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    ElectricAutoimmunityEtiologyandbasisfornontoxicintervention

    inautoimmunediseases

    AnovelviewofcauseandcofactorsinautoimmunedysregulationinRheumatoid

    Arthritis,MultipleSclerosis,AnkylosingSpondylitis, Lupusandotherautoimmune

    syndromes. Thisarticlepresentsanintegratedviewofcellularenergeticfactors

    participateautoimmunetargetingwithBCelllinesthroughTumorNecrosisFactor

    targetingcommon

    toautoimmune

    disease.

    2008

    MarkSquibb

    WholeHealthResearchAlliance,LLC

    4/15/2008

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    ContentsForeword......................................................................... 3

    TheCellMembrane......................................................... 4

    TheBasisofPower...................................................... 4

    pHCulture................................................................... 5

    AutoimmuneDysregulation............................................ 6

    ElectricallyMediatedTNFCancerResponse...............6

    TNFandAutoimmuneDysfunction............................. 6

    PEMFCellularEffectModel............................................ 7

    InVivoPasteurization..................................................... 7

    ObviousversusStealth................................................ 8

    RingingandPathogens................................................ 9

    PulsedMagneticFieldsandBiology............................ 9

    ElectromagneticSensitivityExplained........................ 9

    AutoimmuneCofactors................................................... 9

    ThePotassiumMystery................................................. 10

    TheSecretPotassiumSource.................................... 10

    PotassiumAnabolism................................................ 10

    PotassiumandAutoimmuneSymptoms................... 10

    LupusFootnote......................................................... 11

    TherapeuticResponseModel................................... 11

    AutoimmuneSyndromes.............................................. 12

    RheumatoidArthritis/AnkylosingSpondylitis:........12

    MultipleSclerosis...................................................... 12

    LupusErythematosus................................................ 12

    AutoimmuneCofactorCandidates............................... 13

    ForProtocolInformation.............................................. 13

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    ForewordCellsareelectricaldevices.

    Thisarticlepresentsanelectricallyorientedviewofthe

    cell,withparticularfocusonthecellmembrane.

    Autoimmunity,asconductedbylymphocytes,BCells,T

    Cells,andNaturalKillercells,generallyinvolves

    interactionsofthesecellswithphysicalcontactto

    evaluatethehealthofothercells.

    Autoimmunityisthearmyofcellswhichpatrol,

    evaluate,andeliminateforeignanddisruptive

    elements,inthecontinuingprocessoflifeandhealth.

    Thetraditionalviewthatthesecellularinteractionsare

    chemical,andnotelectrical,isstrikinglylimited.

    Thisarticlesuggestsnovelviewthatmostautoimmune

    disordersreflectacombinationautoelectrical

    dysfunctionscombinedwithimmunological

    insufficiencyagainstanundiagnosedpathogen.

    Thisarticlepresentstheviewthatautoimmune

    interactionsareinteractionsareelectrical,magnetic,

    andparamagnetic,andchemicalinnature. Modern

    researchprovidesatleastfifteendifferentfield

    phenomenon,whichinaggregate,enableastrikingly

    differentmodelingforimmunologicalcellular

    interaction.

    Thetraditionalviewthatautoimmunityischemically

    mediated,andthenearabsenceofcurativeprogressin

    treatmentofautoimmunediseases,suggeststhat

    researchandtreatment,aremisdirected.

    Thepurposeofthisdocumentistoopenanewchapter

    inautoimmunemodeling.

    http://en.wikipedia.org/wiki/Electric_fieldhttp://en.wikipedia.org/wiki/Magnetichttp://en.wikipedia.org/wiki/Paramagnetichttp://en.wikipedia.org/wiki/Paramagnetichttp://en.wikipedia.org/wiki/Magnetichttp://en.wikipedia.org/wiki/Electric_field
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    TheCellMembrane

    Thecellmembraneistheoutsideshellofthecell. It

    isverythin,rangingfrom38nanometers. Ithosts

    millionsorbillionsofchemicalstructuresthat

    implementthecellsroleinthebody.

    Thesubstrateismadeofspeciallipidsorfats,which

    separatetheinsideofthecellfromtheoutsideofthe

    cellinabilayermembrane. Themembraneisa

    specialbecauseitischemicalandelectrical.

    Membranesarestructuralcomponentsofcells,a

    fattyskeletonseparatingwatercompartments. The

    exteriormembraneencapsulatestheexteriorofthe

    cell,andinterfacestocelltobodyfunctions.

    Theexternalmembraneencapsulatesthecell,which

    inturnhousesmanyotherstructures,whichmany

    areencasedincellmembranematerial.

    Phospholipidstructuresprovidethestructureofthe

    mitochondria,whichproduceenergy. Inotherwords,

    cellmembranematerialisafunctionalstructure,

    providingbothformandfunctiontoexteriorand

    interiorcellularfunctions,includingmultipleenergy

    productionprocesses.

    Hereisalinktoatrulyexcellentcompilationof

    mitochondrialfunctionsponsoredbyDrsClarkand

    Cargile. Thisisa22,000articlecompilationofabout

    120,000references. ItIStheplacetostartlearning

    aboutcellularenergyproduction.

    Asanelectricalelement,itisasemiconductor,a

    capacitor,aresistorandabattery. Asachemicalentity,itissimplyindescribable.

