mechanisms of injury
DESCRIPTION
MECHANISMS OF INJURY. THERMAL INJURIES. - PowerPoint PPT PresentationTRANSCRIPT
![Page 1: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/1.jpg)
MECHANISMS OF INJURY
![Page 2: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/2.jpg)
THERMAL INJURIES• SCALDS —70% of burns in children; They also often occur in elderly
people. Water at 140°F (60°C) creates a deep partial-thickness or full-thickness burn in 3 seconds. At 156°F (69°C), the same burn occurs in 1 second. Scald burns from grease or hot oil are usually deep partial-thickness or full-thickness burns, as the oil or grease may be in the range of 400°F (200°C).
![Page 3: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/3.jpg)
• FLAME — often associated with inhalational injury and other concomitant
trauma. Flame burns tend to be deep dermal or full thickness. Flame burns are the second most common mechanism of thermal injury.
• CONTACT —In order to get a burn from direct contact, the object touched must either have been extremely hot or the contact was abnormally long. Burns from brief contact with very hot substances are usually due to industrial accidents. Contact burns tend to be deep dermal or full thickness.
THERMAL INJURIES
![Page 4: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/4.jpg)
• FLASH - Explosions of natural gas, propane, butane, petroleum distillates, alcohols, and other combustible liquids, as well as electrical arcs cause intense heat for a brief time period. Flash burns generally have a distribution over all exposed skin, with the deepest areas facing the source of ignition. Are typically epidermal or partial thickness, their depth depending on the amount and kind of fuel that explodes
THERMAL INJURIES
![Page 5: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/5.jpg)
ELECTRICAL INJURY• Some 3-4% of burn unit admissions are caused by
electrocution injuries (US data, 2005). • Mechanism:
– An electric current will travel through the body from one point to another, creating "entry" and "exit" points.
– The tissue between these two points can be damaged by the current.
– The amount of heat generated, and hence the level of tissue damage, is equal to 0.24x(voltage)2xresistance.
– The voltage is therefore the main determinant of the degree of tissue damage
![Page 6: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/6.jpg)
CHEMICAL INJURY• Chemical injuries are usually results of industrial accidents but may occur
with household chemical products.• These burns tend to be deep, as the corrosive agent continues to cause
coagulative necrosis until completely removed. – Alkalis tend to penetrate deeper and cause worse burns than acids. – Cement is a common cause of alkali burns.
Chemical burn due to spillage of
sulphuric acid
![Page 7: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/7.jpg)
BURN PATHOPHYSIOLOGY
![Page 8: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/8.jpg)
The body’s response to a burn.
• Burn injuries result in both local and systemic responses
![Page 9: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/9.jpg)
Local response
• Zone of coagulation—point of maximum damage., irreversible tissue loss due to protein coagulation
• Zone of stasis— with decreased tissue perfusion, tissue potentially salvageable
• Zone of hyperemia—outermost zone tissue perfusion is increased
![Page 10: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/10.jpg)
Clinical image of burn zones. There is central necrosis, surrounded by the zones of stasis and of hyperaemia
![Page 11: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/11.jpg)
LOCAL RESPONSE
• Loss of tissue in the zone of stasis will lead to the wound deepening as well as widening
![Page 12: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/12.jpg)
SYSTEMIC RESPONSE
• release of cytokines and other inflammatory mediators at the site of injury has a systemic effect once the burn reaches 30% of total body surface area
![Page 13: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/13.jpg)
SYSTEMIC RESPONSE
![Page 14: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/14.jpg)
Electrical injuries• electric current travels through the body from
one point to another, creating “entry” and “exit” points.
• tissue between these 2 points can be damaged by the current
• amount of heat generated = level of tissue damage
• Voltage determines the degree of tissue damage
![Page 15: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/15.jpg)
Electrical Injuries (Type)• Domestic Electricity (Low Voltage): cause
small, deep contact burns at the exit and entry sites
• True” high tension injuries: voltage ≥1000Vextensive tissue damage, often limb losslarge amount of soft and bony tissue necrosis• Flash injuries: tangential exposure to a high
voltage current arc but no current flow through the body
![Page 16: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/16.jpg)
![Page 17: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/17.jpg)
Classification of Burn Depths
• Partial thickness burns: do not extend through all skin layers
Superficial—affects the epidermis but not the dermis (such as sunburn), epidermal burn
Superficial dermal: extends through the epidermis into the upper layers of the dermis, associated with blistering
Deep dermal—extends through the epidermis into the deeper layers of the dermis but not through the entire dermis.
• Full thickness burns: extend through all skin layers into the subcutaneous tissues
![Page 18: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/18.jpg)
Classification of Burn Depths
![Page 19: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/19.jpg)
Assessment of Burn Depth
![Page 20: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/20.jpg)
ASSESMENT OF BURN AREA
![Page 21: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/21.jpg)
WALLACE RULE OF
NINES
![Page 22: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/22.jpg)
Lund and Browder
chart
![Page 23: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/23.jpg)
RESCUSCITATION REGIMEN
![Page 24: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/24.jpg)
RESCUSCITATION REGIMEN
• The starting point for resuscitation is the time of injury, not the time of admission
• High tension electrical injuries require substantially more fluid (up to 9 ml×(burn area)×(body weight) in the first 24 hours) and a higher urine output (1.5-2 ml/kg/hour)
• regimens should be continuously adjusted• according to urine output and other physiological
parameters (pulse, blood pressure, and respiratory rate)
![Page 25: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/25.jpg)
CRITERIA FOR REFERRAL TO A BURN CENTER• Second- and third-degree burns greater than 10%
TBSA in patients under 10 or over 50 years of age • Second- and third-degree burns greater than 20%
TBSA in other age groups • Second- and third-degree burns that involve the
face, hands, feet, genitalia, perineum, and major joints
• Third-degree burns greater than 5% TBSA in any age group
• Electrical burns, including lightning injury
![Page 26: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/26.jpg)
CRITERIA FOR REFERRAL TO A BURN CENTER• Chemical burns • Inhalation injury • Burn injury in patients with pre-existing medical
disorders that could complicate management, prolong recovery, or affect mortality
• Any patients with burns and concomitant trauma (e.g., fractures, blast injury) where burn injury poses the greatest risk of morbidity or mortality.
• Burn injury in children who will require special social/emotional and/or long-term rehabilitative support, including cases involving suspected child abuse or substance abuse
![Page 27: MECHANISMS OF INJURY](https://reader036.vdocument.in/reader036/viewer/2022062305/5681625f550346895dd2b8b5/html5/thumbnails/27.jpg)
REFERENCES
•Initial management of a major burn: II—assessment and resuscitation. Shehan Hettiaratchy, Peter Dziewulski. BMJ VOLUME 329 10 JULY 2004•Pathophysiology and types of burns. Shehan Hettiaratchy, Peter Dziewulski. BMJ VOLUME 328 12 JUNE 2004•American Burn Association. Hospital and Prehospital Resources for Optimal Care of Patients with Burn Injury: Guidelines for Development and Operation of Burn Centers. Journal of Burn Care and Rehabilitation. 1990; 11: 98-104.