powerpoint presentation · 2021. 3. 1. · napier jr. the prehensile movements of the human hand. j...
TRANSCRIPT
3/1/2021
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The Finger Joints
Learning Objectives
At the completion of the course, the learner will be able to
Describe the detailed anatomy of the MP and IP joints
Interpret imaging studies on a basic level
Describe intra-articular, capsular, and tendinous pathologies affecting the digits
Collect a good history and examination for a wide variety of digital joint problems
Communicate effectively with patients and other health care providers regarding digital conditions and treatments
METHODS
• 15 lectures
• question and answer sessions: The DD Forum• Second Saturday of every month in 2021, 8:00-8:45 am Pacific Time
• Find Zoom links on www.doctorsdemystify.com home page
• some repetition between talks
bad news: unavoidable because of multiple speakers
good news: repetition is a good learning tool
Finger Joint Anatomy
Roy A. Meals, MD
MP Joint:Bony Anatomy
• Metacarpal head• Nearly circular when
viewed from side• Wider anteriorly
when viewed end on• Proximal phalanx base
• nearly elliptical• faintly concave
• No bony stability
MP JointLigament Anatomy
• Dorsally: just capsule and tendon, no ligament
• Volar plate
• Thick distally, securely attached to P1• Thin proximally, attached to metacarpal
Volar plate
extensionflexion
MP JointLigament Anatomy
• Volar plate
• Sesamoid bone(s), especially thumb• Mini patellas• Common at 1st MTP jt in foot
• Patients see and think they are pathological
• Location differentiates between simple and complex MCP dislocations
• Rarely fracture, develop arthritis, cause joint locking
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MP JointLigament Anatomy
• Dorsally: just capsule and tendon, no ligament
• Volar plate
• Collateral ligaments
• origin on mc neck, insert on P1• tight in flexion, lax in extension
MP JointLigament Anatomy
• Dorsally: just capsule and tendon, no ligament
• Volar plate
• Collateral ligaments
• Accessory collateral ligaments
• origin on mc neck, insert on volar plate
Video clip: MP joint ligaments
Interphalangeal JointsBony Anatomy
• Head of proximal phalanx: bicondylar
• Base of middle phalanx: oval, faint adaptation to bicondylar nature of proximal phalanx
• No bony stability
• Same at DIP joints
Interphalangeal JointsLigament Anatomy
Identical to MP joint except:
• Volarly
• Less laxity in thin, proximal portion (reduces hyperextensibility)
• Medially and laterally
• Collateral ligaments—some portion is tight in any degree of flexion (no medial/lateral motion)
Video clip, PIP joint ligaments
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Extrinsic Flexor Tendons(extrinsic = originates in forearm)
Sole flexors of IP joints, 2o flexors of MP joints
• FDS
• Superficial in forearm and palm• Splits and wraps around FDP to insert on P2
• FDP and FPL
• Analogous deep origins, course and insertions
• Annular pulleys
• Restrain tendonsVideo clip, flexor tendon sheath
Extensor Mechanism: A complex interweave of tendon fibers controlled by 4 or 5 muscles
Terminal
tendon
Central
slipConjoined
tendon
Lateral band
Extrinsic
Extensor (1-2)
Interossei (2)
Lumbrical (1)
MP Joint Extension
• Solely performed by extrinsic extensors
• Thumb: epb and epl• Index: eip and edc• Middle: edc• Ring: edc• Small: edc and edq
PIP and DIP JointExtension
Working in concert:
• Extrinsic extensors• Intrinsic muscles
• Interosseous muscles• Lumbrical muscles
• Landsmeer’s oblique retinacular ligaments
Review each……
Intrinsic Muscles
• Interossei• Insertion #1: P1
bases: flex and abduct/adduct MP joints
• Insertion #2: dorsal mechanism: flex MP joints and extend PIP/DIP joints
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Intrinsic Muscles
• Lumbricals
• Insert into dorsal mechanism on radial side: flex MP joints and extend PIP/DIP joints
Landsmeer’sOblique Retinacular Ligament
O.R.L.
Passes volar to center of rotation ( ) at PIP joint and
dorsal to center of rotation at DIP joint, therefore
tightens during PIP joint extension and assists with
DIP joint extension
lateral band
MP joints PIP joints DIP joints
extrinsic
flexors
2o flexor sole flexor sole flexor
extrinsic
extensors
sole extensor 2o extensor 2o extensor
intrinsics 1o flexor 1o extensor 1o extensor
Overview of Finger Joint E/FDorsal Tendon Restraints
• Sagittal bands: centralize extrinsic
• extensors at MP joint
• Transverse retinacular ligament (Landsmeer): control conjoined tendon at PIP joint
Video clip: dorsal mechanism
Reading
Wise, K: The anatomy of the mp joints, with observations on the etiology of ulnar drift. J Bone Joint Surg 1975, 57B:485
Gad, P: The anatomy of the volar part of the capsules of the finger joints. J Bone Joint Surg 1967, 49B:362
Kuzcynski, K: The proximal interphalangeal joint. Anatomy and causes of stiffness in the fingers. J Bone Joint Surg 1968, 50B:656
Gigis, P and Kuczynski, K: The distal interphalangeal joints of the human fingers. J Hand Surg 1982, 7:176
Pang EQ et al: Anatomy and Biomechanics of the Finger Proximal Interphalangeal Joint. Hand Clin. 2018 May;34(2):121-126
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Hand Surgeons Providing Excellence in EducationforOccupational and Physical Therapists
www.doctorsdemystify.com
The Finger Joints
Examination and Functional Requirements
Mark Elzik, MD
Mission Viejo, CA
Introduction
- Basic examination:- History, history, history
- Inspection, Palpation, Neurovascular exam, Range of motion
- Normal variants
- Functional requirements
History, History, History
- When you ask the right questions, patient will give the results of his/her exam, for instance:
- “Are you able to fasten buttons?”- Requires median nerve sensibility
- “Is your finger stiff in the morning and does it loosen up under warm water?”
- Check closely for trigger finger
- “Does it hurt when you open jars?”- In patient over 45, suggests basal thumb OA
Inspection
• “Attitude” of the hand:• MCP and IP joints are in
position of slight flexion
• Palm contour
• Fingers parallel
• Compare to other side
Inspection• Number of digits
• Webbing
• Distal palmar crease
• Thenar and hypothenar eminences
• Skin: calluses, grime
• Forearm rotation
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Inspection• Quick check of upper extremity motion
• Hands behind head, behind back
Rough check of full shoulder motion
• Elbow E/F, palms up/down
• Congenital R-U synostosis can be missed for yrs
• Wrist E/F
• Hand open/fist
Palpation
• Tenderness, interruptions, prominences
• Distal radius and ulna
• Anatomic snuffbox
• Skin: Dry? Cool?
• Joints: Hot? Puffy? Unstable?
Palpation
• Carpo-metacarpal joints• Stable 2nd and 3rd versus
• Mobile 4th and 5th:
Required for grip
Stable
Mobile
Palpation
• Palmar aponeurosis• Dupuytren’s contracture
• Flexor tendons • Trigger finger
Trapped!Tendon
Tendon
SheathNodule
Motor Exam
ThumbExtension: palm on table surface
Opposition to all fingers
Adduction, abduction, flexion
Motor Exam
Normal Variant
Anomalous connection between FPL and FDP to the index fingerLinburg and Comstock anomaly
Can cause inability to flex IP joint of thumb without flexing DIP of index finger
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Motor Exam
• Fingers• Abduction/Adduction
• Measured from middle finger
• Perpendicular to plane of thumb
Motor Exam• Fingers
• Isolate joints in order to isolate muscle function
• Flexor Digitorum Superficialis (FDS): • Flexion at PIP• Hold other DIP’s in
hyperextension in order to isolate
Motor Exam• Normal variations
• 10% no independent FDS to small finger
• While examining small finger with ring DIP in extension, small finger FDS will appear to be ruptured
• Must instead hold middle finger in extension, allowing ring and small finger to flex
Motor Exam
• Flexor Digitorum Profundus (FDP):
• Flexion at DIP• Fix PIP of same finger to
isolate FDP
Motor ExamNerve Function
Median: Abductor pollicis brevis
Ulnar: First dorsal interosseous
Radial: Extensor pollicis longus
Riche-CannieuConnection
◼ First web area of palm
◼ Motor branch of ulnar nerve connecting to portion of median nerve
◼ Function incompletely defined
◼ Probably accounts for dual/cross innervation of flexor pollicis brevis
◼ i.e., weak opposition may be possible with complete MN palsy
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Sensory Exam
Median
Ulnar
Radial???, because…
Berettini Connection◼ Sensory branch in
palm between ulnar and median nerves
◼ Present in 80% of hands
◼ Usually: sensory fibers from ulnar nerve passing laterally into classic median distribution
Clemente Atlas
Sensory Exam
Monofilaments: neuropathy
2-point discrimination: nerve injury/recovery
Children/comatose/uncooperative: skin wrinkling in water
Dry skin
Dry skin on thumbNo wrinkling
Wrinkled
Sensory Exam
• Tinel sign:• Tapping over course of nerve….gently!
• Positive in compressive neuropathy• False positive rate of 45%
• Can follow course of regenerating nerve
Vascular Exam
• Pulses: radial and ulnar arteries
• Capillary refill
• Allen test: patency of arteries
• Patient clenches fist• Pressure placed over
both arteries with thumbs • One artery released and capillary
refill assessed• Repeat releasing other artery
Vascular Exam
• Modified Allen for the digital arteries
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Special Tests
• Bunnell-Littler test• Limitation of
Flexion• Intrinsic
Tightness (lumbricals and interossei) vs.
• Joint capsule contractures
Special testsBunnell-Littler
• Hold MCP in extension, if cannot flex PIP intrinsic vs capsular tightness exists
Allow flxn at MCP: relaxes intrinsics
If remains tight, capsular contracture is responsible for limitation of flexion
Special Tests
• Elson’s test• Early test for central slip dysfunction, prior to appearance of
boutonniere deformity
• Hold PIP in flexion
• Ask patient to extend DIP: normally cannot; early dysfunction can
Range of Motion
• Normal finger passive ROM, in degrees
Flexion Extension
MCP 90 -30 to -45
PIP 110 0
DIP 80 -20
Range of Motion
• MCP Joints: • “cam” joint
• lateral motion in extension, none in flexion
• Collateral ligaments
• Importance of proper immobilization
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Range of Motion
• Measurement: degrees of range of motion and contracture
• Objective and reproducible
• Multiple techniques• Total Active Motion, Total Passive Motion (see Addendum)
• Distance of pulp to palm: useful in flexion contractures
FunctionalRequirements
• Grasp: Power gripping
• Hammer, golf club
• Pinch: Fine motor• Shirt buttons, paper clips
Function
• Pinch mechanism• Thumb and index finger, middle finger
• Several muscles involved
• Long flexors and extensors stabilize proximal joints
• Lumbricals and interossei provide fine pinch
Function
• Pinch mechanism• Need adequate index and middle finger
extension for finger pulps to meet thumb pulp
Function
• Grasp• Need adequate ring and
small finger flexion for power grip
• Ring and small carpometacarpal mobility
FunctionalRequirements
• Sensory• Protective sensation: border digits, ulnar aspect of small finger, radial aspect
of index
• Fine motor function: necessitates intact sensation of thumb pulp and radial aspect of index finger
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Summary
• Examination includes thorough testing of motor, sensation, and vasculature
• Be aware of normal variations to avoid mistakes in diagnosis
• Understand what functions are most important for optimal hand use
Addendum 1Neurologic Levels
Motor Sensory
C6 Wrist extension Thumb and index, radial forearm
C7 Wrist flexion, finger extension
Middle finger
C8 Finger flexion Ring and small, ulnar forearm
T1 Finger abduction, adduction
Ulnar arm
Addendum 2Range of Motion: Notation system
Active Passive
MCP 10 to 75 0 to 90
PIP 30 to 75 10 to 95
DIP 0 to 55 0 to 60
Total flexion 205 245
- Total lack of extension
-40 -10
= Total motion Active 165 Passive 235
Further StudyModel Z, Liu AY, Kang L, Wolfe SW, Burket JC, Lee SK. Evaluation of Physical Examination Tests for Thumb Basal Joint Osteoarthritis. Hand (N Y). 2016;11(1):108-112.Rodriguez R, Strauch RJ. The middle finger flexion test to locate the thenar motor branch of the median nerve. J Hand Surg Am. 2013 Aug;38(8):1547-50.Valdés-Flores E, García-Álvarez E, García-Pérez MM, Castro-Govea Y, Santos-Ibarra A, Chacón-Martínez H, Betancourt-Espericueta L, Mecott GA. A Test for the Clinical Evaluation of the Flexor Digitorum Superficialis of the Fifth Finger. Ann Plast Surg. 2019 Feb;82(2):166-168.