    TheBasisofPower

    Thecellmembraneisachemicalandelectrical

    insulator. Theinsideandoutsideofthemembrane

    hostsapHdifferential. ThepHdifferentialcreatesa

    voltage,orelectricity. Thiselectricityisthepower

    sourceformanyessentialfunctionsinthecell

    membrane.

    Anythingthatcompromisesthecellmembrane

    powerisaprobablecauseorcofactorincellular

    malfunction.

    Compromiseofeitherthecellmembranequalityor

    thetransmembrane

    pHdifferentialinhibits

    theelectrical

    functionsofthecell,

    andpreventsoptimalcellfunction.

    Theintracellularand

    extracellularpH

    differentialcreatesa

    voltagewhich

    provideschemical

    potentialorbatteryforcellmembranefunctions.

    http://en.wikipedia.org/wiki/Cell_membranehttp://en.wikipedia.org/wiki/Cell_membranehttp://en.wikipedia.org/wiki/Cell_membranehttp://en.wikipedia.org/wiki/Cell_%28biology%29http://en.wikipedia.org/wiki/Lipidhttp://en.wikipedia.org/wiki/Lipidhttp://en.wikipedia.org/wiki/Lipidhttp://en.wikipedia.org/wiki/Lipidhttp://en.wikipedia.org/wiki/Lipidhttp://en.wikipedia.org/wiki/Bilayer_lipid_membranehttp://en.wikipedia.org/wiki/Bilayer_lipid_membranehttp://en.wikipedia.org/wiki/Bilayer_lipid_membranehttp://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://en.wikipedia.org/wiki/Electricityhttp://en.wikipedia.org/wiki/Semiconductorhttp://en.wikipedia.org/wiki/Capacitorhttp://en.wikipedia.org/wiki/Resistorhttp://en.wikipedia.org/wiki/Battery_%28electricity%29http://en.wikipedia.org/wiki/Electrical_insulationhttp://en.wikipedia.org/wiki/PHhttp://en.wikipedia.org/wiki/Electric_powerhttp://en.wikipedia.org/wiki/Electric_powerhttp://en.wikipedia.org/wiki/PHhttp://en.wikipedia.org/wiki/Electrical_insulationhttp://en.wikipedia.org/wiki/Battery_%28electricity%29http://en.wikipedia.org/wiki/Resistorhttp://en.wikipedia.org/wiki/Capacitorhttp://en.wikipedia.org/wiki/Semiconductorhttp://en.wikipedia.org/wiki/Electricityhttp://www.themitochondria.com/book/http://www.themitochondria.com/book/http://www.themitochondria.com/book/http://en.wikipedia.org/wiki/Bilayer_lipid_membranehttp://en.wikipedia.org/wiki/Lipidhttp://en.wikipedia.org/wiki/Cell_%28biology%29http://en.wikipedia.org/wiki/Cell_membrane
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    Thispowersupplyistheessentialbasisforhormone

    regulation,anabolicenergyproduction,andionic

    cellularrespiration.

    Anythingthatcausesthecellmembranetoleak

    electricitydrainspower. BothinadequatepH

    differential,andpowerleakscausedbygarbage

    lodgedinmembranestructures,interferewithcellularfunctionsthatrequirepower.

    Whencellmembranepowerisdownduetolipid

    toxins,orpHimbalancescelljustdontwork. When

    cellsdontwork,thebodydoesntwork.

    Therearethreemainaspectsofcellmembrane

    powerproduction:

    1. Thelipidsubstratemustinsulatetheinsideandoutsideandnotleakpower;

    2. ThepHdifferentialmustbebalancedtoenableelectricity,justlikeacarbattery;

    3. RawMaterials mustbepresenttocreatechemicalandelectricalstructuresneededfor

    thecelltowork.

    pHCulture

    pHisutterlyimportantbecauseitenablesthecellto

    produceelectricalpowertodrivemembrane

    functions.

    ThereisatendencyinhealthcaretofocusonpH

    withoutconsideringthemembraneintegrity.

    Singularfocusonwaterchemistrytendstoleaveabig

    partofmanypeopleshealthproblemsimproved,but

    unresolved.

    pHandwaterchemistryisrelativelyeasytoshift

    becausethebodywatercompartmentstendtoquicklyexchange. Waterrelatedprotocolsproduce

    beneficial,butshortterm,resultsbecausethey

    temporarilycompensateforothermorestructural,or

    lipiddysfunctions.

    Inotherwords,waternutrientprotocolsincreasecell

    voltagebytemporarilyimprovingtheabilityto

    maintainpHinsideandoutsidethecell.

    Unfortunately, waterchemistryleavesthecellular

    structuralgarbageinplace,resultinginatendency

    forshorttermbenefits.

    Clearingboththemembranetoxins,andthesources,

    andrestoringthecellularprocessofautomatic

    cleansingiscriticaltodurablerestorationofcellular

    health.

    http://en.wikipedia.org/wiki/Cell_(biology)
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    AutoimmuneDysregulation

    Theautoimmunesystemprotectsthebodyfrom

    invasion,andkeepsfriendlyorganismsundercontrol.

    Theautoimmunesystemuseslibraryofinvader

    sensingcapabilities. It

    respondstoinvading

    pathogensor

    overgrowthofsymbiotic

    organisms,cells,

    bacteria,yeast,andfungi,usingmanydifferent,and

    oftenbarelyunderstoodsensingmechanisms.

