(ncm 104)ortho sir pat lao 2

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  • 7/28/2019 (NCM 104)Ortho Sir Pat Lao 2

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    1111111

    http://www.nysora.com/techniques/advanced/sciatic/000190.jpghttp://www.nysora.com/techniques/advanced/sciatic/000187.jpghttp://www.nysora.com/techniques/advanced/sciatic/000181.jpg
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    Preoperative

    Offer support/encouragement Discuss:

    Rehabilitation program & use of prosthesis

    Upper extremity exercise such as push ups in bed

    Crutch walking

    Amputation dressing/cast Phantom limb sensation as a normal occurrence

    Observe stump dressing for signs of hemorrhageand mark outside of dressing so rate of bleedingcan be assessed (tourniquet at bedside)

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    Prevent edema

    Raise extremity with pillow support for first 24 h Prevent hip/knee contractures

    Avoid letting patient sit in chair with hips flexedfor long periods of time

    Have patient assume prone position several timesa day and position hip on extension Avoid elevation of stump after 24 hrs For BKA: hip & knee exercises For AKA: hip exercises

    Pain medication as ordered (phantom limbpain)

    Ensure that stump bandages fit tightly andare applied properly to enhance prosthesisfitting

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    Rheumatoid arthritis chronic systemic

    inflammatory disease

    destruction of connectivetissue and synovial

    membrane within thejoints

    weakens and leads todislocation of the jointand permanent deformity

    Risk Factors exposure to infectious

    agents

    fatigue

    stress

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    Signs and Symptoms Morning stiffness Fatigue Weight loss Joints are warm,

    tender, and swollen

    Swan neckdeformity-lateDiagnostic Studies X-ray Elevated WBC,

    platelet count, ESR*,and positive RFTreatmentNo cure for RA

    Swan neck deformity

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    Aspirin- mainstay of treatment, has bothanalgesic and anti-inflammatory effects

    Nonsteroidal anti-inflammatory drugs(NSAIDs): Indomethacin (Indocin) Phenylbutazone (Butazoldin) Ibuprofen (Motrin) Fenoprofen (Nalfon) Naproxen (Naprosyn) Sulindac (Clinoril)

    Immunosuppressives: Methotrexate Gold Standard for RA treatment Teratogenic

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    Gold compounds

    Injectable form: sodium thiomalate,aurothioglucose; given IM once a week;

    takes 3-6 months to become effective Oral form: auranofin- smaller doses are

    effective; diarrhea is a common sideeffect

    Corticosteroids

    Intra-articular injections

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    Treatment Surgical Procedures: synovectomy,

    arthrotomy, arthrodesis, arthroplasty

    Nursing Management Advised bed rest during acute pain Passive ROM exercise of joints Splint painful joints Heat & Cold application Advised warm bath in the morning Protect from infection Advised well-balanced diet

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    Arthrotomy

    Arthrodesis Arthroplasty

    http://www.ortho-u.net/images/tssr30.jpg
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    Progressivedegeneration of thejoints as a result ofwear and tear affects weight-bearing joints andjoints that receive the

    greatest stress, suchas the knees, toes,and lower spine.

    http://rds.yahoo.com/S=96062883/K=Osteoarthritis+/v=2/l=IVI/*-http://www.arthritis-glucosamine.net/graphics1/cartilage.gifhttp://rds.yahoo.com/S=96062883/K=Osteoarthritis+/v=2/l=IVI/*-http://www.thirdage.com/health/adam/images/ency/fullsize/8882.jpg
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    Risk Factors aging (>50 yr) rheumatoid arthritis arteriosclerosis obesity trauma family historySigns and Symptoms Dull, aching pain,* tenderjoints fatigability, malaise crepitus cold intolerance* joint enlargement presence of Heberdensnodes or Bouchardsnodes weight loss

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    Aspirin inhibits cyclooxygenase enzyme, diminishes the formation

    of prostaglandins anti-inflammatory, analgesic, antipyretic action inhibit platelet aggregation in cardiac disordersAdverse effects Epigastric distress, nausea, and vomiting In toxic doses, can cause respiratory depression Hypersensitivity Reyes syndrome

    Ibuprofen use for chronic treatment of rheumatoid and osteoarthritis less GI effects than aspirinAdverse effects dyspepsia to bleeding headache, tinnitus and dizziness

