vinit orbit
TRANSCRIPT
Presenter
Dr. Vinit Kamble
Moderator
DR. B.P.SINHA
Walls
Apex
Openings
Spaces
Relations
Blood vessels
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Dimensions- conical or quadrangular bony
pyramidal in shape which connect ACF and
maxillary sinus below
Depth- 42 mm on medial ,50 mm on lateral
side
Height- 35 mm
Intermargimal dist.-25mm on medial wall
100mm on lateral
Width- 40mm parts- Apex /notch of pyramid
Base- ante. Most quadrangular11/24/2014 3
Sketch of orbit by Dr Sanjay Shrivastava
Frontal
Ethamoid
Zygomatic
Lesser and Greater
wing of Sphenoid
Maxillary
Lacrimal
Palatine
Optic Foramen
Sup Orbital Fissure
7 BONES USED IN FORMATION OF
ORBITAL CAVITY
ROSTRUM OF SPHENOID
,PERPEND..PLATE OF ETHMOID, FRONTAL
, MAXILLA ,PALATINE, ZYGOMATIC,
LACRIMAL BONES
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Orbital index - height/width x 100
It is 83 to 89 in CAUCASIANs and
<83 in NIGROEs , >89 in orientals
Ratio of orbital vol/globe vol=4.1: 1
Vol 29 ml
Ant- quadrangular called BASE
Post -notch called APEX
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Lateral wall of each orbit lies at angle 45
degree to medial wall
Lateral wall of both orbit 90 degree to
each
other
lateral wall seprate orbit from from MCF and
muscular temporal foss ante.
WALLS – medial , lateral , floor, roof
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4 walls meet at SUP. INTERNAL, SUP.
EXTERNAL, INF. INTERNAL , INF. EXTERNAL
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Roof- is formed by the orbital plate of frontal bone and lesser wing of sphenoid
Floor- is formed by the maxillary bone- orbital plate and maxillary process of zygomatic bone and orbital process of palatine bone
Medial wall- is formed by the lacrimal and ethamoidal bone, frontal process of maxillary bone and body of sphenoid
Lateral wall- is formed by the greater wing of sphenoid and zygomatic bone
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Trangular in shape, formed by frontal bone
Anteriorly and behind by lesser wing of
sphenoid
Ante.ro lateral- fossa for lacrimal gland
Above – frontal lobe of cerebrum+ meninges
Below – LPS, SR,SO,LACRIMAL GLAND, 4th
CN , FRONTAL NERVE , PERIORBITAL
TISSUE.
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M/C - involved in blow out fracture and
invaded by tumour of maxillary antrum
Seen in X ray orbit P/A view.
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Quadrilateral
Formed by- from front to back frontal
process of maxilla , lacrimal bone, body of
sphenoid, orbital plate of ethmoid,
Lacrimal fossa anteriorly with ant. Lacrimal
crest of maxillary bone and post lacrimal
crest of lacrimal bone infe. By NASO
LACRIMAL CANAL
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Medial to lacrimal fossa lie – ant. Ethmodal
sinus in upper part , middle meatus of nose
in lower part ,
Lacrimal fossa consist – sac + fascia
Post.- to post lacrimal crest consist Horners
muscle , septum orbitale, ligament of medial
rectus
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Consist that is towards nose Ethmoid air
sinus , middle meatus of nose , sphenoid
air sinus
Orbital surface of medial wall- superior
obligue in upper part , MR- middle part
In bet. Superior obligue muscle and MR
consist ant ethmoidal nerve , post ethmoidal
nerve, infratrochler nerve
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Thinnest wall , ethmoiditis, with orbital or
preseptal cellulitis in child , eroded by cyst,
Neoplasm, fracture during orbitomy operation
or surgery, injury
Haemorrhage m/c due to injury to ethmoidal
vessels
Xray P/A view recquired for diagnosis