    Variousformsofwhitebloodcells,lymphocytes,

    patrolthebodycontinuouslylookingforimbalanced

    cellsororganisms. Immunepatrollymphocytes,B

    Cells&TCells,andNaturalKillerCellsmaintain

    constantguardforinvadingorovergrownerrant

    organisms.

    TumorNecrosisFactorAlphaisamemberoftheTNF

    familyofcytokines. Thisfamilyofcytokinestagcells

    fordestructionbytheimmunesystem. Theyarecreatedbymacrophagesandotherimmunesystem

    cells.

    TNFAlphaareaspecialclassofproteinscalled

    transmembraneproteins.Insimpleterms,theyreach

    throughthecellmembrane. Thetransmembrane

    proteinandtransmembranepotential sharethe

    membranedimension.

    ThisauthorsuggeststhattheTNFtriggersapoptosis,

    celldeath,whenacellmembranevoltagedrops

    belowatriggerthreshold. TNFisthereforeadefense

    mechanismagainstdiseaseswhichresultfromcells

    whichcannotmaintainmembranepower.

    ElectricallyMediatedTNFCancer

    Response

    Healthycellsexistwithatransmembranepotentialof

    about70mV. Cancercellshaveamembrane

    potentialfrom1530mV. Sincecancercellsexist

    belowtheapoptosistriggervoltage,TNFisafront

    linedefenseforcancer.

    TNFwasnamedafteritmainrole,triggeringdeathof

    cancercellsbecauseoflowcellmembranevoltage.

    TNFandAutoimmuneDysfunction

    Whencellsmembraneintegritydeterioratestoa

    levelnearorbelowtheTNFactivation,the

    autoimmunetargetingofseeminglyhealthycells

    occur,resultinginvariousautoimmunediseases.

    Tissuestargetedbyautoimmunediseasestendto

    sharerelativelylowlevelsofoxygenation.

    Thisphenomenonexplainswhypulsed

    electromagnetictherapiesprovideoftenstriking

    benefitswithautoimmunesyndromes.

    Thistimelapsephotoofanindividualdiagnosedwith

    RheumatoidArthritis. Theindividualshandswere

    exposedforaboutsixminutesperdaythreetimes

    http://en.wikipedia.org/wiki/Lymphocyteshttp://en.wikipedia.org/wiki/B_cellhttp://en.wikipedia.org/wiki/B_cellhttp://en.wikipedia.org/wiki/B_cellhttp://en.wikipedia.org/wiki/T_cellhttp://en.wikipedia.org/wiki/T_cellhttp://en.wikipedia.org/wiki/T_cellhttp://en.wikipedia.org/wiki/Natural_killer_cellhttp://en.wikipedia.org/wiki/Natural_killer_cellhttp://en.wikipedia.org/wiki/Natural_killer_cellhttp://en.wikipedia.org/wiki/Natural_killer_cellhttp://en.wikipedia.org/wiki/Natural_killer_cellhttp://en.wikipedia.org/wiki/Tumor_necrosis_factor-alphahttp://en.wikipedia.org/wiki/Tumor_necrosis_factor-alphahttp://en.wikipedia.org/wiki/Tumor_necrosis_factor-alphahttp://en.wikipedia.org/wiki/Tumor_necrosis_factor-alphahttp://en.wikipedia.org/wiki/Tumor_necrosis_factor-alphahttp://en.wikipedia.org/wiki/Tumor_necrosis_factor-alphahttp://en.wikipedia.org/wiki/Tumor_necrosis_factor-alphahttp://en.wikipedia.org/wiki/Tumor_necrosis_factorshttp://en.wikipedia.org/wiki/Tumor_necrosis_factorshttp://en.wikipedia.org/wiki/Tumor_necrosis_factorshttp://en.wikipedia.org/wiki/Cytokineshttp://en.wikipedia.org/wiki/Macrophageshttp://en.wikipedia.org/wiki/Transmembrane_proteinhttp://en.wikipedia.org/wiki/Transmembrane_proteinhttp://en.wikipedia.org/wiki/Transmembrane_proteinhttp://en.wikipedia.org/wiki/Transmembrane_proteinhttp://en.wikipedia.org/wiki/Transmembrane_proteinhttp://en.wikipedia.org/wiki/Transmembrane_proteinhttp://en.wikipedia.org/wiki/Transmembrane_potentialhttp://en.wikipedia.org/wiki/Transmembrane_potentialhttp://en.wikipedia.org/wiki/Transmembrane_potentialhttp://en.wikipedia.org/wiki/Transmembrane_potentialhttp://en.wikipedia.org/wiki/Apoptosishttp://en.wikipedia.org/wiki/Apoptosishttp://en.wikipedia.org/wiki/Transmembrane_potentialhttp://en.wikipedia.org/wiki/Transmembrane_proteinhttp://en.wikipedia.org/wiki/Transmembrane_proteinhttp://en.wikipedia.org/wiki/Transmembrane_proteinhttp://en.wikipedia.org/wiki/Macrophageshttp://en.wikipedia.org/wiki/Cytokineshttp://en.wikipedia.org/wiki/Tumor_necrosis_factorshttp://en.wikipedia.org/wiki/Tumor_necrosis_factorshttp://en.wikipedia.org/wiki/Tumor_necrosis_factor-alphahttp://en.wikipedia.org/wiki/Natural_killer_cellhttp://en.wikipedia.org/wiki/T_cellhttp://en.wikipedia.org/wiki/B_cellhttp://en.wikipedia.org/wiki/B_cellhttp://en.wikipedia.org/wiki/Lymphocytes
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    perweekforfourmonthstopulsedmagneticfields,

    approximately1microsecond,and25Teslaintensity.