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    Indomethacin inhibits cyclooxygenase enzyme

    more potent than aspirin as an anti-inflammatory agent

    Adverse effects: nausea, vomiting, anorexia, diarrhea

    headache, dizziness, vertigo, light-headedness, and mental confusion

    Hypersensitivity reaction

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    Nursing Intervention Promote comfort: reduce pain, spasms,

    inflammation, swelling Heat to reduce muscle spasm Cold to reduce swelling and pain

    Prevent contractures: exercise, bed rest on

    firm mattress, splints to maintain properalignment Weight reduction Isometric and postural exercisesNursing Diagnosis Pain related to friction of bones in joints Risk for injury related to fatigue Impaired physical mobility related to stiff,

    limited movement

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    Metabolic disorder that

    develops as a result ofprolonged hyperuricemia

    Caused by problems insynthesizing purines or bypoor renal excretion of uricacid.

    Acute onset, typicallynocturnal and usuallymonarticular, often involving

    the first metatarsophalangealjointRisk Factors Men

    Age (>50 years)

    Genetic/familial tendency

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    Signs and Symptoms extreme pain swelling erythema of the

    involved joints fever

    TophiLaboratory Findings elevated serum uric

    acid (>7.0 mg/dl)* urinary uric acid elevated ESR and WBC crystals of sodium

    urate aspirated from atophus confirms thediagnosis*

    http://rds.yahoo.com/S=96062883/K=Tophi/v=2/l=IVI/*-http://meded.ucsd.edu/isp/1994/im-quiz/images/tophi.jpghttp://rds.yahoo.com/S=96062883/K=Tophi/v=2/l=IVI/*-http://www.hopkins-arthritis.som.jhmi.edu/other/images/gout_fig7.gif
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    Allopurinol- a purine analog

    - reduces the production of uric acid by competitively inhibiting uricacid biosynthesis which are catalyzed by xanthine oxidase.

    Effective in the treatment of primary hyperuricemia of gout andhyperuricemia secondary to other conditions (malignancies).

    Adverse effects: hypersensitivity reactions, nausea and diarrhea

    Colchicine Effective for acute attacks Anti-inflammatory activity alleviating pain within 12 hours Adverse effects: nausea, vomiting, abdominal pain, diarrhea,

    agranulocytosis, aplastic anemia, alopecia

    Probenecid/Sulfinpyrazone uricosuric agents increases the renal excretion of uric acid Sulfinpyrazone used as a preventive agent. Adverse effects: nausea, rash & constipation

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    Maintain a fluid intake of at least 2000 to 3000 ml a day to avoidkidney stone. Avoid foods high in purine such as wine, alcohol, organ meats,sardines, salmon, anchovies, shellfish and gravy. Take medication with food. Have a yearly eye examination because visual changes can occurfrom prolonged use of allopurinol Caution client not to take aspirin with these medication because itmay trigger a gout attack and may cause an elevated uric acidlevels. Encourage rest and immobilize the inflamed joints during acuteattacks Avoid excessive alcohol intake Notify physician if rash, sore throat, fever or bleeding develops.

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    COMPARING ARTHRITIS

    Rheumatoid Osteoarthritis Gouty

    Etiology Autoimmune

    + Rh factor

    Degenerative

    senescence

    Metabolic orfamilialpurinemetabolism

    Incidence 35-45 women Men or more inwomen

    Men over 40

    Signs andsymptoms

    Subcutaneaousnodules

    Morning stiffness

    Swan neckdeformity

    Heberdens nodule Tophi

    Areasaffected

    Joints of hands Weight bearingjoint

    Great toe

    Management

    Aspirin, NSAIDs

    Paraffin bath

    Symptomatic Colchicine

    Avoid purine

    dietAllopuyrinol

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    Bacterial Infection ofthe bone and softtissue

    Staphylococcus aureus

    is the most commonpathogen. Hemolytic

    streptococcus

    Other organismsinclude Proteus,Pseudomonas and E.Coli

    http://rds.yahoo.com/S=96062883/K=Osteomyelitis/v=2/l=IVI/*-http://www.nlm.nih.gov/medlineplus/ency/images/ency/fullsize/9712.jpg
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    Risk Factors

    poorly nourished, elderly orobese

    impaired immune systems

    chronic illnesses

    long term corticosteroid therapy

    Open wound

    infection

    http://rds.yahoo.com/S=96062883/K=Osteomyelitis/v=2/l=IVI/*-http://www.microsurgeon.org/images/osteomyelitis_debridement.jpghttp://rds.yahoo.com/S=96062883/K=Osteomyelitis/v=2/l=IVI/*-http://www.microsurgeon.org/images/osteomyelitis.jpg
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    Organism reaches the bone throughan open wound