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Trangular , ant. relation- zygomatic bone
with groove which transmit zygomatic
nerve and vessels
Post . Relation – greater wing of sphenoid,
Origin to LR muscle , seprated from roof
by sup orbital fissure and from floor
by infe orbital fissure
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Protect post half of eyeball
Palpation of retro orbital mass is eazy
through lateral wall
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Annulus of zinn giving rise to origin to extra
ocular muscles
Optic canal- 6 to 11 mm at 4to 5 yr
Part of superior orbital fissure
Optic canal -transmit optic nerve and
ophthalmic artery , lateral wall is shortest
Gret. And lesser wing of sphenoid form sup
orbital fissure..divided in to lateral,
medial,inferior
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It is post notch of pyramid types-
1] orbital end is vertically oval
2] center- circular
3] cranial end- horizontally oval
Optic nerve glioma and meningioma show
Enlargement of optic canal seen best in x
ray
P/A VIEW
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IN ORBITAL APEX SYNDROME - IT IS
VISUAL LOSS FROM OPTIC
NEUROPATHY, OPHTAHLMOPLEGIA,
MULTIPLE CRANIAL NERVE
INVOLVEMENT consist
SOFS/Rohon Duvigneaud syndrome,
THS/unilateral, CST/bilateral diag. from
HRCT,MRI
Involve 3,4,5,6 CN with optic nerve
dysfunction
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Sketch of Apex of Orbit by Dr Sanjay Shrivastava
Sup Orbital Fissure
Annulus of Zinn
Med Rectus Muscle
Inf Rectus Muscle
Lat Rectus Mus
LPS
Sup Oblique Mus
Optic Nerve
Optic canal- optic nerve with meninges and
ophthalmic artery connect orbit to MCF
Superior orbital fissure-
Outside tendinous ring – structures passing
outside are:
Lacrimal nerve –V1
Frontal nerve -V2
Trochlear nerve
Superior and inferior veins
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Inside tendinous ring- structures passing inside
the ring are -
Oculomotor (3rd cranial nerve) upper division
Nasociliary nerve
Abducent nerve (6th cranial nerve)
Oculomotor lower division (3rd cranial nerve)
Inferior orbital fissure-inferior ophthalmic vein
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Foramen rotandum - maxillary nerve
Superior orbital notch-supraorbital nerve and
vessels
Infra orbital foramen-infraorbital nerve and
artery
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Subperiostial space
Peripheral orbital space
Central space
Tenons space
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Space bet orbital bone and periorbita
Tumour arising from bone seprate periorbita
to orbital rim
Dermoid, epidermoid cyst,mucocele,myeloma,
hematoma, fibrous dysplasia are seen in this
space
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Bounded peripherally by periorbita, 4 extra
Ocular muscle, with intermuscular septa
Tumour in this space produse proptosis
E.g malig lymphoma, capiilary haemangioma,
Pseudotumour, neoplasm of lacrimal gland
Contents- SO,IO,LPS, LACRIMAL FRONTAL,
TROCHLEAR, LACRIMAL GLAND
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RETROBULBAR SPACE/ MUSCULAR CONE
Ant- by tenons capsule or fascia bulbi
Periphery-rectus muscles and intramus. Septa
Contents- optic nerve , meninges, 3RD ,6th
,opthalmic artery, ciliary ganglion, central
Fat, sup ophtha. Vein
Cavernous haemangioma,neurofibroma,neuro-
-lemoma ,meningioma,optic nerve glioma
Treat- lateral orbitomy
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Tenons capsule, envelop globe from limbus
To optic disc , inner face lies in contact with
sclera
outer face of fascia bulbi lies in contact with
orbital fat, subconjuctival space anter.