Further StudyAustin GJ el al: Variations of the FDS of the small finger. J Hand Surg 1989, 14A:262-7.Linburg RM, Comstock BE. Anomalous tendon slips from the flexor pollicis longus to the FDP. J Hand Surg 1979, 4:79-83.Napier JR. The prehensile movements of the human hand. J Bone Joint Surg 1956; 38B:902-13.Kuschner SH et al: Tinel’s sign and Phalen’s test in carpal tunnel syndrome. Orthopedics 15:1297-1302, 1992.Meals RA, Shaner M: Variations in digital sensory patterns: a study of the ulnar nerve-median nerve palmar communicating branch.J Hand Surg 1983 8:411-4.
Imaging of the HandMark Greyson, MD
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Objectives:
◼ Historical background
◼ Basic concepts of radiology
◼ Special views of the hand
◼ Fractures
◼ Joints
◼ Bone density
History
• First x-ray in 1895
• Wilhelm Roentgen (1845-1923)
• Wife’s hand
Know the Bones
What are these shadows?
Sesamoid Bones
Pediatric X-rays Basic Views
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Basic Views
Posteroanterior (PA) view
Basic Views
Lateral View
Basic Views
Where’s
the
fracture?
Where’s
the
fracture?
Basic Views
Fracture Concepts
• Cortical breaks
• Radiolucent lines
Special Views
“Standard” pronated oblique view
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Special Views
Supinated oblique view
Special Views
Supinated oblique view – Arthritis & Fracture
Special Views
PA View
Special Views
Brewerton View
Special Views: BrewertonJoints
Osteoarthritis
Arthritis presents with:
• joint space narrowing
• sclerosis
• cysts
• osteophytes
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Joints
Osteoarthritis
Density
UltrasoundCheap and quick
Useful for detecting:• Dynamic changes
Ganglion/Abscess
• Foreign body • May not see XR
• Ligament Tears/Tendinitis
• Guided aspirations
CT Scan• Quick 3D view possible
• Useful for detecting:• Suspected fractures not easily seen on X-ray
• Assess union
• Evaluate cortical integrity with bony tumors
• Study fracture pattern and plan surgery
MRI Scan
• Expensive, timely
• Useful for detecting:• Soft Tissue Anatomy
(mass/swelling)
• Ligament injuries – May use arthrography
• Vascularity (Kienböck’s, Scaphoid Non-union)
Bone Scan
• Technetium-labeled bone scintigraphy• High sensitivity, low specificity.
• Increased uptake indicates increased blood flow (immediate) and bone turnover (delayed phase).
• Useful for detecting:• Osteomyelitis
• CRPS
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Summary
• PA and lateral radiographs depict the hand and fingers in three dimensions
• Special oblique views may be obtained to confirm presence of fractures
• Radiographs can visualize the finger joints
• Density of bone may vary in metabolic bone diseases
References
Hand Surgeons Providing Excellence in Education
forOccupational and Physical Therapists
www.doctorsdemystify.com
DIGITAL
CONGENITAL DIFFERENCES
PROSPER BENHAIM, MD
UCLA Hand Center
CONGENITAL DEFORMITIESUpper Extremity
• Common
• 1 in 626 live births
• Many are single gene disorders
DIFFERENTIATIONOF THE UPPER LIMB
• Limb buds - ventrolateral wall of embryo
• Limb buds appear 4th week (day 26)
• Limb buds develop from day 26-47
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Weeks 5-6• Hand paddle develops
• Nerve ingrowth from spinal
cord rami begins to occur
proximally
d37 d42 d44
Hand Paddle
Weeks 6 - 7
• Fingers begin to separate
• Cartilaginous “bones” form
d48 d51
Weeks 7 — 8
• The UE grows and rotates 90°
• Elbows project posteriorly
• Dorsal mesenchymal stem cells – extensors
• Ventral mesenchymal stem cells - flexors
d53 d55 d58
CONGENITAL HAND ANOMALIESClassification and Examples
I Failure of formation of parts
II Failure of differentiation
III Duplication
IV Overgrowth
V Undergrowth
VI Constriction band syndrome
VII Generalized skeletal abnormalities
FAILURE OF FORMATIONTransverse Deficiency
FAILURE OF FORMATIONLongitudinal Deficiency
Complete
longitudinal failure
phocomelia
Partial
Radial (preaxial)
Central
Ulnar (post-axial)
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HYPOPLASTIC THUMB
Grade I
Mild shortening
Grade II
Moderate shortening
Thenar hypoplasia
TYPE III HYPOPLASTIC THUMB
TYPE IV - FLOATING THUMB
TYPE V - ABSENT THUMB
• Lateral pinch grip between index and middle fingers
• Secondary rotation of index finger
• Pollicization improves grasp and tip-to-tip pinch
CLEFT HAND
TYPICAL
Absent middle finger
Usually bilateral
Assoc w/ cleft feet (50%)
Assoc with cleft lip, palate
TYPICAL CLEFT HAND
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CLEFT HAND
ATYPICAL
Absence of > 1 central digit
LOBSTER CLAW HAND
Thumb and small finger only
ULNAR APLASIA
CONGENITAL HAND ANOMALIESClassification
I Failure of formation of parts
II Failure of differentiation
Syndactyly
Camptodactyly
Clinodactyly
Kirner deformity
SYNDACTYLY
• 1 in 2000 live birth, sporadic vs. familial (10%)
• 50% bilateral
• Middle-ring finger web - most common
• Classification
• Webbing: “complete” vs “incomplete”
• Bone not involved = “simple”, involved = “complex”
SIMPLE SYNDACTYLY
Incomplete Complete
COMPLEX COMPLETE SYNDACTYLY
SIMPLE
COMPLETE
SYNDACTYLY
RF tethering MF
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POLAND’S SYNDROMEsymbrachydactylychest wall deformity
POLAND’S SYNDROMEabsent PIP joints
ACROCEPHALO-SYNDACTYLY
• APERT SYNDROME
• “Spoon-like” hand
• Common nail index, middle &
ring fingers
• Complex bony deformities
Apert
Syndrome
CAMPTODACTYLY
• Flexion contracture PIP
joint
• Small finger - 90%
• ? Abnormal insertion of
lumbrical muscle
• Bilateral - 66%
CAMPTODACTYLY Treatment
• Advise parents to accept the deformity
• Passive stretching, serial splinting
rarely successful
• Consider surgery: Young
child - severe contractureRapid
progression
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CLINODACTYLY
• Radial-ulnar curvature
• Usually small finger
• Usually radial deviation
• Delta phalanx
DELTA PHALANX
• Trapezoidal-shaped phalanx
• Abnormal C-shaped epiphysis
• Usually middle phalanx of the
small finger
Delta Phalanx of ThumbKIRNER DEFORMITY
• Palmar-radial curvature, rotation
• Distal phalanx, usually bilateral
• Associated musculoskeletal anomalies
• No functional limitations usually
• Observe
• Splint
• Correctional osteotomy
CONGENITAL HAND ANOMALIESClassification
I Failure of formation of parts
II Failure of differentiation
III Duplication polydactyly
• Pre-axial Thumb
• Central Index, middle, ring
• Post-axial Small finger
POLYDACTYLY
• Small finger is most commonly involved
• More common in African-American infants
(incidence = 1 in 300)
• More common in females
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SMALL FINGER POLYDACTYLYType 1 – Rudimentary Soft Tissue
SMALL FINGER POLYDACTYLY
Type II – Bifid Digit, One Metacarpal
SMALL FINGER POLYDACTYLY
Type II
SMALL FINGER POLYDACTYLYType III – Metacarpal Duplicated
INDEX FINGER DUPLICATION THUMB DUPLICATION(Type 1: broad pulp and nail)(Type 2: distal phalanx duplication)
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THUMB DUPLICATIONType 3
THUMB DUPLICATIONType 4
TRIPHALANGEAL THUMBType 7
CONGENITAL HAND ANOMALIESClassification
I Failure of formation of parts
II Failure of differentiation
III Duplication
IV Overgrowth
V Undergrowth
VI Constriction band syndrome
VII Generalized skeletal abnormalities
MACRODACTYLY
• 90% unilateral
• Multiple digits vs. single digit = 3 : 1
• Affects radial side of hand
• Index finger most frequently affected
• ? Related to neurofibromatosis
MACRODACTYLY
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MACRODACTYLY
• Staged debulking of soft tissue
• Wedge osteotomies to correct deviation
• Consider amputation
CONGENITAL HAND ANOMALIESClassification
I Failure of formation of parts
II Failure of differentiation
III Duplication
IV Overgrowth
V Undergrowth (brachydactyly)
• Short digit - normal # of bones, 1 is small
• Short metacarpal (brachymetacarpia)
• Short phalanx (brachyphalangia)
BRACHYMETACARPIA BRACHYPHALANGIA
*
*
**
*
*
*
*
CONGENITAL HAND ANOMALIESClassification
I Failure of formation of parts
II Failure of differentiation
III Duplication
IV Overgrowth
V Undergrowth
VI Constriction band syndrome
VII Generalized skeletal abnormalities
CONSTRICTION RING SYNDROME
• Circumferential grooving or transverse
amputation
• Associated anomalies (40-50%)
Club feet; cleft lip and palate
• Associated hand anomalies (80%)
Syndactyly
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CONSTRICTION RING SYNDROME
REFERENCES
1: Chung MS. Congenital differences of the
upper extremity. Clin Orthop Surg. 2011
Sep;3(3):172-7.
2: Bates SJ, et al. Reconstruction of congenital
differences of the hand. Plast Reconstr Surg.
2009 Jul;124(1 Suppl):128e-143e.
3: Oberg KC, et al. Developmental biology and
classification of congenital anomalies. J Hand
Surg Am. 2010 Dec;35(12):2066-76.
REFERENCES
4: Sammer DM, et al. Congenital hand
differences: embryology and classification.
Hand Clin. 2009 May;25(2):151-6.
5: Linder JM, et al. Congenital anomalies of
the hand: an overview. J Craniofac Surg. 2009
Jul;20(4):999-1004.
6: Goldfarb CA. Congenital hand surgery:
what's new and what's coming. Hand Clin.
2009 May;25(2):293-9.
Hand Surgeons Providing Excellence in EducationforOccupational and Physical Therapists
www.doctorsdemystify.com
Collateral Ligament & Volar Plate Injuries
Nicholas E. Rose, M.D.Newport Beach, CA
No vested interest. No off-label FDA uses.
Anatomy: MCP Joint
• Cam effect
• Ligaments• Tight in flexion
• Lax in extension
• JOINT MUST BE SPLINTED IN FLEXION!
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Specialized AnatomyThumb MCP Joint
• Condyloid joint
• Sesamoid bones
• Collateral ligaments
• Static stability• Adductor aponeurosis
• Dynamic stability
Anatomy: PIP Joint• Collateral Ligaments
• Cord-like proper lig.
• Anterior: accessory lig.