    Infection causes bone destruction

    Bone fragment necrose -sequestra

    New bone cells form over thesequestrum during healing

    resulting in nonunion

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    Clinical Manifestation area appears warm, swollen and

    extremely painful ( localized edema)

    systemic manifestations (fever,chills, tachycardia) Bone pain Muscle spasm

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    Diagnostic Studies X-ray

    Bone Scan Blood and wound culture

    Bone Biopsy

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    1. Antibiotics : Cefazolin and Clindamycin

    2. Analgesic : Oxycodone,

    1. 1015 mg of oxycodone produces ananalgesic effect similar to 10 mg of morphine

    3. Vital Signs and Neurovascular status

    4. Diet : High calorie, vitamin C and DCalcium

    5. Activity : bed rest6. Heat therapy

    7. Antipyrectic : aspirin, acetaminophen

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    1. Monitors the neurovascular status of the affectedextremity

    2. Elevation reduces swelling and associateddiscomfort

    3. Pain is controlled with prescribed analgesics andother pain-reducing techniques

    4. Must be protected by immobilization devices andavoidance of stress on the bone

    5. The patient must understand the rationale of forthe activity of restriction

    6. Encourage patient to have a full participation inADLs within the physical limitations to promotegeneral well-being

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    Monitor the patients response to antibiotic therapy

    Observes the IV access for evidence of phlebitis, infection, orinfiltration,

    With long term intensive antibiotic therapy (monitors thepatient for sign of infection like oral or vaginal candidiasis,loose or fouling-smelling stool

    If surgery is necessary (take measures to ensure adequatecirculation to the affected area (wound suction to prevent

    accumulation, elevation of the area to promote venousdrainage, avoidance of pressure on the grafted area) tomaintain needed immobility, and to ensure the patientsadherence of to weight bearing restrictions.

    Changes dressings using aseptic technique (to promote

    healing and to prevent cross-contamination

    Diet high in protein and Vitamin C (promotes a positivenitrogen balance and healing

    Encourage adequate hydration as well

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    Promote comfort

    Immobilized affected bone by maintainingsplinting. Elevate affected leg Administer analgesics as needed.Control infectious process Apply warm, wet soaks 20 min. several

    times a day. Administer antibiotics as prescribed. Use aseptic technique when dressing the

    wound.Encourage participation in ADL within thephysical limitations of the patient.

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    a reduction in bone density and a change inbone structures

    bones become previously porous, brittle and

    fragile

    bones fracture easily under stresses thatwould not break in normal bone

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    Incidence- women older than 80 years old of age is 84%.

    The average 75 years-old woman has lost 25%of her cortical bone and 40% of her trabecular

    bone.

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    Age1. Post-menopause

    2. Advance age

    3. Low testosterone in men

    4. Decreased calcitonin

    Nutrition1. Low calcium intake

    2. Low vitamin D3. High phosphate intake

    4. Inadequate calories

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    Physical exercise Sedentary

    Lack of weight-bearing exercise Low weight and body mass index

    Lifestyle choices Caffeine

    Alcohol Smoking

    Lack of exposure to sunlight

    Medications

    Corticosteroids

    Antiseizure medications Heparin

    Thyroid Hormone

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    Back pain.

    Loss of height and stooped posture.

    A Curved upper back (dowager's hump). Fracture : hip, spine and wrist.

    Compression fractures

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    1. Primary Osteoporosis : women after menopause (usually after 45 and 55

    years) later in men.

    2. Secondary Osteoporosis medications or other conditions and disease that

    affect bone metabolism.

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    Predisposes to low bone

    mass

    Genetics-Caucasian or Asian-Females

    -Family history-Small frameAge-Post-menopause-Advance age-Low testosterone in men-Decreased calcitoninNutrition-Low calcium intake-Low vitamin D-High phosphate intake-Inadequate calories

    Hormones

    (estrogen, calcitonin,and testosterone)inhibit bone loss.

    Reduces nutrients

    needed for the boneremodeling

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    Physical Exercise-Sedentary-Lack of weight-bearing exercise-Low weight and body mass indexLifestyle Choices-Caffeine-Alcohol-Smoking-Lack of exposure to sunlightMedications-Corticosteroids-Antiseizure medications-Heparin-Thyroid Hormone

    Reducesosteogenesis inbone remodeling

    Affects calciumabsorption andmetabolism

    Bones needed

    stress for bone

    maintenance.

