2b-211 msu pres

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Page 1: 2b-211 msu pres
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5. BreastfeedingMilk is Produced by the breast alveoliColostrum contains less fats and carbohydrates than

mature milkInitial duration: 1-2 minutesPurpose: promotes bondingRegular duration: 10 minutesThe amount of breastmilk produced is directly related

to the effectiveness of SUCKINGBreast milk can remain frozen up to 6 monthsIncrease galactogoguesIncrease caloric intake by 500 calories/dayNot allowed in a client receiving COUMADIN

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• Mastitis is an infection of the parenchyma of the mammary gland

• usually occurring 2 to 3 weeks postpartum• Breast engorgement can cause low-grade

fever in the early• Risk factors: breast-feeding, recent weaning,

and fissures of the areola

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• Treatment includes continued breast-feeding or pumping from the affected side

• ice packs, and support

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Domains of Learning

• Cognitive - knowledge• Affective - behavior• Psychomotor - skills

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13. SIGNS OF PREGNANCY

Presumptive symptoms: SUBJECTIVE SYMPTOMS

Probable signs: OBJECTIVE SIGNS

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FHT

• FHT audible via doppler @ 10 – 12 weeks• FHT audible via Fetuscope @ 18 – 20 weeks • FHT audible via stethoscope or w/out

instrument – 20 weeks onwards

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PE

• Vital signs/ IO• Uterus and lochia• abdomen • perineum • Bladder function • breasts • lungs• extremities

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• 1st – onset of labor to full dilation• 2nd – complete dilatation to delivery• 3rd – delivery of baby to delivery of placenta

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First stageDilatations

Frequency Duration

Latent Phase

0-3 cm 5-10 mins 20-40 mins

Encourage walking : shortens 1st stage of laborEncourage to void q 2-3 hrs : full bladder inhibits uterine contractionbreathing (chest breathing technique)

Active 4-8 cm 3-5 mins 30-60 secs

M – edications – have meds readyA – ssessment include: v/s, cervical dilatation & effacement, fetal monitor, etcD – ry lips – oral care (ointment), dry linensBreathing – abdominal breathing

Transition 8-10 cm 2-3 mins 45-90 secs

I – inform of progress (to relieve emotional support)R – restless support her breathing techniqueE – encourage & praise

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SECOND STAGE OF LABOR

• Crowning occurs• PRIMI – transfer to DR @ 10 cm dilatation• MULTI – transfer to DR @ 7 – 8 cm dilatation• Position in lithotomy both legs at the same

time• BULGING OF PERENIUM surest sign of

delivery initiation• PANT & BLOW Breathing, fetal pushing

should be done on an open glottis

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• Respiratory alkalosis– Due to incorrect breathing– Hyperventilation– S/sx

• RR• Lightheadedness• Tingling sensation• Carpopedal spasm• Circumoral numbness

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Preterm Labor

labor after 20 weeks and before 37 weeks

Triad signsPremature contractions every 10-20 minaEffacement of 50 – 80%Dilatation of 2 – 3 cm

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–Rhythmic contractions–Lower abdominal cramping–Low back pain–Pelvic pressure–Increase in vaginal discharge–Bloody vaginal discharge (bloody

show)

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Risk factors:

– Prior preterm delivery– African American race– Poverty – Lack of prenatal care– Exertion or Stress– ↓BMI– Extremes of maternal

age (younger than 18 or older than 40)

– Tobacco use– Substance abuse

– Prior induced abortion

– Prior cervical surgery– Periodontal disease– Uterine

overdistension – Vaginal bleeding

during pregnancy– Uterine anomaly– Anemia– Reproductive tract

infections

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Home Management

– CBR– Avoid Sex– Empty bladder– Drink 3 – 4 Glasses of H2O

• Full bladder inhibit contraction

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Cleft lip/ cleft palate

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PYLORIC STENOSIS

• Thickening of the pylorus muscle

• SURGERY: Pyloromyotomy/Fredet-Ramstedt procedure

ASSESSMENT: • Olive-shape mass• Vomiting

PROJECTILEBLOOD STREAKEDWITHOUT BILE

Peristaltic wavesFailure to thrivedehydreation

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NURSING INTERVENTIONS

PREOPERATIVELY:• Administer replacement fluids and electrolytes• Prevent vomiting:NPO,high Fowlers, minimize

handling• strict I&O • daily weights• urine specific gravity

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NURSING INTERVENTIONS

POSTOPERATIVELYAdvance diet as toleratedFeeding- small, frequent feeding within 24 hoursGradual increase the amount- 48 hoursFeed slowly and burp frequentlyPlace on the right sideElevate headObserve incisionHealth teaching