    Before After4months

    Thesignificantdecreaseinjointdeformationovera4

    monthperiodisthelikelyresultofhealingwhich

    occurredinthesustainedabsenceoflocalized

    autoimmuneoveractivity,andprobablyimprovedoxygenation.

    Theseresultswerethelikelyresultofsynergistic

    effects:

    Eachexposureimprovedthetransmembranepotentialforaperiodof13days,likely

    resultinginatendencytoinhibitautoimmune

    TNFtargeting;

    PEMFexposureincreasestissueoxygenavailabilitythroughavarietyofmeans,likely

    resultinginimprovedtissueoxygenation,

    whichenabledhealingwhichwouldnothave

    beenotherwisepossible;

    PEMFexposurewasnotlimitedtothehands,andtheresultswereconsistentthroughout

    thebody.

    PEMFCellular

    EffectModelPulsedelectromagnetic

    fieldscreateasignificant

    turbulenceatthe

    cellularlevel. Brief,microsecond,pulsesgenerated

    whenelectricitybridgesagap,causeashortcurrentto

    traverseawire.

    Theshortcurrentcausesatendencyforelectricityto

    flowoppositethecurrentinthewireneartheexposure

    Theresultisa

    forward/backward

    electricityflowinthe

    tissuesnearthewire.

    Cellsinthevicinityofthe

    wireappeartoabsorb

    energyusuallyobtaining

    adurableincreasein

    transmembranepotential,usuallyobservedasapparent

    performanceimprovementincellmembrane

    structures,including,butnotlimitedto,TNFsignaling.

    In VivoPasteurizationPulsedfieldsalsotendtoassertantipathogenicactivity

    oncertainmicroorganisms. Clickthelinksbelowto

    reviewNIHreferenceswhichindicatefunctional

    deactivationofvariousmicroorganismsinfood

    products:

    Saccharomycescerevisiae,AlternateArticle SalmonellaEnteritidis SalmonellaEnteritidis,E.coliandL.

    monocytogenes

    Escherichiacoli,ListeriainnocuaandSaccharomycescerevisiae

    ListeriaMonocytogenes Escherichiacoli,SalmonellaTyphimuriumand

    Listeriainnocua

    PseudomonasFluorescens Lactobacillusplantarum KillingofMicroorganismsbypulsedelectric

    fields

    MycobacteriumparatuberculosisAlertreadersmayperceivethattheantimicroorganism

    effectsofpulsedfieldsarelikelynotlimitedtofood

    products.

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    Theseresearchreferencesstronglysuggestthat

    microbialdeactivationisabyproductofPEMF

    exposure. Theresultsoftheexposurearegenerally:

    ProportionaltotheintensityofthePEMFpulse; Inverselyproportionaltothedurationofthe

    PEMFpulsesshorterpulsesexhibitstronger

    antimicrobialeffects.

    Inotherwordshighintensityshortpulsesdoabetter

    jobofdisruptingmicrobiallifecyclethanlowintensity

    longdurationpulses.

    PEMFexposurehasthelikelycobenefitofinhibiting

    pathogenicmicrobes,hencereducingtheloadonthe

    autoimmunesystem.

    Obviousversus

    Stealth

    Autoimmunedisorders

    reflectatendencytoattack

    cellswithnoobvious

    pathogens. Thekeyword

    hereisobvious.

    Thereareawiderangeof

    pathogenicfactorswhich

    remainundetectable/un

    testableusingcommon

    laboratoryassays.

    Viralandothercrittersmay

    infactberesidentinsidecells. Therearemany

    examplesofdormantcells,

    likeHIV,Herpes,andLyme

    parasites,whichremain

    hiddenbeyondthereachof

    autoimmunesystem.

    Inotherwords,theinadequateorincompleteimmune

    responseisalikelycofactorintheongoingautoimmune

    challengesfacedbyindividualswithautoimmune

    conditions.

    Blaminganoveractiveimmunesystemforattacking

    innocentcellsisatbestnave. Thediagnosticinability

    toidentifyapathogendoesntmeanthatthereisnt

    one.

    StealthpathogensmaycreateaSisiphuscurseonthe

    immunesystem,damnedbythegodstopursuethe

    unachievable.

    Allopathicautoimmuneinterventionsreducesymptoms

    bytakingoutpartoftheimmunesystem,normally

    inhibitingBCellsynthesis,orcripplingtheTNF

    mediatedresponse.

    ThealertreaderwillfinditcuriousthatBCell

    interventionstakeout/inhibitBCellautoimmune

    response,whichisdirectlyrelatedwithviraldefense.

    Autoimmunedysregulationstrikinglyresemblesthe

    Sisyphusphenomenon,damnedbythegodstopursue

    theunachievablewithstealthpathogensplayingthe

    roleofTartarus.

