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    SURGICAL RETINA

    D r. J a m s h e d A h m e dM B B S., M C P S., F C P S ( O P H T H )

    ASSISTANT PROFESSOR

    Department of Ophthalmology

    Dow International Medical Collegehttp://www.youtube.com/watch?v=VHzJIUfO3ck

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    Retinal Detachment(R.D)

    R.D. is a separation of the sensory retina from theretinal pigment epithelium(R.P.E) by sub retinalfluid(S.R.F)

    Two main types

    1)Rhegmatogenous or Primary R.D

    2)Nonrhegmatous or SecondaryR.D

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    Types

    Two main types

    1. Rhegmatogenous or Primary R.D

    2. Nonrhegmatous or Secondary R.D Tractional R.D.

    Exudative(Serous)R.D

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    Rhegmatogenous R.D

    In Greek word,Rhegma meaning a rent or fissure

    http://www.youtube.com/watch?v=9Lxu3cUguM0&feature=related

    is a condition in which fluid from vitreous cavitypasses through a full thickness retinal defect into the sub retinal space to cause separation ofthe neural retina from the underlyingR.P.E

    It affects about 1:10,000 of the population eachyear

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    Pathogenesis

    Acute posterior Predisposing

    vitreous retinal degenerations

    detachment AphakiaTrauma retinal break

    degenerated vitreous passes via this breakbetween sensory retina and RPE collected as SRF

    Retinal detachment

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    Clinical Features

    History age: Common age group is 40-70 years

    No age is bar

    Sex: 60 % case comprises by males

    Hereditary: although a no.of pedigree shows

    familial detachments, most cases aresporadic

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    Symptoms :

    Classic premonitory symptoms present in 60% ofpatients with Spontaneous rheg.R.D are

    1) Flashes of lights(Photopsia)

    2) floaters- may be Solitary ring shaped opacity(Weiss Ring)

    Cobwebs Sudden shower of minute red colored or dark spots

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    Contd

    3) localized relative loss in field of vision-early

    less aware of superior than inferior field defect

    highly specific for localization ofR.D

    cause -spread of SRF behind the equator

    perceived by the patient as black curtain

    4) sudden painless loss of vision in case of large or

    centralR

    .D

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    Contd

    Blurring of distant vision

    40-45% R.D occurs in myopic patients

    History of trauma:(Surgical/Accidental)

    Surgical details

    a)Aphakia

    b) Pseudophakia with vitreous loss

    Ocular Trauma

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    Contd

    History of previous ocular disease similar problem in other eye

    glaucoma

    more in pigment dispersion syndromeMiotic drugs

    uveitis, vitreous hemorrhage, diabetic

    retinopathy Cytomegalovirus retinitis

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    Contd

    History of systemic diseases eg.

    Diabetes,tumors,angiomatosis of C.N.S,sickle

    cell disease,leukemia and EclampsiaMarfan's syndrome,

    Family history

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    Signs

    1) VisualAcuity less ifR.D extend up to fovea

    2) Confrontation test;Visual field defect

    3) External examination

    4)Anterior segment examination Cornea is usually clear but descemet's folds

    if hypotony

    Mild flare and cells inAnterior chamber

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    Signs

    5) Pupillary reaction

    Relative afferent pupillary

    defect in extensiveR

    .D6) Posterior segment

    Tobacco dust in retrolental

    vitreous, haemorrhages

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    Signs

    7) Fundus Examinationa Loss of normal fundal glowb)Retinal breaks: Present in about 70% of

    the eyes with tobaccodust.

    appears as reddiscontinuities in theretinal surface

    Upper temporal quadrantis the commonest site forretinal break

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    Signs

    Breaks may be in the form of holes,tears or

    dialysis

    Shape may be variable

    Can locate primary break by the shape of theR.D

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    Shape of R.D in relation to primary break

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    Signs

    Retinal signs depends on

    duration ofR.D

    A) Fresh R.D

    Detached retina has a

    convex configuration,

    slightly opaque and

    corrugated appearance,undulates freely with eye

    movements.

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    Signs

    loss of underlying choroidal pattern

    retinal blood vessels appears darker so color

    contrast vein and artery is less SRF extends up to ora serrata except caused

    by macular hole

    pseudo hole in posterior pole detachment

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    Signs

    B) Long standing retinal detachment

    retinal thinning secondary to atrophy

    secondary intraretinal cysts if more than 1 year sub retinal demarcation lines

    present after 3 months.