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CHD- Congenital Hip Dysplasia

Asymmetrical gluteal folds

Ortolani’s Click

Limited Hip Abduction

Positive Trendelenburg Sign04/18/2023 06:06:12 AM

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• Additional skin folds with knees bent• When lying on abdomen, buttocks of affected

side will be flatter because head of femur falls toward bed from gravity

• T _ _ _ _ _ _ _ _ _ _RG test- used when child is old enough to walk– He have to stand on affected leg only– Pelvis will dip on normal side as child

attempts to stay erect

04/18/2023 06:06:12 AM

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04/18/2023 06:06:12 AM

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CLUBFOOT (TALIPES)• Abnormal rotation of the

ankle• Varus- inward rotation• Valgus- outward rotation• Calcaneous- upward rotation• Equinas- downward rotation

MEDICAL MANAGEMENT• Exercise• Casting• Denise brown shoe• Surgery and casting for

several months

ASSESSMENT:• Foot cannot be manipulated

by PROM into correct position

NURSING INTERVENTIONS:• Provide cast care • Child who is learning to walk

must be prevented from trying to stand

• Assess toes to be sure that cast is not too tight

• Need to monitor special shoes for continued fit throughout treatment

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CLUBFOOT CASTING

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I s o l a t i o n P r e c a u t i o n s

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T i e r s

S t a n d a r d F o r A L L c l i e n t s

T r a n s m i s s i o n – b a s e d F o r S O M E c l i e n t s

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T r a n s m i s s i o n – B a s e d

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A i r b o r n e

L e s s t h a n 5 m i c r o n s

M o r e t h a n 3 f e e t

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A i r b o r n e

R e q u i r e m e n t s N e g a t i v e p r e s s u r e r o o m

F r e q u e n t h a n d w a s h i n g

P a r t i c u l a t e or N – 9 5 M a s k

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A i r b o r n e

I n d i c a t i o n s M e a s l e s

T u b e r c u l o s i s

V a r i c e l l a

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D r o p l e t

M o r e t h a n 5 m i c r o n s

L e s s t h a n 3 f e e t

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D r o p l e t

R e q u i r e m e n t s P r i v a t e r o o m

F r e q u e n t h a n d w a s h i n g

M a s k → w i t h i n 3 f e e t of c l i e n t

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D r o p l e t

I n d i c a t i o n s D i p h t h e r i a

G e r m a n M e a s l e s

M u m p s

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D r o p l e t

I n d i c a t i o n s P e r t u s s i s

P h a r y n g i t i s

P n e u m o n i a

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C o h o r t i n g

P r e s c h o o l e r → S a m e A G E

S c h o o l e r → S a m e S E X

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H a n d w a s h i n g

P r e p a r a t i o n F i n g e r n a i l s → l e s s t h a n ¼ i n c h

A s s e s s for s k i n b r e a k s

R e m o v e A L L j e w e l r y

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H a n d w a s h i n g

R e q u i r e m e n t s R u n n i n g w a t e r → W A R M

S o a p → L i q u i d : 2 – 4 m l or 1 t s p

T i s s u e p a p e r

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H a n d w a s h i n g

K e y p o i n t s 1 0 – 1 5 s e c s

C i r c u l a r m o v e m e n t s

F r i c t i o n

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H a n d w a s h i n g

M e d i c a l H a n d s L o w e r T h a n E l b o w s

D r y in r o t a t i n g m o t i o n f r o m E l b o w s to H a n d s

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H a n d w a s h i n g

S u r g i c a l H a n d s H i g h e r T h a n E l b o w s

D r y in r o t a t i n g m o t i o n f r o m H a n d s to E l b o w s

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D o n n i n g P P E

W a s h h a n d s

G o w n

M a s k

E y e w e a r

G l o v e s

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R e m o v i n g P P E

G l o v e s

W a s h h a n d s

M a s k

G o w n

E y e w e a r

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SICKLE CELL DISEASE• Autosomal recessive inheritance pattern• Sickled RBCs live for 6-20 days only• Usually no symptoms prior to age 6 months• HgbS- sensitive to changes in oxygen content of RBC• Insufficient oxygen cause it to sickle, become

rigid and clumped together--- capillary blood flow

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ASSESSMENT FINDINGS

• First sign in infancy- “COLIC”- due to abdominal pain (Abdominal infarct)

