentially growing killer onan exponentially growing population - soionising radiation is the only biologicalkiller of cancer in the world. UHF merelyensures all the targets can be simultane-ously activated and therefore killed whencorrect temporal sequence related to x-raysis obeyed.

160


748-759.8. Geiger A: Role of glutathione in anaerobic tis-

sue glycolysis. Biochem J, 1935; 29: 811-823.9. Needham J, Nowinski WW: Intermediary car-

bohydrate metabolism in embryonic life.Biochem J, 1937; 31: 1165-1184.

10. Baker Z: Glutathione and the Pasteur reaction.Biochem J, 1937; 31: 980-986, and 1938; 32: 332-341.

11.Warburg O: Is aerobic glycolysis specific for tu-mours? Biochem Zeitschr, 1929; 240: 402: 326-327.

12. In: Clinical Oncology. Springer-Verlag - Berlin.UICC - International Union Against Cancer.1973; 55.

13. Kosower NS, Kosower EM: The glutathione-disulphide system. Free Radicals in Biology.New York. Academic Press. 1976; 2: 67-71.

14. Holum JR: Introduction to Organic and Biologi-cal Chemistry. New York. John Wiley & Sons.1969; 471-478.

15. Holt JAG: The cause of cancer: biochemical de-fects in the cancer cell demonstrated by theeffects of electromagnetic radiation, glucoseand oxygen. Med Hypoth, 1979; 1: 109-144.

16. Holt JAG: The extranuclear control of mitosisand cell function. Med Hypoth, 1980; 6: 145-192.

17. Holt JAG: The glutathione cycle is the creativereaction of life and cancer. Cancer causesoncogenes and not vice-versa. Med Hypoth,1993; 40: 262-266.

18. Gold J: Use of hydrazine sulphate in terminaland pre-terminal cancer patients; results ofinvestigational new drug (IND) study in 84evaluable patients. Oncology, 1975; 32(1): 1-10.

19. Stern H, Timonen S: The position of the cellnucleus in pathways of hydrogen transfer. J GenPhysiol, 1954; 38(1): 41-52.

20. Stern H, Kirk PL: The oxygen consumption ofthe microspores of trillium in relation to themitotic cycle. J Gen Physiol, 1948; 31: 243-248.

21. Holt JAG: Cancer therapy by immobilising mi-totic energy sources. J Orthomol Med, 1996; 11:2: 100-110.

22. Horrobin D: Nucleus or Nucleolus: which runsthe cell? New Scientist, 29 Jan 1981; 266-269.

23. MacCarty WC: The value of the macronucleus inthe cancer problem. Am J Cancer; 1936; 26: 529-535.

24. Busch H, Smetana K: The Nucleolus. New York.Academic Press. 1970; 136 and 549.

25. Needham J: The energy sources in ontogen-esis. J Exper Biol, 1933; 10: 79-87.

26. Dixon KC: The oxidative disappearance of lac-tic acid from the brain and the Pasteur reac-tion. Biochem J, 1935; 29: 499, 973-977.

27. White A, Handler P, Smith EL: Principles of Bio-chemistry. 3rd Edition. New York. McGraw Hill.

1964; 760.28. White A, Handler P, Smith EL: Principles of Bio-

chemistry. 3rd Edition. New York. McGraw Hill.1964; 499-500.

29. Mellanby E: Diet and canine hysteria. Br MedJ, 1946; 2:885-887, and 1947; 2: 288-289.

30. Bentley HR et al: Action of nitrogen trichlo-ride on certain proteins. Proc R Soc B, 1950; 137:402-417, and 1951; 138: 265-272.

31. Holt JAG: The metabolism of sulphur in rela-tion to the biochemistry of cystine and cysteine:its fundamental importance in biology. A cyclicinterchange between their mono- and di-sul-phides is the unique reaction creating life andintelligence. Med Hypoth, 2001; 56(5): 658-676.