Anatomy: PIP Joint
• Cam effect less significant than MCP joint
• Collateral ligaments tight in flex AND ext
Anatomy: Volar Plate
• MCP and PIP Joint• Firm distal attachment
• More flexible proximal attachment
• Separates joint from flexor tendons
• Resists hyperextension
Finger MCP Injuries
• Classification• Collateral ligament injuries• Volar plate injuries• Dislocations• Fracture-dislocations
• Stable vs. unstable
Finger MCP Collateral Ligament Injuries
• Relatively rare compared to PIP injuries
• Diagnosis often missed
• Middle finger most common due to length
• Radial > ulnar collateral injuries
• Index RCL of particular concern
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Finger MCP Collateral Ligament Injuries
• Examination• Subtle swelling in valley between metacarpal heads• Pain with lateral stress with MCP in flexion (MCP normally lax in
extension)
• Radiographs• +/- avulsion fragment
• Sonography
TreatmentAcute Finger MCP Collateral Ligament Injuries
• Splint MCP in 50º flexion x 3w
• Buddy taping
• Operative treatment• >3mm displacement of small avulsion fragment• Fragment involves > 20% of articular surface &
is displaced & rotated• Grossly unstable joints
TreatmentAcute Finger MCP Collateral Ligament Injuries
• Operative treatment• Internal brace
• Suture tape augmentation• Immediate stability• Earlier rehab • Earlier return to activity & sports
TreatmentChronic Finger MCP Collateral Ligament Injuries
• Can be symptomatic for 12-18 months
• Steroid injection trial + buddy taping
• Arthroscopic debridement
• Ligament reconstruction +/- free tendon graft
MCP Volar Plate Injuries
• Hyperextension injury
• Volar plate torn from proximalattachment
MCP Volar Plate InjuriesDorsal Dislocation
• Simple
• Subluxation (articular surfaces in contact)
• Proximal phalanx 60 - 90 degrees hyperextension
• Reducible
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MCP Volar Plate InjuriesDorsal Dislocation
• Complex
• Complete dislocation• Less dramatic clinical appearance• Irreducible
• volar plate interposition
• DeVonta Smith • Alabama receiver• 2021 CFP National Championship
MCP Volar Plate InjuriesComplex Dorsal Dislocation
Volar plate interposition
Thumb MCPGamekeeper’s Thumb
• Sudden abduction stress
• Rupture of UCL & volar plate
• Stener lesion (adductor aponeurosis interposition)
Thumb MCPGamekeepers Thumb
• Stress testing
• Stress radiographs
Thumb MCPTreatment
• UCL Tear (Gamekeepers)• Partial tear
• Gamekeepers brace• Cast
• Complete tear
• Early repair • +/- internal brace
• Late reconstruction with tendon graft
• RCL Tear
• Surgery rarely indicated (no Stener lesion)
PIP Injuries
• Classification• Collateral ligament injuries• Volar plate injuries• Dislocations• Fracture-dislocations
• Stable vs. unstable
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PIPCollateral Ligament Injuries
• Lateral dislocation
• Abduction or adduction force• Usually in extension
• Radial > ulnar collateral
ligament injuries
• Acute (<6 weeks) vs. chronic (>6 weeks)
• Complete vs. incomplete
PIPCollateral Ligament Injuries
• Examination• Tenderness
• Central slip• Volar plate• Radial collateral ligament• Ulnar collateral ligament
Phot
Would be great
PIPCollateral Ligament Injuries
• Examination• Stability
• Radial & ulnar• Angulation > 20 degrees
indicates loss of collateral ligament integrity
• Sonography
Photos here would be great
PIPVolar Plate Injuries
• Examination• Stability
• Volar plate• Integrity tested
by passive hyperextension
PIPCollateral Ligament Injuries
• Examination• Stability
• Radiographic examination / stress testing
• Angulation > 20 degrees indicates loss of collateral ligament integrity
PIPCollateral Ligament Injuries
• Examination• Range of motion
• Active• Inability of patient to actively extend PIP joint against
resistance may indicate central slip rupture• Passive• Boutonniere deformity
• Limited passive DIP flexion with PIP held in maximum extension
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PIPCollateral Ligament Injuries
• Radiographs• PA & Lateral• Oblique view
• Useful for condylar injuries
• Sonography
TreatmentAcute Partial Collateral Ligament Injuries
• Buddy taping
• Taped to normal adjacent digit• Immediate active ROM• Full time for first 3 weeks• During periods of stress for 3 more weeks
• Athlete allowed to continue play during course of treatment
TreatmentAcute Collateral Ligament Injuries
• Surgery
• Rarely indicated• Avulsion of PIP collateral lig w/ buttonholing
of condyle between ligament & central slip
TreatmentAcute Collateral Ligament Injuries
• Surgery (ligament augmentation)
• Internal brace • Suture tape
TreatmentChronic Collateral Ligament Injuries
• Symptoms for 12-18 mo.
• Buddy taping 3-6 weeks
• Cortisone injection
TreatmentChronic Collateral Ligament Injuries
• Repair less satisfactory than in acute injuries
• Ligament shortening (difficult to gauge)• Combined ligament/volar plate reconstruction• Loss of motion• Operative repair for index finger PIP RCL in young
adult (Stability for pinch > full range of motion)
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Collateral Ligament InjuriesLong-term Sequelae
• Swelling
• Stiffness
• Flexion contracture
• Pain
• Chronic instability
• Post-traumatic arthritis
PIP Volar Plate Injuries• Hyperextension force
• Volar plate injury without dislocation• Dorsal dislocation
• Volar plate torn from distal attachment
• Most common PIP dislocation
• Usually reduced by patient, trainer or coach
PIP Volar Plate Injuries
• Volar plate almost always detaches distally (+/- bony avulsion)
TreatmentAcute PIP Volar Plate Injuries
• Most patients regain full ROM
• Symptoms will persist 12-18 months
• Permanent stiffness
PIP Volar Plate InjuriesSequelae
• PIP Flexion Contracture
• Scarring of volar plate• Dynamic splinting• Therapy• Occasional operative treatment
References
• Freiberg A, Pollard BA, Macdonald MR, Duncan MJ. Management of proximal interphalangeal joint injuries. Hand Clin. 2006 Aug;22(3):235-42.
• Hogan CJ and Nunley JA. Posttraumatic Proximal Interphalangeal Joint Flexion Contractures. J Am Acad Orthop Surg. 2006;14:524-33
• Leibovic SJ, Bowers WH. Anatomy of the Proximal Interphalangeal Joint. Hand Clinics 10:169-78, 1994.
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• Merrell G and Slade JF (2011). Dislocations and Ligament Injuries in the Digits. In Green’s Operative Hand Surgery(6th Ed. pp. 291-332). Philadelphia, PA. Elsevier Churchill Livingstone.
• Abbiati G, et al. The Treatment of Chronic Flexion Contractures of the Proximal Interphalangeal Joint. J Hand Surg 20B:385-9, 1995
• Durham JW et al: Acute and Late Radial Collateral Ligament Injuries of the Thumb MP Joint. J Hand Surg 18A:232-7, 1993.
• Lee SJ et al. Thumb ulnar collateral ligament repair with suture tape augmentation. J Hand Surg Asian Pac 25(1):32-38, Mar 2020.
• Draghi F et al. Injuries to the collateral ligaments of the metacarpophalangeal and interphalangeal joints: sonographic appearance. J Ultrasound Med. 37(9):2117-2133, Sept 2018.
Hand Surgeons Providing Excellence in EducationforOccupational and Physical Therapists
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Metacarpal and Phalangeal Intra-articular Fractures
Ali Ghiassi, MDUSC Department of Orthopedic Surgery
Introduction
• Intra-articular fractures are challenging to treat
• Digital joints are predisposed to stiffness• Articular involvement = hemarthrosis =
arthrofibrosis• Predispose to early arthrosis• May present late (10 days)
Principles of Treatment
• Accurate fracture reduction
• Maintenance of motion in uninvolved digits/joints
• Edema control
• Immobilization in safe position
• Early remobilization of the injured finger
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Mechanism of Injury
• Mechanism/time of Injury
• Closed vs. Open
• Angular or rotational deformity
• Stability
• Associated injuries
Associated Injuries
Time of Presentation
• Early presentation and treatment important
• Present beyond 2-3 weeks consider late reconstruction
• Get motion in all other joints
CondylarFractures
• Common athletic injuries
• Misdiagnosed as sprains
• Classified
• Stable nondisplaced• Unstable Unicondylar• Unstable Bicondylar
(comminuted)
Condylar Fractures
• Generally unstable
• Will have angular deformity
• Surgical stabilization for acute presentation
• Gutter splinting for delayed presentation or non-displaced fractures
• Needs close follow up in the acute period
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Condylar Fractures
• Most are unstable and need surgical stabilization
• Consider non-operative for late presentation 2-3 weeks
• High risk of stiffness
Non-operative Treatment
• Use a gutter splint with 3-point molding
• Need weekly fu
• Always risk of displacement
• Used for late presentation to prevent further displacement
Closed Reduction,Percutaneous Pinning(CRPP)
Condylar Fractures
• ORIF
• Should start early unrestricted motion
• Generally extensor mechanism is split or moved to one side
• CRPP
• May start early motion (surgeon’s decision)
• Need to protect wires for 3-4 weeks
Condylar Fractures
• ORIF (open reduction, internal fixation)
• Early edema control
• Tricks and tips
• CRPP (closed reduction, percutaneous pinning)
• Early edema control
• Tricks and tips
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PIP Fracture Dislocations
• Dorsal dislocations are associated with impacted fractures at the base of the middle phalanx
• Stable• Articular fragment < 40%
• Tx: CR and dorsal extension block splinting
• Unstable• Articular fragment > 40%
• Reduction is difficult to achieve and maintain
Non-operative Treatment
Non-operative Treatment
• Gradually work on extension
• One week rest
• Second week flexion in stable arc (90- full)
• Third week flexion in 1/2 stable arc (45-full)
• Fourth week (20-full)
• Week 5-6 d/c splint
• After week 6 work on residual contracture
PIP Fracture Dislocations
• Unstable injuries require operative treatment
• Goal is a congruous reduction of joint• Volar fragment is usually very comminuted• There is usually articular impaction noted• ORIF or Volar plate arthroplasty is indicated
Open Reduction,Internal Fixation
• Need large articular fragment
• Early motion is preferred within stable arc
• No passive strech for 6 weeks
Volar Plate Arthroplasty
• VPA is resurfacing of the depressed articular fragment with the volar plate
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Volar Plate Arthroplasty Volar Plate Arthroplasty
• One week rest
• Second week flexion in stable arc
• Third week divide extension 1/2
• Fourth week divide by 1/2
• Week 5-6 button removed
• Week 6 work on residual contracture
Hemi-Hamate Arthroplasty Hemi-Hamate Arthroplasty
Hemi-Hamate Arthroplasty
• One week rest
• Second week active flexion started with dorsal blocking splint (15-20º)
• Discontinue splint at 6 weeks
• Work on residual contracture
Dynamic Skeletal Traction
• Good for pilon fractures of pip joint
• Unstable volar lip fractures of pip
• Chronic pip dislocations
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Unstable Pip Fracture Dislocation Dynamic Skeletal Traction
Metacarpal Head Fractures
• Rare intra-articular fractures
• Usually comminuted or associated MP dislocation
• Brewerton view to asses CL avulsion
• Treatment is individualized depending on pattern and degree of comminution
• If < 20% of joint
• Buddy tape, protected ROM
• If > 20%
• ORIF• With early motion• Edema control• Buddy taping
• Usually protective splinting is not needed
• Stiffness is main issue
Metacarpal Head Fractures
CMC dislocations
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CMC Dislocations Reading
• Abzug J et al: Pediatric phalanx fractures. J Am AcadOrtho Surg. 2016; 24(11) – pe174-e183
• Carpenter S, Rohde R: Treatment of phalangeal Fractures. Hand Clinics, 2013; 29 (4):519-534, P519-
• Difficult Fractures of the Hand and Wrist, Hand Clinics May 1994
• Caggiano, N et al: Management of Proximal Interphalangeal Joint Fracture Dislocations. Hand Clinics. 2018 May;34(2):149-165
Hand Surgeons Providing Excellence in EducationforOccupational and Physical Therapists
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Wendy Chen, MD, MS
Stiff Fingers, Joint Contractures, and Capsulotomies
Presentation originally prepared by Scott Mitchell, M.D.
“An ever-present menace in hand surgery is the tendency for the hand to stiffen and to stiffen in the position of non-function.”
