ocular trauma mohamad abdelzaher msc. epidemiology 40% of monocular blindness is related to trauma...
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Ocular TraumaMohamad Abdelzaher
MSc
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Epidemiology 40% of monocular blindness is related to trauma
The leading cause of monocular blindness 70-80% injured are males Age: more in children and young age group Incidence of penetrating eye injuries: 3.6/100000 Occupation : construction, industry Sports : boxing , racket sports Motor vehicle accidents
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Classification
Ocular Injuries
Mechanical
Blunt Perforating
Chemical
Thermal Physical
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Assessment
Rule out life threatening injuriesRule out globe threatening injuriesExamine both eyesImagePlan for treatment
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Blunt ocular trauma
Direct force “coop”
Shock waves “counter-coop”
Antero-posterior compression & equatorial expansion
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Orbital blow-out fractures
Fracture Floor
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• Periocular ecchymosis and oedema• Infraorbital nerve anaesthesia
• Ophthalmoplegia - typically in up and down gaze (double diplopia)
Enophthalmos - if severe
Signs of orbital floor blow-out fracture
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Lid
Black “raccoon” eye Lid laceration
Traumatic ptosis Surgical emphysema
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Conjunctiva
Subconjunctival hemorrhage Conjunctival laceration
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Cornea
Corneal abrasion Corneal FB
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Corneal rupture with iris prolapse
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Sclera
Scleral rupture
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Anterior Chamber
Hyphema
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Iris
Iridodialysis
Aniridia
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Ciliary Body
Angle recession
IridocyclitisCB shut down“Hypotony”
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Lens
DislocationCataract
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Vitreous
Vitreous hemorrhage
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Choroid
Choroidal rupture Choroidal detachment
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Retina
Commotio retinae
Retinal detachment
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Penetrating injuries• Without FB retention:- Cut wound: lid, conjunctiva, cornea, …- Infection: corneal, endo, panophthalmitis- Sympathetic ophthalmitis
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• With Intraocular FB:
Mechanical Infection FB reaction
Cut wound: lid, conjunctiva, cornea, …
corneal, endo, panophthalmitis
- Non specific
- Siderosis bulbi “Fe”
- Chalcosis bulbi “Cu”
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Kayser Fleischer’s ring Sunflower cataract
Chalcosis bulbi
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Cemical Ocular InjuriesMost chemical substances that come in contact with the
conjunctiva or cornea cause little harm.
The chief danger comes from alkali-containing compounds found in household cleaning fluids, fertilizers and pesticides. They erode and opacify the cornea.
Acid-containing compounds (battery fluid, chemistry labs) are
somewhat less dangerous.
There are no antidotes to these chemicals. The best you can do is to dilute them immediately with plain water.
The resultant reaction of the tissue causes the damage.
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Treatment should be instituted immediately, even before testing vision.
Emergency treatment:1-copious irrigation of the eyes,
preferably with saline or ringer lactate.
Don’t use acidic solutions to neutralize alkalis or vice versa.
Pull down the lower eyelid and evert the upper eyelid to irrigate the fornices
2-irrigation should be continued until neutral PH is reached.
Except lime
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For mild to moderate burns (during and after irrigation):
• cycloplegic• topical antibiotic• oral pain medication• if increase IOP use drugs to reduce it (acetazolamide,
methazolamide add b blocker if additional IOP control is required)
• frequent use of preservative free artificial tear
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For severe burns (Treatment after irrigation):• Admission to the hospital• Lysis of conjunctival adhesion• Debride necrotic tissue • Topical antibiotic • Topical steroid• Antiglaucoma medication if the IOP is increased or cant be
determined• Frequent use of preservative free artificial tear• Other consideration: Therapeutic contact lenses, collagen, amniotic membrane
transplant IV ascorbate and citrate for alkali burns If any melting of the cornea occurs, collagenase inhibitors
may be used If the melting progresses an emergency patch graft or corneal
transplat may be necessary.
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A hazy cornea following an alkali burn
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Physical InjuriesGlass blower Iron melter Arc flash
Infra Red
Cataract Photophthalmia
UVR
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• Punctate epithelial erosions “photophthalmia”
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