32. Slater JC: Microwave Transmission. 1st Edi-tion. New York. McGraw Hill. 1942; 83.

33. Jellinck E: Resuscitation of lightning injuries.Klinische Wochenschrift, 1932; 45: 33-37.

34. Galvani L: Physics for the Life and Health Sciences.Massachusetts. Addison-Wesley. 1975; 318-319.

35. Schoenlein RW et al: The first step in vision:femtosecond isomerisation of rhodopsin. Sci-ence, 1991; 25: 412-415.

36. Woodard GE: The effect of ultra-violet, radiumand x-ray radiation on glutathione in pure so-lution. J Biol Chem, 1937; 31: 1411-1414.

37. van den Brenk HAS: The treatment of malig-nant disease of the extremities by megavoltageirradiation under tourniquet anoxia. J CollRadiol A’asia, 1963; 7: 142-158.

38. van den Brenk HAS: Fundamental aspects ofradiosensitivity of tumours. Proc Coll RadiolA’asia, 1959; 3:120-135, and 1962; 6:116-121.

39. Holt JAG: The principles of hyperbaric and an-oxic radiotherapy. Br J Radiol, 1975; 48: 819-826.

40. van den Brenk HAS: Radiobiology: A Coursefor Students of Radiotherapy. Melbourne. Can-cer Institute Board. 1957.

41. Nelson AJM, Holt JAG: A report to the RoyalCollege of Radiologists entitled “Radiation in-duced malignancies”. A survey from public andprivate radiation therapy records in WesternAustralia between 1935 and 1985. Unpublished.

42. Hornback NB et al: Preliminary clinical resultsof combined 433 MHz microwave therapy andradiation therapy. Cancer , 1977; 40 6: 2854-2863.

43. Hornback NB et al: Radiation and microwavetherapy in the treatment of advanced cancer.Radiology, 1979; 130: 459-464.

44. Hornback NB, Shupe RE, Marshall C, Baker R:Experience in clinical hyperthermia and radia-tion therapy in patients with advanced cancer.Progress in Radio Oncology II. New York. RavenPress. 1982; 435-442.

45. Holt JAG: Alternative therapy for recurrent

161


Hodgkin’s disease: radiotherapy combined withelectromagnetic radiation to create completeremission in 11 patients without morbidity. BrJ Radiol, 1980; 53: 1061-1067.

46. Holt JAG: The use of UHF radiowaves in can-cer therapy. A’asia Radiol, 1975; 2:223-241.

47. Holt JAG, Nelson AJM: Combined microwavetherapy. Med J Aust, 1978; 2: 88-90, and 1985;13: 707-708.

48. Holt JAG, Stanford RW: The synergism betweenhyperthermia and ionising radiation. Br JRadiol, 1986; 59: 795-796.

49. Nelson AJM, Holt JAG: Microwave adjuvant toradiotherapy and chemotherapy for advancedlymphoma. Med J Aust, 1980; 1: 561.

50. Caldwell WL. Evaluating the Perth experiencein treating with ionising radiations and UHF(434 MHz) radiations. Int J Rad, Onc, Biol, Phys,1979; 5: 1919-1921.

51. Stickland LH: The inhibition of glycolysis byglyceraldehyde. Biochem J, 1941; 35: 859-871.

52. Mendel B: Krebszelle und Glycerinaldehyd.

Klinische Wochenschrift, 1929; 8: 169-170.53. Clark AJ: Applied Pharmacology. 7th Edition.

London. J & A Churchill. 1942; 451-452.54. Memoranda on Medical Disease in Chemical

Warfare. HMSO -RAMC Training Course. Lon-don. 1946.

55. Peters LJ, Withers HR, Thames HD: Radiobio-logical bases for multiple daily fractionation.Progress in Radio Oncology II. New York. RavenPress. 1982; 317-323.

56. Holt JAG: Some characteristics of the glutath-ione cycle revealed by ionising and non ionis-ing electromagnetic radiation. Med Hypoth,1995; 45: 345-368.

57. Fiala S et al: Glutathione and gamma glutamyltranspeptidase in rat liver during chemical car-cinogenesis. J Nat Cancer Inst, 1976; 57: 591-598.

58. Laird AK: Dynamics of Tumour Growth. Br JCancer, 1964; 18: 490-502.

59. Johns HE. The Physics of Radiology. 2nd Edi-tion. Illinois. Charles C Thomas. 1964; 660-668.

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