Sterling Bunnell. Surgery of the Hand, 3rd ed. 1956
• Wrist flexed
• Metacarpal arch flattened
• Intrinsic minus position• MCP joints extended
• Fingers clawed
• Thumb adducted
Position of Non-Function
Proximal (wrist)
Distal (digits)
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Causes of Hand Stiffness Causes of Hand Stiffness
• Prolonged immobilization or Inadequate immobilization
• Trauma
• Infection
• Arthritis
• Dupuytren’s contracture
• Burns
• Congenital anomalies
• Neurologic dysfunction
Pathophysiology: EDEMA is the FIRST RESPONSE
• Affects both injured AND uninjured structures
• Joint capsules distend with fluid
• Pain and swelling lead to characteristic hand posture
• MP joint extension
• IP joint flexion
Pathophysiology: EDEMA is the FIRST RESPONSE
• Affects both injured AND uninjured structures
• Joint capsules distend with fluid
• Pain and swelling lead to characteristic hand posture
• MP joint extension
• IP joint flexion
Pathophysiology: EDEMA is the FIRST RESPONSE
Edema distends JOINT CAPSULE
• MP joint key to swollen hand posture
• Joint assumes position of maximum fluid capacity
• Extended MPJ least stable
• Minimal articular surface contact
• Capsular structures lax
• Fluid drives MP joint into full extension
Pathophysiology: EDEMA is the FIRST RESPONSE
MP joint drives position of IP joints
• MP EXTENSION leads to:• Increased flexor tension
• Decreased extensor tension
• IP joint flexion
• Capsule and collateral ligaments shorten
• Changes become fixed → joint contractures
Edema → Fibrosis → Shortening
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Pathophysiology: EDEMA is the FIRST RESPONSE
Edema increases stiffness via SKIN TENSION
• Normal digit • Dorsal skin over PIP lengthens for
12mm for 90o of flexion
• Swollen digit• 5mm dorsal swelling
• 19mm lengthening required for 90o of flexionEdema → Fibrosis → Shortening
Soft Tissue Injury
• Extent of soft tissue damage influences motion loss
• Combined injuries (bone, tendon, joint) have worse prognosis.
• Internal wounds may be extensive, even with closed fractures.
Soft Tissue Injury• Extent of soft tissue damage influences
motion loss
• Combined injuries (bone, tendon, joint) have worse prognosis.
• Internal wounds may be extensive, even with closed fractures.
The Anatomy of FLEXION Contractures
• Volar scar contracture
• Superficial fascia (Dupuytren’s contracture)
• Flexor tendon sheath
• Flexor tendon adhesions
• Volar plate
• Accessory collateral ligaments
• Bony block/exostosis
• Articular incongruity
Superficial (Skin)
Deep (Bone)
Anatomy
The Anatomy of FLEXION Contractures
• Volar scar contracture
• Superficial fascia (Dupuytren’s contracture)
• Flexor tendon sheath
• Flexor tendon adhesions
• Volar plate
• Accessory collateral ligaments
• Bony block/exostosis
• Articular incongruity
The Anatomy of FLEXION Contractures
• Volar scar contracture
• Superficial fascia (Dupuytren’s contracture)
• Flexor tendon sheath
• Flexor tendon adhesions
• Volar plate
• Accessory collateral ligaments
• Bony block/exostosis
• Articular incongruity
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The Anatomy of FLEXION Contractures
• Volar scar contracture
• Superficial fascia (Dupuytren’scontracture)
• Flexor tendon sheath
• Flexor tendon adhesions
• Volar plate
• Accessory collateral ligaments
• Bony block/exostosis
• Articular incongruity
The Anatomy of EXTENSION Contractures
• Dorsal scar contracture
• Extensor tendon adhesions
• Joint capsule
• Collateral ligament
• Bony block/exostosis
• Articular incongruity
Superficial (Skin)
Deep (Bone)
The Anatomy of EXTENSION Contractures
• Dorsal scar contracture
• Extensor tendon adhesions
• Joint capsule
• Collateral ligament
• Bony block/exostosis
• Articular incongruity
Physical Exam: Active versus Passive Motion
• Passive > active ROM• Extra-articular causes limit
motion• Tendon adhesions
• Passive = active ROM• Intra-articular causes
• Capsular contracture• Tendon adhesions on opposite
side of joint
Physical Exam
Physical Exam: Fixed versus positional motion loss
• Motion varies in response to position of adjacent joints
• Tenodesis effect: • Non-articular contracting structure
spanning > 1 joint
• Intrinsic or extrinsic musculotendinous tightness
Physical Exam: Intrinsic Tightness
• Intrinsic tendons • Flex MP joints• Extend PIP and DIP joints
Credit: Martin PosnerHand 50 Lecture on Extensor Tendons
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Physical Exam: Intrinsic Tightness
• Intrinsic tendons • Flex MP joints
• Extend PIP and DIP joints
• Tight intrinsics limit composite digital flexion (IP joint flexion)
Credit: Martin PosnerHand 50 Lecture on Extensor Tendons
Physical Exam: Intrinsic Tightness
• Intrinsic tendons • Flex MP joints
• Extend PIP and DIP joints
• Tight intrinsics limit composite digital flexion (IP joint flexion)
• Motion loss more severe if MP joint is extended
Physical Exam: Intrinsic Tightness
Bunnell Test for Intrinsic Tightness• Tests passive PIP flexion with the MP joint flexed then
extended.
• MP flexion
• relaxes intrinsics
• PIP easily flexes
Physical Exam: EXTRINSIC Tightness
• Long flexor tightness• Wrist and MP joints extended• Test passive IP extension
•Extrinsic extensor tightness•Wrist and MP joints flexed•Test passive IP flexion
Capsular Contracture•Loss of both active and passive motion
•Not affected by adjacent joint position
•Exceptions:
•Tendon adhesions on both sides of joint
•Bony blocks to motion
•Joint incongruity
•Disclaimer:
•Capsular contractures may *mask* extra-articular causes of motion loss
Is it the collateral ligaments?
If collateral ligaments are the only limitation, it won’t matter whether the MCPJs are flexed or extended—
the PIPJ will be the same degree of stiff.
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Treatment of Stiff Fingers•Prevention
•Judicious immobilization
•Reduce edema
•Pain control
•Early active and passive motion
•Splinting
•Art of therapy
Treatment of Stiff Fingers•Prevention
•Judicious immobilization
•Reduce edema
•Pain control
•Early active and passive motion
•Splinting•Art of therapy
Treatment of Stiff FingersSplinting: Counteract tendency to assume intrinsic minus posture
•Intrinsic plus position
•MCP joints flexed•Stretches capsule and collaterals •Balances flexor/extensor tone
•IP joints extended
•Prevents volar plate contracture
•Thumb abducted
Treatment of Stiff FingersSplinting:•Intrinsic plus position
•MCP joints flexed
•Stretches capsule and collaterals
•Balances flexor/extensor tone
•IP joints extended
•Prevents volar plate contracture
•Thumb abducted
Treatment of Stiff FingersTherapy to Restore Motion•Loss of active motion
•Active and active-assisted motion
•Tendon gliding
•Blocking exercises
•Resisted motion
Treatment of Stiff FingersSplinting: 87% of MPJ and PIPJ contractures can be managed conservatively•Static splint
•Dynamic splint
•Static progressive splint
•Serial cast/splint
Combine daytime dynamic splint or static progressive splint + nighttime static splint.
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Treatment of Stiff Fingers: PIP EXTENSION Splints Splinting: PIP EXTENSION Splints
Treatment of Stiff Fingers: PIP FLEXION Splints Splinting: PIP FLEXION Splints
Treatment of Stiff Fingers: EXPECTATIONS
•Nonoperative treatment of fixed flexion deformity of the PIPJ. Hunter et al. JHS Br 1999.
•Average PIP motion arc improved from 24-67 to 8-98o
Treatment of Stiff Fingers: EXPECTATIONS
•Dynamic splinting for the stiff hand after trauma. Glasgow C. J Hand Ther2011
•Time spent in splint is key
•Better outcomes with:
•Less pretreatment stiffness•Shorter time since injury (<12 weeks)•Flexion rather than extension deficits
Surgery
Indications
• No absolutes
• Motion loss > 30 degrees
• Plateau in therapy
•Firm vs soft end point
•May require 5-6 months of non-op treatment
• Tissue equilibrium
• Final benefit anticipated
Contraindications
• Neurovascular compromise
• Arthritis
• Incongruent joint surfaces
• Deficient motors
• Chronic, severe flexion deformities
MP extension contracture release
• Dorsal approach
• Extensor tendon split
• Extensor tenolysis
• Dorsal capsulectomy
• Collateral ligament release
• Splint in flexion
• Final motion 50% of operative ROM
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MP extension contracture release
• Dorsal approach
• Extensor tendon split
• Extensor tenolysis
• Dorsal capsulectomy
• Collateral ligament release
• Splint in flexion
• Final motion 50% of operative ROM
MP extension contracture release
• Dorsal approach
• Extensor tendon split
• Extensor tenolysis
• Dorsal capsulectomy
• Collateral ligament release
• Splint in flexion
• Final motion 50% of operative ROM
PIP extension contracture release
• Dorsal approach
• Extensor tenolysis
• Hardware removal
• Dorsal capsulotomy
• Dorsal collateral ligament release
• Distal intrinsic release
• Check active flexion
PIP extension contracture release
• Dorsal approach
• Extensor tenolysis
• Hardware removal
• Dorsal capsulotomy
• Dorsal collateral ligament release
• Distal intrinsic release
• Check active flexion
PIP extension contracture release
• Dorsal contractures may mask flexor adhesions
• Traction test to evaluate flexor system following dorsal release
• Confine surgery to one side of joint if flexion limited
• Therapy to maximize passive ROM
• Staged flexor tenolysis
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PIP FLEXION contracture release
• Sequential approach• Flexor tenolysis
• Proximal volar plate
• Accessory collateral
• Excision of collateral ligaments
• Check active extension
• Watch for vasospasm
• Outcomes worse with more structures requiring release
PIP FLEXION contracture release• Sequential approach
• Flexor tenolysis
• Proximal volar plate
• Accessory collateral
• Excision of collateral ligaments
• Check active extension
• Watch for vasospasm
• Outcomes worse with more structures requiring release
PIP FLEXION contracture release
• Sequential approach• Flexor tenolysis
• Proximal volar plate
• Accessory collateral
• Excision of collateral ligaments
• Check active extension
• Watch for vasospasm
• Outcomes worse with more structures requiring release
PIP FLEXION contracture release
• Sequential approach• Flexor tenolysis
• Proximal volar plate
• Accessory collateral
• Excision of collateral ligaments
• Check active extension
• Watch for vasospasm
• Outcomes worse with more structures requiring release
Digit Widget•Dynamic external
fixator
•Extension torque transmitted through pins in the middle phalanx
•Avoids skin pressure
•Accommodates additional soft tissue procedures
•Allows active motion
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Summary
•Traumatized hand tends toward a stiff, poorly functional position
•Prevent contractures with appropriate immobilization and early motion
•Physical exam to identify causes of stiffness
•Early therapy, splint for motion
•Surgery offers modest ROM gains, may improve function
ReferencesWang ED, Rahgozar P. The Pathogenesis and Treatment of the Stiff Finger. Clin Plast Surg. 2019 Jul;46(3):339-345. doi: 10.1016/j.cps.2019.02.007. Epub 2019 Apr 19. PMID: 31103078.
Yang G, McGlinn EP, Chung KC. Management of the stiff finger: evidence and outcomes. Clin Plast Surg. 2014 Jul;41(3):501-12. doi: 10.1016/j.cps.2014.03.011. PMID: 24996467; PMCID: PMC4124823.
Catalano LW 3rd, Barron OA, Glickel SZ, Minhas SV. Etiology, Evaluation, and Management Options for the Stiff Digit. J Am Acad Orthop Surg. 2019 Aug 1;27(15):e676-e684. doi: 10.5435/JAAOS-D-18-00310. PMID: 30475280.
Tuffaha SH, Lee WPA. Treatment of Proximal Interphalangeal Joint Contracture. Hand Clin. 2018 May;34(2):229-235. doi: 10.1016/j.hcl.2017.12.012. PMID: 29625642.
