regurg & vomiting
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Regurgitation andRegurgitation and
VomitingVomiting
Regurgitation andRegurgitation and
VomitingVomiting
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REGURGITATIONREGURGITATIONDefinition:
Non forceful expulsion of food & secretions from esophagus
or stomach through the
mouth
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VOMITINGVOMITINGDefinition:
Forceful expulsion of stomach
contents through the mouth May be accompanied by nausea &
retching
May be evidence of gastro-duodenal reflux (bile stained )
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Differences :Differences :Regurgitation is:
« NOT accompanied by nausea & retching
« NO forceful contraction of abd. muscles
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Physiological RegurgitationPhysiological Regurgitation
P osseting P osseting
Happy Spitter Happy Spitter In early weeks of life
± 50% of infants 0-3 months
± 67% of infants at 4th month
Short time after feeds
Small amounts of milk
Baby is calm and happy
Weight gain is NORMAL
No treatment needed Reassure parents
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Causes of Regurgitation:Causes of Regurgitation:
Physiological regurg. (posseting)
Faulty feeding techniques
Gastroesophageal reflux (GER) Hiatal hernia
Achalasia of cardia
Congenital: E. atresia + TE fistula
Foreign body, Esophagitis, Stricture
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Esophagus: AnatomyEsophagus: Anatomy
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Normal Intraluminal pressuresNormal Intraluminal pressures
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Hiatal HerniaHiatal Hernia
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Gastroesophageal refluxGastroesophageal reflux
³³G.E.R or ChalaziaG.E.R or Chalazia´́Causes:
Hypotonia of Lower E. Sphincter (LES)
Transient LES relaxations at inappro- priate
moments
Failure of LES to adapt to intragastric
pressure during crying or straining
Abolished effect of gravity
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PHY
SIOLOGIC AL
PHY
SIOLOGIC AL G.E.R.G.E.R.
GER is a NORMAL phenomenon
Occurring virtually in everyone
Characteristically :
± Brief
± + 5 times in post-prandial hour ± Then frequency decrease rapidly to Zero
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IN P ATHOLOGIC AL
G.E.R.
D.IN
P ATHOLOGIC AL
G.E.R.
D.
There is increased frequency of GER
± During fasting ± During sleep
Volumes are large (soak clothes)
Weight gain is affected
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GER:Clinical presentations
GER:Clinical presentations
Regurgitation / Vomiting.....................97 %
Failure to thrive, malnutr., anemia.....20 %
Respiratory complications..................12 % Esophagitis & its complications......... 5 %
Silent Esophagitis
Dysphagia, Heartburn, excessive cry
Hematemesis Anemia
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R espiratory complicationsR espiratory complications Aspiration of refluxate
N octurnal cough
Asthma
Recurrent aspiration pneumonia
Vagal stimulation
Stridor, croup
Apnea
Bradycardia S.I.D.S.
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Acid Acid--Related AsthmaRelated Asthma
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Acid Acid--Related Asthma (cont )Related Asthma (cont )
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Diagnosis of G.E.R Diagnosis of G.E.R Clinical : suspect, weight curve
Barium swallow fluoroscopy Esophageal pH monitoring
Isotope scan (Gamma camera) Endoscopy + Biopsy
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Management of
G.E.R Management of
G.E.R
Phase I :
M ilk thickening , Positioning
Avoid certain foods at bed time
Phase II : all phase I + Domperidone
Phase III : all phase I & II +
Cimetidine, Ranitidine, etc.
Phase IV: Surgery
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Positional Treatment
Old method
GER
Anti-Trendlenberg
position at 30 o
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Causes of VomitingCauses of Vomiting Stimulation of supramedullary centers
Stimulation of Chemoreceptive T. Z.
Stimulation of peripheral receptors
Obstruction of Gastrointestinal tract
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Supramedullary Centers Stim.Supramedullary Centers Stim. Increased Intracranial pressure
Psychogenic vomiting (school phobia..)
Vestibular disease (motion sickness...)
Vascular causes (migraine, B.P. )
Seizures
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Stimulation of C.T.ZStimulation of C.T.Z Drug overdose: opiate, digoxin, aspirin..
M etabolic : acidosis, ketosis, uremia.. Hypervitaminosis A & D
Endocrinal : diabetes, adrenal insuf.,..
Irradiation
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GIT obstr.&
P
eriph. Stim.G
IT obstr.&P
eriph. Stim. Pharyngeal: P. N .D, self induced, etc.
Esophageal: G.E.R, achalasia, etc. Gastric: gastritis, ulcer, dysmotility, bezoar
Intestinal: infection, appendicitis, obstruction
Hepatobiliary: hepatitis, cholecystitis, etc.
Pancreatic: pancreatitis, etc.
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Dietetic: Cow¶s milk & food allergies
Improper feeding techniques
Cardiovascular : intestinal ischemia
Renal: Pyeloneph., calculi, hydroneph.
Respir.: Common cold, sinusitis
Tonsilopharyngitis,Otitis media
Pneumonia, Severe cough M iscellaneous: infectious fevers,
malaria, sepsis, peritonitis, cyclic vomiting,
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Main causes in NewbornMain causes in Newborn Birth trauma (I.C.Hge), septicemia
M econium aspiration
Congenital G.I.T malformations
Inborn errors of metabolism
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Main causes in InfancyMain causes in Infancy Gastroenteritis, dehydration, acidosis
G.E.R & other functional disorders
Infectious fevers, septicemia, etc.
Overfeeding & other dietetic causes
Hypervitaminosis A or D
Anatomic obstruction: pyloric stenosis
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Main causes in ChildrenMain causes in Children Gastroenteritis, dehydration, acidosis
Tonsillopharyngitis, otitis media, etc.
Systemic infections
Psychogenic ( relief by hospitalization)
Drugs, chemicals & toxins
Pertussis syndrome
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Main causes in AdolescentsMain causes in Adolescents Gastroenteritis
Systemic infections
Toxic ingestion, drugs, medication
Psychogenic, bulimia
Appendicitis
M igraine
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Cong. Hyp. Pyloric StenosisCong. Hyp. Pyloric Stenosis
HOW TO PALPATE ?HOW TO PALPATE ?
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DiagnosticE
valuationDiagnosticE
valuation Vomiting: Age of onset, frequency, duration,
timing, precipitating factors (cough, food..)
Associated S & S: fever, coryza, headache,diarrhea, cough, pain, constipation, jaundice,
seizures, dysuria, polydipsia, weight loss
History of drug intake, chemical, irradiation Examination of G I T
Exam.of other systems: CVS, Resp., C N S..
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InvestigationsInvestigations
Depending on suspicion:Depending on suspicion: Stools (parasites, melena, pus cells, etc.)
Urine (pus cells, bacteria, casts, etc.)
Liver function tests: hepatitis
C.B.C + differential count
Blood electrolytes, pH, Urea, Ketones
Skull X-ray & fundus : ? I.C.T.
Imaging studies: ulcers, obstructions, etc.
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Treatment:G
eneral linesTreatment:G
eneral lines Diet: stop oral intake or only fluid sips
Correct water, electrolyte & acid-base
Drugs:
¥ Prokinetic: metoclopramide, domperidone
¥ Anticholinergic: atropine, scopolamine
¥ Antihistamines: dimenhydrinate
¥Chlorpromazine (potent, use cautiously)
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