• Spleenomegaly- due to hemolysis and phagocytosis– later part is due to firbrosis from repeated infarct

• Leg ulcers in adolescent– due to blockage of blood supply to skin of legs

• Delayed growth and development

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PRECIPITATING FACTORSFEVEREMOTIONAL/ PHYSICAL STRESS INFECTIONDEHYDRATIONHYPOXIAEXTREME FATIGUEEXTREME CHANGES IN ALTITUDE

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VASO-OCCLUSIVE CRISIS• Most common• Painful• Obstructs blood vessels• Treatment: hydration, oxygen, electrolyte

replacement, bed rest, avoid tight clothing, keep wounds dry and clean

SPLENIC SEQUESTRATION• Life-threatening- causing decrease blood volume and

shock• Treatment: transfusion and spleenectomy

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CYSTIC FIBROSIS

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membrane conductance regulator protein (chloride channel in exocrine tissues)

Chloride transport problems

thick, viscous secretions in the lungs, pancreas, liver,

intestine, and reproductive tract

increased salt in sweat gland secretions

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• pancreatic insufficiency, recurrent pancreatitis, weight loss)

• recurrent abdominal pain• biliary cirrhosis• vitamin deficiencies, • genitourinary problems (male and female

infertility),• clubbing of the extremities

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bronchial plugging/bronchial wall thickening due to inflammation

bronchial infections

Airflow obstruction

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•CYSTIC FIBROSIS multisystem disorder transmitted as autosomal recessive trait affecting the EXOCRINE gland.

SX:•    high level of NaCl in the sweat• “salty kissed”• increased viscosity of mucous

glands secretion

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DIAGNOSTIC TEST

SWEAT CHLORIDE TEST“PILOCARPINE IONTOPHORESIS”-production of sweat is stimulated-sweat is collected -sweat electrolytes are measuredNORMAL: LESS THAN ____mEq/LABNORMAL:MORE THAN ____mEq/LHIGHLY SUGGESTIVE:_____mEq/L requires a repeat test

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TRACTIONS• Pulling force exerted on bones

to reduce or immobilize fractures, reduce muscle spasm, correct or prevent deformities

• SKIN TRACTION-Buck’s traction-Russell traction-Cervical traction-Pelvic Traction

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BUCK’S TRACTION

• Exerts straight pull in the extremity

• Generally used to immobilize the leg in a client with hip fracture

• Shockblocks at the foot of the bed

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RUSSELL TRACTION

• Knee is suspended in a sling attached to a rope and pulley on a Balkan frame– creates an upward pull from the knee

• Weights are attached at the foot of the bed– creating a horizontal force exerted on the tibia and fibula

• Generally used to stabilize fractures on the femoral shaft

• Head of bed: Flat

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Cervical traction pelvic traction

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NURSING CARE FOR PATIENTS WITH TRACTION

• Check traction apparatus• Bed in proper position• Line of traction within the axis of the

bone• Do not rest affected limb against foot

of the bed• Perform neurovascular checks:• Prevent foot drop

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Primary dysmenorrhea • no identifiable pelvic pathology• menarche or shortly thereafter• crampy pain before or shortly after the onset of

menstrual flow - 48 to 72 hours• excessive production of prostaglandins• Psychological factors, such as anxiety and tension

Secondary dysmenorrhea• pelvic pathology such as endometriosis,tumor, or pelvic inflammatory disease (PID) exists

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mgt

• Relieve anxiety• Aspirin every 4 hrs• NSAIDS• Continuous low-level local heat • mechanism is not clear• increases blood flow and may counteract

constriction and• muscle contraction• continue usual activities