References
Hogan CJ, Nunley JA. Post-traumatic proximal interphalangeal joint flexion contractures. Journal of the American Academy of Orthopaedic Surgeons 2008;14(9), 524-33
Curtis RM. Capsulectomy of the interphalangeal joints of the fingers. J Bone Joint Surg Am 1954;36:1219-1232.
Prosser R. Splinting in the management of PIP joint flexion contracture. J Hand Ther 1996 9:378-86.
Hunter et al. Nonoperative treatment of fixed flexion deformity of the PIP joint. J Hand Surg Br 1999 Jun;24(3):281-3.
Houshian S. Chronic flexion contractures of the PIP joint treated with the compass hinge external fixator. J Hand Surg Br 2002;27B(4)356
Soft-tissue distraction vs checkrein ligament release for Dupuytren PIP joint contractures. Plast Reconstr Surg. 2011 128:1107-13
Hand Surgeons Providing Excellence in Education
forOccupational and Physical Therapists
www.doctorsdemystify.com
Finger InfectionsClifton Meals, M.D.
Introduction
• 35% of admissions to hand surgeons
• Most result from neglected minor trauma
• ~2/3 of infections caused by Staph aureus
• Prompt evaluation and treatment
• Antibiotics for uncomplicated cellulitis
• Pus requires surgical drainage
• A few zebras
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Bacteria
• Staphylococcus Aureus • Gram + cocci in clusters• cellulitis (no pus)• abscesses (pus)
• MRSA (methicillin-resistant S. aureus)
• More difficult to treat• More soft tissue damage
Bacteria
• Polymicrobial infections• Often include Gram neg. • Intravenous drug use• Bite wounds• Farm injuries• Diabetes mellitus
Viruses Fungi
History
• Injury?
• Timing
• Occupation / Exposures
• Health status (DM, HIV, RA, Gout?)
• Tetanus status
• Other infections?
• Medication allergies
Physical Exam
• Rule out threats to life, limb
• Rule out threats to • Joint, tendon
• Cellulitis?
• Abscess?
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Cellulitis
• No pus
• Infection of sub-q fat
• Less destructive
• Lymphatic streaking
Abscess
• Pus under pressure
• Locally destructive
• Difficult for antibiotic to penetrate
Treatment
• Antibiotics• Should cover Staph A.
• Rest • Minimizes spread
• Heat• Vasodilation / immune response
• Elevation• Reduces swelling
• Surgery?
Acute Paronychia
• Cellulitis / abscess of soft tissue around nail
• Most common infection of the hand
Paronychium
Eponychium
Hyponychium
Acute Paronychia
• Cellulitis: warm soaks & oral Abx
• Pus: use scalpel to elevate paronychial fold from nail plate
• Pus underneath nail: remove part of nail
• 7-10 days of oral antibiotics
Chronic Paronychia
• Indurated eponychium• Long duration• Dishwashers• DM, HIV• Candida albicans• Eliminate exposure• Topical / Oral Abx• Occasionally Surgery
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Felon
• Abscess of pulp
Felon• Incision & drainage
• Leave open / pack
• Warm soaks BID x ~ 5 days
• Antibiotics ~ 5 days
Flexor Tenosynovitis
• Kanavel Signs• Flexed posture
• Pain w/ passive stretch
• TTP along tendon sheath
• Fusiform swelling
Flexor Tenosynovitis
• Abscess of tendon sheath• Destructive
• May ascend rapidly
• Difficult for immune system
• Difficult for Abx
• Surgical emergency
Treatment Principles
• Minimize soft tissue trauma
• Direct access to tendon sheath
• Permit easy extension
• Avoid high-contact areas
• Loose closure
• + / - drain
• Postop immobilization
Incisions
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Incisions Bite Wounds
• Dog > Cat > Human
• Open wounds left open
• Abscesses drained
• Cover gram positive, gram negative, anaerobes
• Augmentin / Unasyn
Septic Arthritis
• Rule out gout, pseudogout, OA
• Often penetrating trauma
• Fist vs. teeth → infected MPJ
• Pain with joint motion
• Rapid destruction of cartilage
• Surgical emergency
• Abx
Osteomyelitis
• Abscess of bone
• Local spread
• Hematogenous seeding
• X-Ray findings often subtle
• Often a diagnosis of exclusion
• + / - biopsy / culture
• IV Abx
• Surgery if necessary
Mycobacterium& Chronic FTS
• Milder, Chronic FTS
• Exposure to salt water
• Rice bodies on exam
• No pus
• Mycobacterium marinum• Granulomas
• Special stains
• Long course IV Abx
• + / - surgery
Herpetic Whitlow
• Virus
• May mimic paronychia / felon
• Think exposure
• Clear vesicles
• Self-limiting (3 weeks)
• Anti viral Rx may help
• Do not operate
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Gout
• Crystal arthropathy
• Commoly mimics infection
• Most common in men
• H/o recurrent episodes
• Any joint
• Treatment: NSAIDs, colchicine, allopurinol
• Rarely surgery
Summary
• ~2/3rds of infections caused by Staph A.
• Several emergencies• necrotizing fasciitis
• Flexor tendon sheath
• septic joint
• Antibiotics for uncomplicated cellulitis
• Pus requires surgical drainage
• Mimics: gout, virus, mycobacteria
References
• Koshy JC, Bell B. Hand Infections. J Hand Surg 2019; 44(1): 46-54.
• Stevanovic MV, Sharpe F: Acute infections. In Wolfe SW, Hotchkiss RN, Pederson WC, Kozin SH (eds): Green’s Operative Hand Surgery, Philadelphia, Elsevier, 2017; 17-61.
• Kann SE, Jacquemin J and Stern PJ. Simulators of hand infections. Instr Course Lect, 1997; 46: 69-82.
The Snapping FingerClifton Meals, MD
Always a trigger finger? Always a trigger finger?
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Always a trigger finger?
• Trigger finger• Subluxing EDC / sagittal band injury
• Lateral band subluxation at the PIP• MP osteoarthritis with snapping collateral ligament• Sesamoid at thumb IP after IP hyperextension injury
Trigger Finger
Trigger Finger
• Stenosing tenosynovitis
• Nodule in tendon and/or thickening of A1 pulley cause tendon to catch
Symptoms
• Pain and in the palm
• Can’t make a tight fist
• Morning stiffness
• Locking in flexion
Associated Conditions
• Diabetes mellitus
• Gout
• Rheumatoid arthritis
• Amyloid deposition
• Carpal tunnel syndrome
Exam
• Demonstrate triggering
• Palpate nodules
•Palmodigital crease is half way between
the PIP crease and the proximal edge of A1
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Treatment
• NSAIDS
• Splinting
• Static MP extension• 3-6 weeks• Approx 50% success
Non-operative Treatment
• Cortisone injection• 0.5 ml Kenalog 40
• Success rates• Single injection: 50%
• Second injections: 75-90%
Repeat injections in short
time span may risk tendon
rupture
Surgery Complications
• Digital nerve injury
• Bowstringing
• Stiffness
Subluxing EDC
• Sagittal bands centralize EDC
• Stressed when MP flexed
Subluxing EDC
• Unilaterally injured sagittal band allows EDC to sublux
• Usually, radial sagittal band injured, EDC subluxesulnarly
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Mechanism of Injury
• Direct blow – “boxer’s knuckle”
• Flicking injury
• Laceration
• Rheumatoid arthritis
Subluxing EDC
• EDC can stick in subluxed position
• Difficulty initiating extension
• Snapping
Treatment of acute injuries
• Splinting of MP in extension
• hand-based P1 blocker• relative motion splint• 4-6 weeks
Treatment of chronic cases
• Can try splinting – less successful
• Surgical repair of the sagittal band
Post-op Rehab
• Relative motion splint for 4 weeks
• Protected flexion / night splinting for 2 weeks
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References
• Kuczmarski AS, Harris AP, Gil JA, Weiss AC. Management of Diabetic Trigger Finger. J Hand Surg Am. 2019 Feb;44(2):150-153.
• Gil JA, Hresko AM, Weiss AC. Current Concepts in the Management of Trigger Finger in Adults. J Am Acad OrthopSurg. 2020 Aug 1;28(15):e642-e650.
• Strauch RJ. Extensor Tendon Injury. In Wolfe SW, Hotchkiss RN, Pederson WC, Kozin SH (eds). Green’s Operative Hand Surgery ed 7, Philadelphia: Elsevier, 2017; 152-82.
Hand Surgeons Providing Excellence in EducationforOccupational and Physical Therapists
www.doctorsdemystify.com
Dislocations:MP, PIP, and DIP Joints
Tze C. Ip, M.D.Newport Orthopaedic Institute
Finger Dislocations
• PIP joint most commonly injured joint in the hand
• 2004-2008: over 166,000 ED visits
• Spectrum of injury• “Jammed Finger”
• Irreducible Fracture Dislocation
Finger Dislocations
• Goals• Maintain a congruent joint
• Focus on early stable ROM
• Edema control
Finger Dislocations
• Primary Stabilizers:
Joint surface
Collateral ligaments
• Secondary Stabilizers:
Volar plate
Accessory collateral ligaments
Soft tissues
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Evaluation
Clinical:
• Swelling
• Skin Puckering
• Deformity
• Other injuries
Evaluation
Clinical:
• Concentric reduction with motion
• Active stability
• Passive stability
Central Slip Evaluation
Maintenance of full PIP extension
Elson Test:
-PIP is held flexed at 90⁰ and the patient is asked to extend the DIP
-Rigidity at the DIP indicates an injury to the central slip
Evaluation
Three Grades of Injuries:
• Grade I: Microscopic tear (stable injury)
• Grade II: Intact ligament but with laxity (stable injury)
• Grade III: Complete disruption of ligaments
Dislocations
Three Types (point of reference is
the location of the middle phalanx):
• Dorsal
• Lateral
• Volar
Dislocations
Dorsal:• Hyperextension
• Longitudinal compression
• Can result in soft tissue and/or bony injury
• Instability with extension
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Dislocations2) Type II (Dorsal dislocation)
• volar plate injury
• bilateral split to the collateral ligaments
• usually results in bayonette apposition
For 2021: better image needed
Dislocations
Treatment:
• Types I and II
• 2-3 days of protection
• begin early range of motion
Dislocations
• Type III (Stable)• Dorsal Blocking splint at 20-30 degrees of flexion
for 7-14 days
• Early active motion
• Buddy taping afterwards
Dislocations
• Type III (Unstable)
• Dynamic skeletal traction
• Extension block splinting
• Open reduction internal
fixation
Dislocations
Lateral Dislocation:
• Clinical Test
• Stress at full extension
• Stress at 30 degrees of flexion
Dislocations
Lateral Dislocation:
• Treatment
• Buddy Taping
• Greater than 20 degrees laxity Repair in an
athlete?
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Dislocations
Volar Dislocations:
• Rotatory longitudinal compression
• Semi-flexed middle phalanx
Dislocations
Volar Dislocations:
• Rare
• Unstable in flexion
• Can be unreducible
• Volar plate or collateral ligaments can be
interposed
MCP JointsCapsule
• Extends from the metacarpal neck to the base of the proximal phalanx
Volar Plate
• continuous with the deep transverse metacarpal ligament
Dorsal
• reinforced with common extensors
MCP Joints
Dorsal Dislocation• Uncommon
• Most frequent digits involved are the index and small digit
MCP Joints
Simple: reducible
• Begin early ROM and dorsal blocking splint
MCP Joints
Complete (Complex):
• Treatment
• Open reduction with the release of the A1 pulley
• Retrieve displaced volar plate interposed in the joint
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MCP Joints
Volar Dislocations:
• Extremely Uncommon
• Two structure blocking reduction
• dorsal capsule
• volar plate
DIP Joints
Dislocations• Usually dorsal or lateral
• Usually reducible
References
• Miller EA, Friedrich JB. Management of Finger Joint Dislocation and Fracture-Dislocations in Athletes. Clin Sports Med. 2020 Apr;39(2):423-442.
• Saitta BH, Wolf JM. Treating Proximal Interphalangeal Joint Dislocations. Hand Clin. 2018 May;34(2):139-148.