    ResearchdatadoesnotpresentlysupportthatPEMF

    exposureisdirectlyantiviralinthemicrosecondpulsewidthrange.

    Newdatahoweverstronglyindicatesthatatshorter

    pulsewidths,intherangeof200ns,exhibitsurprising

    effectsonmelanomas.

    ItisconceivablethatsharperPEMFexposurehelpsthe

    immunesystemby:

    Disablingpathogensthatmatchthewaveformofthepulses,largepathogens,bacteriaas

    discussedearlier,respondtocoarsepulses;

    Smallerpathogensareincreasinglydisruptedbysmallerdurationmoreintensepulses;

    Reducingthelargerpathogenicformssupportstheimmunesystembyreducingoverall

    autoimmuneload,freeingresourcesforother

    antipathogenactivities.

    http://en.wikipedia.org/wiki/Sisyphushttp://en.wikipedia.org/wiki/Sisyphushttp://en.wikipedia.org/wiki/Tartarushttp://www.pubmedcentral.nih.gov/articlerender.fcgi?&pubmedid=16545779http://www.pubmedcentral.nih.gov/articlerender.fcgi?&pubmedid=16545779http://www.pubmedcentral.nih.gov/articlerender.fcgi?&pubmedid=16545779http://www.pubmedcentral.nih.gov/articlerender.fcgi?&pubmedid=16545779http://www.pubmedcentral.nih.gov/articlerender.fcgi?&pubmedid=16545779http://www.pubmedcentral.nih.gov/articlerender.fcgi?&pubmedid=16545779http://www.pubmedcentral.nih.gov/articlerender.fcgi?&pubmedid=16545779http://www.pubmedcentral.nih.gov/articlerender.fcgi?&pubmedid=16545779http://www.pubmedcentral.nih.gov/articlerender.fcgi?&pubmedid=16545779http://www.pubmedcentral.nih.gov/articlerender.fcgi?&pubmedid=16545779http://www.pubmedcentral.nih.gov/articlerender.fcgi?&pubmedid=16545779http://en.wikipedia.org/wiki/Tartarushttp://en.wikipedia.org/wiki/Sisyphushttp://en.wikipedia.org/wiki/Sisyphus
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    WholeHealthResearchAlliance MarkSquibb P a g e |9

    RingingandPathogens

    Biologicaltissuesstimulatedwithrawpulseenergy

    resonateattheirnaturalfrequency.

    Reinforcingthenaturalresonancestrengthensthe

    dominantorganism. Similarly,strongmaster

    resonancecreatesanoftendebilitatingstrong

    energeticdisadvantageforpathogenicorganisms,by

    disruptingnonharmonic,pathogenicelements.

    PEMFexposurealsoprovidesantipathogeniceffects

    mostclearlydocumentedintheabilitytousePEMF

    asasterilization

    andpasteurization

    technique,NIH

    ReferencesHere.

    Itsveryhandytobe

    abletodo

    invivo

    sterilization,andto

    strengthenthehost

    organism.

    PulsedMagneticFieldsandBiology

    Thesituationinbiologicalorganismsissimilar. The

    pulseistheringer,causingthebodytoringstrongat

    itsnaturalfrequency. Anythingwhichdoesntring

    along,likepathogens,experiencestress,and

    encounteranenvironmentaldisadvantage.

    ElectromagneticSensitivityExplained

    Electricallyweakindividualswillringloudly. This

    potentringingcreatesstrongsensations.

    Individualswithelectricallyweakcellstendtobe

    moresensitivetopulsedfieldsbecausetheircells

    respondmorereadilytobothbeneficialandharmful

    radiation.

    Theytendtogainenergyrapidlyfrompulsedfields

    whichsupportscellularmetabolism. Likewisethey

    tendtoresonatewithharmfulradiations. Useof

    pulsedmagneticfieldstendstodecreasesensitivity

    todetrimentalelectromagneticradiationbystrengtheningthenativebiofield.

    AutoimmuneCofactors

    Diseasesandthemisstargetedcells:

    MultipleSclerosisAttacksthecellmembraneofnervescausingdegenerationin

    themyelinsheath. 1000+NIHarticleslinking

    TNFandMS.

    RheumatoidArthritisAttacksjoints,typicallythesynovium,appearinglikethe

    immunesystemhasgoneawry. Clickhereto

    review5000+NIHarticleslinkingTNFandRA.

    Lupusseeminglyattackscellsatrandomaroundthebody. Clickheretoreview700+

    NIHarticleslinkingTNFandRA.

    AnkylosingSpondylitisAttacksthespinecausingdegeneration. Clickheretoreview

    435+NIHarticleslinkingTNFandAS.Eachoftheseautoimmunedisorderssharesseveral

    curiousattributes:

    Individualswithautoimmunediagnosistendtohaveexhibitpotassiumdeficiency; Click

    heretoreview208+NIHarticleslinking

    autoimmunediseasesandpotassium

    deficiency.

