bojana gardijan 4th year march 16, 2010 mentor: a. Žmegač horvat
TRANSCRIPT
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Bojana Gardijan4th year
March 16, 2010Mentor: A. Žmegač Horvat
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destruction of heart tissue resulting from obstruction of the blood supply to the heart muscle
part of acute coronary syndrome, ACS non- STEMI
non ST-elevation MI STEMI
ST-elevation MI
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diabetes hyperlipoproteinemia,
especially high LDL and low HDL
high blood pressure family history of
ischemic heart disease
obesity BMI>30 kg/m² age M>45, F>55 stress alcohol
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a manifestation of coronary artery disease, also called ischemic heart disease
most common triggering event: disruption of an atherosclerotic plaque in an coronary artery clotting cascade sometimes results in total occlusion of the artery
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chest pain (typically radiating to the left arm or left side of the neck)
shortness of breath (dyspnea) nausea, vomiting palpitations sweating anxiety
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history and physical examination EKG CBC cardiac markers, preferably troponin T
(and complete biochemistry) differential diagnosis includes pulmonary
embolism, aortic dissection, pericardial effusion causing cardiac tamponade, tension pneumothorax, pancreatitis and esophageal rupture
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MONA as soon as possiblemorphine, oxygen, nitrates, aspirin
clopidogrel, heparin, eptifibatide procedure of choice : PCI (percutaneous
coronary intervention) if unavailable: fibrinolysis
postinfarctial therapy: SAABstatins, ACEI, aspirin, beta blockers
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varies greatly, depending on a person’s health, the extent of the heart damage and the treatment given
time to reperfusion is of great importance
a quick reaction saves lives
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http://en.wikipedia.org/wiki/Myocardial_infarction
http://www.escardio.org