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    Dual-energy X-ray Absorptiometry (DEXA)-Means of measuring bone mineral densities

    (BMD)

    Two xray beams with differing energy levels are

    aimed at the patient bones.X ray. Serum calcium Serum phosphataseUrine calcium excretion

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    1. Hormone replacement therapy (HRT)1. Raloxifene (Evista) preserving BMD w/o

    estrogenic effect on the uterus ( prevention and

    treatment for osteoporosis

    2. Bishosphonates

    3. Alendronate - inc. bone mass by inhibiting

    osteoclast function.

    4. Calcitonin

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    Nursing Diagnosis

    1. Deficient knowledge about the osteoporotic

    process and treatment regimen

    2. Acute pain related to fracture and musclespasm

    3. Risk for constipation related to immobility or

    development of ileus

    4. Risk for injury:additional fractures related to

    osteoporosis

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    Nursing Inteventions

    Promoting and Understanding of Osteoporosis and the

    Treatment Regimen

    1. Adequate dietary or supplemental calcium (1200-

    1500 mg/day) and vitamin D

    2. Regular weight-bearing exercise

    3. Modification of lifestyle like cessation of smoking,

    reduce the use of caffeine and alcohol4. Help to maintain bone mass

    5. Instruct to take the calcium supplements with meals

    6. Teach patient to drink adequate fluids to reduce the

    risk of renal calculi7. For Alendronate users, it must be taken on an empty

    stomach with water and the patient must not

    consume foods or liquids for 30-60 minutes.

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    Relieving Pain1. Rest in bed in a supine position or side-

    lying position several times a day. Themattress should be firm and non-sagging.

    2. Intermittent local heat and back rubspromote muscle tension

    3. Instruct to move the trunk as a unit andavoid twisting.

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    primary malignant neoplasm of bones

    Also known as Osteogenicsarcom

    Occurs between 10-25 years of age, with Paget's

    disease and exposure to radiation.

    Exhibits a moth-eaten pattern of bone destruction.

    Most common sites: metaphysis of long bones

    especially the distal femur, proximal tibia andproximal humerus.

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    Risk Factors

    Cause is unknown. family history / inherited cancers :

    Li-Fraumeni SyndromeRetinoblastoma

    being tall for specific agePrevious treatment with radiation for another cancer

    That usually occurs in children younger than 4 yrs.

    P h h i

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    1.Osteosarcoma occurs mainly in the metaphyses oflong bones, sites of active epiphyseal growth.Distal femurProximal tibiaProximal humerus1.As a tumor of mesenchymal cells, osteosarcoma

    demonstrates production of osteoid cells.2.It is a bulky tumorthat extends beyond the bone the

    bone into a soft tissue.3.This may encircle the bone and destroy the

    trabeculae of affected area.4.Osteosarcoma disseminates through bloodstream,usually to the lung.

    5.Other sites of metastatic spread include otherbones and visceral organs

    Pathophysiology:

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    High grade mesenchymaltumorDistal femurProximal tibia

    Proximal humerusFormation of

    osteoid

    Lungs, bones,visceral organs

    Metastasize throughblood streams

    Bulky tumor thatdestroys trabeculaeof diseased area

    Predisposing Factors

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    Clinical Manifestation local signs pain ( dull,

    aching and intermittent innature), swelling, limitationof motion

    palpable mass near the endof a long bone

    systemic symptoms:

    malaise, anorexia, andweight loss

    Diagnostic Findings Biopsy- confirms the

    diagnosis

    X-ray MRI Bone Scan Increase alkaline

    phosphatase

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    Radiation Chemotherapy (methotrexate(leucovorin/Adriamycin) Cytoxan, Ifosfamide Surgical management

    amputation limb salvage procedures

    Prognosis: poor prognosis (rapid growth rate)

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    Promote understanding of the diseaseprocess and treatment regimen

    Promote pain relief Prevent pathologic fracture Assess for potential complications(infection, complications of immobility). Encourage exercise as soon as possible(1st or 2nd post-op day)

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    a plastic surgery

    that involvesremoval of thehead of the femurfollowed by

    placement of aprosthetic implant

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    Teach client how to use crutches

    Teach client mechanics of transferring.

    Discuss importance of turning and positioning post-op.