• Prucz RB, Friedrich JB. Finger joint injuries. Clin Sports Med. 2015 Jan;34(1):99-116.
• Glickel and Barron: Proximal interphalangeal joint fracture dislocations. Hand Clin. 2000 Aug;16(3):333-44. Review.
References
• Bindra and Foster: Management of proximal interphalangeal joint dislocations in athletes.
• Hand Clin. 2009 Aug;25(3):423-35. Review• Calfee and Sommerkamp TG. Fracture-dislocation about the
finger joints. J Hand Surg 2009 34A:1140-7. Review.• Dinh et al: Management of proximal interphalangeal joint
injuries. J Am Acad Orthop Surg. 2009: 17(5):318-24. Chinchalkarand Gan: Management of proximal interphalangeal joint fractures and dislocations. J Hand Ther. 2003 Apr-Jun;16(2):117-28. Review.
• Freiberg, et al: Management of proximal interphalangeal joint injuries. Hand Clin. 2006 Aug;22(3):235-42. Review.
Hand Surgeons Providing Excellence in EducationforOccupational and Physical Therapists
www.doctorsdemystify.com
Mallet Fingersand
Distal Phalanx Fractures
Roy A. Meals, MD
Disclaimer: RM receives royalties for Mallet Mender Splint
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Mallet Fingeretiology: sudden forced dip joint flexion
• Hand stationary: baseball, volleyball, basketball• Bony or tendinous
• Hand moving: tucking in slip cover or bed spread, pushing off sock, cleaning upholstery
• Tendinous
Mallet deformityExamination
• dip joint • medial/lateral stability• FDP tendon function • neurovascular status• nail
• pip joint• m/l stability• active e/f• hyperextensibility
• x-ray
Mallet FingerDifferential Diagnosis
• fracture or dislocation
• osteoarthritis
• flexor tendon tightness (especially after graft)
• Dupuytren disease can rarely contract DIP joint
Mallet FingerNatural History
• patient intuitively splints it
• The bone always heals
• rarely a functional deficit
• unsightly to some
• may lead to hyperextension at PIP joint (swan neck deformity)
• actual benefits• reduce or correct dip extensor lag
• Prevent/minimize swan neck deformity
• purported benefit of surgery• prevent osteoarthritis (never occurs)
Mallet FingerTreatment
Mallet FingerComplications of TreatmentJHS 1988, 13A:341
• Splinting 38/84 fingers (45%)• Skin maceration, necrosis, nail deformity, pain, tape allergy
• 1/38 complications permanent (nail groove)
• Surgery 24/45 (53%)• Infection, reoperation, nail deformity, joint deformity
• 18/24 complications permanent
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So if we can
avoid these
problems,
splinting is best.
DIP Joint Hyperextension
• causes loss of capillary circulation dorsally
• avoid
Methods of Splinting
any rigid material+tape
MethodsofSplinting
aluminum/foam
(closed or open
pore)
plaster
Mallet Mender splint
Stack splint
Videoclip:Mallet Mender Application
• BonyWeeks 1-4: full time
• Tendinous• Weeks 1-6: full time• Week 7: night time• Week 8: if persistent extension lag:
resume full time splinting
• Both injury types at week 9: blocking and passive flexion exercises
Splinting Acute Mallet Injuries
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Splint Treatment for Late Presentation
• Effective up to 6 months after tendinous injuries• For each month of delay in starting splinting, splint a week
longer
• e.g., 3 month delay: 6 weeks + 3 weeks
• Not effective for bony injuries
Surgical Treatment for Early Presentation
K-wire holding dip joint in extension, cut off below skin.
Possibly useful for surgeons, dentists
Surgical Treatment for Late Presentation
• Fowler procedure (release central slip)
• Spiral oblique retinacular ligament reconstruction
• Fusion
Distal Phalanx Fractures
• Intra-articular• Dorsal lip—bony mallet injuries
• Volar lip—FDP avulsion
• Shaft and tuft• Lever arm is short
• Mechanism: crush• Car doors usually do not fracture distal phalanx
Distal Phalanx Fractures
• Intra-articular• Volar lip—FDP avulsion, usually with only fleck of distal phalanx
and preservation of joint
Joint surface restored
FDP reattached
Gentle splinting of shaft fx
adequate since it does not
experience deforming
forces from tendons
Distal Phalanx Fractures
• Epiphyseal—may retard growth but finger lengths are different anyway
• Shaft and tuft—dramatic but innocuous• Tendons attach more proximally
• Gentle taping or splinting for comfort
• Frequent pulp, nail bed injuries associated with tuft and shaft fxs
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References: Mallet Fingers
• Lamaris G, Matthew M: The Diagnosis and Management of Mallet Finger Injuries. Hand (N Y). 2017 May; 12(3): 223–228.
• Grunberg A and Reagan D: Central slip tenotomy for chronic mallet finger deformity. J Hand Surg 1987, 12A:545
• Wehbe, M and Schneider L: Mallet fractures. J Bone Joint Surg 1984, 66A:658
• Chao JD et al: Central slip tenotomy for chronic mallet finger. An anatomic study. J Hand Surg 2004, 29A: 216
• Kleinman W and Peterson D: Oblique retinacular ligament reconstruction for chronic mallet finger deformity. J Hand Surg 1984, 9A:399
• Stern P and Kastrup J: Complications and prognosis of treatment of mallet finger. J Hand Surg 1988, 13A:329
References
Intra-articular Distal Phalanx Fractures
Wieschhoff G et al: Traumatic Finger Injuries: What the Orthopedic
Surgeon Wants to Know. RadioGraphics; 2016;36 (4) Published
Online: Jul 11 2016
Carpenter S, Rohde R: Treatment of Phalangeal Fractures. Hand Clin
29 (2013) 519–534
Bartelmann, U. et al. Handchir Mikrochir Plast Chir 2001 (screws)
Sauerbier M et al. Handchir Mikrochir Plast Chir. 1999 (tension
band)
Lubahn, J. D., Hood, J. M. Clin Orthop 1996
Schneider, L. H. Hand Clinics 1994
Schneider, L. H., Wehbe, M. A., J Bone Joint Surg Am 1988
Stark HH et al. J Bone Joint Surg Am. 1987
Resource
• Mallet mender splint blanks and instruction sheets for application and for patient:
• George Tiemann and Company• 1 800 TIEMANN (843-6266)
• www.georgetiemann.com
Hand Surgeons Providing Excellence in EducationforOccupational and Physical Therapists
www.doctorsdemystify.com
Osteoarthritis and Post-traumatic Arthritis of the Digits
Paul A Ghareeb MD
Assistant Professor, Emory Hand Surgery
Osteoarthritis (OA)
• Joint inflammation and destruction due to “wear and tear”
• Most common joint disorder
• Symptoms often wax and wane
• Almost universal in elderly
• Traumatic OA tends to occur in younger patients
• Diagnosis: Clinical exam + Imaging
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Traumatic Arthritis
• Intra-articular fracture leading to joint incongruity
• Amount of incongruity that leads to arthritis is unknown
• Dislocation
• Subluxation/instability
OA of the Fingers
• DIP, PIP, and thumb CMC joints are most frequently affected
• 50+ y/o -- females > males
• 50% genetically linked
• Estrogen may be protective
• Increased with repetitive forceful use - factory workers, farmers
Symptoms of OA
• “Gelling” after prolonged rest
• Morning stiffness
• Pain following activity, relieved by rest
• Weakness - usually mild and localized
Signs of OA
• Tenderness to palpation of the joint
• Effusion with little synovitis
• Heat and redness not common
• Bone spurs/osteophytes
• +/- crepitus or grinding
DIP Joints• Heberden nodes
• Mucous cysts
• Decreased motion
• +/- instability due to bone erosion
• Angulation at joint due to pinching, i.e. ulna deviation
• Pain
Treatment- OA DIP Joints• Heat, paraffin baths, splinting,
NSAIDS, Therapy
• Fusion in 0-20 degrees with wires, pins, screws
• Takes 6-8 weeks to heal, some get a fibroarthrodesiswith some motion still present
• Splint DIP, mobilize PIP/MCP joints
• Arthroplasty- typically unsuccessful, 10-30 extensor lag, 25-30 motion, lacks stability
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OA PIP Joints
• Bouchards nodes• Decreased motion• +/- instability due
to bone erosion• Angulation at joint
due to pinching, i.e. ulna deviation
• Pain
OA PIP Joints
• Heat, paraffin baths, splinting, NSAIDS, Therapy
• Joint de-innervation
• Fusion (plate and screws, interosseous wiring, tension band)
• 40-50 degrees, cascade, takes 6-8 wks to heal, mobilize MCP and DIP joints, maintain strength
• Arthroplasty-silicone
• fibrous encapsulation, get motion, not strength or stability
• older and low demand patients
• not index finger, due to ulna deviation with pinch
Joint Deinnervation
• Jimenez et al JHS 2020• 11 cases
• Improvement in pain from 7.8 to 1.4
• ROM improved from 52 to 79 degrees
• DASH scores significantly improved
Fusion vs Arthroplasty
Silicone PIPJ Arthroplasty -Postop
• Guarded active flexion/extension days 5-20
• Protect collateral ligaments
• Buddy tape to intact radial digit
• Isolate and increase flexion by extension splinting of MCP + DIP joints
• Passive ROM at 3weeks
• Flexion 0-60 degrees
Treatment of OA MCP Joints
• More common in rheumatoid arthritis
• Heat, paraffin baths, cortisone injections, NSAIDS, Therapy
• Fusion- young high demand patients 20-30 degrees
• increased strength
• can not spread finger
• Silicone implants-low demand/elderly patients
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Silicone MCP ArthroplastyPost - op
• 3-5 days p/o start ROM
• Daytime dynamic splinting
• Nighttime static splint
• Limit flexion to 45-60 degrees for stability and durability
• Can add flexion outrigger at 3 weeks if stiff in extension
Pyrocarbon Joint Arthroplasty
Pyrocarbon Joint Arthroplasty
• Wagner et al 2018:• No change in PIP joint motion
• Significant improvement in joint pain
• 34% reoperation rate• 21% implant revision rate
• 13% secondary procedure rate
Thumb OA• IP joint- Fuse 5-20 degrees
• MCP joint- Fuse 5-25 degrees• May occur after radial/ulna
ligament injury with instability
• Arthroplasty- low demand/elderly, not often indicated
Thumb CMC OA• Most common location of
symptomatic arthritis in the hand
• Joint laxity may predispose
• Eaton classification to describe severity of XR findings
• Pain with activity (can’t open a jar)
• + CMC Grind test
• Therapy, NSAIDs, short opponens splinting, steroid injection
Thumb CMC Joint
• Fusion
• young, high demand, post Bennett's fx, subluxation
• Place metacarpal in opposition
• May develop OA of Scapho-trapezial joint
• Arthroplasty – Many different techniques
• Trapeziectomy alone
• Ligament Reconstruction with Tendon Interposition (LRTI)
• Suspensionplasty
• Tightrope
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CMC
Fusion Excision ArthroplastyKnuckle Cracking!
• Joint subluxes causing decreased intra-articular pressure
• Gas formation and vaporization of joint fluid
• Audible crack
• Takes 30 minutes for gas to resorb- can repeat
When to Treat OA with Therapy Rather Than Surgery
• Mild deformity but good function
• Minimal pain
• Active patients frustrated by normal decline of strength and endurance with ageing
• Patients need to understand normal limitations of OA- need instructions in ergonomics
References
• EW, JW, MH et al. Medium term outcomes with pyrocarbon proximal interphalangeal arthroplasty: A study of 170 consecutive patients. J Hand Surg Am 2018;43:797-805
• Pickrell BB, Eberlin KR. Thumb Basal Joint Arthritis. Clin Plast Surg. 2019 Jul;46(3):407-413
• Yamamoto M, Malay S, Fujihara Y, Zhong L, Chung KC. A Systematic Review of Different Implants and Approaches for Proximal Interphalangeal Joint Arthroplasty. Plast Reconstr Surg. 2017 May;139(5):1139e-1151e
• Jiménez I, Marcos-García A, Muratore G, Caballero-Martel J, Medina J. Denervation for Proximal Interphalangeal Joint Osteoarthritis. J Hand Surg Am. 2020 Apr;45(4):358.e1-358.e5
• Operative Hand Surgery- Green
• Arthritis and Allied Conditions- Koopman
• Kelley’s Textbook of Rheumatology
Hand Surgeons Providing Excellence in EducationforOccupational and Physical Therapists
www.doctorsdemystify.com
Rheumatoid Arthritis
in the Finger Joints
Emil Dionysian, M.D.
