nargess tavakoli
DESCRIPTION
alopecia areataTRANSCRIPT
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Alopecia Areata
Nargess Tavakoli
Guilan university of medical sciences
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• A nonscarring type of alopecia
• Many different patterns
• The true cause ?
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HistoryAcute onset
can affect any hair-bearing area
scalp
Sigle/multiple
Itch?
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Physical Exam
• Alopecia pattern in AA?
• Other findings?
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Pattern of alopecia
• Patchy• Ophiasis• sisaipho (ophiasis spelled
backwards) • diffuse
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Ophiasis
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Alopecia Totalis
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AT
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Alopecia universalis
complete loss of hair on all hair-bearing areas
Rare
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Nail involvement
• Pitting
• Several other abnormalities have been reported
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Epidemiology• Prevalence: 0.1-0.2% in the
general population• Sex: F = M• Age: can occur at any age / more
common before 40yo,esp. childhood & adolescence
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physiopathology
• AA progresses as a wave of follicles which enter telogen prematurely
• Autoimmune/T_cell mediated/genetic
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Etiology
• The true cause: unknown
• autoimmune?• Genetic?• Infection?• Emotional stress?
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Evidence supports the autoimmune hypothesis
• Other autoimmune disease
• Changes in Immune system
(cellular/homoral) • Experiments on mice
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Genetic
• Familial? 4_27%• AD?• HLA• Atopia• Down’s synd.
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•Infection? CMV?
•Emotional stress?
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Diagnosis•Clinicaly•Biopsy?•Thyroid tests?
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ChronicRegrowth(+/- therapy) usuallyHow much does it take?
Recurrence may occur
Prognosis
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White hair regrowth in alopecia areata.
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Adverse prognosis factors
onset at a young age severe forms + FH of AAnail dystrophy Ophiasis
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Treatment
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Treatment
• No medication?• Local steroidsatrophy• Antralen• Systemic steroids• Irritant dermatitis• False hair to cover?
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