    Place affected leg in an abducted position and straightalignment following surgery

    Prevent hip flexion of more than 90 degrees.

    Apply support stockings

    Advise client to avoid external/internal rotation of affectedextremity for 6 months to 1 year after surgery

    Instruct client to avoid excessive bending, heavy lifting,jogging, jumping

    Encourage intake of foods rich in Vitamin C, protein, and

    iron. Administer prescribed medications.

    Metallic implant

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    Infection Hemorrhage Thrombophlebitis Pulmonary embolism Prosthesis dislocation Prosthesis loosening

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    condition in which

    the head of thefemur is

    improperly seatedin the acetabulum,or hip socket, ofthe pelvis.

    Congenital ordevelop after birth

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    Neonates: laxity of theligaments around thehip, allowing thefemoral head to bedisplaced from theacetabulum uponmanipulation.

    Implementation: Splinting of the hipswith Pavlik harness tomaintain flexion andabduction and externalrotation (neonatalperiod) Pavlik harness

    http://images.google.com/imgres?imgurl=www.musckids.com/health_library/orthopaedics/images/1harnessff.gif&imgrefurl=http://www.musckids.com/health_library/orthopaedics/ddh.htm&h=500&w=530&prev=/images%3Fq%3DDysplasia%2Bof%2BHip%26start%3D20%26svnum%3D10%26hl%3Den%26lr%3D%26ie%3DUTF-8%26oe%3DUTF-8%26sa%3DN
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    Infants Asymmetry of the gluteal and

    thigh skin folds when thechild is placed prone and thelegs are extended against theexamining table. Limited range of motion inthe affected hip. Asymmetric abduction of theaffected hip when the child isplaced supine with the kneesand hips flexed. apparent short femur on theaffected side

    http://images.google.com/imgres?imgurl=www.orthoweb.be/brochures/pediatric/images/pavlik.jpg&imgrefurl=http://www.orthoweb.be/brochures/pediatric/pavlik_uk.htm&h=244&w=163&prev=/images%3Fq%3DDysplasia%2Bof%2BHip%26start%3D20%26svnum%3D10%26hl%3Den%26lr%3D%26ie%3DUTF-8%26oe%3DUTF-8%26sa%3DN
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    Implementation Traction and/or

    surgery to releasemuscles andtendons

    Following surgery,positioning and

    immobilization in aspica cast untilhealing is achieved.

    http://rds.yahoo.com/S=96062857/K=+spica+cast/v=2/SID=w/l=II/R=3/*-http://images.search.yahoo.com/search/images/view?back=http%3a//images.search.yahoo.com/search/images%3fsrch=1%26p=%2bspica%2bcast%26ei=UTF-8%26n=20%26fl=0&h=231&w=428&imgurl=showcase.netins.net/web/eulemaraja/img8.jpg&name=img8.jpg&p=+spica+cast&rurl=http://showcase.netins.net/web/eulemaraja/page5.html&type=&no=3&tt=65http://rds.yahoo.com/S=96062857/K=+spica+cast/v=2/SID=w/l=II/R=1/*-http://images.search.yahoo.com/search/images/view?back=http%3a//images.search.yahoo.com/search/images%3fsrch=1%26p=%2bspica%2bcast%26ei=UTF-8%26n=20%26fl=0&h=719&w=899&imgurl=www.ucsf.edu/orthopaedics/patientedu/img/ddh4.jpg&name=ddh4.jpg&p=+spica+cast&rurl=http://www.ucsf.edu/orthopaedics/patientedu/peds_ddh.html&type=&no=1&tt=65
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    The walking child minimal to

    pronouncedvariation in gait with

    lurching toward theaffected side;positiveTrendelenburg sign

    Positive Barlow orOrtolanis maneuverOrtolanis maneuver Barlow maneuver

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    Caring forPatient with

    http://www.gamasutraexchange.com/Previews/Content_on_10_8_2001_16_16_50%5Cbone_thumb.jpgDD4B6947-A91F-4B2A-8E420366AA969116.jpgLarge.jpg
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    Osteomalaciainvolves softeningof

    the bones caused bya deficiency of vitamin Dor problems with themetabolism of this

    vitamin.

    http://www.capitolmarket.net/photosh1/bone.jpghttp://www.capitolmarket.net/photosh1/bone.jpg
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    In children, the

    condition is calledrickets and isusually caused by adeficiency of

    vitamin D .