    TestsshowelevationsinTumorNecrosisFactor;

    SymptomsrespondtodrugswhichsuppressTNF;

    SymptomsareeasedbyfactorsthatsuppressTNF,curcumin;

    SymptomsrespondtodrugsthatinhibitBCelresponses;

    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earch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(%25ra%20or%20rheumatoid%20arthritis)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(%25ra%20or%20rheumatoid%20arthritis)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(%25ra%20or%20rheumatoid%20arthritis)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(%25ra%20or%20rheumatoid%20arthritis)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(%25ra%20or%20rheumatoid%20arthritis)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(%25ra%20or%20rheumatoid%20arthritis)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(%25ra%20or%20rheumatoid%20arthritis)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(%25ra%20or%20rheumatoid%20arthritis)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(%25ra%20or%20rheumatoid%20arthritis)%20)http://en.wikipedia.org/wiki/Lupus_erythematosushttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(lupus)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(lupus)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(lupus)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(lupus)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(lupus)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(lupus)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(lupus)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(lupus)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(lupus)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(lupus)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(lupus)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(lupus)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(lupus)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(lupus)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(lupus)%20)http://en.wikipedia.org/wiki/Ankylosing_spondylitishttp://en.wikipedia.org/wiki/Ankylosing_spondylitishttp://en.wikipedia.org/wiki/Ankylosing_spondylitishttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(ankylosing%20or%20spondylitis)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(ankylosing%20or%20spondylitis)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(ankylosing%20or%20spondylitis)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(ankylosing%20or%20spondylitis)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(ankylosing%20or%20spondylitis)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(ankylosing%20or%20spondylitis)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(ankylosing%20or%20spondylitis)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=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or%20necrosis%20factor)%20and%20(ankylosing%20or%20spondylitis)%20)http://en.wikipedia.org/wiki/Ankylosing_spondylitishttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(lupus)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(lupus)%20)http://en.wikipedia.org/wiki/Lupus_erythematosushttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(%25ra%20or%20rheumatoid%20arthritis)%20)http://en.wikipedia.org/wiki/Synoviumhttp://en.wikipedia.org/wiki/Rheumatoid_arthritishttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(%20ms%20or%20multiple%20sclerosis)%20)http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%20(%20(%20ttnf%20or%20tumor%20necrosis%20factor)%20and%20(%20ms%20or%20multiple%20sclerosis)%20)http://en.wikipedia.org/wiki/Multiple_sclerosishttp://en.wikipedia.org/wiki/Multiple_sclerosishttp://en.wikipedia.org/wiki/Multiple_sclerosishttp://en.wikipedia.org/wiki/In_vivohttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%28%20pemf%20OR%20%28pulsed%20and%20%28magnetic%20or%20electromagnetic%29%29%20and%20%28%28pasteurization%20or%20sterilization%29%29http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=PureSearch&db=pubmed&term=%28%20pemf%20OR%20%28pulsed%20and%20%28magnetic%20or%20electromagnetic%29%29%20and%20%28%28pasteurization%20or%20sterilization%29%29http://en.wikipedia.org/wiki/Pasteurization
  • 8/6/2019 Electric Autoimmunity

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    WholeHealthResearchAlliance MarkSquibb P a g e |10

    ThePotassiumMysteryObscurereferencesdocumenttherelationshipfor

    systemicpotassiumdeficiencyandrheumatoidarthritis.

    Depressedsystempotassiumlevelsareconsistentwith

    virtuallyallautoimmunedisorders. Themostvisible

    telltaleforanabolicmetabolismdysfunctionisvisiblein

    bloodtests:

    Highserumpotassium,above4.5mEq)asthebodyineffectivelyelevatescirculating

    potassiuminattempttoincreasecellularlevels

    (Revici),above;

    Highredbloodcellsedimentationrate,above15ml/hr;

    LowEosinophilialevels,below100cmm; LowurinarypH,below6.4;

    Lowurinary

    surface

    tension;

    Lowcalciumorchlorideexcretion.Generally,autoimmunedisordersarepredicatedbya

    longtermhistoryofanabolicmetabolismdysfunction.

    Inmanycasesthereisahistoryofpoorsleepinwomen,

    andacceleratedaginginmen. Unlikewomen,menwith

    acellularanabolicimbalancetendtosleepbecause

    elevatedtestosteronetendstoenablesleep.

    TheSecretPotassiumSourceCellularpotassiumcomesfromthecoldfusionof:

    Na+O >K+energy.

    Thiswelldocumentedsecretwasdescribedand

    confirmedbyLouisKervran,andconfirmedbySolomon

    GoldfeinworkingfortheUSArmy. Goldfeinalso

    confirmedthefusionof:

    Mg+O >Ca+energy

    Thesetwomineralconversionequationsprovidea

    RosettaStoneforirresolvablemetabolicsyndromes,

    includingautoimmunedisordersandseveralformsof

    chronicfatigue.

    PotassiumAnabolism

    ThePotassiumfusionreaction,Na+O K, isamajor

    driverintheanabolicenergyproduction.

    Asanabolicenergyproductiondegenerates,cellularand

    systemicpotassiumlevelsdecline,resultinginarange

    ofconditionsrelatingto:

    AnabolicDysfunctionHealingdeficiency; CellularPotassiumDeficiencysyndromes

    particularlyneurologicaltransmission

    dysfunctionsresultingfromaxonpotassium

    deficiency;

    Neural

    hyper

    excitability

    syndromes,

    epilepsy,

    tinnitus,resultingfromattenuatedneuralsignal

    toneuralnoiseratios;

    SystemicPotassiumDeficiencysyndromes.Inthemeantime,cellularmitochondriacompensate,

    overworking,anddepletingMagnesiumreserves,and

    otheroxygenrelatedmetabolites.