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    Signs

    Macula

    pigment of the macula may become more

    evident as a yellow colour due to intraretinaloedema or sub retinal fluid

    macular involvement is high in superior or

    temporal break or if in equatorial region

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    Signs

    Apparent holes in macula are relatively common

    Macular breaks leading toR.D are common instaphyloma of high myopia or following trauma

    Associated retinal degenerations,

    Intraocular pressure-low

    Systemic evaluation

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    Signs

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    Differential diagnosis

    Includes R.D from other causes and retinoschisis

    1) Tractional R.D

    Symptoms Photophobia and floaters usually absent

    Visual field defect progressive

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    Contd

    Signs concave configuration ,

    breaks absent

    SRF - shallower, seldomextends to ora serrata

    highest elevation at sites

    of vitreo-retinal traction

    severely reduced retinalmobility , shifting fluid

    absent

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    Contd

    2) ExudativeRD : Symptoms

    photopsia is absent

    VF defects develop suddenly

    and progress rapidly

    Signs-

    convex configuration

    breaks are absent smooth surface, SRF deep, shifting fluid phenomenon present

    Leopard spots in resolved RD

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    Contd

    Degenerative retinoschisis : Symptoms

    photopsia and floaters absent

    VF defects absolute scotoma

    Signs

    Elevation is convex, thin, smooth , relativelyimmobile

    Demarcation lines and secondary cyst absent

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    Management

    Diagnosis/Treatment

    Diagnosis

    1. Clinical Feature

    2. Investigations

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    USG- B scan

    RoutineTC, DC, Hb, ESR, Blood sugar,

    Urine R/M

    ERG (Brightflash)

    Investigations

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    Prophylaxis: Retinal Breaks

    INDICATIONS

    A. Characteristics of break

    B. Predisposing Peripheral retinal degenerations

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    High risk Breaks

    1)Tears more dangerous than holes

    2) Large break

    3)Symptomatic tears4) Superior breaks

    5) Equatorial breaks

    6) Sub clinical R.D

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    Other Considerations

    Break with or without symptoms:

    Aphakia

    Myopia

    One eyed

    Family History

    Systemic Diseases e.g. Marfan Syndrome,

    Stickler Syndrome, Ehlers DanlosSyndrome

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    Prophylaxis

    Predisposing Peripheral retinal

    degenerations (Lattice/Snail track)

    R.D in the fellow eye Aphaka or pseudophakia

    High Myopia

    Strong family history of R.D

    Systemic diseases

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    Prophylaxis Contd.

    White without pressure with fellow eye -

    giant retinal tears

    Fellow eye aphakic retinal detachment (

    360 degree cryotherapy 4 weeks prior to

    cataract surgery)

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    Prophylaxis

    Treatment Options

    C seals the retina with the RPE and choroid

    around the break,C 2-3 rows of nearly confluent laser burns

    around e.g. break in lattice.

    C

    firm chorioretinal scar forms in 7-10 days.

    Laser photocoagulation

    Transcleral cryotherapy

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    Cryotherapy/Laser

    Therapy

    Location of lesion

    Clarity of the media

    Pupil size

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    Retinal Detachment Surgery

    Historical Review:1920:T/t of R.D began in rational lines,

    Jules Gonin-first man to appreciate the pathologicalsignificance and therapeutic effect of closing retinal

    breaksT/t of R.D by sealing of the retinal breaks andevacuating the inter retinal fluid by puncture with agalvanocautery.

    1930 : Rosengren tried intravitreal air injection in

    upper half detachments.

    1933: Deutschmann tried cryopexy for R.D

    .

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    History contd..

    1949 Custodis : concept of ScleralBuckling

    1957 Schepens : refined scleral buckling techniqueand devised encircling operation in 1960.

    1949: Goldman devised three mirror funduscontact lens

    1950 Schepens : Indirect ophthalmoscope1962 Cibis introduced silicon oil to tamponade

    Lincoff and co-workers: Silicon sponge explant andcryotherapy

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    History contd

    1964 Lincoff revived Cryotherapy

    1940 AmslerSRF distribution to find

    probable hole, later in 1971 Lincoffprovided detail description.

    1985 Gilbert and McLeod successfully tried

    D-ACE procedure for upper half bullous R.D

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    Preoperative considerations &

    patient counseling

    IfRRD threatening to detach the macula;surgery urgently necessary

    Ifmacula is involved prompt surgery within 2-3 days

    recovers reasonable central vision

    macula off> 2 months postoperative Va usually poor

    Ifmacula is not involved, most eyes maintain preop VA

    Simple pathology and principles ofsurgery has to explained

    Anatomic success does not equal visual success,

    occassionally second surgery may be needed

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    Anaesthesia

    C Local or general anesthesia

    Advantages of LA Shorter operating time

    Quicker postoperative recovery

    Decreased morbidity and mortality

    C Peribulbar / retrobulbar

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    Purpose of R.D surgery

    To close the retinal hole

    To relieve any vitreoretinal traction.

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    Treatment options: RRD

    C Permanent scleral buckle, with/withoutdrainage- radial, segmental or encirclage

    C Temporary scleral buckle- Lincoff balloon, absorbable buckles

    C Pneumatic retinopexyC Primary vitrectomy

    C Combinations of aboveC Observation rarely

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    Scleral buckling

    C Purpose: to close retinal breaks

    : to reduce VR traction

    C Explants: material sutureddirectly onto the sclera

    Implants: intrascleral placementof buckle

    C Explant configuration1. Radial

    2. Segmental circum

    3. Encirclage

    C Explant size1. Buckle width/length

    2. Buckle height

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    Scleral buckling

    Indications for radial buckle

    - Large U-tears

    - relatively posterior

    breaks

    Indications for segmental buckle

    - single or closely

    spaced breaks