    http://www.capitolmarket.net/photosh1/bone.jpghttp://www.capitolmarket.net/photosh1/bone.jpg
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    In adult, the conditionis usually caused by:

    1. Inadequate dietaryintake of vitamin D

    2. Inadequate exposureto sunlight (ultravioletradiation)

    3. Malabsorption ofvitamin D

    http://www7.nationalacademies.org/germanbeyonddiscovery/VitaminD_7-2.jpghttp://www.msd.com.hk/images/health_info/disease_info/osteoporosis/e_picture6b_1a.gifhttp://www.capitolmarket.net/photosh1/bone.jpghttp://www.capitolmarket.net/photosh1/bone.jpg
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    Other conditions:1. Hereditary or acquired disorders

    of vitamin D metabolism

    2. Kidney failure and acidosis ,3. PO4 depletion associated with

    low dietary intake or kidneydisease

    4. Side effects ofmedications usedto treat seizures .

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    Risk factors are related tothe causes.

    In the elderly, there is anincreased risk for those whotend to remain indoors andwho avoid milk because oflactose intolerance

    The incidence is 1 in 1000people.

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    Deficiency of activated VITAMIN D

    (Calcitriol)

    Decreased absorption, malabsorption or excessive loss ofCALCIUM

    Inadequate

    MINERALIZATION of bone

    SOFTENING of the

    BONE

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    diffuse bone pain , especially in the hips muscle weakness

    symptoms associated with low calcium1. numbness around the mouth & of

    extremities2. Carpopedal spasms3. Bowing of legs4. Waddling or limping GAIT5. Decrease in height/ Spinal Deformities (i.e.

    KYPHOSIS)

    k

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    In children, symptoms ofricketsinclude:

    delayed sitting, crawling, and walking; pain whenwalking; and the development ofbowlegs orknock-

    knees.

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    1. Bone biopsy: (+) increase in osteoid

    2. Bone X-ray or CT scan of lumbosacral spineshows demineralization.

    3. Studies of the vertebrae: (+) compression fx

    4. Low serum vitamin D level

    5. Low serum calcium &phosphate levels

    6. Elevated ALP (Alkaline Phosphatase)

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    1. Adequate dietaryintake of dairy

    products that arefortified withvitamin D

    2. Adequate exposure

    of the body tosunlight

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    Oral supplementsof vitamin D ,calcium, and

    phosphorus

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    Large doses of Vitamin Dwith exposure to sunlightmay be indicated inpeople with intestinal

    malabsorption .

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    Monitoring of blood levelsofphosphorus andcalcium may be indicatedwith some underlying

    conditions. Braces or surgery to

    correct deformities

    30

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    30

    Protrusion of the nucleus of the disk into thefibrous ring of the disk with subsequent nervecompression

    May occur in any portion of the vertebralcolumn

    Signs & Symptoms1. Pain

    2. Sensory changes3. Loss of reflex

    4. Muscle weakness

    77

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    30

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    1. Cervical Pain/ Stiffness head, neck & upper extremities Paresthesia, numbness

    Weakness

    2. Lumbar Low back pain radiating to the buttocks and leg Postural deformity of the spine

    (+) Straight-Leg Raise test

    Weakness & Asymmetric reflexes

    Sensory loss

    Nursing Alert: Perform repeated assessments ofsensorimotor

    functions/ reflexes to determine progression of condition80

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    30

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    30

    Alleviating pain1. Anti-inflammatory drugs, muscle

    relaxants, and narcotic analgesics2. Use ofbed boards under the mattress3. Bed restsupine or low fowlers or side

    lying position with slight knee flexion andpillows between knees.

    4. Moist heat application5. Relaxation techniques

    Nursing Interventions

    82

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    Surgical incision of part of posterior arch ofvertebrae and removal of protruded disc Nursing InterventionPreoperative

    Teach patient log rolling and use of bedpanPostoperative Position as ordered Lower spinal surgery- flat Cervical spine surgery: slight elevation of

    head of bed Proper body alignment- cervical spinal

    surgery: avoid flexion of neck and applycervical collar

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    Avoid: Acute hip flexion (bending, stooping, crossing the

    legs

    Prolonged sitting/standing

    Running, jogging, horseback riding Back- strengthening exercises

    Prone position

    Walk in seawater

    Lie in side- lying with hip flexion

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    Patient teaching and Discharge Planning Wound care

    Good posture and proper body mechanics

    Activity level as ordered

    Recognition and reporting of complications such aswound infection, sensory or motor deficits

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