Lower part of fascia bulbi is thick takes in
Formation of sling / hammcock on which globe
rest called as suspensory liga of
LOCKWOOD
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Frontal sinus
Sphenoidal sinus
Maxillary sinus
Ethamoidal air cells
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Eyeball, extra ocular muscles, muller muscles
Optic nerve, occulomotor,trochlear,abducent,
Trigeminal,fat,fascia, lacrimal sac gland
Ophthalmic artery, and its branches
Orbital fascia, reticular tissue
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Proptosis
Exophthalmos- endrocrinal
Enophthalmos
Pseudoproptosis-slight prominence of eyes like
myopia, paralysis of extra ocular muscles,
obese people, mullers stimulation by cocain
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Abnormal protrusion of eye ball is called
proptosis or exophthalmos.
The term exophthalmos is reserved for
prominence of the eye secondary to thyroid
disease
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Abnormal protrusion of globe
It may be Unilateral or Bilateral
Unilateral – caused by orbital cellulitis, idiopathic orbital inflammatory disease, thrombosis of orbital vein, arterio-venous aneurysms, tumors of structures of orbit , orbital haemorrahge , emphysema.
Bilateral – endocrine exophthalmos , cavernous sinus thrombosis , symmetrical orbital tumors, oxycephaly - diminished orbital volume
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Dermoid and epidermoid cyst
Capillary haemangioma
Optic nerve glioma
Rhabdomyosarcoma
Leukaemias
Metastatic neuroblastoma
Plexiform neurofibromatosis
Lymphomas
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Metastases – (of malignancy) from breast,
lung, GIT
Cavernous haemangiomas
Mucocele
Lymphoid tumors
Meningiomas
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Types of Proptosis
Axial proptosis - eye is pushed directly
forwards – lesions situated in optic nerve
and central space
Non axial- situated elsewhere in orbit
pushes eye in opposite direction
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Extra conal lesions Intra conal lesions Muscular disorders
Dermoid cyst Cavernous haemangioma Thyroid
ophthalmopathy
Rhabdomyosarcoma Optic nerve glioma Pseudo tumor
Extension of nasal
/sinus diseases
Meningioma Cysticercosis
A-V malformations Lymphoproliferative
disorder
Rhabdomyosarcoma
Static- as seen usually in congenital causes
Increasing – fast- as in cases of
Rhabdomyosarcoma, neuroblastoma,
haemopoetic
Gradual- as in cases of meningiomas
Pulsatile- as in cases of carotid cavernous
fistula
Intermittent- as in cases of orbital varicosity
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Impaired mobility
Diplopia
Papilloedema
Optic atrophy
Hertel exophthalmometry – measures more
than 18 mm
Difference in two eyes of more than 2 mm is
considered positive
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• Careful history recording
• Systemic examination
• ENT examination
• Biochemical and haematological investigations
• Imaging of bony structures- plain x ray
• Imaging of soft tissues –CT scan, MRI
• Vascular study- orbital venography, carotid
angiography, MR angiography, digital
subtraction angiography
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Definition: Purulent inflammation of the cellular
tissue of the orbit
Causes of Orbital Cellulitis:
Spread of infection from neighbouring
structures like nasal sinuses, eyelids, eyeball
(like in case of panophthalmitis) facial erysiplas
etc
Also due to deep penetrating injuries (specially
in cases of retained Foreign body) and
metastatic infection in cases of pyaemia
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Two types- pre septal cellulitis and orbital
cellulitis
Pre septal –structures anterior to orbital
septum, characterized by erythema, chemosis,
conjunctival discharge without restriction of
ocular movements and visual impairment
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Orbital – behind orbital septum,
characterized severe pain, fever,
diminution of vision (due to retrobulbar
neuritis or compression of optic nerve and
/or its blood supply), massive swelling of
lids, chemosis, proptosis, restriction of
ocular movements, diplopia, an abscess
may form pointing somewhere in the skin
of the lid near the orbital margin or fornix
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Panophthalmitis
Extension into brain through meninges , cavernous sinus thrombosis may develop
In diabetic patients fungal superinfection may develop
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Culture and sensitivity of pus, if present and of blood
Treatment –Broad spectrum Intravenous antibiotics , and anti inflammatory
If abscess has formed – Incision and Drainage under cover of antibiotics
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Due to extension of thrombosis from various feeding
vessels
Superior and inferior ophthalmic vein enter in front
Superior and inferior Petrosal sinus leave from
behind
Cavernous sinus communicates with facial veins,
lateral sinus, jugular vein, Mastoid emmisary vein-
lateral sinus- superior petrosal sinus
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Cavernous sinus on one side communicates
with other side through transverse sinus
Because of connection with mastoid through
mastoid emmisary vein, mastoid tenderness is
diagnostic feature of cavernous sinus
thrombosis
Orbital veins - as in cases of eryiepelas, septic
lesion of face, orbital cellulitis , infective
condition of face, mouth, nose, sinuses
Furuncle of upper lip – dangerous area of face
Metastatic infection or septic condition
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Patient may present with symptoms and signs
of Orbital cellulitis, there is sever supra-orbital
pain
Systemic features – headache, fever ,altered
sensorium, vomiting and cerebral symptoms
Transference of symptoms and signs to other
eye (bilateral orbital cellulitis with which it may
be confused is very rare clinical condition).