    Cellularsleepdegeneratesasmitochondrialenergy

    productiondominatesthelifefunctions. Restisrare

    andofpoorquality.

    PotassiumandAutoimmuneSymptoms

    Thesereferencescorrelatedepressedsystemic

    potassiumlevelswithRheumatoidArthritis:

    Boneturnoverinearlyrheumatoidarthritis LaCelle,P.L.,etal.1964"AnInvestigationof

    TotalBodyPotassiuminPatientswith

    http://dshedu.com/Research/MembranePower/SupportPages/USArmyGoldfeinReport.htmhttp://dshedu.com/Research/MembranePower/SupportPages/USArmyGoldfeinReport.htmhttp://dshedu.com/Research/MembranePower/SupportPages/USArmyGoldfeinReport.htmhttp://dshedu.com/Research/MembranePower/SupportPages/USArmyGoldfeinReport.htmhttp://dshedu.com/Research/MembranePower/SupportPages/USArmyGoldfeinReport.htmhttp://dshedu.com/Research/MembranePower/SupportPages/USArmyGoldfeinReport.htmhttp://dshedu.com/Research/MembranePower/SupportPages/USArmyGoldfeinReport.htmhttp://dshedu.com/Research/MembranePower/SupportPages/USArmyGoldfeinReport.htmhttp://dshedu.com/Research/MembranePower/SupportPages/USArmyGoldfeinReport.htmhttp://dshedu.com/Research/MembranePower/SupportPages/USArmyGoldfeinReport.htmhttp://dshedu.com/Research/MembranePower/SupportPages/USArmyGoldfeinReport.htmhttp://en.wikipedia.org/wiki/Rosetta_stonehttp://en.wikipedia.org/wiki/Rosetta_stonehttp://en.wikipedia.org/wiki/Rosetta_stonehttp://www.pubmedcentral.nih.gov/picrender.fcgi?artid=1001712&blobtype=pdfhttp://www.pubmedcentral.nih.gov/picrender.fcgi?artid=1001712&blobtype=pdfhttp://www.pubmedcentral.nih.gov/picrender.fcgi?artid=1001712&blobtype=pdfhttp://www.pubmedcentral.nih.gov/picrender.fcgi?artid=1001712&blobtype=pdfhttp://www.pubmedcentral.nih.gov/picrender.fcgi?artid=1001712&blobtype=pdfhttp://www.pubmedcentral.nih.gov/picrender.fcgi?artid=1001712&blobtype=pdfhttp://www.pubmedcentral.nih.gov/picrender.fcgi?artid=1001712&blobtype=pdfhttp://www.pubmedcentral.nih.gov/picrender.fcgi?artid=1001712&blobtype=pdfhttp://www.pubmedcentral.nih.gov/picrender.fcgi?artid=1001712&blobtype=pdfhttp://www.pubmedcentral.nih.gov/picrender.fcgi?artid=1001712&blobtype=pdfhttp://www.pubmedcentral.nih.gov/picrender.fcgi?artid=1001712&blobtype=pdfhttp://www.pubmedcentral.nih.gov/picrender.fcgi?artid=1001712&blobtype=pdfhttp://www.pubmedcentral.nih.gov/picrender.fcgi?artid=1001712&blobtype=pdfhttp://en.wikipedia.org/wiki/Rosetta_stonehttp://dshedu.com/Research/MembranePower/SupportPages/USArmyGoldfeinReport.htm
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    RheumatoidArthritis."Proc.Ann.MeetingoftheAm.RheumatismAssoc.Arth.Rheum.7;321.

    LupusFootnote

    LupusisweaklydifferentiatedfromRheumatoid

    Arthritis. Symptomaticdifferentiationbetweenthetwo

    conditionsrelatestothelocationtendencyof

    inflammationsymptoms. Lupusfloatswhile

    RheumatoidArthritisaffectsprimarilyjointtissue.

    Inallcasesautoimmunediseasesareprecededby

    breakdownincellularanabolicperformance,and

    consequentlycellularpotassiumdeficiencyis

    guaranteedinautoimmunedisorders.80%

    ofadults

    with

    rheumatoid

    arthritis

    test

    positive

    for

    rheumatoidfactor. Moreover,individualswithLupus

    frequentlytestpositiveforrheumatoidfactor.Thecriticaloversightinresearchliteratureisthat

    cellularpotassiumisabyproductofanabolic

    metabolism.TherapeuticResponseModel

    Theapproachsuggestedinthisessayseeksto

    improvethecellmembranevoltagebycorrecting

    systemicandnutritionalfactorswhichcompromise

    cellmembranepower.

    Dietarypotassiumsourcesweaklyinfluencecellular

    potassiumlevelsbecausethecellularpotassium

    channelsareonewayout,notin.

    Modestexceptionsoccurwhendietarypotassiumis

    encapsulated,chelatedintoaspartate,orotateforms,or

    lipidstructureswhichintegratewiththecellmembrane.

    TNFautoimmunetriggeringtargetselectricallyweak

    cells. Electricallyweakcellsresultfromdeficiencyin

    cellularpower. Restorationofnormalcellularenergy

    isbroadlyeffectiveatrestoringautoimmunemiss

    targeting.