Kaiser Permanente, Orange County
Slide preparation assisted by Neil Harness, M.D.
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Introduction
• Systemic autoimmune disorder
• Chronic, symmetric erosive synovitis of peripheral joints
• Progressive deformity
• Nonarticular manifestations
Initial Evaluation
• Systematic approach
• Document ROM over time
• AROM, PROM, Deformity
• Wrist: synovitis
• Thumb: CMC, MP, IP
• Digits: MP, PIP, DIP
Nodules
Flail IPs both thumbs
Marked Z collapse
thumb
RA Deformities
Stages
• I Synovitis without deformity
• II Synovitis with passively correctible deformity
• III Fixed deformity without joint changes
• IV Articular destruction
Non-operative Rx
• Rest: • Decreases synovitis
• Exercise:• Decreases stiffness
• Increases tendon motion, strength
• Short frequent exercise best
• Therapist
• Joint protection instruction
Lifestyle modification (anti-inflammatory)
Diet. Fasting followed by plant based
Exercise: 30 minutes per day
Sleep. Regularity and duration
Stress Management / coping technique
Connection and support
Avoid smoking and Alcohol
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The disease that can cause deformityOpen with left hand
Close with right hand
Diminish stresses that promote ulnar drift
Assistive device for turning knobs
Pamper the joints• A wheeled cart or island in the kitchen
helps move food from cooktop to tabletop..
• Lever-style faucets or tap turners make turning taps easier.
• Larger handle utensils and pots and pans
Non-operative Rx
• Splinting: wrist and thumb
• Steroid Injections: • CTS
• Tenosynovitis
• Joints not responding to medical Rx
• Only inject area 2 to 3 x per year• Risk of tendon rupture
• Steroid induced arthropathies
Non-operative Rx
• Not all patients will require surgery
• Goals: pain relief, preservation of function• NSAIDs
• resting splints
• local steroid injection
• Disease modifying anti-rheumatic drugs
• Tumor necrosis factor inhibitors
• eg Enbrel, Arava, etc.
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Indications
• Deformity alone is not an indication
• Loss of function that can be dramatically changed with surgery
• Pain
• Avoid surgery in:• Healthy, active patients with loss of function (ie. sports)
• Older patients with minimal pain who function well
Medical Considerations
• Cervical spine clearance
• Pulmonary involvement
• Felty’s syndrome (low WBC)
• Steroids and Methotrexate• Affect on wound healing
• NSAIDS• Affect platelet aggregation
Surgical Goals
• Alleviation of pain
• Improvement of function
• Retardation of progression of the disease
• Improvement in appearance
• Motion, dexterity and weakness remain problematic
Surgical Treatment
• Categories• Synovectomy
• Tenosynovectomy
• Tendon surgery
• Arthroplasty
• Arthrodesis
• Goals according to stage of disease• Preventative
• Corrective
• Salvage
Preoperative Planning
• Surgical planning• address painful areas first
• reliable/predictable procedures
• address nerve compression
• Complex deformities• proximal to distal
• staged vs. single stage reconstruction
Synovectomy
• Rarely indicated
• Consider in a patient with:• Smoldering, slowly progressing disease
• Responding to med Rx
• Responds to steroids
• Little radiographic joint destruction
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MP Joint Deformity
• Synovitis deforms joint capsule/ ligaments
• Radial sagittal band attenuation
• Flexor/Extensor imbalance
• Influence of wrist deformity
• Intrinsic contractures
Treatment
• Splinting
• Synovectomy
• Soft-tissue reconstruction• requires adequate articular
surfaces
• MP arthrodesis
• MP silicone arthroplasty
MP silicone arthroplasty
• Swanson implant commonly used
• Average 45 deg motion
• Good pain relief
• Good ability to realign joint
• Implant fx < 30% & often asymptomatic
• Infection 1-9%
MP Joint Arthroplasty
• Silicone implants• Gained popularity in the 1970’s
• first designed in 1962
• On long term follow-up high implant fracture rate (30%) and osteolysis.
• 2% failure rate per year
• Newer designs • pre-flexed 15 deg. And volar hinge
• Pyrolytic carbon implants
Therapy
• Post-operative care and rehabilitation
• Early protected motion PIP
• Early immobilization for MP
• MCP dynamic support
• Active and static splinting
• Exercises under the supervision of a hand therapist
• Result of arthroplasty 50% surgery: 50% therapy
Therapy
• Dressings removed 2 days after surgery
• A/AAROM starts POD 3-5
• Therapist• Maintains alignment during motion• 0 to 70 degrees at 2 weeks• Limit IF, MF to 45-60 degrees• Dynamic extension splints w/ digit alignment slings• Resting extension splint at night• Therapy x 3 months
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MP Joint ArthroplastyTrail IA et.al, JBJS Br. 2004 Sep. 86(7) 1002-6
17 year survivorship analysis
MP joint silastic replacement
1336 implants
2/3 fracture
63% survivorship
Improved with Soft tissue balancing and wrist realignment
MP Joint Arthroplasty
• Long Term Results (Best study/honest)• Goldfarb & Stern, JBJS 2003
• 36 patients (208 joints) w/ Avg. 14 yr f/u
• Motion improved (30 to 36 deg.)
• Ulnar drift (26 to 16 deg.)
• 63% implants broken
• 38% of patients expressed satisfaction
• 27% pain free
Silicone MCP implants
• Modification of Swanson Design• Bass, Stern, Nairus, JHS 1996
• Sutter silicone MP implants
• Retrospective review
• 42 hands, 168 implants
• 45% fractured if followed >3 yrs
PIP Joints• Swan Neck and Boutonniere
• Motion more important than at DIP joint
• Motion important in the more ulnar digits
• Stability more important in border digits
• Arthrodesis vs. Arthroplasty
General Rules
• Index finger arthrodesis perferred• Lateral stability needs to be maintained for pinch
• PIP arthrodesis preferred in setting of MP arthroplasty, although PIP/MP arthroplasty may give good results when staged
General Rules
• Limited role for arthroplasty in flexion boutonniere deformity
• PIP arthroplasty ok in swan neck if hyperextension deformity corrected (sublimis tenodesis)
• Poor lateral ligamentous support and/or associated flexor tendon ruptures relative indication for fusion.
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PIP Arthrodesis
• PIP cascade• 40 deg. Index to 55 deg. Small finger
• Fixation• 90-90 wiring
• Tension band
• Compression screws
• Plate and screws
PIP Arthroplasty
• Traditionally performed w/ Swanson silicone implants
• New pyrocarbon/metal implants gaining popularity for osteoarthritis
• Soft tissue balancing/stability remain difficult in RA
PIP Arthroplasty
• A systemic review of different implants and approaches for PIP joint arthroplasty
• Yamamoto M et. Al Plast Reconstr Surg
• 2017 May; 139(5): 1139e-1151e
• “Silicone implant with volar approach showed the best Active arc of motion with less extension lag and fewer complication”
DIP Joints - Arthrodesis
• Position: 0 to 10 deg flexion
• Relative unimportance of DIP motion
Arthroplasty/ArthrodesisSummary
• DIP/Thumb IP Arthrodesis
• PIP/Thumb MP Arthrodesis/Arthroplasty
• MP Arthroplasty
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References
• Feldon, Rheumatoid Arthritis and other connective tissue diseases, in Green’s Operative Hand Surgery, 7th ed. (2017)
• Amadio & Shin, Small joint arthrodesis and arthroplasty , in Green’s Operative Hand surgery, 7th ed. (2017)
• Chung KC, Pushman AG, Current concepts in management of rheumatoid hand. J.Hand Surg. 2011
• Akinson, JH, Chung, KC: “Advances in small joint arthroplasty of the hand” Plast. Reconst. Surg. 2014 dec.134(6): 1260-1268
Hand Surgeons Providing Excellence in EducationforOccupational and Physical Therapists
www.doctorsdemystify.com
Boutonniere and Swan Neck Deformities
Brian A. Pinsky, MD
The Long Island Plastic Surgical Group
Anatomy of the extensor mechanism
• Complex system of tendons/ligaments
• Combination of two separate systems
• Extrinsic - radial n. • Intrinsic - median and
ulnar n.