Mastoid edema and tenderness is present.
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In case of infection spreading to other eye, the
first sign is involvement of lateral rectus of
other eye
Papilloedema
Emergency
Broad spectrum Intra Venous antibiotics
Anti coagulants
Neurophysicians to be consulted
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Endocrine exophthalmos : Graves
Ophthalmopathy (dysthyroid eye disease) is
the commonest cause of uniocular or bilateral
proptosis in age groups between 25 and 50
years
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Consists of Exophthalmos, and all signs of
thyrotoxicosis (i.e. tachycardia, muscular
tremors and raised BMR)
In early stage the presentation may be
unilateral, becomes bilateral. Palpabral
aperture is wide open due to lid retraction
(Dalrymple sign). Upper lid fail to follow
downward movement of eye (von Graefe sign)
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Lid retraction
Lid lag (upper and lower
Infrequent blinking and incomplete closure of lids (Stellwag
sign)
Lid edema
Exophthalmos
Conjunctival congestion over the insertion of recti muscles
and chemosis
Convergence insufficiency (Mobius sign) and Diplopia
Raised intraocular tension may be present
Superior limbic keratopathy
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Grade 0 – No signs or symptom
Grade 1 – Only sign (lid retraction)
Grade 2 – Soft tissue involvement (Chemosis)
Grade 3 – Proptosis (which may be minimum
<23, moderate , marked >28)
Grade 4 – Extraocular muscle involvement
Grade 5 – Corneal involvement
Grade 6 – Sight loss
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Is proptosis with external ophthalmoplegia
Usually seen in middle aged people , it is of
insidious onset, typically assymetrical limiting
upward movement and abduction due to
swollen, pale edematous, infiltrated ocular
muscles . There is irreducible exophthalmos
with risk of exposure keratitis , globe
dislocation mechanical compression of optic
nerve and ophthalmic vessels
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Disease is self limiting with intermissions and
relapses, usually not affected by any treatment
. Spontaneous resolution may take place which
rarely is complete
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Short term oral steroid therapy (with dose of
40-60 mg) with radiotherapy (1000 rad ) are
effective in controlling soft tissue inflammation
Exposed cornea should be protected by doing
tarsorrhaphy in less severe cases , by orbital
decompression in more severe cases. Lateral
tarsorrhaphy may also be needed.
Residual muscle palsy is dealt with muscle
adjustment surgery.
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• Type – I : Characterized by symmetrical mild
proptosis with lid retraction usually associated
with thyrotoxicosis
• Type – II : Characterized by extreme
exophthalmos, compressive neuropathy and
extraocular muscle involvement. This form may
be associated with any state of thyroid function,
but usually with hypothyroidism, seen after
thyroidectomy.
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Due to edema, lymphocytic infiltration anf
fibrosis of orbital contents and extra-ocular
muscles
Lid retraction is due to contraction of Muller
muscle
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