    Cellularpotassium,andeventuallysystemicpotassium

    levelsdropbecausetheprocesswhichcreatesthe

    potassiumfails,notbecauseofpotassiumintakeis

    deficient. Generally,protocolsthatrestorecellularpower

    productionreducethetendencyforimmunemiss

    targeting.Thebiasedviewthatpotassiumrelatedissuesisalways

    relatedtopotassiumchanneldysfunctionreflectthe

    dominantoversightthatcellularpotassiumisa

    metabolicbyproduct.Cell power restoration

    is broadly effective at

    treating autoimmune

    disorders.

    WholeHealthResearchAlliance MarkSquibb P a g e |11

    http://dshedu.com/Research/PEMF/index.phphttp://dshedu.com/Research/PEMF/index.phphttp://dshedu.com/Research/PEMF/index.phphttp://dshedu.com/Research/PEMF/index.phphttp://dshedu.com/Research/PEMF/index.phphttp://dshedu.com/Research/PEMF/index.phphttp://dshedu.com/Research/PEMF/index.phphttp://dshedu.com/Research/PEMF/index.phphttp://dshedu.com/Research/PEMF/index.php
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    WholeHealthResearchAlliance MarkSquibb P a g e |12

    AutoimmuneSyndromes

    Thepatterninautoimmunesyndromesisgenerally

    thesame:

    1. Toxinsorotherfactorscausecompromisedcellmembraneperformance;

    2. Resultinginlowcellmembranevoltage(lowmembranevoltageiscommontomostcancercells);

    3. LowcellmembranevoltagecreatesvulnerabilitytoTumorNecrosisFactor,TNF,

    autoimmuneresponse;

    4. TNFactivationcausesinflammatoryandimmuneresponse.

    Neurologicalautoimmunesyndromesusuallyhave

    symptomsfromneuralpotassiumdeficiency,

    resultingfromcompromisedanabolism. Neuralpotassiumdeficiencycausesdegenerateimpulse

    transmissionthroughnerves.

    RheumatoidArthritis/Ankylosing

    Spondylitis:

    1. Synoviumcellssurroundingjointsbecomeweakeneddueage,stressorother

    environmentalfactors;

    2. Resultinginadisruptedanabolicenergyproduction;

    3. ResultingincellmembranepotentialfallingbelowthetriggerthresholdforTNFtriggering;

    4. Bcellsinitiateimmuneresponseontaggedcellscausingdestructionofthesecells;

    5. Thebodydepositscalciumintoisolatetherestofthebodyfromthediseasedcells.

    MultipleSclerosis

    1.

    Nervecell

    sheaths

    become

    weakened

    due

    to

    pathogenicorsystemictoxinaccumulation;

    2. Resultinginadisruptedanabolicenergyproduction;

    3. Whichcausesadeclineinintraneuralpotassium;

    4. Whichcausesadecreaseintheabilityofthenervetocarryimpulses;

    5. Astheconditionworsensduetosystemicdegenerationandtoxinaccumulatinginthe

    nervesheath(cellmembrane),intracellular

    potassiumdeficiencyaccumulates;

    6. Resultinginacontinuousdeteriorationofneuralfunction.

    7. Also,resultingincellmembranepotentialfallingbelowthetriggerthresholdforTNFactivation;

    8. Bcellsinitiateimmuneresponseontaggedcellscausingdestructionofthesecells

    9. Acceleratingneuraldestructioninadvanceddiseasestages.

    LupusErythematosus

    1. Agroupofcellsbecomeweakeneddueage,stressorotherenvironmentalfactors;

    2. Resultinginadisruptedanabolicenergyproduction;

    3. ResultingincellmembranepotentialfallingbelowthetriggerthresholdforTNFtriggering

    Bcellsinitiateimmuneresponseontaggedcells

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    WholeHealthResearchAlliance MarkSquibb P a g e |13

    AutoimmuneCofactorCandidates

    Cofactor Role Supports

    CellMembraneIntegrity Definestheabilityforcellsto

    maintainelectricalintegrity.

    Allhormonalandelectricalintegrity

    ofthecell.

    Intra/ExtracellularpH

    differential

    Createsenergyforcellmembrane

    functions,hormonereceptors,and

    autoimmunecellevaluation.

    Autoimmunesensingofdiseased

    cells.

    AmbientAutoimmuneLoad Determinestheresourcesavailableto

    supportnormalautoimmune

    performance.

    Autoimmuneresourcestoavailable

    toeradicatestealthandother

    pathogens.

    StealthPathogenPrevalence Absorbsautoimmuneresources.

    1,25dihydroxyvitaminDin

    responsetointracellular

    bacteria

    Describesmaskingmechanismused

    byintracellularbacteria/mycoplasma

    evadeautoimmuneeradication.

    SeeMarshallprotocol

    ForProtocolInformationContactWholeHealthResearchAlliance,LLC,9703724344,oremail:[email protected]

    http://www.marshallprotocol.com/forum32/1263.htmlhttp://www.marshallprotocol.com/forum32/1263.htmlhttp://www.marshallprotocol.com/forum32/1263.htmlhttp://www.marshallprotocol.com/forum32/1263.htmlhttp://www.marshallprotocol.com/forum32/1263.htmlmailto:[email protected]:[email protected]:[email protected]://www.marshallprotocol.com/forum32/1263.html