• Coordinates movements btw joints
Anatomy
• Extrinsics function to extend the MCP joint, flex PIP and DIP joints
• Intrinsics flex MCP and extend PIP and DIP
• Pathologic posture is related to imbalance in intrinsic/extrinsic flexor and extensor mechanisms
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Boutonniere Deformity
• Definition: Flexed PIP -> hyper-extended DIP -> hyper-extended MCP
• Imbalance between muscle and tendon units across finger joints
• Deformity begins at the PIP, adjacent joint changes are secondary
• Test DIP extension to look @ competence of central slip (Elson’s Test)
Boutonniere Deformity
• Etiology: Dorsal disruption of the extensor mechanism central slip at the PIP
• Laceration (trauma), • Attenuation (RA - synovitis, Dupuytren’s) • Dorsal burn• Attrition • Rupture of the central tendon
Boutonniere
• Lateral bands fall volar to PIP axis; PIP extensor becomes a flexor
• Initially presents w/ PIP flexion
• DIP hyperextension follows in several weeks
Boutonniere DeformityMechanisms
• ACUTE• Traumatic - open vs. closed
• Full active flexion maintained
• Full passive extension
• If PIP passively placed in full extension, can maintain position actively
• CHRONIC• Shortened, scarred lateral bands
• Thickened/shortened oblique retinacular ligament limits active DIP flexion
• Can see secondary joint changes - OA
4 Stages of the Boutonniere
• Dynamic imbalance (muscle and tendon)• Stage I - Mild lag at PIP, min deficit
• Stage II - Mod lag (10-30 deg), Passively correctable PIP
• Static extensor mechanism imbalance• Stage III – Fixed PIP flex contracture
• DIP stiff in extension
• Oblique retinacular ligament tightness
• Fixed joint contractures • Stage IV
• Collateral ligament/volar plate scarring
• Cartilage loss/ arthrosis
Treatment of BoutonniereGoals
• Reduce swelling and pain
• Prevent/minimize PIP flexion contracture
• Prevent lateral band subluxation
• Restore AROM and PROM of MP/PIP/DIP
• Restore hand function
• Patient MUST understand that some degree of extensor lag @ PIP expected even w/ good outcome
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Treatment of BoutonniereNon-Surgical
• Most boutonnieres will respond to splinting and therapy
• First-line treatment in closed injury -• Splint 6-8 wks constant extension @ PIP with DIP flxn exercises, 4-6
wks intermittent pm splinting
• The earlier, the better
• Allows central slip to heal
• Most flexible/correctable boutonnieres can be treated non-surgically
Exercise/splinting for the boutonniere
• Key in treatment is full passive ROM at PIP and DIP joints
• PIP Contracture -> Consider dynamic splinting, static-progressive splinting, or serial casting
• Monitor skin integrity
• Expect a small amt of PIP extensor lag and DIP hyperextension even if splinted early, no functional deficit
A multitude of options… Principles of Surgical Treatment
• Differentiate between acute and chronic injury
• Rebalancing of extensor system - divert power from distal joint to prox joint
• Best when joint has full passive mobility• Can be done in 2 stages: 1) release joint
contracture 2) extensor rebalancing
Surgical Principles Continued…
• Done in conjunction with a splinting and exercise program
• If arthritic changes, must combine with arthroplasty or fusion
• Most patients have good function WITH this deformity, careful to not worsen finger function
Surgical TreatmentChronic Boutonniere
• TERMIAL TENDON TENOTOMY• Must have full PROM @ PIP
• Post-op management• DIP extension splintremove for AROM• Splint for 6-8 weeksthen gradual wean
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Surgical procedures for boutonniere
• Tendon graft procedures
• Lateral band transfer
• Transfer 1-2 lat bands tocentral slip• Can restrict PIP flexion
Surgical procedures for boutonniere
• Staged Approach • 1) Extensor and transverse ligament tenolysis
• 2) Transverse ligament division• 3) Terminal tenotomy (if mild lag present)• 4) Central slip advancement
Complications after boutonniere surgery
• Recurrent deformity
• Incomplete recovery of flexion after prolonged splinting program and/ or surgery
• Secondary mallet (after terminal tenotomy)
• Surgical results unpredictable
Swan Neck Deformity
• PIPJ is hyperextended and the DIPJ is flexed
• Lack of voluntary DIP extension
• Function loss relative to PIP flexibility
• All swan-neck deformities are NOT the same
Etiology - Variable
1. Disruption of terminal extensor insertion leading to extensor tendon imbalance
2. Disruption or laxity of volar plate, FDS, or retinacular ligament @ PIP resulting in PIP hyperextension and compensatory DIP flexion
3. Intrinsic muscle tightness or spasticity from systemic diseases (RA) or CNS dysfunction
Etiology - Rheumatoid
1. If Mallet –type injury: Ext tendon attrition or rupture @ DIP
2. Synovitis resulting in PIP volar plate laxity or FDS rupture
3. MCP joint subluxation/dislocation causing intrinsic tightness, can decrease PIP ROM in certain positions only
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Swan Neck Pathophysiology
• Altered functional dynamics
• If PIP hyperextends, the lateral bands ride dorsally→ decreases distal tendon tension→results in droop of DIP into flexion
• Excess Extensor force @ PIP causes volar plate to stretch over time
• PIP and DIP motion are interdependent, lack or excess motion at one joint will reciprocally affect the other joint
Treatment
• GOAL = PREVENT PIP HYPEREXTENSION
• Treatment depends on the status of the PIP joint
• Important to treat early to avoid PIP stiffness, improves overall function and outcome
• Multiple procedures described
• Deformity tends to be worse at the joint that initiates deformity
Classification
• Type 1 No loss of PIP joint flexion• Tx aimed at preventing or correcting PIP
hyper-extension
• Type 2 Intrinsic Tightness
• Type 3 Limited PIP ROM
• Type 4 PIP arthritis
Non-operative Tx
• Splinting can be definitive depending on patient desires
• DOES NOT USUALLY RESPOND TO A SPLINTING AND EXERCISE PROGRAM
• splinting necessary preop to relieve fixed contractures/ mobilize joint/ relieve intrinsic tightness but volar plate laxity will persist
DIP Arthrodesis
• Use when deformity initiated by mallet injury
• Recommend fusion in neutral position
• Still need to address PIP joint
Flexor tenodesis• Strong internal splint to PIP
hyperextension
• One slip FDS divided proximal to MCP, left in tact distally
• FDS Sutured to itself around A2 pulley
• Suture in 20 deg flexion contracture
• Post-op: Early flexion, extension-block splint for 4-6 wks
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Oblique Retinacular Ligament Reconstruction
• Multiple techniques, complicated
• Require passively correctable deformity and preserved joint
• Volar PIP tenodesis - passively pulls the DIP into extension as the PIP actively extends
Type IIIntrinsic Tightness
• PIP flexion limited secondary to MP extension/ radial deviation
• Can be seen in untreated swan-neck or Rheumatoid pt with MP joint pathology
• Correct intrinsic tightness with therapy or surgical release
• Ex: If pathology at MCP => intrinsic release +/- MP arthroplasty, extensor centralization
• Then treat PIP hyperextension
Type IIILimited PIP ROM
• PIP flexion limited in all positions (fixed deformity)
• Usually soft tissue contracture, joint space maintained until late-stage disease
• Passive motion restricted by extensor mechanism, collateral ligaments, skin
• Treatment = Closed manipulation, stretch soft tissue• Can be done at time of MP arthroplasty or
DIP fusion, pin in 90 deg flexion
Type III
• Lateral Band Release – if closed manipulation fails
• Concept – restore PROM, then AROM
• Operation assumes in tact flexor tendon w/o adhesions
Type IVStiff PIP, arthritis
• Arthroplasty vs. arthrodesis
Complications after swan neck surgery
• Stretching/ rupture of tenodesis→ recurrence
• Too tight tenodesis→ excessive PIPJ flexion and potential boutonniere
• Loss of joint flexion
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Take home points
• Extensor mechanism is complex
• Deformity at one joint will result in opposite deformity of adjacent joints
• Boutonniere is best treated with splinting and exercise program
• Swan neck can be treated with splinting if pt desires but surgery is only permanent correction
• Reconstruction only possible if deformity is passively correctable!
Lin JS, Samora JB. Surgical and Non-surgical management of Mallet Finger: a Systematic Review. Journal Hand Surg. Vol 43 (2) Nov 2017
Strickland JW, Boyer MI: Swan-neck Deformity.pp.459-470. In Strickland JW (ed): Master Techniques in OrthopaedicSurgery: The Hand. Lippincott-Raven, Philadelphia, 1998
Nalebuff EA,Millender EH: Surgical Treatment of the Swan-neck Deformity in Rheumatoid Arthritis. Ortho Clinic NA, 6(3), 7/75
Souter WA: The problem of Boutonniere Deformity. Clin Ortho 1974; 104
To P, Watson JT: Boutonniere Deformity. J Hand Surg 2011, 36A (1)
References
Grau L et al: Biomechanics of the Acute Boutonniere Deformity. Journal Hand Surg. Vol 43, issue 1. Sept 2017.
Aulicino PL: Extensor Tendon Injuries.pp.149-158. In Light TR (ed): Hand Surgery Update II. AAOS, Rosemont, IL. 1999.
Burton RI, Melchior JA: Extensor Tendons-Late Reconstruction.pp.1988-2021.In Green DA: Operative Hand Surgery, 4th edition. Churchill Livingstone, Philalphia 1999.
Doyle JR: Boutonniere Deformity.pp.539-554. In Strickland JW (ed): Master Techniques in Orthopaedic Surgery: The Hand. Lippincott-Raven, Philadelphia. 1998.
References
Hand Surgeons Providing Excellence in EducationforOccupational and Physical Therapists
www.doctorsdemystify.com
Digital Tumors
Roy A. Meals, M.D.
Finger Tumors
• “tumor” = “swelling”• “neoplasm” = “new growth”
• inflammation
• cysts
• chemical deposits
• foreign bodies tumorsinfections
neoplasms
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Questions to Ask the Patient
How long present?
How fast came up?
Wax and wane in size?
Recall injury?
Similar findings elsewhere?
Now or previously?
General health problems?
Family history of similar?
Questions to Ask Yourself
Soft, firm, hard?
Hot, tender?
Pulsatile?
Movable on skin, deep structures?
Shape: spherical, spindle, flat, irregular?
Follow path of nerve? tendon? blood vessel?
Overlying skin changes? redness? ulcer?
Localized or diffuse?
Soft tissue or bone?
Classification of Finger Tumors
• Neoplasm• Soft tissue
• Benign
• Malignant
• Bony• Benign
• Malignant
• Inflammation • Infectious
• Traumatic
• Acellular • Cysts
• Deposits
• Foreign bodies
Neoplasm
or
Inflammation
or
Acellular?
Soft tissue
or
Bony?
Localized
or
Systemic?
Benign
or
Malignant?
Choose 1 characteristic
from each column
Neoplasm, soft tissue, benign, localized
Now ask, which tissue?
• synovium/tenosynovium
• skin
• nerve
• blood vessel
• connective tissue
• fat
Neoplasm, soft tissue, benign, localized
• synovium/tenosynovium• giant cell tumor of tendon sheath
• skin: wart, keratoacanthoma, many others
• nerve: nerilemmoma (Schwann cell tumor)
• blood vessel• arteriovenous malformation• glomus tumor• pyogenic granuloma
• connective tissue: Dupuytren’s
• fat: lipoma
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Neoplasm, soft tissue, benign, localized: TENOSYNOVIUM
giant cell tumor of tendon sheath
most common neoplasm in the hand
firm, lobulated, near tendon
Neoplasm, soft tissue, benign, localized: SKIN
wart, keratoacanthoma, many others
Neoplasm, soft tissue, benign, localized: NERVE
nerilemmoma (Schwann cell tumor)spindle-shaped swelling in digital nervemoves more side to side than proximal to distal
Neoplasm, soft tissue, benign, localized: BLOOD VESSEL
1. arteriovenous malformation: nest of vessels2. glomus tumor: overgrown A-V shunt3. pyogenic granuloma: reaction to injury
1
1 2
3
Neoplasm, soft tissue, benign, localized: CONNECTIVE TISSUE
• Dupuytren’s• rare in digit without affecting palm
Neoplasm, soft tissue, benign, systemic
Now ask, which tissue?
• connective tissue
• blood vessel
• nerve• neurofibomas—rare in fingers
• synovium/tenosynovium
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Neoplasm, soft tissue, benign, systemic: CONNECTIVE TISSUE
• Dupuytrens: palms, feet, knuckles, penis
• rheumatoid nodules: fingertips and elbow
Neoplasm, soft tissue, benign, systemic: BLOOD VESSEL
• arteriovenous malformations• diffuse; intravascular calcifications
Neoplasm, soft tissue, benign, systemic: SYNOVIUM
• rheumatoid synovitis
• rheumatoid tenosynovitis
Neoplasm, soft tissue, MALIGNANT, LOCALIZED
1 squamous cell carcinoma
2 malignant melanoma
(differentiate from subungual hematoma 3)
1 3 2
Neoplasm, soft tissue, MALIGNANT, SYSTEMIC:• metastatic
• lung; any tissue possible
• finger pulp location can be first indication of disease
Neoplasm, bony, benign, localized
• enchondroma• metaphyseal, expand but doesn’t destroy cortex, calcific stippling
interiorly, often identified because of pathologic fracture
•
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Neoplasm, bony, benign, systemic
• enchondromas• multiple: Ollier’s disease
• multiple w/ hemangiomas: Mafucci’s syndrome
• considerable risk of malignant degeneration
Neoplasm, bony, benign, systemic
• osteoarthritis (bone spurs)• DIP joints (Heberden’s nodes) very common
• PIP joints (Bouchard’s notes) less common
Neoplasm, bony, aggressive
• giant cell tumor of bone• high local recurrence rate, esp. in hand
• can metastasize
• treat aggressively• wide, local excision
• amputation
Neoplasm, bony, malignant
• localized • osteosarcoma
• chondrosarcoma
• systemic• multiple myeloma
Inflammation
infection, soft tissueparonychia
felontenosynovitis
infection, boneosteomyelitis
common bacterialtuberculosis
syphilis
Inflammation
• trauma • sprain
• acute
• chronic
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Acellular: cysts• ganglion and mucous cysts
• volar midline at proximal flexion crease
• at dip joint off dorsal midline
• other locations--transilluminate
Acellular: cysts
• inclusion cysts• epithelium gets pushed inside, keeps producing keratin
• hard; usually on contact surfaces
• do not transilluminate
Acellular: Chemical Deposits
Calcium: tendon, artery, soft tissue (lupus)
Acellular: Chemical Deposits
• uric acid (gout) • gouty deposits called tophus/tophi
• joint erosions, especially DIP joints
Acellular: Foreign Bodies Estimated Relative Occurrences
• soft tissue• ganglions and mucous cysts 800• giant cell tumors of tendon sheath 30• all other benign, including infections 150• malignant 5
• bone• enchondroma 10• other benign 5• malignant <<1
• total 1000
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Reading
Nepal Pet al: Common Soft Tissue Tumors Involving the Hand with Histopathological Correlation. J Clin Imaging Sci. 2019; 9: 15.
Henderson M et al: Hand Tumors I. Skin and Soft-Tissue Tumors of the Hand. Plas Recon Surg 2014; 133(2): 154e-164e
Schultz R and Kearns R: Tumors in the hand. J Hand Surg 1983, 8:803
Johnson J et al: Tumorous lesions of the hand. J Hand Surg 1985, 10A:384
Binu P et al: Malignant tumours of the hand and wrist. Indian J Plast Surg. 2011 May-Aug; 44(2): 337–347.
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