third trimester bleeding you must fear anything that can ... campbell- third trimester...
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![Page 1: Third Trimester Bleeding You Must Fear Anything That Can ... Campbell- Third Trimester Bleeding.pdf · You Must Fear Anything That Can BleedThat Much and LIVE! Objectives •Identify](https://reader031.vdocument.in/reader031/viewer/2022041123/5d24cd2888c993cd7d8c1b4a/html5/thumbnails/1.jpg)
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Third Trimester Bleeding
Teri Campbell RNFlight NurseUniversity of Chicago Aeromedical Network
You Must Fear Anything ThatCan Bleed That Much and
LIVE!
Objectives• Identify normal physiologic hemodynamic changes in pregnancy
• Discuss the assessment and care of placenta previa
• Review the assessment and care of placenta abruptio
• Identify hemorrhage risks associated with maternal trauma
• Discuss DIC considerations for the pregnant patient
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Cardiovascular
• Blood- volume- coagulation factors- fibrinogen
• Vital Signs
• Heart Sounds
Reproductive
• Uterus: intrabdominal
• Increased vascularity
800cc !
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Third Trimester Bleeding
• Placenta Abruptio
• Placenta Previa
Third Trimester Bleeding
Placenta Previa
• Types- Complete- Partial- Low lying
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Placenta Previa
• Initiation of the bleed• Reoccurrence
Placenta Previa
• Presentation- Usually bright red blood- Usually painless
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Placenta Previa
No Vaginal Exams!
Third Trimester Bleeding
Treatment / Management
• Assess bleeding objectively
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Third Trimester Bleeding
• Monitor fetus for tachycardia
• Assess for signs of shock
• IVF bolus’
• Assess for contractions
Chicken or the Egg?
• Bleeding causes contractions…
• Contractions cause bleeding…
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Third Trimester Bleeding
Abruptio Placenta
• Definition
• Types
• Causes
Third Trimester Bleeding
Abruptio Placenta
• Presentation- Acute pain- Tetanic contractions- Usually dark red, painful bleeding- Shock- * Board-like abdomen
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Third Trimester Bleeding
Abruptio Placenta
• Management- Check for vaginal bleeding
- Save chux pads- IVF bolus’- Left lateral tilt- Delivery
The Mama Trauma
• Blunt
• Penetrating
• Burns
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The Mama Trauma…complications
• Abruptio• liver laceration• spleen rupture• pelvic fractures • fetal skull fractures / other fetal insults
I’m all choked up…• Pulmonary emboli
• Virchow’s Triad…for a green piece of pie?
• ↑ BMI = ↑ risk of DVT
• Prolonged immobilization
• Prevention
9/26/2016Free Template from
www.brainybetty.com 18
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Trauma complications
• Uterine rupture
Post mortum assessment
Maternal / Fetal Assessment
• A,B,C’s• Positioning• R/O hypovolemia vs IVCS
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Maternal / Fetal assessment
• Palpate the uterus• Fundal height assessment
Fundal Height• Gross estimation of fetal age
• Tenderness? Firm? UC’s?
• FM?
• Mark it with a sharpie
9/26/2016Free Template from
www.brainybetty.com 22
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They need the “red stuff”
• Lots and lots of it
• PRBCs, FFP and Cryo
• ↑ risk of DIC
• Petechia (B/P cuff, periorbital)
• Oozing sites
• Red top (test)
Pregnancy Hemostasis
Hypercoaguable state
Protective mechanism developed in the placenta to protect mother from
hemorrhage after delivery
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DIC Made Easy!Long Name?...NEVER GOOD!
Disseminated:
Intravascular:
Coagulation:
“Disorganized”
Occurs within the vascular bed
Clot formation and clot lysing
Intrinsic and Extrinsic Pathways
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Keep It Simple
Intrinsic and Extrinsic Pathway
Intrinsic: Initiated by blood vessel damage
Extrinsic: Initiated by tissue damage
PTT
PT
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Clotting Pathology (A), (B), (C)
(A): Precipitating event (i.e. sepsis, abruption)
releases excessive amount of Thrombin
Initiates both the intrinsic and extrinsic pathways. ↑Thrombin→Fibrinogen→Fibrin
= ↑ CLOTS
innumerable clots clog microcirculation leading to hypoxia and tissue ischemia
Clotting Pathology (B) and (C)
(B): Injured platelets and RBC’s release
Phospholipids. Potent clotting factorsthat causes even more clots!
(C): Decreased clearing ability of the
reticuloendothelial system
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Consumptive Coagulopathy
(A): ↑ Thrombin release leading to increasedclot production. Occluding microcirculation.
(B): Injured blood cells release phospholipids,
potent clotting factors.
(C): ↓ clearing ability of the reticuloendothelialsystem.
When all the clotting factors have been consumed…
A+ B +C= CONSUMPTIVE COAGULOPATHY
DIC Pathophysiology
DIC is a positive feedback system where
CLOTTING is the problems and
HEMORRHAGEis the finding
PROBLEM: Excessive Thrombin affects both COAGULATION
as well asFIBRINOLYSIS
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Second Half of DIC…
HEMORRHAGESimultaneous initiation of the fibrinolytic system.
FIBRINOLYSIS→lyses→CLOT→resulting in→FSP’S AND FDP’S
FSP and FDP’s: Potent anticoagulants
Fibrinolysis + FSP anticoagulants + consumed clot factors
=Hemorrhage and rapid exsanguination
Mommy! Make it go AWAY
• Delivery
• Blood Productsmay run out
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Blood Products
PRBC’S: Indication: need to increase blood volume & clotting factors. Each unit increases HgB by 1g/dl & the Hct by 3%
Platelets:Indication: Need to increase platelet # or function.Usually given for platelet count of ↓ 50K if bleeding & ↓ 20K if not bleeding. Each unit raises platelets by 5,000-8,000 / unit.
Blood Products
Cryoprecipitate:Indications: Decreased fibrinogen. Each unit contains 200mg of fibrinogen.
Fresh Frozen Plasma (FFP):Indications: Need for clotting factors as well as volume expanders.
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Call the OR!
Possible hysterectomy
Pre-hospital / Inter-facility transfer
DLABOOH !
FLABOOH!
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Her Emergency not YOURS
In Conclusion….
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Wrapping It All Up
• Fix underlying problem…..?• Maternal compensatory mechanism• Objective EBL• Low threshold for blood products• Early assessment of DIC
Questions???
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Third Trimester Bleeding
Teri Campbell RNFlight NurseUniversity of Chicago Aeromedical Network
You Must Fear Anything ThatCan Bleed That Much and
LIVE!